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1 biofilm formation by the periodontopathogen Aggregatibacter actinomycetemcomitans.
2 se caused by Cdt-producing organisms such as Aggregatibacter actinomycetemcomitans.
3 omyces naeslundii, Streptococcus mutans, and Aggregatibacter actinomycetemcomitans.
4 scherichia coli and leukotoxin A (LtxA) from Aggregatibacter actinomycetemcomitans.
5 4 to 5 mm and >/= 6 mm, BOP, and ABL, except Aggregatibacter actinomycetemcomitans.
6 files, especially those testing positive for Aggregatibacter actinomycetemcomitans.
7 .884) had a decreased colonization risk with Aggregatibacter actinomycetemcomitans.
8 in that is secreted from the oral bacterium, Aggregatibacter actinomycetemcomitans.
9 the remaining five implants, per group, with Aggregatibacter actinomycetemcomitans.
10 odontopathogens Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans.
11 usobacterium nucleatum (a middle colonizer), Aggregatibacter actinomycetemcomitans (a late colonizer)
13 high orange" bacterial complexes, and "high" Aggregatibacter actinomycetemcomitans (Aa) colonization.
14 duce the proinflammatory response induced by Aggregatibacter actinomycetemcomitans (Aa) in human coro
15 ent species in a bacterial biofilm of Sg and Aggregatibacter actinomycetemcomitans (Aa) in terms of h
16 of periodontal disease (PD) associated with Aggregatibacter actinomycetemcomitans (Aa) infection.
19 nd quantifying Fusobacterium nucleatum (Fn), Aggregatibacter actinomycetemcomitans (Aa), Porphyromona
20 tal amount of Porphyromonas gingivalis (Pg), Aggregatibacter actinomycetemcomitans (Aa), Tannerella f
21 n (Ig) G response, and alveolar bone loss in Aggregatibacter actinomycetemcomitans (Aa)-inoculated Fa
23 gnificant differences in serum IgG levels to Aggregatibacter actinomycetemcomitans among the four dia
25 xamined in vivo and in vitro colonization by Aggregatibacter actinomycetemcomitans, an organism highl
26 lus systems from several bacteria, including Aggregatibacter actinomycetemcomitans and Caulobacter cr
27 mice were infected on days 0, 2, and 4 with Aggregatibacter actinomycetemcomitans and divided into g
29 oad, whereas PCT3 and PCT5 were dominated by Aggregatibacter actinomycetemcomitans and Porphyromonas
31 was observed against the periodontopathogens Aggregatibacter actinomycetemcomitans and Porphyromonas
32 by a 3-log reduction in periodontopathogenic Aggregatibacter actinomycetemcomitans and Porphyromonas
33 In the present study, the aim is to measure Aggregatibacter actinomycetemcomitans and Porphyromonas
35 ofilm consisting of the periodontal pathogen Aggregatibacter actinomycetemcomitans, and a commensal S
36 iodontal bacteria: Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans, and Fusobacterium
37 susceptibility of Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans, and Fusobacterium
38 d substantial evidence of the association of Aggregatibacter actinomycetemcomitans, and its highly le
39 phyromonas gingivalis, Tannerella forsythia, Aggregatibacter actinomycetemcomitans, and total bacteri
40 formation and virulence of the oral pathogen Aggregatibacter actinomycetemcomitans, and we previously
42 by Staphylococcus aureus, S. epidermidis and Aggregatibacter actinomycetemcomitans, but not by A. ple
43 cy strains were clustered into eight groups: Aggregatibacter actinomycetemcomitans, Campylobacter spp
45 parainfluenzae, Aggregatibacter aphrophilus, Aggregatibacter actinomycetemcomitans, Cardiobacterium h
46 ng toxin (Cdt) from the periodontal pathogen Aggregatibacter actinomycetemcomitans causes extensive d
47 the phagocytic ability of mast cells against Aggregatibacter actinomycetemcomitans compared with macr
48 arrest in lymphocytes after exposure to the Aggregatibacter actinomycetemcomitans cytolethal distend
49 est in lymphocytes following exposure to the Aggregatibacter actinomycetemcomitans cytolethal distend
50 s physiologic support, and pathogens such as Aggregatibacter actinomycetemcomitans display resource p
51 growth with streptococci, the oral pathogen Aggregatibacter actinomycetemcomitans displays enhanced
54 onstrate that CDTs from Haemophilus ducreyi, Aggregatibacter actinomycetemcomitans, Escherichia coli,
57 s protease-deficient mutant KDP128, and live Aggregatibacter actinomycetemcomitans In contrast, infec
58 carrier rate of JP2 and non-JP2 genotypes of Aggregatibacter actinomycetemcomitans in the Ghanaian ad
60 injections of lipopolysaccharide (LPS) from Aggregatibacter actinomycetemcomitans in wild-type (WT)
61 es of the Gram-negative periodontal pathogen Aggregatibacter actinomycetemcomitans include serotype a
63 d with pathogens Fusobacterium nucleatum and Aggregatibacter actinomycetemcomitans, increased GCF-IL-
66 (Cdt), expressed by the periodontal pathogen Aggregatibacter actinomycetemcomitans, inhibits the prol
68 esin A (EmaA) of the Gram-negative bacterium Aggregatibacter actinomycetemcomitans is a fibrillar col
78 iofilm formation by the periodontal pathogen Aggregatibacter actinomycetemcomitans is dependent upon
82 gram-negative fastidious human oropharyngeal Aggregatibacter actinomycetemcomitans is implicated in t
85 s were found to inhibit biofilm formation by Aggregatibacter actinomycetemcomitans, Klebsiella pneumo
86 obacterium nucleatum (middle colonizer), and Aggregatibacter actinomycetemcomitans (late colonizer).
