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1 e of infected female mosquitoes of the genus Aedes.
2 ransmitted mainly by mosquitoes of the genus Aedes.
4 ity inhibitor of Anopheles (An.) gambiae and Aedes (Ae.) aegypti Kir1 channels that incapacitates adu
6 uence of the yellow fever and Dengue vector, Aedes aegypti (Aa), has enabled a comparative phylogenom
7 thione S-transferase (GST) from the mosquito Aedes aegypti (aagste2), selected in the field as a majo
8 omain D7 proteins from Anopheles gambiae and Aedes aegypti (AeD7), respectively, were shown to bind b
12 tein-coding genes is 22% larger than that of Aedes aegypti and 52% larger than that of Anopheles gamb
13 d-borne viruses, is transmitted to humans by Aedes aegypti and A. albopictus mosquitoes in tropical a
14 ype 1 [DENV-1] to DENV-4) are transmitted by Aedes aegypti and A. albopictus mosquitoes, causing up t
15 -emerging arbovirus transmitted to humans by Aedes aegypti and Ae. albopictus mosquitoes, causes debi
17 nya virus (CHIKV) is primarily spread by the Aedes aegypti and Aedes albopictus mosquito vectors.
18 Chikungunya virus (CHIKV) is transmitted by Aedes aegypti and Aedes albopictus mosquitoes and causes
19 virus (DENV1-DENV4) are spread primarily by Aedes aegypti and Aedes albopictus mosquitoes, whose geo
21 porary distribution of their shared vectors, Aedes aegypti and Aedes albopictus remains incomplete an
22 ly includes two key mosquito vector species: Aedes aegypti and Aedes albopictus The model was paramet
23 , we highlight biological characteristics of Aedes aegypti and Aedes albopictus, 2 invasive mosquito
24 s, include the release of Wolbachia-infected Aedes aegypti and Aedes albopictus, for either its virus
25 A unique feature of the R7 photoreceptors in Aedes aegypti and Anopheles gambiae is the extreme apica
30 ediculus humanus humanus, Anopheles gambiae, Aedes Aegypti and Culex pipiens quinquefasciatus is nota
32 mosquitoes (three Anopheles gambiae genomes, Aedes aegypti and Culex quinquefasciatus), tick (Ixodes
37 p and physiology of aaNATs from the mosquito Aedes aegypti and serve as a reference for studying the
38 AT transposons, AeBuster1, from the mosquito Aedes aegypti and TcBuster from the red flour beetle Tri
39 tionally, recombinant ZIKV is infectious for Aedes aegypti and thus provides a means to examine virus
41 idemic spread widely to many countries where Aedes Aegypti as the main transmitting vector is endemic
42 age-specific and density dependent change in Aedes aegypti behaviour towards host cues when exposed t
46 fitness of two clades of DENV serotype 2 in Aedes aegypti cells and mosquitoes collected from the re
48 imensional solution structures of SCP-2 from Aedes aegypti determined by NMR spectroscopy in its liga
52 In this study behavioral assays identified Aedes aegypti females that were insensitive to DEET, and
55 h DENV serotype 2 strain 1232 at sites where Aedes aegypti had or had not probed immediately prior.
