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1 mum spatial resolution of the malaria vector Anopheles.
2 robiota can impede Wolbachia transmission in Anopheles.
3 ding Drosophila and the mosquitoes Aedes and Anopheles.
5 m the salivary glands of the malaria vector, Anopheles albimanus The inhibitor was purified from sali
6 Haiti from May to September, 2012, where the Anopheles albimanus vector bites primarily outdoors and
7 VU041, a submicromolar-affinity inhibitor of Anopheles (An.) gambiae and Aedes (Ae.) aegypti Kir1 cha
8 ce of the mechanisms regulating fertility in Anopheles, and identify HPX15 as a target for vector con
9 smitted to humans by mosquitoes of the genus Anopheles, and these insects are the targets of innovati
13 Anopheles gambiae sensu stricto (s.s.), and Anopheles arabiensis were estimated, and their effects w
15 ons on the possible use of natural Wolbachia-Anopheles associations to limit malaria transmission.
17 CO(2)-sensitive ORN in the malaria mosquito Anopheles can also be inhibited by excitation of an adja
18 ce of Wolbachia in females from a laboratory Anopheles coluzzii (A. gambiae, M form) colony experimen
20 es and consequences of a hybrid zone between Anopheles coluzzii and Anopheles gambiae in Guinea-Bissa
21 istance between two African malaria vectors, Anopheles coluzzii and Anopheles gambiae, have been attr
22 e chemical composition and attractiveness to Anopheles coluzzii mosquitoes of skin odours from partic
23 ed Anopheles gambiae s.l. males (all M form (Anopheles coluzzii)) were collected from mating swarms i
24 time-series analyses, the seasonal cycles of Anopheles coluzzii, Anopheles gambiae sensu stricto (s.s
25 frican malaria vectors Anopheles gambiae and Anopheles coluzzii, as well as Aedes aegypti, the cosmop
28 Eilat virus (EILV), isolated from a pool of Anopheles coustani mosquitoes from the Negev desert of I
31 es of the New World species A. albimanus and Anopheles darlingi) also played some functionally releva
33 Moreover, human landing catches of sylvan Anopheles demonstrated the propensity of these three vec
34 sporozoites in two Southeast Asian vectors, Anopheles dirus and Anopheles minimus, and the major Afr
38 control of major malaria vectors, including Anopheles funestus Effective management of resistance re
40 this insecticide class in the malaria vector Anopheles funestus is spreading rapidly across Africa, t
44 s) may induce a switch in biting behavior in Anopheles funestus, a major malaria vector in Africa.
46 major malaria vectors Anopheles gambiae and Anopheles funestus, with a higher frequency of resistanc
47 % of which have their closest counterpart in Anopheles gambiae and 21% have highest similarity with o
51 e most important vectors of human pathogens (Anopheles gambiae and Aedes aegypti) imbibing multiple b
53 b-Saharan Africa, two major malaria vectors, Anopheles gambiae and An. coluzzii, breed in distinct la
54 of key vector species from Africa and Asia (Anopheles gambiae and An. stephensi) to transmit the hum
55 They include the African malaria vectors Anopheles gambiae and Anopheles coluzzii, as well as Aed
56 f Plasmodium berghei and Plasmodium vivax to Anopheles gambiae and Anopheles dirus, respectively.
