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1 ter formation of cutaneous immune complexes (Arthus reaction).
2 d extravasation of neutrophils in a reversed Arthus reaction.
3 mune complexes, which can trigger a type III Arthus reaction.
4 with the classical histologic features of an Arthus reaction.
5 e DIC model and a guinea pig reverse passive Arthus reaction.
6 n immune complex-mediated FcgammaR-dependent Arthus reaction.
7 uired for development of the reverse passive Arthus reaction.
10 we re-examined the cutaneous reverse passive Arthus reaction and observed that IC-activated neutrophi
11 ssary for neutrophil influx in skin and lung Arthus reactions and agonist-induced neutrophilia in the
12 exaggerated responses to reverse peritoneal Arthus reaction characterized by increased infiltration
13 erleukin 6 in the peritoneal reverse passive Arthus reaction compared to their wild-type littermates.
17 ted processes are of major importance in the Arthus reaction in rats and guinea pigs, and suggest tha
18 e C5aR and its ligand in the reverse passive Arthus reaction in the lung and a synergistic role toget
21 injury in mice, as modeled by the cutaneous Arthus reaction, requires receptors for the Fc portion o
24 ed with viable T. pallidum; and 2) classical Arthus reactions when challenged intradermally with eith
25 disease, as well as from the reverse passive Arthus reaction, with functional overlap between the thr
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