56 le as the primary vector for dengue viruses, Aedes aegypti has a long history as a genetic model orga
62 eloped for the major mosquito disease vector Aedes aegypti Here, we describe the generation of multip
63 S1 was successfully detected in spiked adult Aedes aegypti homogenate over a broad dynamic range with
64 s in JH responsive Aag-2 cells revealed that Aedes aegypti homologues of both Met and SRC are require
66 nt populations of the yellow fever mosquito, Aedes aegypti in the southeastern United States and in B
67 pression of a sodium channel, AaNav1-1, from Aedes aegypti in Xenopus oocytes, and the functional exa
68 terfering RNAs (viRNAs), 21 nt in length, in Aedes aegypti infected with the mosquito-borne virus, Si
70 gal-specific immune response in the mosquito Aedes aegypti involves the Toll immune pathway transduce
81 irus (SINV) strain MRE16 efficiently infects Aedes aegypti midgut epithelial cells (MEC), but laborat
85 in displays sequence identities of 70% to an Aedes aegypti mosquito TA receptor, followed by 60% to a
88 V) is primarily transmitted to humans by the Aedes aegypti mosquito, human-to-human transmission has
89 us-Zika virus-spread by the same vector, the Aedes aegypti mosquito, that also carries dengue, yellow
93 n in mosquitoes, we manipulated apoptosis in Aedes aegypti mosquitoes by silencing the expression of
95 nitiated infection and transmission rates in Aedes aegypti mosquitoes comparable to those of the prim
96 n from symptomatic dengue cases (n = 208) to Aedes aegypti mosquitoes during 407 independent exposure
97 f the endosymbiotic bacterium Wolbachia into Aedes aegypti mosquitoes has the potential to greatly re
98 Chikungunya virus is mainly transmitted by Aedes aegypti mosquitoes in tropical and subtropical reg
102 tive disposal of nitrogen waste in blood-fed Aedes aegypti mosquitoes requires alanine aminotransfera
103 ), Cecropin A, and Defensin A, in transgenic Aedes aegypti mosquitoes results in the cooperative anti
104 nate metabolic pathway for urea synthesis in Aedes aegypti mosquitoes that converts uric acid to urea
105 f Wolbachia can reduce the permissiveness of Aedes aegypti mosquitoes to disseminated arboviral infec
108 Field-collected and laboratory-colonized Aedes aegypti mosquitoes were fed on blood containing ea
110 osis during arbovirus infection by infecting Aedes aegypti mosquitoes with a Sindbis virus (SINV) clo
111 achia spreads rapidly through populations of Aedes aegypti mosquitoes, and strongly inhibits infectio
112 ication of dengue virus when introduced into Aedes aegypti mosquitoes, as well as to stimulate chroni
113 ilar rates of infection and dissemination in Aedes aegypti mosquitoes, suggesting differing roles for
114 and DENV together in the saliva of infected Aedes aegypti mosquitoes, these findings suggest a mecha
122 is required for DENV-2 replication in adult Aedes aegypti mosquitos implying that the requirement fo
124 Florida, the mosquitoes Aedes albopictus and Aedes aegypti often co-occur in water-filled containers
125 NV) are transmitted to humans by the bite of Aedes aegypti or Aedes albopictus mosquitoes, with milli
126 t hairpin RNAs (shRNAs) corresponding to the Aedes aegypti orthologs of fasciculation and elongation
127 el driven by meteorological data to simulate Aedes aegypti populations and dengue cases in 23 locatio
130 N(1575)Y + L(1014)F were introduced into an Aedes aegypti sodium channel, AaNav1-1, and the mutants
132 tion; particularly, for the 1.3 GB genome of Aedes aegypti the mean value of prediction Sensitivity a
134 COPI functions in the Yellow Fever mosquito Aedes aegypti to interfere with blood meal digestion.
136 imfast (Slif) from the yellow-fever mosquito Aedes aegypti using codon-optimized heterologous express
137 ful completion of the infection cycle in the Aedes aegypti vector, which is initiated in the midgut t
139 sed of large bacterial-type proteins that in Aedes aegypti were implicated as receptors for Plasmodiu
140 ciatus, and Cx. pipiens) and bridge vectors (Aedes aegypti) have differential impacts on viral mutati
141 rs of human pathogens (Anopheles gambiae and Aedes aegypti) imbibing multiple bloodmeals during a gon
144 antennal lobe of the yellow fever mosquito, Aedes aegypti, a major vector of arboviral diseases.
