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1 ea squirt (Ciona savignyi) and the mosquito (Aedes aegypti).
2 e transmitted by the disease vector mosquito Aedes aegypti.
3 t maintenance in the dengue mosquito vector, Aedes aegypti.
4 n (PE) development in the fat body of female Aedes aegypti.
5 feeding and egg development in the mosquito Aedes aegypti.
6 f fruit flies and the yellow fever mosquito, Aedes aegypti.
7 pora allata-corpora cardiaca of the mosquito Aedes aegypti.
8 cally relevant range for humans and infected Aedes aegypti.
9 ivergent relative, the yellow fever mosquito Aedes aegypti.
10 2 cDNA clone from the yellow fever mosquito, Aedes aegypti.
11 ation of Toll immune pathway in the mosquito Aedes aegypti.
12 in some important mosquito vectors, notably Aedes aegypti.
13 was discovered in the yellow fever mosquito, Aedes aegypti.
14 ene from transgenic yellow fever mosquitoes, Aedes aegypti.
15 ogous to Drosophila Relish from the mosquito Aedes aegypti.
16 n biosynthesis in the yellow fever mosquito, Aedes aegypti.
17 ant than MITEs in the yellow fever mosquito, Aedes aegypti.
18 imilar kinetics as seen in the mosquito host Aedes aegypti.
19 th distinct N-termini, occur in the mosquito Aedes aegypti.
20 alivary glands of the yellow fever mosquito, Aedes aegypti.
21 m the hemolymph of bacteria-challenged adult Aedes aegypti.
22 anticoagulant of the yellow fever mosquito, Aedes aegypti.
23 e to the biology and behavior of its vector, Aedes aegypti.
24 embryonic soma of the yellow fever mosquito, Aedes aegypti.
25 his mosquito species using RFLP markers from Aedes aegypti.
26 are described in the yellow fever mosquito, Aedes aegypti.
27 isolated from a cDNA library of the mosquito Aedes aegypti.
28 emale fat body cDNA library of the mosquito, Aedes aegypti.
29 Habrobracon) hebetor and a diploid mosquito. Aedes aegypti.
30 micro-injection into embryos of the mosquito Aedes aegypti.
31 quefasciatus, and the yellow fever mosquito, Aedes aegypti.
32 y arthropods, generally, using the mosquito, Aedes aegypti.
33 homeostasis and dengue virus replication in Aedes aegypti.
34 E-binding proteins from the mosquito species Aedes aegypti.
35 es from the mosquitoes Anopheles gambiae and Aedes aegypti.
36 alanine aminotransferase (ALAT) in blood-fed Aedes aegypti.
37 eles stephensi and the yellow fever mosquito Aedes aegypti.
38 he male and female germline, in the mosquito Aedes aegypti.
39 of spermatozoa in the yellow fever mosquito, Aedes aegypti.
41 the impact of transgenesis on the fitness of Aedes aegypti, a mosquito that transmits yellow fever.
42 smodium protozoan agent causing malaria, and Aedes aegypti, a vector for the flaviviral agents causin
43 splicing in Manduca sexta, Bombyx mori, and Aedes aegypti: A C-terminally amidated ITP and a C-termi
44 uence of the yellow fever and Dengue vector, Aedes aegypti (Aa), has enabled a comparative phylogenom
46 thione S-transferase (GST) from the mosquito Aedes aegypti (aagste2), selected in the field as a majo
47 midgut of the Yellow Fever vector mosquito, Aedes aegypti (aeAAT1, AAR08269), which primarily suppli
48 l crystal structure of AGT from the mosquito Aedes aegypti (AeAGT) and structures of its complexes wi
49 omain D7 proteins from Anopheles gambiae and Aedes aegypti (AeD7), respectively, were shown to bind b
51 in immunity by generating a total of 11,952 Aedes aegypti and 12,790 Armigeres subalbatus expressed
52 tein-coding genes is 22% larger than that of Aedes aegypti and 52% larger than that of Anopheles gamb
53 d-borne viruses, is transmitted to humans by Aedes aegypti and A. albopictus mosquitoes in tropical a
54 ype 1 [DENV-1] to DENV-4) are transmitted by Aedes aegypti and A. albopictus mosquitoes, causing up t
55 -emerging arbovirus transmitted to humans by Aedes aegypti and Ae. albopictus mosquitoes, causes debi
57 smitted among humans by the mosquito vectors Aedes aegypti and Aedes albopictus are hypothesized to h
58 nya virus (CHIKV) is primarily spread by the Aedes aegypti and Aedes albopictus mosquito vectors.
