1 ble PQQ-dependent glucose dehydrogenase from Acinetobacter calcoaceticus.

2 ins from Synechocystis sp. strain PCC6803 or Acinetobacter calcoaceticus.

3 c compound degradation by the soil bacterium Acinetobacter calcoaceticus.

4 lus subtilis and the Gram-negative bacterium Acinetobacter calcoaceticus.

5 equence type (MLST) analysis, and WGS on 148 Acinetobacter calcoaceticus-A. baumannii complex bloodst

6 r spp. belonging to the clinically important Acinetobacter calcoaceticus-Acinetobacter baumannii comp

7 ng hospital in Houston, Texas, from whom the Acinetobacter calcoaceticus-Acinetobacter baumannii comp

8 uconate, and the beta-ketoadipate pathway in Acinetobacter calcoaceticus ADP1 (BD413).

9 erse bacteria, including Pseudomonas putida, Acinetobacter calcoaceticus, Agrobacterium tumefaciens,

10                                              Acinetobacter calcoaceticus also offers the advantage of

11                                              Acinetobacter calcoaceticus BD413 accumulates wax esters

12                  Akkermansia muciniphila and Acinetobacter calcoaceticus, both increased in MS patien

13 pase (LipA) and lipase chaperone (LipB) from Acinetobacter calcoaceticus RAG-1 were cloned and sequen

14 protein is conserved between E. coli Asd and Acinetobacter calcoaceticus soluble glucose dehydrogenas

15 monas putida, Agrobacterium tumefaciens, and Acinetobacter calcoaceticus, whereas wild-type R995 is s

16 oluble glucose dehydrogenase (PQQ-sGDH) from Acinetobacter calcoaceticus with osmium-based redox poly