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1 enyls that are nonmutagenic (inactive in the Ames test).
2 ity using the Salmonella mutagenicity assay (Ames test).
3 zole metabolite that would be negative in an Ames test.
4  antimutagenic activity was evaluated by the Ames test.
5  4-aminobiphenyls that are not active in the Ames test.
6  microM and was inactive as a mutagen in the Ames test.
7 parations to activate IQ to a mutagen in the Ames test.
8 phimurium TA102 strain, as determined by the Ames test.
9 st (i.e., human lungs) and not only with the Ames test.
10 s anti-mutagenic potential by performing the Ames test against heterocyclic amines (HCAs), in continu
11 c extract did not showed mutagenic effect by Ames test against Salmonella typhimurium TA98 and TA100
12  channel blockage testing, negativity in the Ames test, and 5/5 cure @ <15 mpk x 3 in mice infected w
13 ic or phototoxic effects, is negative in the Ames test, and affects cytochrome P450-dependent enzymes
14 odent liver TD(50) and lethal dose (LD(50)), Ames tests, and Comet assays for in vitro DNA damage.
15     Furthermore, 14 was also negative in the AMES test for genotoxicity.
16 While many institutions use a version of the Ames test in the undergraduate genetics laboratory, stud
17 n the educational content of the traditional Ames test teaching laboratory, while simultaneously enha
18                         We have extended the Ames test to include both quantitative analysis of rever
19 o found to be a direct-acting mutagen in the Ames test using Salmonella typhimurium tester strains TA
20 pound revealed bacterial mutagenicity in the Ames test using TA97a Salmonella strain, and subsequent
21                                          The Ames test was used to show that propionaldehyde is a mut
22             Comparative mutagenicity assays (Ames test) were performed on 1 versus 1NO2, 5 versus 5NO
23 he biological activity of flavonoids, by the Ames test, with Salmonella typhimurium strains TA98, TA1

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