59 Chikungunya virus (CHIKV) is transmitted by Aedes aegypti and Aedes albopictus mosquitoes and causes
60 virus (DENV1-DENV4) are spread primarily by Aedes aegypti and Aedes albopictus mosquitoes, whose geo
62 porary distribution of their shared vectors, Aedes aegypti and Aedes albopictus remains incomplete an
63 ly includes two key mosquito vector species: Aedes aegypti and Aedes albopictus The model was paramet
64 , we highlight biological characteristics of Aedes aegypti and Aedes albopictus, 2 invasive mosquito
65 s, include the release of Wolbachia-infected Aedes aegypti and Aedes albopictus, for either its virus
67 A unique feature of the R7 photoreceptors in Aedes aegypti and Anopheles gambiae is the extreme apica
72 ediculus humanus humanus, Anopheles gambiae, Aedes Aegypti and Culex pipiens quinquefasciatus is nota
74 mosquitoes (three Anopheles gambiae genomes, Aedes aegypti and Culex quinquefasciatus), tick (Ixodes
79 s assumption using the yellow fever mosquito Aedes aegypti and malaria parasite Plasmodium gallinaceu
80 reduced the infectivity of P. gallinaceum to Aedes aegypti and P. falciparum to Anopheles mosquitoes.
81 p and physiology of aaNATs from the mosquito Aedes aegypti and serve as a reference for studying the
82 or Anopheles gambiae and a D7 long form from Aedes aegypti and showed by isothermal microcalorimetry,
83 AT transposons, AeBuster1, from the mosquito Aedes aegypti and TcBuster from the red flour beetle Tri
84 tionally, recombinant ZIKV is infectious for Aedes aegypti and thus provides a means to examine virus
85 er accession numbers AY 431103 to AY 433788 (Aedes aegypti) and AY 439334 to AY 441440 (Armigeres sub
86 om two other insects (Drosophila virilis and Aedes aegypti) and three vertebrates (Homo sapiens, Mus
89 nt region has expanded in Anopheles gambiae, Aedes aegypti, and Tribolium castaneum, while the PF rep
90 nome information for three mosquito species: Aedes aegypti, Anopheles gambiae and Culex quinquefascia
91 e sequence data are available for 3 species, Aedes aegypti, Anopheles gambiae, and Culex quinquefasci
93 idemic spread widely to many countries where Aedes Aegypti as the main transmitting vector is endemic
94 age-specific and density dependent change in Aedes aegypti behaviour towards host cues when exposed t
95 Aegyptin, a secreted salivary protein from Aedes aegypti, binds collagen and inhibits platelet aggr
98 uced local populations of the dengue vector, Aedes aegypti, but challenges remain in scale and in sep
100 enin (Vg) gene in the yellow fever mosquito, Aedes aegypti, by EcR/USP and E74 in response to an elev
103 fitness of two clades of DENV serotype 2 in Aedes aegypti cells and mosquitoes collected from the re
105 including DENV and Zika virus transmitted by Aedes aegypti, continue to be a threat to global health
106 Overexpressing wild-type AeSCP-2 in the Aedes aegypti cultured Aag-2 cells resulted in an increa
109 e nuclear factor-4 (HNF-4) from the mosquito Aedes aegypti, designated AaHNF-4a, AaHNF-4b, and AaHNF-
110 imensional solution structures of SCP-2 from Aedes aegypti determined by NMR spectroscopy in its liga
112 ted that several mosquito species, including Aedes aegypti, do not develop beyond the first instar wh
113 ets, S6 kinase, of the yellow fever mosquito Aedes aegypti during egg development in adult females.
117 In this study behavioral assays identified Aedes aegypti females that were insensitive to DEET, and
120 tory region of the vitellogenin (Vg) gene of Aedes aegypti for its ability to express potential antip
121 We tested female yellow fever mosquitoes (Aedes aegypti) for responses to 8-hydroxy-8-methyl-6-(2'
123 Two approaches were used to correlate the Aedes aegypti genetic linkage map to the physical map.
124 h DENV serotype 2 strain 1232 at sites where Aedes aegypti had or had not probed immediately prior.