57 s after initiation of treatment to determine Anopheles gambiae and Anopheles funestus survival and in
58 he last 3 y in the two major malaria vectors Anopheles gambiae and Anopheles funestus, with a higher
59 -kDa) protein related to the gSG7 protein of Anopheles gambiae and Anopheles stephensi Recombinant al
61 the lifecycle of the malaria vector mosquito Anopheles gambiae and are initiated by peripheral signal
62 uts in an area of Benin where the mosquitoes Anopheles gambiae and Culex quinquefasciatus are resista
63 FREP1) is critical for parasite infection in Anopheles gambiae and facilitates Plasmodium invasion in
64 residues of the ortholog of human ICMT from Anopheles gambiae and observed reduced or undetectable c
65 gen vectors such as the malaria-transmitting Anopheles gambiae and to correct deleterious mutations i
66 al kinase (JNK) pathway is a key mediator of Anopheles gambiae antiplasmodial responses to P. falcipa
67 The Y chromosome of the human malaria vector Anopheles gambiae appears to be involved in sex determin
68 mately 13,000 genes listed in VectorBase for Anopheles gambiae are predictions that have still not be
69 NAi-mediated depletion of Kto and Skd in the Anopheles gambiae cell line L5-3 resulted in a decrease
70 ga-Mali) was identified in mosquitoes of the Anopheles gambiae complex collected in the Malian villag
71 n male mating behavior, recent data from the Anopheles gambiae complex suggests that, apart from the
75 ulations of the major African malaria vector Anopheles gambiae Contact with LLINs reduced the immedia
76 genome of the major malaria vector mosquito Anopheles gambiae contains at least seven putative AQP s
77 of an Orco family agonist, VUAA1, using the Anopheles gambiae coreceptor (AgOrco) and other ortholog
78 We developed and characterized an efficient Anopheles gambiae densovirus (AgDNV) over-expression sys
79 transmission by the African mosquito vector Anopheles gambiae depends on finely tuned vector-parasit
80 e monitored miRNA expression in the mosquito Anopheles gambiae during the 72-h period immediately aft
83 sts data for nine genomes: mosquitoes (three Anopheles gambiae genomes, Aedes aegypti and Culex quinq
85 PHs) are positive and negative regulators of Anopheles gambiae immune responses mediated by the compl
87 a hybrid zone between Anopheles coluzzii and Anopheles gambiae in Guinea-Bissau, where high hybridisa
88 ne expression to develop transgenic lines of Anopheles gambiae in which olfactory receptor neurons ex
92 9-based homing system for the suppression of Anopheles gambiae is encouraging; however, with current
96 hough the presence of Pe. chrysogenum in the Anopheles gambiae midgut does not affect mosquito surviv
97 termine the antiviral immune pathways of the Anopheles gambiae midgut, the initial site of viral infe
99 he microbial community within the gut of the Anopheles gambiae mosquito, a major malaria vector in Af
105 ingested blood enhance the susceptibility of Anopheles gambiae mosquitoes to malaria infection by dis
112 iled analysis of the resistance situation in Anopheles gambiae on Bioko Island after pyrethroid resis
114 h Plasmodium falciparum infection in natural Anopheles gambiae populations at malaria endemic areas i
115 h previously described mating preferences of Anopheles gambiae populations could be exploited to mani
117 ll this knowledge gap, coupled and uncoupled Anopheles gambiae s.l. males (all M form (Anopheles colu
120 ssed by passive case detection and number of Anopheles gambiae sensu lato mosquitoes collected per li
121 , the seasonal cycles of Anopheles coluzzii, Anopheles gambiae sensu stricto (s.s.), and Anopheles ar
122 iphenyltrichloroethane (DDT) in field-caught Anopheles gambiae sensu stricto homozygous for the kdr m
124 that in the primary African malaria vector, Anopheles gambiae sensu stricto, a single enzyme, CYP6M2
126 nversion is a widespread polymorphism in the Anopheles gambiae species complex, the major African mos
128 bosomal gene sequences of the malaria vector Anopheles gambiae that are located exclusively on the mo
129 rotein from the major African malaria vector Anopheles gambiae that specifically, tightly, and quickl
130 nding site of an OR from the malaria vector, Anopheles gambiae The closely related odorant-specificit
131 actometer, we investigated the attraction of Anopheles gambiae to 50 Kenyan children (aged 5-12 years
132 ngs of heterologously expressed and purified Anopheles gambiae TRPA1 (AgTRPA1), with and without the
135 ome of the primary African malaria mosquito, Anopheles gambiae We find that the An. gambiae Y consist
136 ob, for the M factor in the malaria mosquito Anopheles gambiae Yob, activated at the beginning of zyg
137 g in two other mosquitoes (Culex pipiens and Anopheles gambiae) and the bed bug, Cimex lectularius, s
