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1 mum spatial resolution of the malaria vector Anopheles.
2 robiota can impede Wolbachia transmission in Anopheles.
3 ding Drosophila and the mosquitoes Aedes and Anopheles.
4               As previously reported for the Anopheles albimanus orthologue anophelin, cE5 binds both
5 m the salivary glands of the malaria vector, Anopheles albimanus The inhibitor was purified from sali
6 Haiti from May to September, 2012, where the Anopheles albimanus vector bites primarily outdoors and
7 VU041, a submicromolar-affinity inhibitor of Anopheles (An.) gambiae and Aedes (Ae.) aegypti Kir1 cha
8 ce of the mechanisms regulating fertility in Anopheles, and identify HPX15 as a target for vector con
9 smitted to humans by mosquitoes of the genus Anopheles, and these insects are the targets of innovati
10  to differ in their innate host preferences: Anopheles arabiensis and An. gambiae sensu stricto.
11                          We show that gravid Anopheles arabiensis are attracted and oviposit in respo
12                                              Anopheles arabiensis is the major malaria vector in Chad
13  Anopheles gambiae sensu stricto (s.s.), and Anopheles arabiensis were estimated, and their effects w
14  increased prevalence of the malaria vector, Anopheles arabiensis, in sub-Saharan Africa.
15 ons on the possible use of natural Wolbachia-Anopheles associations to limit malaria transmission.
16 measure the risk among humans of exposure to Anopheles bites.
17  CO(2)-sensitive ORN in the malaria mosquito Anopheles can also be inhibited by excitation of an adja
18 ce of Wolbachia in females from a laboratory Anopheles coluzzii (A. gambiae, M form) colony experimen
19 heles minimus, and the major African vector, Anopheles coluzzii (formerly Anopheles gambiae M).
20 es and consequences of a hybrid zone between Anopheles coluzzii and Anopheles gambiae in Guinea-Bissa
21 istance between two African malaria vectors, Anopheles coluzzii and Anopheles gambiae, have been attr
22 e chemical composition and attractiveness to Anopheles coluzzii mosquitoes of skin odours from partic
23 ed Anopheles gambiae s.l. males (all M form (Anopheles coluzzii)) were collected from mating swarms i
24 time-series analyses, the seasonal cycles of Anopheles coluzzii, Anopheles gambiae sensu stricto (s.s
25 frican malaria vectors Anopheles gambiae and Anopheles coluzzii, as well as Aedes aegypti, the cosmop
26 quito populations of a major malaria vector, Anopheles coluzzii, in Burkina Faso.
27  content and permissiveness to Plasmodium of Anopheles coluzzii.
28  Eilat virus (EILV), isolated from a pool of Anopheles coustani mosquitoes from the Negev desert of I
29                                              Anopheles darlingi diverged from the African and Asian m
30                                              Anopheles darlingi is the principal neotropical malaria
31 es of the New World species A. albimanus and Anopheles darlingi) also played some functionally releva
32  accessible at www.labinfo.lncc.br/index.php/anopheles-darlingi.
33    Moreover, human landing catches of sylvan Anopheles demonstrated the propensity of these three vec
34  sporozoites in two Southeast Asian vectors, Anopheles dirus and Anopheles minimus, and the major Afr
35  119 falciparum-infected Cambodian adults to Anopheles dirus mosquitoes by membrane feeding.
36 nd Plasmodium vivax to Anopheles gambiae and Anopheles dirus, respectively.
37 r) to pyrethroids/DDT resistance observed in Anopheles funestus across Africa.
38  control of major malaria vectors, including Anopheles funestus Effective management of resistance re
39                                              Anopheles funestus is one of three major African vectors
40 this insecticide class in the malaria vector Anopheles funestus is spreading rapidly across Africa, t
41                     Pyrethroid resistance in Anopheles funestus is threatening malaria control in Afr
42      Up to 15% of morphologically identified Anopheles funestus mosquitoes in insecticide resistance
43 treatment to determine Anopheles gambiae and Anopheles funestus survival and infection rates.
