ライフサイエンスコーパス: Alcar

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1 e (K(i) = 0.05 +/- 0.02 microM for 4-N-allyl-AlCAR).

2 d after feeding old rats acetyl-L-carnitine (ALCAR).

3 tochondrial metabolites, acetyl-l-carnitine (ALCAR) [0.5% or 0.2% (wt/vol) in drinking water], and/or

4 the dietary addition of acetyl-l-carnitine [ALCAR, 1.5% (wt/vol) in the drinking water] and/or (R)-a

5 nificantly restored CAT-binding affinity for ALCAR and CoA, and CAT activity.

6 in CAT-binding affinity for both substrates, ALCAR and CoA.

7 These results suggest that feeding ALCAR and LA to old rats improves performance on memory

8 Dietary supplementation with ALCAR and/or LA improved memory, the combination being t

9 copic studies in the hippocampus showed that ALCAR and/or LA reversed age-associated mitochondrial st

10 Dietary administration of ALCAR and/or LA significantly reduced the extent of oxid

11 s with the CAT substrate acetyl-l-carnitine (ALCAR) and/or the mitochondrial antioxidant precursor R-

12 n, while restoration of SIRT3 with AICAR and ALCAR improved cisplatin-induced mitochondrial dysfuncti

13 Feeding ALCAR in combination with LA increased metabolism and lo

14 rats were given a 1.5% (wt/vol) solution of ALCAR in their drinking water for 1 mo, were sacrificed,

15 ALCAR increases cellular oxygen consumption, which decli

16 Feeding old rats LA or LA plus ALCAR inhibited lipid peroxidation but did not decrease

17 To determine whether ALCAR+LA also affected indices of oxidative stress, asco

18 ALCAR+LA also increased ambulatory activity in both youn

19 ALCAR+LA partially reversed the age-related decline in a

20 n old versus young rats, also declined after ALCAR+LA supplementation and was not significantly diffe

21 estored to the level seen in young rats when ALCAR+LA was given.

22 Feeding ALCAR or ALCAR plus LA to old rats significantly restore

23 Preincubation of CAT with ALCAR or CoA prevented malondialdehyde-induced dysfuncti

24 also greater when compared with old rats fed ALCAR or LA alone.

25 Feeding ALCAR or ALCAR plus LA to old rats significantly restored CAT-bin

26 or the antioxidant agent acetyl-l-carnitine (ALCAR) restored SIRT3 expression and activity, improved

27 ALCAR supplementation increases ambulatory activity sign

28 untreated old rats, further indicating that ALCAR supplementation might increase oxidative stress.

29 ALCAR supplementation significantly reverses the age-ass

30 Thus, ALCAR supplementation to old rats markedly reverses the

31 % and 50% lower, respectively, in cells from ALCAR-supplemented old rats than in untreated old rats,

32 n WT animals and died, and neither AICAR nor ALCAR treatment prevented death in Sirt3-/- AKI mice.

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