139 eding arthropods, Pediculus humanus humanus, Anopheles gambiae, Aedes Aegypti and Culex pipiens quinq
140 Draft genome sequences are available for Anopheles gambiae, Aedes aegypti, and Culex quinquefasci
141 biology of two SGSs in the malaria mosquito, Anopheles gambiae, and demonstrate their involvement in
142 ated with Plasmodium falciparum infection in Anopheles gambiae, and FREP1 is important for Plasmodium
143 main, ZLD orthologs from Drosophila virilis, Anopheles gambiae, and Nasonia vitripennis activate tran
144 natural populations of the malaria mosquito Anopheles gambiae, blood-fed females direct nutritional
145 nthetically in the main human malaria vector Anopheles gambiae, by selectively destroying the X-chrom
146 three major disease-transmitting mosquitoes: Anopheles gambiae, Culex quinquefasciatus and A. aegypti
147 ocidal to important vector species including Anopheles gambiae, Culex quinquefasciatus, and Aedes aeg
148 nsects, such as the malaria vector mosquito, Anopheles gambiae, depend upon chemoreceptors to respond
149 ican malaria vectors, Anopheles coluzzii and Anopheles gambiae, have been attributed to assortative m
150 the dispersal of its most efficient vector, Anopheles gambiae, in order to target interventions and
152 s application using genome variation data of Anopheles gambiae, Plasmodium falciparum and Plasmodium
153 Afrotropical human malaria vector mosquito, Anopheles gambiae, remains a significant threat to globa
154 e mechanisms in the principle malaria vector Anopheles gambiae, remains largely uncharacterized in Bu
156 s have been identified in the malaria vector Anopheles gambiae, the crystal structures of only six of
160 island (PRI) of the African mosquito vector, Anopheles gambiae, was mapped to five genomic regions co
161 rated in Caenorhabditis elegans and later in Anopheles gambiae, we show here that an acellular gut ba
162 ble exception is the female malaria mosquito Anopheles gambiae, which after sex loses her susceptibil
163 n applied widely to the major malaria vector Anopheles gambiae, which has proved more difficult to ge
175 ly understood in many species, including the Anopheles genus of mosquitoes-an emerging model system f
176 ra is required for full viral infectivity to Anopheles, in contrast to malaria infection, where the p
178 of male-biased genes on the X Chromosomes in Anopheles is a conserved feature in this genus and can b
180 resistance is a major obstacle to control of Anopheles malaria mosquitoes in sub-Saharan Africa and r
183 : Anopheles vinckei, Anopheles moucheti, and Anopheles marshallii Their role in transmission was conf
184 cal value of increasing the understanding of Anopheles mating preferences in the wild as a means to h
186 Southeast Asian vectors, Anopheles dirus and Anopheles minimus, and the major African vector, Anophel
187 ction of distance from the hotspot boundary, Anopheles mosquito density, mosquito breeding site produ
188 Malaria is transmitted when an infected Anopheles mosquito deposits Plasmodium sporozoites in th
189 ium must complete a complex lifecycle in its Anopheles mosquito host, the main vector for Plasmodium.
190 th injection of Plasmodium sporozoites by an Anopheles mosquito into the skin of the mammalian host.
191 n of malaria-refractory transgenes into wild Anopheles mosquito populations that would limit their ca
192 n vectorial capacity for human malaria among Anopheles mosquito species is determined by many factors
196 ods to control malaria is by controlling the Anopheles mosquito vectors that transmit the parasites.
198 ctor dynamics of Plasmodium falciparum in an Anopheles mosquito with unbiased random mating and incom
203 erated substantial numbers of sporozoites in Anopheles mosquitoes and diverged from NF54 parasites by
204 ug that can block Plasmodium transmission to Anopheles mosquitoes and eliminate liver-stage hypnozoit
209 ate (HBR), expressed as the number of female Anopheles mosquitoes collected per house-night of collec
212 infections can reduce Plasmodium numbers in Anopheles mosquitoes in the laboratory, however, natural
213 eeding cultured P. falciparum gametocytes to Anopheles mosquitoes in the presence of the test antibod
214 known that infants who are highly exposed to Anopheles mosquitoes infected with Plasmodium are at gre
215 ion of Plasmodium species between humans and Anopheles mosquitoes is a major contributor to the globa
216 oson-based forward-genetics technologies for Anopheles mosquitoes not only will accelerate our unders
219 asmodium parasites and insecticide-resistant Anopheles mosquitoes, and first generation vaccines offe
220 ocytic gametocytes that are transmissible to Anopheles mosquitoes, and human-to-mosquito infectivity.