44 s) may induce a switch in biting behavior in Anopheles funestus, a major malaria vector in Africa.
45                  In the major malaria vector Anopheles funestus, little is currently known about the
46  major malaria vectors Anopheles gambiae and Anopheles funestus, with a higher frequency of resistanc
47 % of which have their closest counterpart in Anopheles gambiae and 21% have highest similarity with o
48 nophelines were collected, of which 388 were Anopheles gambiae and 629 An. ziemanni.
49                                              Anopheles gambiae and A. stephensi larvae were bred unde
50                                              Anopheles gambiae and Aedes aegypti have evolved a stron
51 e most important vectors of human pathogens (Anopheles gambiae and Aedes aegypti) imbibing multiple b
52  essential AChE1 enzymes from the mosquitoes Anopheles gambiae and Aedes aegypti.
53 b-Saharan Africa, two major malaria vectors, Anopheles gambiae and An. coluzzii, breed in distinct la
54  of key vector species from Africa and Asia (Anopheles gambiae and An. stephensi) to transmit the hum
55     They include the African malaria vectors Anopheles gambiae and Anopheles coluzzii, as well as Aed
56 f Plasmodium berghei and Plasmodium vivax to Anopheles gambiae and Anopheles dirus, respectively.
57 s after initiation of treatment to determine Anopheles gambiae and Anopheles funestus survival and in
58 he last 3 y in the two major malaria vectors Anopheles gambiae and Anopheles funestus, with a higher
59 -kDa) protein related to the gSG7 protein of Anopheles gambiae and Anopheles stephensi Recombinant al
60 ploying two different species of mosquitoes, Anopheles gambiae and Anopheles stephensi.
61 the lifecycle of the malaria vector mosquito Anopheles gambiae and are initiated by peripheral signal
62 uts in an area of Benin where the mosquitoes Anopheles gambiae and Culex quinquefasciatus are resista
63 FREP1) is critical for parasite infection in Anopheles gambiae and facilitates Plasmodium invasion in
64  residues of the ortholog of human ICMT from Anopheles gambiae and observed reduced or undetectable c
65 gen vectors such as the malaria-transmitting Anopheles gambiae and to correct deleterious mutations i
66 al kinase (JNK) pathway is a key mediator of Anopheles gambiae antiplasmodial responses to P. falcipa
67 The Y chromosome of the human malaria vector Anopheles gambiae appears to be involved in sex determin
68 mately 13,000 genes listed in VectorBase for Anopheles gambiae are predictions that have still not be
69 NAi-mediated depletion of Kto and Skd in the Anopheles gambiae cell line L5-3 resulted in a decrease
70 ga-Mali) was identified in mosquitoes of the Anopheles gambiae complex collected in the Malian villag
71 n male mating behavior, recent data from the Anopheles gambiae complex suggests that, apart from the
72                            Mosquitoes of the Anopheles gambiae complex were identified to species usi
73                       As with members of the Anopheles gambiae complex, An. funestus shows marked gen
74 nces restricted to the member species of the Anopheles gambiae complex.
75 ulations of the major African malaria vector Anopheles gambiae Contact with LLINs reduced the immedia
76  genome of the major malaria vector mosquito Anopheles gambiae contains at least seven putative AQP s
77  of an Orco family agonist, VUAA1, using the Anopheles gambiae coreceptor (AgOrco) and other ortholog
78  We developed and characterized an efficient Anopheles gambiae densovirus (AgDNV) over-expression sys
79  transmission by the African mosquito vector Anopheles gambiae depends on finely tuned vector-parasit
80 e monitored miRNA expression in the mosquito Anopheles gambiae during the 72-h period immediately aft
81                Plasmodium falciparum infects Anopheles gambiae efficiently at low densities (4% mosqu
82      The high-affinity adenosine kinase from Anopheles gambiae efficiently converts adenosine to aden
83 sts data for nine genomes: mosquitoes (three Anopheles gambiae genomes, Aedes aegypti and Culex quinq
84                         The M and S forms of Anopheles gambiae have been the focus of intense study b
85 PHs) are positive and negative regulators of Anopheles gambiae immune responses mediated by the compl
86  we show evidence of Wolbachia infections in Anopheles gambiae in Burkina Faso, West Africa.
