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1 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans).
2 ccus radiodurans, Pasteurella multocida, and Actinobacillus actinomycetemcomitans.
3 virulence locus of the periodontal pathogen Actinobacillus actinomycetemcomitans.
4 the Gram-negative capnophilic coccobacillus Actinobacillus actinomycetemcomitans.
5 ntal pathogens: Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans.
6 ng LPS derived from the periodontal pathogen Actinobacillus actinomycetemcomitans.
7 ontal pathogens Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans.
8 he 14-gene Widespread Colonization Island of Actinobacillus actinomycetemcomitans.
9 ve high levels of IgG2 that is reactive with Actinobacillus actinomycetemcomitans.
10 inst a strain of the gram-negative bacterium Actinobacillus actinomycetemcomitans.
11 and biofilm formation by the human pathogen Actinobacillus actinomycetemcomitans.
12 e main causative organism of this disease is Actinobacillus actinomycetemcomitans.
13 R995 Delta incC could not be established in Actinobacillus actinomycetemcomitans.
14 ffinity-purified human anti-PC to PC-bearing Actinobacillus actinomycetemcomitans.
18 Porphyromonas gingivalis (P. gingivalis) and Actinobacillus actinomycetemcomitans (A. actinomycetemco
19 counter known periodontal pathogens, such as Actinobacillus actinomycetemcomitans (A.a.) and, therefo
20 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), a capnophilic fac
26 in a polymorphic region of the chromosome of Actinobacillus actinomycetemcomitans, a predominant oral
27 tle vector that is capable of replicating in Actinobacillus actinomycetemcomitans (Aa) and Escherichi
28 nes from a lambda gt11 expression library of Actinobacillus actinomycetemcomitans (Aa) genomic DNA th
30 ing protein lactoferrin (LF) may either kill Actinobacillus actinomycetemcomitans (Aa) or interfere w
31 s of four randomized treatment modalities on Actinobacillus actinomycetemcomitans (Aa), Porphyromonas
32 study evaluated the reinfection incidence by Actinobacillus actinomycetemcomitans (Aa), Porphyromonas
33 dentify the following target microorganisms: Actinobacillus actinomycetemcomitans (Aa), Tannerella fo
34 on dioxide (CO(2)) is required for growth of Actinobacillus actinomycetemcomitans (Aa), the reputed c
35 tion has tested the hypothesis that HGF from Actinobacillus actinomycetemcomitans (Aa)-infected patie
36 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans [Aa]), and divided
38 red gene required for tenacious adherence of Actinobacillus actinomycetemcomitans, also has significa
39 microorganisms, Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, also invade the pe
41 ntigens associated with host immunity (using Actinobacillus actinomycetemcomitans and Porphyromonas g
42 antigens associated with host immunity (with Actinobacillus actinomycetemcomitans and Porphyromonas g
43 es to the periodontitis-associated pathogens Actinobacillus actinomycetemcomitans and Porphyromonas g
44 acteria, including the periodontal pathogens Actinobacillus actinomycetemcomitans and Porphyromonas g
45 G2 dominates responses to carbohydrates from Actinobacillus actinomycetemcomitans and Porphyromonas g
46 lms produced by the human periodontopathogen Actinobacillus actinomycetemcomitans and the porcine res
47 annerella forsythensis, Treponema denticola, Actinobacillus actinomycetemcomitans) and dental caries
48 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
49 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
50 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
51 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans) and Porphyromonas
52 monas gingivalis, Bacteroides forsythus, and Actinobacillus actinomycetemcomitans) and serum antibody
53 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), and Campylobacter
54 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), and Porphyromonas
55 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), and Streptococcus
56 usobacterium nucleatum, Eikenella corrodens, Actinobacillus actinomycetemcomitans, and Campylobacter
57 sed a multi-species (Streptococcus gordonii, Actinobacillus actinomycetemcomitans, and Fusobacterium
58 es of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitans are naturally compe
62 uced by the Gram-negative periodontopathogen Actinobacillus actinomycetemcomitans as well as by the G
63 ridement is recommended for the treatment of Actinobacillus actinomycetemcomitans-associated periodon
64 4 subjects were analyzed for the presence of Actinobacillus actinomycetemcomitans, Bacteroides forsyt
65 eled synthetic oligonucleotides specific for Actinobacillus actinomycetemcomitans, Bacteroides forsyt
67 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans)-biofilm colonizing
68 tivation by microbial or protein Ags (namely Actinobacillus actinomycetemcomitans, bovine insulin, an
69 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Campylobacter rec
70 entrations and the IgG2 antibody response to Actinobacillus actinomycetemcomitans can be influenced b
71 CEK (species other than Haemophilus species, Actinobacillus actinomycetemcomitans, Cardiobacterium ho
73 , Capnocytophaga, Propionibacterium, yeasts, Actinobacillus actinomycetemcomitans, coagulase-negative
74 Gram-negative bacterial species, such as Actinobacillus actinomycetemcomitans, contain lipopolysa