221 cytes by Plasmodium sporozoites deposited by Anopheles mosquitoes, and their subsequent transformatio
239 cted with ape Plasmodium: Anopheles vinckei, Anopheles moucheti, and Anopheles marshallii Their role
241 erratia bacterium strain (AS1) isolated from Anopheles ovaries that stably colonizes the mosquito mid
242 tissues of four vector species spanning the Anopheles phylogeny to explore the genomic and evolution
243 1 (TEP1), leucine-rich repeat protein 1, and Anopheles Plasmodium-responsive leucine-rich repeat prot
245 evidence of Wolbachia infections in natural Anopheles populations promotes further investigations on
247 To test this in the field, we quantified Anopheles responses to olfactory, visual and thermal sti
248 we address the relevance of using a specific Anopheles salivary biomarker to measure the risk among h
252 tified by PCR in five species of mosquitoes (Anopheles sinensis, Armigeres subalbatus, Aedes albopict
253 into other anthrophillic vectors (Culex and Anopheles sp. mosquitoes), and, importantly, close conta
254 ively associated with the abundance of total Anopheles species and primary malaria vectors and the EI
256 1 is highly conserved (>90% identical) among Anopheles species from different continents, suggesting
257 rphological identifications recognized eight Anopheles species while 18 distinct sequence groups or s
258 tant parasites to infect such highly diverse Anopheles species, combined with their higher gametocyte
259 d analysis of whole genome assemblies for 16 Anopheles species, with genomic flexibility a key emerge
263 umans and other vertebrates, can be toxic to Anopheles spp. mosquitoes when they take a blood meal fr
264 vates the insulin/IGF-1 signaling pathway in Anopheles stephensi and increases the susceptibility of
265 vides insights into the molecular anatomy of Anopheles stephensi and the distribution and localizatio
266 mosquito species, the Asian malaria mosquito Anopheles stephensi and the yellow fever mosquito Aedes
267 omics analysis to compare SGs mRNA levels in Anopheles stephensi fed on non-infected and P. berghei-i
270 e NF54 laboratory strain of P. falciparum in Anopheles stephensi mosquitoes using the standard membra
271 ulses to kill or disable anesthetized female Anopheles stephensi mosquitoes, which were chosen as a r
277 to the gSG7 protein of Anopheles gambiae and Anopheles stephensi Recombinant albicin was produced in
278 mission-based study of Plasmodium berghei in Anopheles stephensi to assess the impact of a transmissi
279 Very recently, the Asian malaria vector (Anopheles stephensi) was stably transinfected with the w
280 bassiana are highly attractive to females of Anopheles stephensi, a major anopheline mosquito vector
281 ene-drive system in the Asian malaria vector Anopheles stephensi, adapted from the mutagenic chain re
282 osome gene of a major urban malaria mosquito Anopheles stephensi, confers 100% female lethality when
283 h for analysis of the genome and proteome of Anopheles stephensi, which is one of the most important
290 nuclease to cleave in a gene associated with Anopheles sterility and another to cleave near a mutatio
292 insect vector, mosquito species in the genus Anopheles, targeting mosquito midgut antigens that serve
293 lus high contrast visual stimuli caught more Anopheles than traps with odour alone, showing that desp
295 d ookinetes, which invade and transverse the Anopheles vector mosquito midgut at around 18-36 h after
298 tidase N (AnAPN1) is highly conserved across Anopheles vectors and is a putative ligand for Plasmodiu
299 les were found infected with ape Plasmodium: Anopheles vinckei, Anopheles moucheti, and Anopheles mar
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