88 eponema denticola, Tannerella forsythia, and Aggregatibacter actinomycetemcomitans levels in subgingi
89 ontrol lean rats by periodontal injection of Aggregatibacter actinomycetemcomitans lipopolysaccharide
94 oral pathogens Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans perturbed the tran
95 e chain reaction determined total bacterial, Aggregatibacter actinomycetemcomitans, Porphyromonas gin
96 species were assessed at baseline, including Aggregatibacter actinomycetemcomitans, Porphyromonas gin
97 d total and quantitative bacterial counts of Aggregatibacter actinomycetemcomitans, Porphyromonas gin
100 gation is to quantify periodontal pathogens (Aggregatibacter actinomycetemcomitans, Porphyromonas gin
103 tive real-time polymerase chain reaction for Aggregatibacter actinomycetemcomitans, Porphyromonas gin
104 Subgingival plaque samples were analyzed for Aggregatibacter actinomycetemcomitans, Porphyromonas gin
105 (tooth and site) factors on the detection of Aggregatibacter actinomycetemcomitans (previously Actino
106 lowed to incubate in a bacterial solution of Aggregatibacter actinomycetemcomitans (previously Actino
108 for serum immunoglobulin G (IgG) antibody to Aggregatibacter actinomycetemcomitans (previously Actino
109 rase chain reaction determined the number of Aggregatibacter actinomycetemcomitans (previously Actino
110 iodontitis are associated with infections by Aggregatibacter actinomycetemcomitans (previously Actino
112 sly T. forsythensis), Prevotella intermedia, Aggregatibacter actinomycetemcomitans (previously Actino
113 serum immunoglobulin G antibody response to Aggregatibacter actinomycetemcomitans (previously Actino
114 esent study evaluated seven target bacteria, Aggregatibacter actinomycetemcomitans (previously Actino
115 igher frequency of Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans (previously Actino
116 l animal model to study the host response to Aggregatibacter actinomycetemcomitans (previously Actino
117 ove the minimal inhibitory concentration for Aggregatibacter actinomycetemcomitans (previously Actino
118 ide PMNs could enhance their ability to kill Aggregatibacter actinomycetemcomitans (previously Actino
120 w cytometry in response to Escherichia coli, Aggregatibacter actinomycetemcomitans (previously Actino
121 odontal pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans [previously Actino
126 similarity to the Escherichia coli LsrB and Aggregatibacter actinomycetemcomitans RbsB proteins that
130 pecific pathogens (Porphyromonas gingivalis, Aggregatibacter actinomycetemcomitans, Tannerella forsyt
132 ted with aggressive periodontitis-associated Aggregatibacter actinomycetemcomitans triggered a type I
133 lla intermedia, Fusobacterium nucleatum, and Aggregatibacter actinomycetemcomitans was examined using
134 votella intermedia, Treponema denticola, and Aggregatibacter actinomycetemcomitans was identified by
135 g toxin (Cdt), from the periodontal pathogen Aggregatibacter actinomycetemcomitans, was conjugated to
137 a/nigrescens, Streptococcus constellatus, or Aggregatibacter actinomycetemcomitans, were resistant in
138 gens, including Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans, were similarly re
139 al activity against the periodontal pathogen Aggregatibacter actinomycetemcomitans when the bacteria
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