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1 d after 95 days by a microbiota dominated by Actinobacillus.
5 Porphyromonas gingivalis (P. gingivalis) and Actinobacillus actinomycetemcomitans (A. actinomycetemco
6 counter known periodontal pathogens, such as Actinobacillus actinomycetemcomitans (A.a.) and, therefo
7 tle vector that is capable of replicating in Actinobacillus actinomycetemcomitans (Aa) and Escherichi
8 nes from a lambda gt11 expression library of Actinobacillus actinomycetemcomitans (Aa) genomic DNA th
10 ing protein lactoferrin (LF) may either kill Actinobacillus actinomycetemcomitans (Aa) or interfere w
11 s of four randomized treatment modalities on Actinobacillus actinomycetemcomitans (Aa), Porphyromonas
12 study evaluated the reinfection incidence by Actinobacillus actinomycetemcomitans (Aa), Porphyromonas
13 dentify the following target microorganisms: Actinobacillus actinomycetemcomitans (Aa), Tannerella fo
14 on dioxide (CO(2)) is required for growth of Actinobacillus actinomycetemcomitans (Aa), the reputed c
15 tion has tested the hypothesis that HGF from Actinobacillus actinomycetemcomitans (Aa)-infected patie
16 lla nigrescens (OR=1.7; 95% CI, 1.1 to 2.6), Actinobacillus actinomycetemcomitans (OR=1.7; 95% CI, 1.
18 5 (PG-2, PG-3, and PG-5) were tested against Actinobacillus actinomycetemcomitans (three strains) and
20 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans [Aa]), and divided
21 [IgG Bf], Porphyromonas gingivalis [IgG Pg], Actinobacillus actinomycetemcomitans [IgG Aa]), and asse
23 acteria, including the periodontal pathogens Actinobacillus actinomycetemcomitans and Porphyromonas g
24 G2 dominates responses to carbohydrates from Actinobacillus actinomycetemcomitans and Porphyromonas g
26 ntigens associated with host immunity (using Actinobacillus actinomycetemcomitans and Porphyromonas g
27 antigens associated with host immunity (with Actinobacillus actinomycetemcomitans and Porphyromonas g
28 es to the periodontitis-associated pathogens Actinobacillus actinomycetemcomitans and Porphyromonas g
29 lms produced by the human periodontopathogen Actinobacillus actinomycetemcomitans and the porcine res
30 es of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitans are naturally compe
34 uced by the Gram-negative periodontopathogen Actinobacillus actinomycetemcomitans as well as by the G
35 entrations and the IgG2 antibody response to Actinobacillus actinomycetemcomitans can be influenced b
37 enomonas noxia, Fusobacterium nucleatum, and Actinobacillus actinomycetemcomitans could be identified
39 that treatment of human lymphocytes with the Actinobacillus actinomycetemcomitans cytolethal distendi
42 ells of the gram-negative periodontopathogen Actinobacillus actinomycetemcomitans express a surface-e
43 ere, we report that the periodontal pathogen Actinobacillus actinomycetemcomitans expresses a homolog
44 es of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitans form tenaciously ad
46 eficient mutants of the periodontal pathogen Actinobacillus actinomycetemcomitans from a library of r
48 risk factor for periodontitis in adults, and Actinobacillus actinomycetemcomitans has been implicated
50 utilized to screen for exported proteins of Actinobacillus actinomycetemcomitans in Escherichia coli
51 ducer 2 (AI-2) produced by the oral pathogen Actinobacillus actinomycetemcomitans influences growth o
56 The cytolethal distending toxin (CDT) of Actinobacillus actinomycetemcomitans is a typical member
57 The cytolethal distending toxin (Cdt) of Actinobacillus actinomycetemcomitans is an atypical A-B-
58 The oral commensal Gram-negative bacterium Actinobacillus actinomycetemcomitans is believed to be t
62 own to confer a hyperleukotoxic phenotype in Actinobacillus actinomycetemcomitans IS1, but the mechan
67 lasts (HGF) were incubated for 24 hours with Actinobacillus actinomycetemcomitans lipopolysaccharide
68 linical isolates of the periodontal pathogen Actinobacillus actinomycetemcomitans live as autoaggrega
69 IL-1beta, Porphyromonas gingivalis LPS, and Actinobacillus actinomycetemcomitans LPS, MIP-1alpha mRN
