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1 lass in the management of diseases caused by Aspergillus.
2  affecting the quantity of asexual spores in Aspergillus.
3 ared with that for the Bruker library alone (Aspergillus, 93% versus 69%; Fusarium, 84% versus 42%; a
4                                    Utilizing Aspergillus, a fungal genus known for its rich secondary
5 digested with endo-polygalacturonase M2 from Aspergillus aculeatus and visualized by AFM.
6 ere misidentified by MALDI-TOF MS (including Aspergillus amoenus [n = 2] and Aspergillus calidoustus
7 found to expand during antifungal treatment (Aspergillus amstelodami, Epicoccum nigrum, and Wallemia
8 he most representative pathogenic molds (154 Aspergillus and 136 non-Aspergillus isolates) with the B
9 o identify selected toxigenic species of the Aspergillus and Fusarium genera isolated from grapes and
10 xin-producing fungal species from the genera Aspergillus and Fusarium using solid-state voltammetry i
11                                              Aspergillus and Mucorales species cause severe infection
12 a natural carcinogenic mycotoxin produced by Aspergillus and Penicillium fungi and so it commonly app
13                           The prevalences of Aspergillus and Scedosporium species were 40.8% and 5.2%
14                                              Aspergillus and Serratia were somewhat more common in lo
15 d therapy had significantly higher levels of Aspergillus and total fungus in their bronchoalveolar la
16  with optimized commercial preparations from Aspergillus and Trichoderma.
17 ntifungal immunity (primarily to Candida and Aspergillus) and will also examine the emerging field of
18 f asthma symptoms suggests that Penicillium, Aspergillus, and Cladosporium species pose a respiratory
19 ed ceftriaxone administration and culturable Aspergillus, and demonstrated significantly increased in
20    The presence of Cladosporium, Alternaria, Aspergillus, and Penicillium species increased the exace
21                    Cladosporium, Alternaria, Aspergillus, and Penicillium species were found to be pr
22  this study, we investigated whether certain Aspergillus antigens are more allergenic than others, as
23 spf2, Aspf3, and Aspf4, as well as two other Aspergillus antigens, Crf1 and Catalase1.
24 y which human neutrophils recognize and kill Aspergillus are poorly understood.
25 th a pro-inflammatory TH17-like phenotype in Aspergillus-associated immunopathology, and identifies k
26 vasive and granulomatous rhinosinusitis, and aspergillus bronchitis.
27 S (including Aspergillus amoenus [n = 2] and Aspergillus calidoustus [n = 1]) although 10 (3.1%) of t
28 ung or airways infection by endemic fungi or aspergillus can be diagnosed with respiratory sample cul
29 ciated with increased levels of Penicillium, Aspergillus, Cladosporium, and Alternaria species, altho
30 ndings reveal distinct killing mechanisms of Aspergillus conidia and hyphae by human neutrophils, lea
31          Human platelets were incubated with Aspergillus conidia and hyphae, isolated wall components
32  for effective host defense against Candida, Aspergillus, Cryptococcus, and others, with specific ele
33 d host immunity is fundamental to combatting Aspergillus diseases.
34                              During disease, Aspergillus DNA burdens in blood are often at the limits
35 hain reaction (PCR)-based detection of serum Aspergillus DNA for the early diagnosis and therapy of i
36 ed monitoring strategy based on serum GM and Aspergillus DNA was associated with an earlier diagnosis
37 (bmGT) are secondary metabolites produced by Aspergillus during invasive, hyphal growth and may prove
38 s relatively low (<10%), despite unavoidable Aspergillus exposure in patients with a potentially simi
39 two separate allergic stimuli (ovalbumin and Aspergillus extract), genetic removal of Muc5ac abolishe
40 (Ccl11) RNAs following pulmonary exposure to Aspergillus extract.
41                  Penicillium roquefortii and Aspergillus flavipes presented optimum activity at tempe
42 aris essential oil (TEO) were evaluated upon Aspergillus flavus "in vitro".