87 a hybrid zone between Anopheles coluzzii and Anopheles gambiae in Guinea-Bissau, where high hybridisa
88 ne expression to develop transgenic lines of Anopheles gambiae in which olfactory receptor neurons ex
89    Resistance to pyrethroids in the mosquito Anopheles gambiae is a growing problem.
90                                              Anopheles gambiae is a major mosquito vector responsible
91                                              Anopheles gambiae is a major vector mosquito for Plasmod
92 9-based homing system for the suppression of Anopheles gambiae is encouraging; however, with current
93                                          The Anopheles gambiae L3-5 refractory (R) line melanizes mos
94 African vector, Anopheles coluzzii (formerly Anopheles gambiae M).
95                         Pyrethroid resistant Anopheles gambiae malaria vectors are widespread through
96 hough the presence of Pe. chrysogenum in the Anopheles gambiae midgut does not affect mosquito surviv
97 termine the antiviral immune pathways of the Anopheles gambiae midgut, the initial site of viral infe
98 nvestigate the function of P. berghei P47 in Anopheles gambiae mosquito infections.
99 he microbial community within the gut of the Anopheles gambiae mosquito, a major malaria vector in Af
100                                          The Anopheles gambiae mosquito, which is the vector for Plas
101                                              Anopheles gambiae mosquitoes are major African vectors o
102        Plasmodium falciparum transmission by Anopheles gambiae mosquitoes is remarkably efficient, re
103                                   A study of Anopheles gambiae mosquitoes shows that a molecule invol
104                                              Anopheles gambiae mosquitoes that transmit Plasmodium fa
105 ingested blood enhance the susceptibility of Anopheles gambiae mosquitoes to malaria infection by dis
106                                  Exposure of Anopheles gambiae mosquitoes to Plasmodium infection enh
107                                         Here Anopheles gambiae mosquitoes, the primary malarial vecto
108 asite to evade the mosquito immune system of Anopheles gambiae mosquitoes.
109 be a powerful molecular tool for research in Anopheles gambiae mosquitoes.
110 . bacterium was isolated from the midguts of Anopheles gambiae mosquitoes.
111 nd Plasmodium berghei expressing PfCelTOS in Anopheles gambiae mosquitoes.
112 iled analysis of the resistance situation in Anopheles gambiae on Bioko Island after pyrethroid resis
113 throid resistance in various Ae. aegypti and Anopheles gambiae populations around the world.
114 h Plasmodium falciparum infection in natural Anopheles gambiae populations at malaria endemic areas i
115 h previously described mating preferences of Anopheles gambiae populations could be exploited to mani
116                Tracking multiple free-flying Anopheles gambiae responding to human-occupied bed nets
117 ll this knowledge gap, coupled and uncoupled Anopheles gambiae s.l. males (all M form (Anopheles colu
118                                              Anopheles gambiae s.s. mosquitoes are efficient vectors
119         Here, we show that the attraction of Anopheles gambiae s.s. to plant odors increased by 30% a
120 ssed by passive case detection and number of Anopheles gambiae sensu lato mosquitoes collected per li
121 , the seasonal cycles of Anopheles coluzzii, Anopheles gambiae sensu stricto (s.s.), and Anopheles ar
122 iphenyltrichloroethane (DDT) in field-caught Anopheles gambiae sensu stricto homozygous for the kdr m
123                                              Anopheles gambiae sensu stricto is the most important ve
124  that in the primary African malaria vector, Anopheles gambiae sensu stricto, a single enzyme, CYP6M2
125                           Gene expression in Anopheles gambiae shows a deficiency of testis-expressed
126 nversion is a widespread polymorphism in the Anopheles gambiae species complex, the major African mos
127 genomic islands of divergence separating the Anopheles gambiae species pair.
