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1 stratum lacunosum-moleculare of field CA1 of Ammon's horn.
2 tons and somata in the stratum oriens of the Ammon's horn.
3 the ventricular and subventricular zones of Ammon's horn.
4 yramidal cells in the CA1 and CA3 regions of Ammon's horn.
5 r to projection cells found in the mammalian Ammon's horn.
6 te gyrus and remodelling of dendrites in the Ammon's horn.
7 e cortex (1.8), CA1, CA2, and CA3 regions of Ammon's horn (1.6-2.0), dentate gyrus (1.7), laminar VI
8 dala, perirhinal and entorhinal cortices, or Ammon's horn and dentate gyrus all initially displayed f
9 the ventricular and subventricular zones of Ammon's horn and from the prime germinal zone of the den
10 uld be observed in the stratum oriens of the Ammon's horn and subiculum, fewer were seen in the denta
12 ound degeneration of neurons and synapses in Ammon's horn and the dentate gyrus of the hippocampal fo
13 nt elevations in BDNF mRNA were localized in Ammon's horn areas 1 (CA1) and 4 (CA4) of the hippocampu
15 cal circuit neurons in the dentate gyrus and Ammon's horn contained GAT-1 immunoreactivity associated
17 s, which coincides with the CA2 subregion of Ammon's horn in the hippocampus, stains intensely for bF
18 ssion in the dentate gyrus and CA3 region of Ammon's horn, increases that were inhibited by tianeptin
19 fundamental property of neuronal networks in Ammon's horn is that each area comprises a single glutam
21 1-2 and NMDAR1-2 hybridization densities for Ammon's horn pyramids and (ii) similar or numerically de
22 (ii) increased AMPA GluR3 mRNA densities for Ammon's horn pyramids; and (iii) similar or numerically
25 is a long-standing controversy as to whether Ammon's horn sclerosis is the result or the cause of sev
26 tis, the pathology of status epilepticus and Ammon's horn sclerosis, and the systematic investigation
27 lective labeling of only apical dendrites in Ammon's horn subregion la (CA1a) to a diffuse band of pu
28 (granule cell layer), and the CA1 region of Ammon's horn, which is the principal site of hippocampal
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