43                                              Aspergillus flavus (8), however was the commonest amongs
44 over, treatment with voriconazole due to the Aspergillus flavus and meropenem due to the Pseudomonas
45 s, a qualitative analytical method to detect Aspergillus flavus in food samples, based on the identif
46                                              Aspergillus flavus is a filamentous fungus found in natu
47                      Aflatoxin production by Aspergillus flavus is exacerbated by drought stress in t
48 cies in all pulmonary syndromes, followed by Aspergillus flavus which is a common cause of allergic r
49 ol, and perillyl alcohol were tested against Aspergillus flavus, Aspergillus ochraceus, Fusarium oxys
50 xin contamination, caused by fungal pathogen Aspergillus flavus, is a major quality and health proble
51        Aflatoxins are mycotoxins secreted by Aspergillus flavus, which can colonize the respiratory t
52 n of conidiation in Aspergillus fumigatus or Aspergillus flavus.
53 hogens, including real-time visualization of Aspergillus fumigatus (5 d for culturing, 1-2 d for imag
54 HDM-exposed mothers, the magnitude of HDM or Aspergillus fumigatus (AF) extract-induced airway hyperr
55 osure in mice sensitised and challenged with Aspergillus fumigatus (Af).
56  relevant fungi such as Candida albicans and Aspergillus fumigatus also form biofilms during infectio
57  and induction of innate immune responses to Aspergillus fumigatus and Candida albicans.
58 s, caused most commonly by Candida albicans, Aspergillus fumigatus and Cryptococcus neoformans, resul
59 ad of agriculturally derived azole-resistant Aspergillus fumigatus and emerging threats such as multi
60 onse to alpha-(1,3)-glucan polysaccharide of Aspergillus fumigatus and ensuing CD4+ T-cell polarizati
61 lungs of mice sensitized and challenged with Aspergillus fumigatus and evaluated ex vivo in tissue cu
62 cy of pH- and calcium-mediated signalling in Aspergillus fumigatus and found that calcium chelation s
63 d juvenile Scnn1b-Tg and wild-type mice with Aspergillus fumigatus and house dust mite allergen and c
64 ides produced by the opportunistic pathogens Aspergillus fumigatus and Pseudomonas aeruginosa, respec
65 n the organotypic bronchiole and cultures of Aspergillus fumigatus and Pseudomonas aeruginosa.
66 ere highly susceptible to aeroallergens from Aspergillus fumigatus and the house dust mite, resulting
67 onditions may result from the interaction of Aspergillus fumigatus and the immune system of its human
68                             Sensitisation to Aspergillus fumigatus and/or Penicillium chrysogenum was
69      Infections caused by triazole-resistant Aspergillus fumigatus are associated with a higher proba
70 breakthrough infections with fungal pathogen Aspergillus fumigatus are associated with caspofungin pr
71  in the growth and virulence of the pathogen Aspergillus fumigatus are unknown.
72                                              Aspergillus fumigatus can exploit the hypoxic microenvir
73 S (7-dimethylallyl tryptophan synthase) from Aspergillus fumigatus catalyze C(4)- and C(7)-prenylatio
74     The most common fungus in asthmatics was Aspergillus fumigatus complex and this taxon accounted f
75 ere asthmatics and the most common fungus is Aspergillus fumigatus complex.
76 % of the cell wall of the filamentous fungus Aspergillus fumigatus comprises polysaccharides.
77 T) formation, are involved in the killing of Aspergillus fumigatus conidia and hyphae, using neutroph
78 d cell death with apoptosis-like features in Aspergillus fumigatus conidia, the most prevalent human
79  following subchronic inhalation exposure to Aspergillus fumigatus conidia.
80                         Homology modeling of Aspergillus fumigatus DHODH has identified a predicted b
81  Here we report a fluorogenic probe to image Aspergillus fumigatus directly in human pulmonary tissue
82 elevance of the SeptiFast assay in detecting Aspergillus fumigatus DNA in whole blood samples from 38
83 (S) utilization by the human-pathogenic mold Aspergillus fumigatus during invasive growth.
84 ermatitis, we examined the effect of topical Aspergillus fumigatus extract exposure in wild-type and
85                                        In an Aspergillus fumigatus extract-induced inflammation model
86 rdingly, beta-glucan surface exposure during Aspergillus fumigatus germination activates an Atg5-depe
87                          Azole resistance in Aspergillus fumigatus has emerged as a global health pro
88                                The genome of Aspergillus fumigatus has four arsM genes encoding ArsMs
89 ndida albicans, Cryptococcus neoformans, and Aspergillus fumigatus have transitioned from a rare curi
90 /-) mice have an impaired ability to inhibit Aspergillus fumigatus hyphal growth in vitro and in infe
91                              Colonization by Aspergillus fumigatus in patients with cystic fibrosis (
92 mmune defense against the opportunistic mold Aspergillus fumigatus In this study, we investigated the
93 ere we show that the peroxiredoxin Asp f3 of Aspergillus fumigatus inactivates ROS.