128 bosomal gene sequences of the malaria vector Anopheles gambiae that are located exclusively on the mo
129 rotein from the major African malaria vector Anopheles gambiae that specifically, tightly, and quickl
130 nding site of an OR from the malaria vector, Anopheles gambiae The closely related odorant-specificit
131 actometer, we investigated the attraction of Anopheles gambiae to 50 Kenyan children (aged 5-12 years
132 ngs of heterologously expressed and purified Anopheles gambiae TRPA1 (AgTRPA1), with and without the
133 icide resistance in the major malaria vector Anopheles gambiae was assessed.
134                                              Anopheles gambiae was genetically partitioned into inlan
135 ome of the primary African malaria mosquito, Anopheles gambiae We find that the An. gambiae Y consist
136 ob, for the M factor in the malaria mosquito Anopheles gambiae Yob, activated at the beginning of zyg
137 g in two other mosquitoes (Culex pipiens and Anopheles gambiae) and the bed bug, Cimex lectularius, s
138                                           In Anopheles gambiae, a likely candidate for sexual selecti
139 eding arthropods, Pediculus humanus humanus, Anopheles gambiae, Aedes Aegypti and Culex pipiens quinq
140     Draft genome sequences are available for Anopheles gambiae, Aedes aegypti, and Culex quinquefasci
141 biology of two SGSs in the malaria mosquito, Anopheles gambiae, and demonstrate their involvement in
142 ated with Plasmodium falciparum infection in Anopheles gambiae, and FREP1 is important for Plasmodium
143 main, ZLD orthologs from Drosophila virilis, Anopheles gambiae, and Nasonia vitripennis activate tran
144  natural populations of the malaria mosquito Anopheles gambiae, blood-fed females direct nutritional
145 nthetically in the main human malaria vector Anopheles gambiae, by selectively destroying the X-chrom
146 three major disease-transmitting mosquitoes: Anopheles gambiae, Culex quinquefasciatus and A. aegypti
147 ocidal to important vector species including Anopheles gambiae, Culex quinquefasciatus, and Aedes aeg
148 nsects, such as the malaria vector mosquito, Anopheles gambiae, depend upon chemoreceptors to respond
149 ican malaria vectors, Anopheles coluzzii and Anopheles gambiae, have been attributed to assortative m
150  the dispersal of its most efficient vector, Anopheles gambiae, in order to target interventions and
151                          The malaria vector, Anopheles gambiae, is associated with a latitudinal clin
152 s application using genome variation data of Anopheles gambiae, Plasmodium falciparum and Plasmodium
153  Afrotropical human malaria vector mosquito, Anopheles gambiae, remains a significant threat to globa
154 e mechanisms in the principle malaria vector Anopheles gambiae, remains largely uncharacterized in Bu
155                   After injection into adult Anopheles gambiae, some strains of Wolbachia invade the
156 s have been identified in the malaria vector Anopheles gambiae, the crystal structures of only six of
157 ructs that function as gene drive systems in Anopheles gambiae, the main vector for malaria.
158                                              Anopheles gambiae, the primary African vector of malaria
159             In the African malaria mosquito, Anopheles gambiae, this is complemented by a strong publ
160 island (PRI) of the African mosquito vector, Anopheles gambiae, was mapped to five genomic regions co
161 rated in Caenorhabditis elegans and later in Anopheles gambiae, we show here that an acellular gut ba
162 ble exception is the female malaria mosquito Anopheles gambiae, which after sex loses her susceptibil
163 n applied widely to the major malaria vector Anopheles gambiae, which has proved more difficult to ge
164 IIS6, in pyrethroid-resistant populations of Anopheles gambiae.
165 f specific ORs of the African malaria vector Anopheles gambiae.
