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1 and the full-length acetyltransferase WeeI (Acinetobacter baumannii).
2 erobacteriaceae, Pseudomonas aeruginosa, and Acinetobacter baumannii).
3 designed an assay for the emerging pathogen Acinetobacter baumannii.
4 ella pneumoniae, Pseudomonas aeruginosa, and Acinetobacter baumannii.
5 ns caused by Gram-negative pathogens such as Acinetobacter baumannii.
6 consensus method for the active screening of Acinetobacter baumannii.
7 ed protein (Bap) in a bloodstream isolate of Acinetobacter baumannii.
8 ctivity do not address MDR pathogens such as Acinetobacter baumannii.
9 (ESBL)-producing Enterobacteriaceae and MDR Acinetobacter baumannii.
10 t defense against the opportunistic pathogen Acinetobacter baumannii.
11 istant strains of Pseudomonas aeruginosa and Acinetobacter baumannii.
12 ncy against the opportunistic human pathogen Acinetobacter baumannii.
13 lecular Microbiology, structural analysis in Acinetobacter baumannii 17978 revealed that a pentasacch
17 siella pneumoniae [10], carbapenem-resistant Acinetobacter baumannii [8], carbapenem- and quinolone-r
20 sted against the class C cephalosporinase of Acinetobacter baumannii [Acinetobacter-derived cephalosp
22 options exist for extensively drug resistant Acinetobacter baumannii, an emerging threat in ICUs worl
23 l monomicrobial necrotizing fasciitis due to Acinetobacter baumannii, an unusual finding that may be
24 Pathogenic Acinetobacter species, including Acinetobacter baumannii and Acinetobacter nosocomialis,
28 gues when used alone lacked activity against Acinetobacter baumannii and Klebsiella pneumoniae; howev
29 ilar protocols have been proposed to include Acinetobacter baumannii and Pseudomonas aeruginosa (SNAP
30 ymyxin resistant recent clinical isolates of Acinetobacter baumannii and Pseudomonas aeruginosa .
31 ority bacteria included carbapenem-resistant Acinetobacter baumannii and Pseudomonas aeruginosa, and
33 e-scaffolds would be more effective against Acinetobacter baumannii and Staphylococcus aureus biofil
36 tive (Staphylococcus aureus), Gram-negative (Acinetobacter baumannii), and fungal (Candida albicans)
37 tive (Staphylococcus aureus), gram-negative (Acinetobacter baumannii), and fungal (Candida albicans)
38 lococcus epidermidis, Enterococcus faecalis, Acinetobacter baumannii, and Candida albicans Catheters
39 Klebsiella pneumonia, Enterobacter cloacae, Acinetobacter baumannii, and methicillin-resistant Staph
40 d 100 well-characterized Enterobacteriaceae, Acinetobacter baumannii, and Pseudomonas aeruginosa isol
41 ococcus spp., members of the orange complex, Acinetobacter baumannii, and Pseudomonas aeruginosa were
43 cally troublesome pathogens, the prokaryote, Acinetobacter baumannii, and the eukaryote, Candida albi
44 Infections caused by the bacterial pathogen Acinetobacter baumannii are a mounting concern for healt
45 essential genes in Acinetobacter lwoffii and Acinetobacter baumannii are active in vitro and in vivo.
46 esistant forms of the Gram-negative pathogen Acinetobacter baumannii are an emerging threat to human
49 -negative bacilli Pseudomonas aeruginosa and Acinetobacter baumannii are increasingly acquiring carba
51 yed for the treatment of multidrug-resistant Acinetobacter baumannii, as it can rapidly develop resis
52 ein 33 (Omp33) is an outer membrane porin of Acinetobacter baumannii associated with carbapenem resis
53 plified samples were Prevotella tannerae and Acinetobacter baumannii at frequencies between 89 and 10
54 activity against the Gram-negative pathogen Acinetobacter baumannii ATCC 17961 (MIC = 0.0078 muM).