94                  Our study demonstrates that Aspergillus fumigatus induces regulatory T-cells with a
95 nism different from that employed to control Aspergillus fumigatus infections.
96                          Azole resistance in Aspergillus fumigatus is an increasing problem.
97                                              Aspergillus fumigatus is an opportunistic fungal pathoge
98                                              Aspergillus fumigatus is an opportunistic human pathogen
99  the protection against fungal infections by Aspergillus fumigatus is essential but not fully underst
100 is due to a TR46/Y121F/T289A azole-resistant Aspergillus fumigatus is reported.
101                                              Aspergillus fumigatus is the causative agent of allergic
102                                              Aspergillus fumigatus is the most virulent species withi
103                                              Aspergillus fumigatus is the opportunistic fungal pathog
104                          Heterogeneity among Aspergillus fumigatus isolates results in unique virulen
105 illus species as well as azole resistance in Aspergillus fumigatus Its performance has been validated
106 cteria Pseudomonas aeruginosa and the fungus Aspergillus fumigatus M-CSF treatment during engraftment
107 -18 structures and a structural model of the Aspergillus fumigatus mtTyrRS showed that the overall to
108  and precocious activation of conidiation in Aspergillus fumigatus or Aspergillus flavus.
109 , CD8, and natural killer (NK) cells against Aspergillus fumigatus over 5 time points and compared th
110        The resistance assay was performed on Aspergillus fumigatus PCR-positive samples when a suffic
111  images of an in vivo mouse disease model of aspergillus fumigatus pneumonia.
112  elicited by epicutaneous sensitization with Aspergillus fumigatus protein extract.
113                                              Aspergillus fumigatus remains the most common species in
114 racterized the fungal opportunistic pathogen Aspergillus fumigatus Sch9 homologue (SchA).
115                                              Aspergillus fumigatus siderophore (SidA), a member of cl
116 utcomes following repeated inhalation of dry Aspergillus fumigatus spores aerosolized at concentratio
117                                              Aspergillus fumigatus spores were delivered to the lungs
118 man pathogenic fungus Aspergillus fumigatus, Aspergillus fumigatus tetramycovirus-1 (AfuTmV-1), which
119                                      We used Aspergillus fumigatus to induce EoE in TRAIL-sufficient
120   Sensitization to animal dander, pollen, or Aspergillus fumigatus was associated with asthma.
121                                              Aspergillus fumigatus was the most commonly identified s
122 as aeruginosa, Streptococcus pneumoniae, and Aspergillus fumigatus when mice were heavily engrafted w
123                        Neosartorya fumigata (Aspergillus fumigatus) is the most common cause of invas
124 exposed to spores of the environmental mould Aspergillus fumigatus, a major opportunistic pathogen.
125                                        Using Aspergillus fumigatus, a respiratory pathogen, we charac
126                                              Aspergillus fumigatus, a ubiquitous human fungal pathoge
127                               In contrast to Aspergillus fumigatus, A. terreus infections are associa
128 gE or skin prick test response positivity to Aspergillus fumigatus, Alternaria alternata, or Cladospo
129  pathogenic fungi including Candida species, Aspergillus fumigatus, and Cryptococcus neoformans.
130 hreatening lung disease caused by the fungus Aspergillus fumigatus, and is a leading cause of invasiv
131 cluding Pseudomonas aeruginosa, Haemophilus, Aspergillus fumigatus, and nontuberculous mycobacteria.
132 acoccidioides brasiliensis, and occasionally Aspergillus fumigatus, are primary pulmonary pathogens o
133 us isolated from the human pathogenic fungus Aspergillus fumigatus, Aspergillus fumigatus tetramycovi
134 ons with the opportunistic pathogenic fungus Aspergillus fumigatus, called aspergillosis.
135 llosis, an infection caused predominantly by Aspergillus fumigatus, have increased due to the growing
136      C57BL/6 and BALB/c mice were exposed to Aspergillus fumigatus, O3, or both (3 ppm for 2 hours).
137 ffectors against the widely distributed mold Aspergillus fumigatus, which is a major threat for immun
138  been shown to suppress house dust mite- and Aspergillus fumigatus-induced allergic inflammation in m
139 lycerol (HOG) pathway in the fungal pathogen Aspergillus fumigatus.
