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1 ain, and they may represent novel species of Actinomyces.
2 re confirmed as members of genera other than Actinomyces.
3 ng these organisms from other genera such as Actinomyces.
4 r of strains tested) used in this study were Actinomyces (32), Anaerobiospirillum (8), Bacteroides (3
7 ic line that is related to but distinct from Actinomyces, Actinobaculum, Arcanobacterium, and Mobilun
9 dependent intercellular interactions between Actinomyces and oral streptococci as well as host cells
11 EP-HN019, there were greater proportions of Actinomyces and Streptococcus-like species and lower pro
12 l interactions between oral streptococci and actinomyces and their adherence to tooth surface and the
13 elucidate the fimbrial assembly pathways in Actinomyces and their function in the pathogenesis of di
15 ivity were also characterized by Prevotella, Actinomyces, and Capnocytophaga species and those free o
21 , for example, between oral streptococci and actinomyces, are central to dental plaque development.
22 revented recolonization of S. mutans but not Actinomyces, as compared with a control peptide or salin
23 ate coaggregation with oral streptococci and Actinomyces biofilm development requires the class C sor
25 between 1988 and 2014, and cases describing Actinomyces breast infections published in the medical l
26 onsistent with their assignment to the genus Actinomyces, but they did not appear to correspond to an
27 of Prevotella, Fusobacterium, Streptococcus, Actinomyces, Capnocytophaga, Selenomonas, and Veillonell
30 to frequent requests of bronchoscopists for Actinomyces cultures combined with a change in microbiol
36 we also demonstrated a complexity within the Actinomyces genus that compromises the biochemical ident
37 th and, in order of decreasing cell numbers, Actinomyces gerencseriae, Bifidobacterium, S. mutans, Ve
38 founders, were observed for A. naeslundii I, Actinomyces gerencseriae, C. gingivalis, E. corrodens, C
41 rst case of disseminated infection with both Actinomyces graevenitzii and Mycobacterium tuberculosis
43 er, Prevotella, Capnocytophaga, Selenomonas, Actinomyces, Granulicatella, and Atopobium were increase
46 types of adhesive fimbriae are expressed by Actinomyces; however, the architecture and the mechanism
48 n intergeneric coaggregation with human oral actinomyces, indicating the specificity of the mutation
49 ive lacrimal gland ductulitis, commonly from Actinomyces infection, should be considered in patients
52 only coisolated with a particular bacterium: Actinomyces israelii was the only Actinomyces spp. coiso
53 a case of spinal cord compression caused by Actinomyces israelii with the coisolation of Fusobacteri
54 species included Actinomyces naeslundii II, Actinomyces israelii, Actinomyces odontolyticus, Veillon
55 ococcus micros, Streptococcus milleri group, Actinomyces israelii, and Arcanobacterium haemolyticum.
56 d coinfection of Fusobacterium nucleatum and Actinomyces israelii, resolving these diagnostic discrep
57 treptococcus micros, Actinomyces naeslundii, Actinomyces israelii, Streptococcus sanguis, Streptococc
59 f a previously undescribed catalase-negative Actinomyces-like bacterium were recovered from human cli
62 between oral viridans group streptococci and actinomyces may play an important role in microbial colo
63 acillus (Haemophilus) actinomycetemcomitans; Actinomyces meyeri was coisolated with Peptostreptococcu
64 gravenitzii, Actinomyces odontolyticus, and Actinomyces meyeri were isolated from respiratory specim
65 bacterium alocis, Actinomyces odontolyticus, Actinomyces meyeri, and Bifidobacterium dentium were all
68 sequence of the chromosomal DNA flanking the Actinomyces naeslundii (formerly A. viscosus) T14V type
70 he Orange-Blue cluster score (which included Actinomyces naeslundii and Eubacterium nodatum) was inve
73 Biofilms of S. mutans, alone or mixed with Actinomyces naeslundii and Streptococcus oralis, were in
74 hogen Streptococcus mutans UA159, as well as Actinomyces naeslundii ATCC 12104 and Streptococcus oral
77 tobacillus acidophilus, Lactobacillus casei, Actinomyces naeslundii genospecies (gsp) 1 and 2, total
78 immune response in saliva to colonization by Actinomyces naeslundii genospecies 1 and 2 was studied i
82 in the presence of Streptococcus oralis and Actinomyces naeslundii steadily formed exopolysaccharide
84 ive with antibody against type 2 fimbriae of Actinomyces naeslundii T14V (anti-type-2) were much less
85 saliva of two human oral commensal bacteria, Actinomyces naeslundii T14V and Streptococcus oralis 34,
90 organisms such as Streptococcus gordonii and Actinomyces naeslundii to the saliva-coated tooth surfac
92 ide to support fimbriae-mediated adhesion of Actinomyces naeslundii was explained by the position of
94 monas gingivalis, Peptostreptococcus micros, Actinomyces naeslundii, Actinomyces israelii, Streptococ
96 eptococcus sanguis, Haemophilus aphrophilus, Actinomyces naeslundii, Fusobacterium nucleatum, and A.