77 at the oral bacterium and periodontopathogen Actinobacillus actinomycetemcomitans secretes an immunos
78 ip exists between antibody reactive with the Actinobacillus actinomycetemcomitans serotype b lipopoly
79 le periodontitis (LJP) patients colonized by Actinobacillus actinomycetemcomitans serotype b often co
80 r gene probe hybridized to Southern blots of Actinobacillus actinomycetemcomitans strain JP2 (serotyp
85 ded by the tad (for tight adhesion) locus in Actinobacillus actinomycetemcomitans that is involved in
86 ere assayed for Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans using a 16S rRNA po
87 a membrane protein of the periodontopathogen Actinobacillus actinomycetemcomitans was achieved by con
91 pe b-specific carbohydrate antigen (SbAg) of Actinobacillus actinomycetemcomitans Y4 is reported to b
92 annerella forsythensis, Treponema denticola, Actinobacillus actinomycetemcomitans) and dental caries
93 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
94 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
95 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
96 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
97 monas gingivalis, Bacteroides forsythus, and Actinobacillus actinomycetemcomitans) and serum antibody
98 coccus mutans, Porphyromonas gingivalis, and Actinobacillus actinomycetemcomitans) selected based upo
99 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), a capnophilic fac
100 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), and Campylobacter
101 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), and Porphyromonas
102 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), and Streptococcus
103 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Campylobacter rec
104 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Porphyromonas gin
105 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Porphyromonas gin
106 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Porphyromonas gin
107 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Prevotella interm
108 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans)-biofilm colonizing
115 in a polymorphic region of the chromosome of Actinobacillus actinomycetemcomitans, a predominant oral
116 red gene required for tenacious adherence of Actinobacillus actinomycetemcomitans, also has significa
117 microorganisms, Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, also invade the pe
118 usobacterium nucleatum, Eikenella corrodens, Actinobacillus actinomycetemcomitans, and Campylobacter
119 sed a multi-species (Streptococcus gordonii, Actinobacillus actinomycetemcomitans, and Fusobacterium
120 4 subjects were analyzed for the presence of Actinobacillus actinomycetemcomitans, Bacteroides forsyt
121 eled synthetic oligonucleotides specific for Actinobacillus actinomycetemcomitans, Bacteroides forsyt
123 tivation by microbial or protein Ags (namely Actinobacillus actinomycetemcomitans, bovine insulin, an
124 CEK (species other than Haemophilus species, Actinobacillus actinomycetemcomitans, Cardiobacterium ho
125 , Capnocytophaga, Propionibacterium, yeasts, Actinobacillus actinomycetemcomitans, coagulase-negative
126 Gram-negative bacterial species, such as Actinobacillus actinomycetemcomitans, contain lipopolysa
127 pathogens (such as Porphyromonas gingivalis, Actinobacillus actinomycetemcomitans, etc.) into the lun
128 levels 8 to 20 h after infection with either Actinobacillus actinomycetemcomitans, F. nucleatum, or P
129 BD-3 against a collection of oral organisms (Actinobacillus actinomycetemcomitans, Fusobacterium nucl
130 ed to distinguish among clinical isolates of Actinobacillus actinomycetemcomitans, Haemophilus aphrop
133 ntal pathogens, Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, may contribute to
135 ned risk indicators for oral colonization by Actinobacillus actinomycetemcomitans, Porphyromonas ging
137 d a 16S rRNA PCR detection method identified Actinobacillus actinomycetemcomitans, Porphyromonas ging