140 sults were available in 1 patient and showed Aspergillus fumigatus.
141  cluster in the opportunistic human pathogen Aspergillus fumigatus.
142 essential role in antifungal defense against Aspergillus fumigatus.
143  5 null (5) or Asp f 13 null (13) strains of Aspergillus fumigatus.
144 zed by an allergic immunological response to Aspergillus fumigatus.
145 le species, the opportunistic human pathogen Aspergillus fumigatus.
146  Netherlands might harbor triazole-resistant Aspergillus fumigatus.
147 infectious agents Listeria monocytogenes and Aspergillus fumigatus.
148           They are activated by contact with Aspergillus fumigatus; putative consequences include ant
149                               The utility of Aspergillus galactomannan (GM) and beta-D-glucan (BG) in
150 ma was spiked with various concentrations of Aspergillus genomic DNA before extraction following inte
151 s were spiked with various concentrations of Aspergillus genomic DNA for extraction, following intern
152 atus is the most virulent species within the Aspergillus genus and causes invasive infections with hi
153                          Molecular tests for Aspergillus have been limited historically by lack of st
154 minate, the extracellular destruction of the Aspergillus hyphae needs opsonization by Abs and involve
155  lacking both AIM2 and NLRP3 fail to confine Aspergillus hyphae to inflammatory foci, leading to wide
156 es the complexities of molecular testing for Aspergillus in clinical mycology.
157                                              Aspergillus incidence was 55% in the lower superoxide-pr
158           Allergic manifestations of inhaled Aspergillus include allergic bronchopulmonary aspergillo
159 6944 or rs419598 was associated with reduced Aspergillus-induced interleukin 1beta and tumor necrosis
160 ld reverse the pro-inflammatory phenotype of Aspergillus-induced regulatory T-cells.
161                          Enrichment of GT in Aspergillus-infected neutropenic lung correlated with fu
162 nd two occurred in the everolimus group (one aspergillus infection and one pneumonia aspiration).
163        Severe hypoxia was observed following Aspergillus infection in both models and correlated with
164 riazoles are the mainstay of therapy against Aspergillus infections for treatment and prophylaxis.
165     This is illustrated by the prevalence of Aspergillus infections in patients with neutropenia or p
166                        The manifestations of Aspergillus infections include invasive aspergillosis, c
167 y a pivotal role in the host defense against Aspergillus infections.
168 to delineate antifungal drug efficacy in non-Aspergillus invasive mold infections (NAIMIs).
169                             The fungal genus Aspergillus is of critical importance to humankind.
170 athogenic molds (154 Aspergillus and 136 non-Aspergillus isolates) with the BMD and Etest methods.
171  PCR assay based on amplification of the pan-Aspergillus ITS1/5.8S ribosomal operon.
172 at had PK or EK for failed TPK conducted for Aspergillus keratitis showed better outcomes in terms of
173 perienced repeated pulmonary infections with Aspergillus, leading to multiple hospitalizations and lo
174                             Mechanistically, Aspergillus melanin inhibits NADPH oxidase-dependent act
175 gnostic setting, but failure to identify non-Aspergillus molds limits its usefulness.
176 nbreakthrough IMIs shows a shift towards non-Aspergillus molds with a significantly increased proport
177                                          The Aspergillus nidulans 7-TMD receptor PalH senses alkaline
178 crystal structures of C-terminally truncated Aspergillus nidulans and Coccidioides posadasii mtTyrRSs
179 terised all Fur proteins of the model fungus Aspergillus nidulans and discovered novel functions and
180 educe the secondary metabolite background in Aspergillus nidulans and minimize the rediscovery of com
181                                     However, Aspergillus nidulans and vertebrate Nup2 also locate to
182               Hyphal tip cells of the fungus Aspergillus nidulans are useful for studying long-range
183                    In the filamentous fungus Aspergillus nidulans BrlA triggers the central developme
184 on of a 237 residue deacetylase (AnCDA) from Aspergillus nidulans FGSC A4.