55 The efficacy of antimicrobial therapy for Acinetobacter baumannii bacteremia has been difficult to
57 al targets in 51% of all wound samples, with Acinetobacter baumannii being the most frequently detect
58 ut in vitro screen to identify inhibitors of Acinetobacter baumannii biofilms using a library of natu
59 thogens that express a T2S system(s) include Acinetobacter baumannii, Burkholderia pseudomallei, Chla
60 smic domains from Pseudomonas aeruginosa and Acinetobacter baumannii by BOCILLIN FL, aztreonam, merop
61 the susceptibilities of 107 isolates of the Acinetobacter baumannii-calcoaceticus complex to amikaci
63 infected or not with Staphylococcus aureus, Acinetobacter baumannii, Candida albicans, or Aspergillu
64 aphylococcus aureus, Pseudomonas aeruginosa, Acinetobacter baumannii, carbapenem-resistant Enterobact
67 cteriaceae spp., Pseudomonas aeruginosa, and Acinetobacter baumannii chosen to provide extreme diagno
68 cient killing by GA was also demonstrated in Acinetobacter baumannii clinical isolates and approximat
72 s, from whom the Acinetobacter calcoaceticus-Acinetobacter baumannii complex was isolated over a 14-m
73 ically important Acinetobacter calcoaceticus-Acinetobacter baumannii complex were found to be more re
74 ydroxyphenylacetate (HPA) 3-hydroxylase from Acinetobacter baumannii consists of a reductase componen
77 ruginosa (CP-PA) and carbapenemase-producing Acinetobacter baumannii (CP-AB) and perform a multicente
78 anisms of resistance of carbapenem-resistant Acinetobacter baumannii (CRAB) were determined in hospit
80 ecium, and silver/copper combination against Acinetobacter baumannii demonstrated antimicrobial syner
81 with MenC or another Gram-negative bacteria, Acinetobacter baumannii, did not inhibit augmented PS-sp
82 jor protein species in the outer membrane of Acinetobacter baumannii does not belong to the high-perm
85 es its properties to those published for the Acinetobacter baumannii enzyme, an example of the altern
86 igm of clinical infectious disease research, Acinetobacter baumannii, Escherichia coli, Klebsiella pn
89 s with molecular methods used to distinguish Acinetobacter baumannii from other members of the A. cal
90 We performed WGS on longitudinal isolates of Acinetobacter baumannii from patients undergoing colisti
91 ideal sampling method for identification of Acinetobacter baumannii from the health care environment
92 comparative analysis on currently completed Acinetobacter baumannii genomes revealed extensive and d
93 the in-hospital mortality was higher in the Acinetobacter baumannii group (16% vs. 13%; odds ratio,
94 cute liver failure were more frequent in the Acinetobacter baumannii group compared to the control gr
99 t emergence of multidrug resistance (MDR) in Acinetobacter baumannii has raised concern in health car
101 rk done over the past 5 years has shown that Acinetobacter baumannii has the remarkable capability to
102 multidrug-resistant, opportunistic pathogen, Acinetobacter baumannii, has spread swiftly through hosp
104 of both drug-susceptible and drug-resistant Acinetobacter baumannii in a soft-tissue infection model
105 fect of the presence of carbapenem-resistant Acinetobacter baumannii in accordance with surveillance
106 domonas aeruginosa, and carbapenem-resistant Acinetobacter baumannii in the pediatric population.
107 re unit length of stay, and complications of Acinetobacter baumannii including multidrug-resistant st
108 e or clinical indifference, the impact of an Acinetobacter baumannii infection on mortality is inconc
110 intensive care unit patients with confirmed Acinetobacter baumannii infection were defined as cases.
112 s for treatment of multidrug-resistant (MDR) Acinetobacter baumannii infections are extremely limited
114 ulfonate (CMS) to treat carbapenem-resistant Acinetobacter baumannii infections, colistin resistance
115 s Diseases (NSRB) against BasE, an AAAE from Acinetobacter baumannii involved in production of the si
135 enem-associated outer membrane protein) from Acinetobacter baumannii is a small OM protein that has b
157 CPNFs), including Pseudomonas aeruginosa and Acinetobacter baumannii, is necessary to prevent their d
158 se collection of epidemiologically unrelated Acinetobacter baumannii isolates to compare the robustne
160 rbapenems (imipenem and meropenem); 90.8% of Acinetobacter baumannii isolates were susceptible to min
162 or 99 isolates of Pseudomonas aeruginosa, 26 Acinetobacter baumannii isolates, and 11 Stenotrophomona
164 Moraxella catarrhalis, and most recently in Acinetobacter baumannii, it has become increasingly appa
165 important Gram-negative pathogens including Acinetobacter baumannii, Klebsiella pneumoniae, Pseudomo
166 stant clinical isolates of Escherichia coli, Acinetobacter baumannii, Klebsiella pneumoniae, Pseudomo
167 am-negative bacteria, but colistin-resistant Acinetobacter baumannii lacking lipid A were isolated af
170 ers for identifying carbapenem resistance in Acinetobacter baumannii, methicillin resistance in Staph
171 To successfully establish an infection, Acinetobacter baumannii must overcome the iron starvatio
173 mophilus influenzae, Pseudomonas aeruginosa, Acinetobacter baumannii, Neisseria meningitidis, Bactero
174 c acid (A/C) for 100 consecutive isolates of Acinetobacter baumannii obtained from various clinical s
180 6, Pseudomonas aeruginosa, pandrug-resistant Acinetobacter baumannii (PDRAB), Staphylococcus aureus,
185 in Escherichia coli, Klebsiella pneumoniae, Acinetobacter baumannii, Pseudomonas aeruginosa, and Ste
186 panresistant Gram-negative bacilli, such as Acinetobacter baumannii, requires consideration of nonan
188 n of NAD metabolism in the emerging pathogen Acinetobacter baumannii revealed unique features suggest
189 Investigating the opportunistic pathogen Acinetobacter baumannii's response to Zn starvation, we
190 ork, we show that the opportunistic pathogen Acinetobacter baumannii senses and responds to blue ligh
192 ial released by treatment of cell walls from Acinetobacter baumannii strain O11 with hot, aqueous phe
193 e and rapid spreading of multidrug-resistant Acinetobacter baumannii strains has become a major healt
195 (3) CFU mL(-1) of Pseudomonas aeruginosa and Acinetobacter baumannii strains that were undetectable u
198 t strains of some bacterial species, such as Acinetobacter baumannii strains, cannot be reliably iden
200 o MDR isolates of Pseudomonas aeruginosa and Acinetobacter baumannii through in vitro disk diffusion,
202 a representative nosocomial human pathogen, Acinetobacter baumannii, to chlorhexidine to identify th
204 comial pathogens, Pseudomonas aeruginosa and Acinetobacter baumannii, was also detected using this me
208 l intensive care unit stay was prolonged for Acinetobacter baumannii when compared to controls (media
210 d carbacephalosporinase producing strains of Acinetobacter baumannii, which have been listed by the W
211 erobacteriaceae, Pseudomonas aeruginosa, and Acinetobacter baumannii with various beta-lactamase susc
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