126 le as the primary vector for dengue viruses, Aedes aegypti has a long history as a genetic model orga
132 ciatus, and Cx. pipiens) and bridge vectors (Aedes aegypti) have differential impacts on viral mutati
133 eloped for the major mosquito disease vector Aedes aegypti Here, we describe the generation of multip
134 S1 was successfully detected in spiked adult Aedes aegypti homogenate over a broad dynamic range with
135 s in JH responsive Aag-2 cells revealed that Aedes aegypti homologues of both Met and SRC are require
137 rs of human pathogens (Anopheles gambiae and Aedes aegypti) imbibing multiple bloodmeals during a gon
139 nt populations of the yellow fever mosquito, Aedes aegypti in the southeastern United States and in B
140 pression of a sodium channel, AaNav1-1, from Aedes aegypti in Xenopus oocytes, and the functional exa
141 ards the vertebrate host and, in the case of Aedes aegypti, increased sensitivity to skin odours.
142 rior studies with the yellow fever mosquito, Aedes aegypti, indicated blood feeding stimulates egg pr
143 terfering RNAs (viRNAs), 21 nt in length, in Aedes aegypti infected with the mosquito-borne virus, Si
145 gal-specific immune response in the mosquito Aedes aegypti involves the Toll immune pathway transduce
156 quitoes of the major vector of Dengue fever, Aedes aegypti, is cyclic because of its dependence on bl
157 an YF, owing to transmission of the virus by Aedes aegypti, is increasing in Africa, as is the potent
162 of movement of the primary mosquito vector, Aedes aegypti, local human movements may be an important
165 on hosts and peridomestic mosquitoes, mainly Aedes aegypti, mediate human-to-human transmission.
166 irus (SINV) strain MRE16 efficiently infects Aedes aegypti midgut epithelial cells (MEC), but laborat
170 ragment length polymorphisms of four natural Aedes aegypti mosquito populations from Trinidad and Tob
172 in displays sequence identities of 70% to an Aedes aegypti mosquito TA receptor, followed by 60% to a
175 V) is primarily transmitted to humans by the Aedes aegypti mosquito, human-to-human transmission has
176 us-Zika virus-spread by the same vector, the Aedes aegypti mosquito, that also carries dengue, yellow
178 eudoobscura, and homologs have been found in Aedes aegypti mosquitoes and in four other insect orders
181 ntitrypsin antibodies blocked infectivity of Aedes aegypti mosquitoes by Plasmodium gallinaceum.
182 n in mosquitoes, we manipulated apoptosis in Aedes aegypti mosquitoes by silencing the expression of
184 nitiated infection and transmission rates in Aedes aegypti mosquitoes comparable to those of the prim
185 n from symptomatic dengue cases (n = 208) to Aedes aegypti mosquitoes during 407 independent exposure
186 f the endosymbiotic bacterium Wolbachia into Aedes aegypti mosquitoes has the potential to greatly re
187 Chikungunya virus is mainly transmitted by Aedes aegypti mosquitoes in tropical and subtropical reg
191 tive disposal of nitrogen waste in blood-fed Aedes aegypti mosquitoes requires alanine aminotransfera
192 ), Cecropin A, and Defensin A, in transgenic Aedes aegypti mosquitoes results in the cooperative anti
193 nate metabolic pathway for urea synthesis in Aedes aegypti mosquitoes that converts uric acid to urea
194 ogaviridae) strain MRE16 efficiently infects Aedes aegypti mosquitoes that ingest a blood meal contai
195 f Wolbachia can reduce the permissiveness of Aedes aegypti mosquitoes to disseminated arboviral infec
198 Field-collected and laboratory-colonized Aedes aegypti mosquitoes were fed on blood containing ea
200 osis during arbovirus infection by infecting Aedes aegypti mosquitoes with a Sindbis virus (SINV) clo
201 say systems (using human dendritic cells and Aedes aegypti mosquitoes) for measuring differences in v
202 achia spreads rapidly through populations of Aedes aegypti mosquitoes, and strongly inhibits infectio
203 ication of dengue virus when introduced into Aedes aegypti mosquitoes, as well as to stimulate chroni
204 ilar rates of infection and dissemination in Aedes aegypti mosquitoes, suggesting differing roles for
205 these MAbs on P. gallinaceum infectivity for Aedes aegypti mosquitoes, the addition of MAbs 1A6 and 2
206 and DENV together in the saliva of infected Aedes aegypti mosquitoes, these findings suggest a mecha
215 is required for DENV-2 replication in adult Aedes aegypti mosquitos implying that the requirement fo
219 epidemics presumably involve transmission by Aedes aegypti, no direct evidence of vector involvement
220 report on the characterization in transgenic Aedes aegypti of two mosquito gut-specific promoters.