166 are involved in anti-Plasmodium responses in Anopheles gambiae.
167 pression system for the major malaria vector Anopheles gambiae.
168 uman skin odorants in both Aedes aegypti and Anopheles gambiae.
169 required for efficient Plasmodium killing in Anopheles gambiae.
170 munity that enhance nitric oxide toxicity in Anopheles gambiae.
171 genic mosquitoes of the human malaria vector Anopheles gambiae.
172 Ago1 is the predominant carrier of miRNAs in Anopheles gambiae.
173  larvae of the mosquitoes, Aedes aegypti and Anopheles gambiae.
174 uestions regarding SGs of the malaria vector Anopheles gambiae.
175 ly understood in many species, including the Anopheles genus of mosquitoes-an emerging model system f
176 ra is required for full viral infectivity to Anopheles, in contrast to malaria infection, where the p
177                                              Anopheles interactions with viruses have been little stu
178 of male-biased genes on the X Chromosomes in Anopheles is a conserved feature in this genus and can b
179 sia that is transmitted by mosquitoes of the Anopheles leucosphyrus group.
180 resistance is a major obstacle to control of Anopheles malaria mosquitoes in sub-Saharan Africa and r
181 ly repetitive content of the Y chromosome in Anopheles malaria mosquitoes.
182                     Here we show that in the Anopheles malaria vector, IMD and Toll pathways mediate
183 : Anopheles vinckei, Anopheles moucheti, and Anopheles marshallii Their role in transmission was conf
184 cal value of increasing the understanding of Anopheles mating preferences in the wild as a means to h
185 ; Ano pheles Spatially-Explicit) to simulate Anopheles metapopulation dynamics.
186 Southeast Asian vectors, Anopheles dirus and Anopheles minimus, and the major African vector, Anophel
187 ction of distance from the hotspot boundary, Anopheles mosquito density, mosquito breeding site produ
188      Malaria is transmitted when an infected Anopheles mosquito deposits Plasmodium sporozoites in th
189 ium must complete a complex lifecycle in its Anopheles mosquito host, the main vector for Plasmodium.
190 th injection of Plasmodium sporozoites by an Anopheles mosquito into the skin of the mammalian host.
191 n of malaria-refractory transgenes into wild Anopheles mosquito populations that would limit their ca
192 n vectorial capacity for human malaria among Anopheles mosquito species is determined by many factors
193                                        Among Anopheles mosquito species, these phenotypic differences
194  present in the genomes of Aedes, Culex, and Anopheles mosquito species.
195                     Six pyrethroid-resistant Anopheles mosquito strains from across Africa were expos
196 ods to control malaria is by controlling the Anopheles mosquito vectors that transmit the parasites.
197 a elimination is the effective management of Anopheles mosquito vectors.
198 ctor dynamics of Plasmodium falciparum in an Anopheles mosquito with unbiased random mating and incom
199 igate vector of the Plasmodium parasite, the Anopheles mosquito.
200 asmodium begins with the bite of an infected Anopheles mosquito.
201 the transmission of malaria parasites by the Anopheles mosquito.
202                                        Using Anopheles mosquitoes (which are naturally uninfected by
203 erated substantial numbers of sporozoites in Anopheles mosquitoes and diverged from NF54 parasites by
204 ug that can block Plasmodium transmission to Anopheles mosquitoes and eliminate liver-stage hypnozoit
205                                              Anopheles mosquitoes are vectors of malaria, a potential
206                                              Anopheles mosquitoes are vectors of the human malaria pa
207 " trap caught nearly ten times the number of Anopheles mosquitoes caught by a human collector.
208                                         Male Anopheles mosquitoes coagulate their seminal fluids via
209 ate (HBR), expressed as the number of female Anopheles mosquitoes collected per house-night of collec
210 nhibit parasite development when ingested by Anopheles mosquitoes during blood meals.
211 eers are bitten by 5 malaria-infected female Anopheles mosquitoes in a controlled environment.