147 nt region has expanded in Anopheles gambiae, Aedes aegypti, and Tribolium castaneum, while the PF rep
148 nome information for three mosquito species: Aedes aegypti, Anopheles gambiae and Culex quinquefascia
149 e sequence data are available for 3 species, Aedes aegypti, Anopheles gambiae, and Culex quinquefasci
150 Aegyptin, a secreted salivary protein from Aedes aegypti, binds collagen and inhibits platelet aggr
152 uced local populations of the dengue vector, Aedes aegypti, but challenges remain in scale and in sep
153 including DENV and Zika virus transmitted by Aedes aegypti, continue to be a threat to global health
154 ted that several mosquito species, including Aedes aegypti, do not develop beyond the first instar wh
156 ards the vertebrate host and, in the case of Aedes aegypti, increased sensitivity to skin odours.
157 rior studies with the yellow fever mosquito, Aedes aegypti, indicated blood feeding stimulates egg pr
158 quitoes of the major vector of Dengue fever, Aedes aegypti, is cyclic because of its dependence on bl
159 of movement of the primary mosquito vector, Aedes aegypti, local human movements may be an important
160 on hosts and peridomestic mosquitoes, mainly Aedes aegypti, mediate human-to-human transmission.
161 epidemics presumably involve transmission by Aedes aegypti, no direct evidence of vector involvement
163 erkingdom cue for the yellow fever mosquito, Aedes aegypti, seeking blood-meals as well as ovipositio
164 s gambiae and Anopheles coluzzii, as well as Aedes aegypti, the cosmopolitan vector of dengue, chikun
167 opheles gambiae, Culex quinquefasciatus, and Aedes aegypti, the latter an important Zika and Dengue v
168 flavivirus that is primarily transmitted by Aedes aegypti, the mosquito vector also important in tra
169 We present a draft sequence of the genome of Aedes aegypti, the primary vector for yellow fever and d
172 -mediated cassette exchange (RMCE) system to Aedes aegypti, the vector of dengue, chikungunya, and Zi
174 thologous gene of the yellow fever mosquito, Aedes aegypti, to control sex- and tissue-specific expre
175 ignated OX3604C, of the major dengue vector, Aedes aegypti, was engineered to have a repressible fema
178 tissue from larvae of the non-target insect Aedes aegypti, we isolated a number of phage for further
179 ransferred from Drosophila into the mosquito Aedes aegypti, where it can block the transmission of de
180 ines ZIKV infectivity in its mosquito vector Aedes aegypti, which acquires ZIKV via a blood meal.
181 the family of 30-kDa salivary allergens from Aedes aegypti, whose function remained elusive thus far.
203 rium Wolbachia, deliberately introduced into Aedes aegyptimosquitoes, have been shown to be able to s
204 e, and the range of invasive species such as Aedes albopictus (Asian Tiger Mosquito) is expanding.
208 hropod cell lines (derived from An. gambiae, Aedes albopictus and Drosophila melanogaster) and six mo
209 utogenous strain in the Asian tiger mosquito Aedes albopictus and examined an F(1) intercross populat
211 licate in mammalian BHK-21 cells or mosquito Aedes albopictus cells and rapidly reverted catalyticall
213 irus (CHIKV) to infect and be transmitted by Aedes albopictus has increased the geographical range at
215 ive CHIKV strains to an atypical vector, the Aedes albopictus mosquito that is ubiquitously distribut
217 (CHIKV) is transmitted by Aedes aegypti and Aedes albopictus mosquitoes and causes febrile illness w
218 rent Wolbachia strains: wAlbB (isolated from Aedes albopictus mosquitoes) and wStri (isolated from th
219 4) are spread primarily by Aedes aegypti and Aedes albopictus mosquitoes, whose geographic range cont
220 ed to humans by the bite of Aedes aegypti or Aedes albopictus mosquitoes, with millions of infections
223 n of their shared vectors, Aedes aegypti and Aedes albopictus remains incomplete and is complicated b
224 near the homes of coinfected patients, and 1 Aedes albopictus specimen was found to be positive for b
225 y mosquito vector species: Aedes aegypti and Aedes albopictus The model was parameterized and calibra