97 d of species found in healthy oral biofilms (Actinomyces naeslundii, Lactobacillus casei, Streptococc
99 he oral key pathogens Enterococcus faecalis, Actinomyces naeslundii, Streptococcus mutans, and Aggreg
100 Streptococcus sanguis and type 2 fimbriated Actinomyces naeslundii, which bound terminal sialic acid
101 and characterized the urease gene cluster of Actinomyces naeslundii, which is one of the pioneer orga
109 ally significant urinary tract infections by Actinomyces neuii (21%) and Corynebacterium aurimucosum
113 apnocytophaga species, Fusobacterium alocis, Actinomyces odontolyticus, Actinomyces meyeri, and Bifid
114 ctinomyces lingnae, Actinomyces gravenitzii, Actinomyces odontolyticus, and Actinomyces meyeri were i
115 nomyces naeslundii II, Actinomyces israelii, Actinomyces odontolyticus, Veillonella parvula, Capnocyt
118 As a pioneer colonizer of the oral cavity, Actinomyces oris expresses proteinaceous pili (also call
119 glycosylation pathway in the oral bacterium Actinomyces oris in which sortase and LCP enzymes operat
120 n a lectin-like activity associated with the Actinomyces oris type 2 fimbria, a surface structure ass
121 iphtheriae and FimA of the type 2 pilus from Actinomyces oris unfold and extend at forces that are th
123 ral microbes, including the coaggregation of Actinomyces oris with Streptococcus oralis that helps to
124 itial colonizers, Streptococcus gordonii and Actinomyces oris, as well as with Veillonella sp. (early
128 the housekeeping sortase SrtA was lethal for Actinomyces oris; yet, all of its predicted cell wall-an
129 ytophaga species and those free of caries by Actinomyces, Prevotella, Selenomonas, Streptococcus, and
131 he 2.4-kb plasmid pAP1 from Arcanobacterium (Actinomyces) pyogenes had sequence similarity within the
133 lytic exotoxin expressed by Arcanobacterium (Actinomyces) pyogenes, is a member of the thiol-activate
138 a hitherto unknown species within the genus Actinomyces related to, albeit distinct from, a group of
139 hes among the subject groups (Streptococcus, Actinomyces, Rothia, and Atopobium), but there were no i
140 tive adolescents were typically colonized by Actinomyces, Selenomonas, Prevotella, and Capnocytophaga
141 r, Streptococcus, and Neisseria and fewer in Actinomyces, Selenomonas, Veillonella, Campylobacter, an
143 ray included: Gemella sanguinis (p = 0.002), Actinomyces sp. HOT 448 (p = 0.003), Prevotella cluster
144 overabundant in the caries-active group were Actinomyces sp. strain B19SC, Streptococcus mutans, and
146 ntified bacterium showed some resemblance to Actinomyces species and related taxa, but biochemical te
148 hydrophobicity, deficient in binding to oral Actinomyces species and to human fibronectin, and unable
150 ity or membrane localization also eliminated Actinomyces species biofilm development and bacterial co
151 tinomycosis is a chronic infection caused by Actinomyces species characterized by abscess formation,
152 , our data suggest that among the mechanisms Actinomyces species employ to persist in the oral cavity
153 med clinically significant included multiple Actinomyces species in wounds, Propionibacterium species
155 ctions in NHS Lothian between 2005 and 2013, Actinomyces species isolated from breast infections refe
156 data suggest that A. gerencseriae and other Actinomyces species may play an important role in caries
160 a hitherto unknown subline within a group of Actinomyces species which includes Actinomyces bovis, th
161 forsythus, three Capnocytophaga species, six Actinomyces species, four Propionibacterium species, and
162 31 (70%) were clearly assigned to recognized Actinomyces species, including 94 isolates assigned to s
163 had higher prevalences of Streptococcus and Actinomyces species, while A. actinomycetemcomitans-posi
164 een high frequency of SUP and high levels of Actinomyces spp, Streptococcus spp., members of the oran
165 es of incidence and clinical associations of Actinomyces spp. add to the currently sparse knowledge o
171 bacterium: Actinomyces israelii was the only Actinomyces spp. coisolated with Actinobacillus (Haemoph
173 We determined the frequency distribution of Actinomyces spp. recovered in a routine clinical laborat
174 and 299 stored clinical isolates of putative Actinomyces spp. referred to the Anaerobe Reference Unit
175 nd disease associations for 21 genogroups of Actinomyces spp. that provide greater insights into appr
179 erella, and Veillonella and 36 of 37 (97.3%) Actinomyces strains, 42 of 46 (91.3%) B. fragilis group
181 enotypic characterization, we found that the Actinomyces/Streptococcus coaggregation is only abolishe
182 ompromises the biochemical identification of Actinomyces that can be performed in most clinical labor
188 ve moieties of FimA, the shaft fimbrillin of Actinomyces type 2 fimbriae, which uniquely mediates the
189 neric coaggregation between streptococci and actinomyces, type 2 fimbriae of actinomyces recognize RP
190 Animals were desalivated, infected with Actinomyces viscosus and Streptococcus mutans (sobrinus)
191 The oral bacteria Actinomyces naeslundii and Actinomyces viscosus are known to contribute to the init
192 e protein containing the catalytic domain of Actinomyces viscosus sialidase, removes cell surface sia
194 ferences in hydrophobicity or coadherence to Actinomyces viscosus were detected between the mutants a
195 e of a genospecies 2 A. naeslundii, formerly Actinomyces viscosus, a portion of a homologue of this g
196 ticola, and Tannerella forsythia, as well as Actinomyces viscosus, Campylobacter rectus/showae, Prevo
197 s from hamsters which had been infected with Actinomyces viscosus, fed a caries-promoting diet, and r
199 ldanamycin (GA) is an antibiotic produced by Actinomyces, which specifically inhibits the function of
201 uently, the interactions of streptococci and actinomyces with the mucinlike domains of these mammalia
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