212  infections can reduce Plasmodium numbers in Anopheles mosquitoes in the laboratory, however, natural
213 eeding cultured P. falciparum gametocytes to Anopheles mosquitoes in the presence of the test antibod
214 known that infants who are highly exposed to Anopheles mosquitoes infected with Plasmodium are at gre
215 ion of Plasmodium species between humans and Anopheles mosquitoes is a major contributor to the globa
216 oson-based forward-genetics technologies for Anopheles mosquitoes not only will accelerate our unders
217                                              Anopheles mosquitoes transmit malaria, a major public he
218                            A total of 21,899 anopheles mosquitoes were collected for 26 months before
219 asmodium parasites and insecticide-resistant Anopheles mosquitoes, and first generation vaccines offe
220 ocytic gametocytes that are transmissible to Anopheles mosquitoes, and human-to-mosquito infectivity.
221 cytes by Plasmodium sporozoites deposited by Anopheles mosquitoes, and their subsequent transformatio
222   However, many important species, including Anopheles mosquitoes, are uninfected.
223                        After transmission by Anopheles mosquitoes, Plasmodium sporozoites travel to t
224 he genetic architecture of osmoregulation in Anopheles mosquitoes, vectors of human malaria.
225 es are frequently overexpressed in resistant Anopheles mosquitoes, were analyzed.
226 olism of Plasmodium-infected and -uninfected Anopheles mosquitoes.
227 l and potential biological control agent for Anopheles mosquitoes.
228 thenate transport in malaria transmission to Anopheles mosquitoes.
229 n the skin, immediately after inoculation by Anopheles mosquitoes.
230 ium falciparum that is transmitted by female Anopheles mosquitoes.
231 he transmission of Plasmodium from humans to Anopheles mosquitoes.
232  takes place, and in gametocytes that infect Anopheles mosquitoes.
233 ibition of mature gametocyte transmission to Anopheles mosquitoes.
234 d reduced oocyst infection and prevalence in Anopheles mosquitoes.
235 nsecticide susceptibility phenotype of adult Anopheles mosquitoes.
236 nthesis and malaria parasite transmission to Anopheles mosquitoes.
237 chrysogenum, from the midgut of field-caught Anopheles mosquitoes.
238 um berghei during its life cycle in mice and Anopheles mosquitoes.
239 cted with ape Plasmodium: Anopheles vinckei, Anopheles moucheti, and Anopheles marshallii Their role
240                     Specimens of neotropical Anopheles (Nyssorhynchus) were collected and identified
241 erratia bacterium strain (AS1) isolated from Anopheles ovaries that stably colonizes the mosquito mid
242  tissues of four vector species spanning the Anopheles phylogeny to explore the genomic and evolution
243 1 (TEP1), leucine-rich repeat protein 1, and Anopheles Plasmodium-responsive leucine-rich repeat prot
244 derstanding the environmental forces driving Anopheles population dynamics.
245  evidence of Wolbachia infections in natural Anopheles populations promotes further investigations on
246 p of closely related mosquitoes known as the Anopheles punctulatus group.
247     To test this in the field, we quantified Anopheles responses to olfactory, visual and thermal sti
248 we address the relevance of using a specific Anopheles salivary biomarker to measure the risk among h
249                             We identified an Anopheles salivary gland protein, named CSP-binding prot
250               Here, we use defined panels of Anopheles samples from West Africa to test two experimen
251                                              Anopheles sinensis is the most important vector of malar
252 tified by PCR in five species of mosquitoes (Anopheles sinensis, Armigeres subalbatus, Aedes albopict
253  into other anthrophillic vectors (Culex and Anopheles sp. mosquitoes), and, importantly, close conta
254 ively associated with the abundance of total Anopheles species and primary malaria vectors and the EI
255                                              Anopheles species assignments based on ribosomal DNA ITS
256 1 is highly conserved (>90% identical) among Anopheles species from different continents, suggesting
257 rphological identifications recognized eight Anopheles species while 18 distinct sequence groups or s
258 tant parasites to infect such highly diverse Anopheles species, combined with their higher gametocyte
259 d analysis of whole genome assemblies for 16 Anopheles species, with genomic flexibility a key emerge
260 n of multiple Plasmodium species to multiple Anopheles species.