229 dbis produced from three different mosquito (Aedes albopictus) cell lines; one other insect cell line
230 2011, a population of Asian tiger mosquito (Aedes albopictus) was discovered in Los Angeles (LA) Cou
231 logical characteristics of Aedes aegypti and Aedes albopictus, 2 invasive mosquito species and primar
232 ica and Asia, the adaptation of the virus to Aedes albopictus, a mosquito species with an almost worl
233 ecause of genetic adaptation of the virus to Aedes albopictus, a species that thrives in temperate re
234 wMel strain of Drosophila melanogaster into Aedes albopictus, a vector of dengue and other arbovirus
236 s (Anopheles sinensis, Armigeres subalbatus, Aedes albopictus, Culex quinquefasciatus and Cu. tritaen
237 ease of Wolbachia-infected Aedes aegypti and Aedes albopictus, for either its virus-blocking capabili
240 ficiency in a historically secondary vector, Aedes albopictus, leading to speculation that this was a
242 y, the highly invasive Asian tiger mosquito, Aedes albopictus, rapidly displaced resident populations
243 owed CHIKV to exploit a new epidemic vector, Aedes albopictus, via an A226V substitution in the E1 en
250 de classes were effective against strains of Aedes and Culex mosquitoes, demonstrating that electrost
252 the virus genome showed that EPEV roots the Aedes-associated mosquito-borne flaviviruses, including
253 Yokose virus, and Sokoluk virus and also the Aedes-associated mosquito-borne flaviviruses, which incl
254 enyan populations of the yellow fever vector Aedes bromeliae and its relative Aedes metallicus, and i
255 mic incompatibility, or both; the release of Aedes carrying dominant lethal genes, such as the OX513A
260 to genera, with viromes of mosquitoes of the Aedes genus exhibiting substantially less diversity and
263 erence (RNAi) approach, we demonstrated that Aedes HR3 plays a critical role in a timely termination
265 ence containing an E-box-like motif from the Aedes Kr-h1 gene promoter specifically interacted with a
267 ever vector Aedes bromeliae and its relative Aedes metallicus, and in Mansonia uniformis and Mansonia
268 pipientis wMel is a novel strategy to reduce Aedes mosquito competency for flavivirus infection.
270 ic expansion is attributed to the success of Aedes mosquito vectors, but local epidemiological driver
271 ika Forest and in crushed suspensions of the Aedes mosquito, which is one of the vectors for Zika vir
272 ng infectious disease that is transmitted by Aedes mosquitoes and causes severe health and economic b
273 cycle between nonhuman primates and arboreal Aedes mosquitoes in Southeast Asia and West Africa.
275 between miRNA gene clusters in Anopheles and Aedes mosquitoes, and in D. melanogaster suggest the los
276 al signaling activity is likely conserved in Aedes mosquitoes, because genetic or pharmacologic manip
277 one of the most used insecticides to control Aedes mosquitoes, despite the development of pyrethroid
278 ation as well as source-finding behaviour in Aedes mosquitoes, even after the odour is no longer pres
279 transmitted from human to human by bites of Aedes mosquitoes, recent evidence indicates that ZIKV ca
280 ered in Uganda in 1947 and is transmitted by Aedes mosquitoes, which also act as vectors for dengue a
286 thropod-borne virus transmitted primarily by Aedes mosquitos and is major cause of disease in tropica
287 d in a mixture of bovine red blood cells and Aedes physiological saline, with ATP as a phagostimulant
288 s of mosquitoes to eliminate or modify local Aedes populations are being developed, with several curr
289 thought to be due to failure to control the Aedes populations, uncontrolled urbanization, population
290 eviously was isolated from Culex pipiens and Aedes rossicus mosquitoes in the Czech Republic, and phy
296 irus (DENV) and its primary mosquito vectors Aedes spp. have spread to every continent except Antarct
297 ead of other arboviruses carried by invasive Aedes spp., such as Chikungunya and Zika, seem to be fol
298 consider the potential role of the mosquito Aedes taeniorhynchus in maintaining the flavivirus West
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