138 the following 3 major periodontal pathogens: Actinobacillus actinomycetemcomitans, Porphyromonas ging
139 imultaneous detection and differentiation of Actinobacillus actinomycetemcomitans, Porphyromonas ging
141 obability for detecting oral colonization by Actinobacillus actinomycetemcomitans, Porphyromonas ging
142 Previously, we reported that intracellular Actinobacillus actinomycetemcomitans, Porphyromonas ging
143 croscopy to detect the periodontal pathogens Actinobacillus actinomycetemcomitans, Porphyromonas ging
144 nce of known periodontal pathogens including Actinobacillus actinomycetemcomitans, Prevotella interme
145 t bacteriostatic effect, particularly on the Actinobacillus actinomycetemcomitans, Prevotella oris, S
146 oral pathogens Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, skin commensal Sta
149 ridement is recommended for the treatment of Actinobacillus actinomycetemcomitans-associated periodon
150 The purpose of this study was to evaluate Actinobacillus actinomycetemcomitans-responsive B lympho
151 amma is clearly associated with (i) enhanced Actinobacillus actinomycetemcomitans-specific RANKL-expr
165 gatibacter actinomycetemcomitans [previously Actinobacillus actinomycetemcomitans], Prevotella interm
167 The periodontal pathogen Aggregatibacter (Actinobacillus) actinomycetemcomitans has an irregular o
168 required for the growth of Aggregatibacter (Actinobacillus) actinomycetemcomitans in culture under c
172 oral and systemic pathogen Aggregatibacter (Actinobacillus) actinomycetemcomitans produces a leukoto
174 s that localized juvenile and other forms of Actinobacillus-associated periodontitis are primarily as
175 d average time to detection for Haemophilus, Actinobacillus, Cardiobacterium, Eikenella, and Kingella
176 as the only Actinomyces spp. coisolated with Actinobacillus (Haemophilus) actinomycetemcomitans; Acti
179 y be applied to other members of Haemophilus-Actinobacillus-Pasteurella family, where genetic manipul
181 previous studies demonstrated that Adh from Actinobacillus pleuropneumoniae (A. pleuropneumoniae) is
182 a coli (Ec) was constructed by modifying the Actinobacillus pleuropneumoniae (Ap) plasmid pYG53.
184 of its ability to confer NAD independence on Actinobacillus pleuropneumoniae and H. influenzae, has b
185 ting of two fastidious veterinary pathogens, Actinobacillus pleuropneumoniae and Haemophilus somnus,
190 Here we report the crystal structure of Actinobacillus pleuropneumoniae HMW1C (ApHMW1C), a funct
199 s, which differentiates serovars 3, 6, and 8 Actinobacillus pleuropneumoniae isolates, is described.
201 directly quantify glycopeptide formation by Actinobacillus pleuropneumoniae NGT and determine its su
203 t a vaccine prepared from outer membranes of Actinobacillus pleuropneumoniae serotype 5 can elicit pr
205 ee extracts isolated from colony biofilms of Actinobacillus pleuropneumoniae serotype 5 were found to
207 tiplex PCR assays were developed to identify Actinobacillus pleuropneumoniae serotypes 1, 2, and 8.
210 -ray crystal structure of the Cu,Zn SOD from Actinobacillus pleuropneumoniae, a major porcine pathoge
211 steurellaceae indicates that M. haemolytica, Actinobacillus pleuropneumoniae, and Haemophilus ducreyi
212 genic Neisseriaceae, Haemophilus influenzae, Actinobacillus pleuropneumoniae, and M. catarrhalis.
214 Haemophilus parasuis, Pasteurella multocida, Actinobacillus pleuropneumoniae, Bordetella bronchisepti
220 apsule type 3), Streptococcus zooepidemicus, Actinobacillus spp., and Mycoplasma equirhinis and equin
221 biosuccinic acid using the bacterial strain Actinobacillus succinogenes 130 Z, and simultaneously pr
222 pathway and the central carbon metabolism of Actinobacillus succinogenes for the production of succin
223 ethod of sampling is applied to the organism Actinobacillus succinogenes for the production of succin
225 ling either cells or membranes purified from Actinobacillus succinogenes to drive electron transfer a
227 19 mm and resistance at < or =11 mm, and for Actinobacillus suis, Erysipelothrix rhusiopathiae, and S
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