221 Florida, the mosquitoes Aedes albopictus and Aedes aegypti often co-occur in water-filled containers
222 NV) are transmitted to humans by the bite of Aedes aegypti or Aedes albopictus mosquitoes, with milli
223 t hairpin RNAs (shRNAs) corresponding to the Aedes aegypti orthologs of fasciculation and elongation
226 hree different vector-parasite combinations, Aedes aegypti-Plasmodium gallinaceum, Anopheles stephens
227 el driven by meteorological data to simulate Aedes aegypti populations and dengue cases in 23 locatio
228 nctioned as a potent ligand for the mosquito Aedes aegypti receptor complex (AaEcR-USP), significantl
231 erkingdom cue for the yellow fever mosquito, Aedes aegypti, seeking blood-meals as well as ovipositio
233 N(1575)Y + L(1014)F were introduced into an Aedes aegypti sodium channel, AaNav1-1, and the mutants
236 test this hypothesis, germ-line-transformed Aedes aegypti that express luciferase (LUC) from the mos
237 tion; particularly, for the 1.3 GB genome of Aedes aegypti the mean value of prediction Sensitivity a
239 s gambiae and Anopheles coluzzii, as well as Aedes aegypti, the cosmopolitan vector of dengue, chikun
240 e waves of invasion of the vector mosquitoes Aedes aegypti, the Culex pipiens Complex, and, most rece
241 sion and that, in the yellow fever mosquito, Aedes aegypti, the expression of the ferritin heavy-chai
244 opheles gambiae, Culex quinquefasciatus, and Aedes aegypti, the latter an important Zika and Dengue v
245 flavivirus that is primarily transmitted by Aedes aegypti, the mosquito vector also important in tra
246 We present a draft sequence of the genome of Aedes aegypti, the primary vector for yellow fever and d
249 -mediated cassette exchange (RMCE) system to Aedes aegypti, the vector of dengue, chikungunya, and Zi
252 (QTL) affecting the ability of the mosquito Aedes aegypti to become infected with dengue-2 virus wer
253 COPI functions in the Yellow Fever mosquito Aedes aegypti to interfere with blood meal digestion.
254 thologous gene of the yellow fever mosquito, Aedes aegypti, to control sex- and tissue-specific expre
255 determining susceptibility of the mosquito, Aedes aegypti, to the malarial parasite, Plasmodium gall
257 Feeding of soybean trypsin inhibitor and Aedes aegypti trypsin modulating oostatic factor affecte
258 imfast (Slif) from the yellow-fever mosquito Aedes aegypti using codon-optimized heterologous express
259 An antiserum raised against recombinant Aedes aegypti V-ATPase B subunit indicated that the majo
260 ful completion of the infection cycle in the Aedes aegypti vector, which is initiated in the midgut t
261 aper, we analyzed the upstream region of the Aedes aegypti Vg gene in order to identify regulatory el
264 ve linkage map of the yellow fever mosquito, Aedes aegypti, was constructed using single-strand confo
265 ignated OX3604C, of the major dengue vector, Aedes aegypti, was engineered to have a repressible fema
268 tissue from larvae of the non-target insect Aedes aegypti, we isolated a number of phage for further
269 sed of large bacterial-type proteins that in Aedes aegypti were implicated as receptors for Plasmodiu
270 pheles gambiae and the yellow fever mosquito Aedes aegypti were searched by Blast against each Plus-C
272 ransferred from Drosophila into the mosquito Aedes aegypti, where it can block the transmission of de
273 ines ZIKV infectivity in its mosquito vector Aedes aegypti, which acquires ZIKV via a blood meal.
274 oid X receptor homologue, from the mosquito, Aedes aegypti, which form a functional complex capable o
275 the family of 30-kDa salivary allergens from Aedes aegypti, whose function remained elusive thus far.
276 t Sindbis virus was used to transduce female Aedes aegypti with a 567-base antisense RNA targeted to
277 ional structure of the complex of SCP-2 from Aedes aegypti with a C16 fatty acid to 1.35-A resolution
278 ted immune deficiency (RMID) by transforming Aedes aegypti with the Delta Rel transgene driven by the
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