75 enomonas noxia, Fusobacterium nucleatum, and Actinobacillus actinomycetemcomitans could be identified
77 that treatment of human lymphocytes with the Actinobacillus actinomycetemcomitans cytolethal distendi
80 pathogens (such as Porphyromonas gingivalis, Actinobacillus actinomycetemcomitans, etc.) into the lun
81 ells of the gram-negative periodontopathogen Actinobacillus actinomycetemcomitans express a surface-e
82 ere, we report that the periodontal pathogen Actinobacillus actinomycetemcomitans expresses a homolog
83 levels 8 to 20 h after infection with either Actinobacillus actinomycetemcomitans, F. nucleatum, or P
84 es of the gram-negative periodontal pathogen Actinobacillus actinomycetemcomitans form tenaciously ad
86 eficient mutants of the periodontal pathogen Actinobacillus actinomycetemcomitans from a library of r
87 BD-3 against a collection of oral organisms (Actinobacillus actinomycetemcomitans, Fusobacterium nucl
89 ed to distinguish among clinical isolates of Actinobacillus actinomycetemcomitans, Haemophilus aphrop
90 risk factor for periodontitis in adults, and Actinobacillus actinomycetemcomitans has been implicated
93 The periodontal pathogen Aggregatibacter (Actinobacillus) actinomycetemcomitans has an irregular o
95 [IgG Bf], Porphyromonas gingivalis [IgG Pg], Actinobacillus actinomycetemcomitans [IgG Aa]), and asse
96 utilized to screen for exported proteins of Actinobacillus actinomycetemcomitans in Escherichia coli
97 required for the growth of Aggregatibacter (Actinobacillus) actinomycetemcomitans in culture under c
98 ducer 2 (AI-2) produced by the oral pathogen Actinobacillus actinomycetemcomitans influences growth o
103 The cytolethal distending toxin (CDT) of Actinobacillus actinomycetemcomitans is a typical member
104 The cytolethal distending toxin (Cdt) of Actinobacillus actinomycetemcomitans is an atypical A-B-
105 The oral commensal Gram-negative bacterium Actinobacillus actinomycetemcomitans is believed to be t
113 own to confer a hyperleukotoxic phenotype in Actinobacillus actinomycetemcomitans IS1, but the mechan
118 lasts (HGF) were incubated for 24 hours with Actinobacillus actinomycetemcomitans lipopolysaccharide
119 linical isolates of the periodontal pathogen Actinobacillus actinomycetemcomitans live as autoaggrega
120 IL-1beta, Porphyromonas gingivalis LPS, and Actinobacillus actinomycetemcomitans LPS, MIP-1alpha mRN
121 ntal pathogens, Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, may contribute to
124 lla nigrescens (OR=1.7; 95% CI, 1.1 to 2.6), Actinobacillus actinomycetemcomitans (OR=1.7; 95% CI, 1.
125 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Porphyromonas gin
126 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Porphyromonas gin
127 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Porphyromonas gin
129 d a 16S rRNA PCR detection method identified Actinobacillus actinomycetemcomitans, Porphyromonas ging
130 the following 3 major periodontal pathogens: Actinobacillus actinomycetemcomitans, Porphyromonas ging
131 imultaneous detection and differentiation of Actinobacillus actinomycetemcomitans, Porphyromonas ging
133 obability for detecting oral colonization by Actinobacillus actinomycetemcomitans, Porphyromonas ging
134 Previously, we reported that intracellular Actinobacillus actinomycetemcomitans, Porphyromonas ging
135 croscopy to detect the periodontal pathogens Actinobacillus actinomycetemcomitans, Porphyromonas ging
136 ned risk indicators for oral colonization by Actinobacillus actinomycetemcomitans, Porphyromonas ging
138 gatibacter actinomycetemcomitans (previously Actinobacillus actinomycetemcomitans), Prevotella interm
139 nce of known periodontal pathogens including Actinobacillus actinomycetemcomitans, Prevotella interme
140 t bacteriostatic effect, particularly on the Actinobacillus actinomycetemcomitans, Prevotella oris, S
141 gatibacter actinomycetemcomitans [previously Actinobacillus actinomycetemcomitans], Prevotella interm
147 oral and systemic pathogen Aggregatibacter (Actinobacillus) actinomycetemcomitans produces a leukoto
148 The purpose of this study was to evaluate Actinobacillus actinomycetemcomitans-responsive B lympho
149 at the oral bacterium and periodontopathogen Actinobacillus actinomycetemcomitans secretes an immunos
150 coccus mutans, Porphyromonas gingivalis, and Actinobacillus actinomycetemcomitans) selected based upo
151 ip exists between antibody reactive with the Actinobacillus actinomycetemcomitans serotype b lipopoly
152 le periodontitis (LJP) patients colonized by Actinobacillus actinomycetemcomitans serotype b often co
154 oral pathogens Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans, skin commensal Sta
155 amma is clearly associated with (i) enhanced Actinobacillus actinomycetemcomitans-specific RANKL-expr
156 r gene probe hybridized to Southern blots of Actinobacillus actinomycetemcomitans strain JP2 (serotyp
161 ded by the tad (for tight adhesion) locus in Actinobacillus actinomycetemcomitans that is involved in
164 5 (PG-2, PG-3, and PG-5) were tested against Actinobacillus actinomycetemcomitans (three strains) and
165 ere assayed for Porphyromonas gingivalis and Actinobacillus actinomycetemcomitans using a 16S rRNA po
166 a membrane protein of the periodontopathogen Actinobacillus actinomycetemcomitans was achieved by con
170 pe b-specific carbohydrate antigen (SbAg) of Actinobacillus actinomycetemcomitans Y4 is reported to b
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