261 ain about its key protagonists, a handful of Anopheles species.
262 gs of which are found thus far only in other Anopheles species.
263 umans and other vertebrates, can be toxic to Anopheles spp. mosquitoes when they take a blood meal fr
264 vates the insulin/IGF-1 signaling pathway in Anopheles stephensi and increases the susceptibility of
265 vides insights into the molecular anatomy of Anopheles stephensi and the distribution and localizatio
266 mosquito species, the Asian malaria mosquito Anopheles stephensi and the yellow fever mosquito Aedes
267 omics analysis to compare SGs mRNA levels in Anopheles stephensi fed on non-infected and P. berghei-i
268                             Analysis of 6860 Anopheles stephensi mosquitoes across 68 experimental fe
269                                              Anopheles stephensi mosquitoes expressing m1C3, m4B7, or
270 e NF54 laboratory strain of P. falciparum in Anopheles stephensi mosquitoes using the standard membra
271 ulses to kill or disable anesthetized female Anopheles stephensi mosquitoes, which were chosen as a r
272 tely blocked mouse-to-mouse transmission via Anopheles stephensi mosquitoes.
273 natomical structure of rodent malaria vector Anopheles stephensi mosquitoes.
274 pletely prevented oocyst formation in female Anopheles stephensi mosquitoes.
275  cultured P. falciparum NF54 gametocytes and Anopheles stephensi mosquitoes.
276 ssion of mature P. falciparum gametocytes to Anopheles stephensi mosquitoes.
277 to the gSG7 protein of Anopheles gambiae and Anopheles stephensi Recombinant albicin was produced in
278 mission-based study of Plasmodium berghei in Anopheles stephensi to assess the impact of a transmissi
279     Very recently, the Asian malaria vector (Anopheles stephensi) was stably transinfected with the w
280 bassiana are highly attractive to females of Anopheles stephensi, a major anopheline mosquito vector
281 ene-drive system in the Asian malaria vector Anopheles stephensi, adapted from the mutagenic chain re
282 osome gene of a major urban malaria mosquito Anopheles stephensi, confers 100% female lethality when
283 h for analysis of the genome and proteome of Anopheles stephensi, which is one of the most important
284                                           In Anopheles stephensi, Wolbachia infection elicited massiv
285 species of mosquitoes, Anopheles gambiae and Anopheles stephensi.
286 ia infection in an important malaria vector, Anopheles stephensi.
287 integrations in the malaria vector mosquito, Anopheles stephensi.
288 ori, Tribolium casteneum, Aedes aegypti, and Anopheles stephensi.
289  in the germline of the human malaria vector Anopheles stephensi.
290 nuclease to cleave in a gene associated with Anopheles sterility and another to cleave near a mutatio
291 creation of conditional male-only transgenic Anopheles strains for malaria control programs.
292 insect vector, mosquito species in the genus Anopheles, targeting mosquito midgut antigens that serve
293 lus high contrast visual stimuli caught more Anopheles than traps with odour alone, showing that desp
294  responsible for the infection of the vector Anopheles, thus perpetuating the plasmodial cycle.
295 d ookinetes, which invade and transverse the Anopheles vector mosquito midgut at around 18-36 h after
296 e the abundance and seasonal dynamics of the Anopheles vector.
297 peripheral blood, available to uptake by the Anopheles vector.
298 tidase N (AnAPN1) is highly conserved across Anopheles vectors and is a putative ligand for Plasmodiu
299 les were found infected with ape Plasmodium: Anopheles vinckei, Anopheles moucheti, and Anopheles mar
300            Among them, only three species of Anopheles were found infected with ape Plasmodium: Anoph

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