185        We have developed a genetic screen in Aspergillus nidulans for negative regulators of fungal S
186                                          The Aspergillus nidulans genome encodes 16 putative GPCRs, b
187 the melanin pathway, we utilized an advanced Aspergillus nidulans heterologous system for the express
188 the model fungi Saccharomyces cerevisiae and Aspergillus nidulans However, the roles of myosins in th
189 ges during mitosis, which in vertebrates and Aspergillus nidulans involves movement of Nup2 from NPCs
190 ment (conidiation) in the filamentous fungus Aspergillus nidulans is governed by orchestrated gene ex
191                                       During Aspergillus nidulans mitosis peripheral nuclear pore com
192                    In the filamentous fungus Aspergillus nidulans SPBs and septum-associated MTOCs we
193 is work, we investigated the contribution of Aspergillus nidulans sphingolipid Delta8-desaturase (Sde
194            We show that the nearly essential Aspergillus nidulans syntaxin PepA(Pep12) , present in a
195                             The model fungus Aspergillus nidulans synthesizes numerous secondary meta
196                                 Tolerance of Aspergillus nidulans to alkalinity and elevated cation c
197 riptional response of the filamentous fungus Aspergillus nidulans to the presence of high and low glu
198                 Using the filamentous fungus Aspergillus nidulans we found that hitchhiking is mediat
199 xternal intron (experimentally confirmed for Aspergillus nidulans).
200 discovery of VezA, a vezatin-like protein in Aspergillus nidulans, as a factor critical for early end
201  of mitochondrial morphology and function in Aspergillus nidulans, systematic characterization was ca
202 cent protein fusions of four SAC proteins in Aspergillus nidulans, the homologs of Mad2, Mps1, Bub1/B
203 tracking MT +end-binding proteins (+TIPS) in Aspergillus nidulans, we find that MTs are regulated to
204                                        Using Aspergillus nidulans, we show that AP-2 has a clathrin-i
205    This investigation focuses on clathrin in Aspergillus nidulans, with the aim of understanding its
206 cence microscopy that KlpA-a kinesin-14 from Aspergillus nidulans-is a context-dependent bidirectiona
207 rine transporter from the filamentous fungus Aspergillus nidulans.
208  the last few decades--Neurospora crassa and Aspergillus nidulans.
209 zae but was dissimilar to the non-oleaginous Aspergillus nidulans.
210 F-kB like fungal regulators VosA and VelB in Aspergillus nidulans.
211 ate the usage of a prolyl endopeptidase from Aspergillus niger (An-PEP) for HDX-MS.
212 ependent glucose dehydrogenase, derived from Aspergillus niger (AnGDH), was characterized.
213                                              Aspergillus niger also precipitated lead oxalate during
214                                              Aspergillus niger and Paecilomyces javanicus grew in 5 m
215  industry) and brandy distillery wastes with Aspergillus niger and Rhizopus oligosporus were investig
216                          In the present work Aspergillus niger beta-glucosidase is immobilized within
217                              Trichoderma and Aspergillus niger cellulases activities were determined
218 (Cu2 (CO3 )(OH)2 ) and bioaccumulated within Aspergillus niger colonies when grown on different inorg
219 nd purified alpha-casein (alphas-CN) with an Aspergillus niger derived prolyl endoproteinase (An-PEP)
220 sing Escherichia coli expressing recombinant Aspergillus niger epoxide hydrolase as the model enzyme
221                                        Using Aspergillus niger Fdc1 as a model system, we reveal that
222  the emulsions assays were conducted against Aspergillus niger given its strong resistance and its re
223  as a dough and bread improver, similarly to Aspergillus niger glucose oxidase (GOX).
224 gh resolution x-ray structures of a PME from Aspergillus niger in deglycosylated and Asn-linked N-ace
225 d and identified as Penicillium oxalicum and Aspergillus niger respectively in this study.
226 tnC from the kotanin biosynthetic pathway of Aspergillus niger was expressed in Saccharomyces cerevis
227 alyze the process, and beta-glucosidase from Aspergillus niger was selected.
228 ausing parasite Plasmodium vivax, the fungus Aspergillus niger, and the TEM-family of beta-lactamase
229  successful history of citrate production in Aspergillus niger, the molecular mechanism of citrate ac
230  enzymes (CAZymes) of the filamentous fungus Aspergillus niger.
231                      The direct detection of Aspergillus nucleic acid in clinical specimens has the p
232 al resolution was used to collect spectra of Aspergillus ochraceus, a mold producer of ochratoxin A (
233 ohol were tested against Aspergillus flavus, Aspergillus ochraceus, Fusarium oxysporum, Saccharomyces
234  of life were not detected for patients with Aspergillus or other non-Fusarium species as the causati
235 ucoid P. aeruginosa (OR, 0.77; P = .013) and Aspergillus (OR, 0.47; P = .039), but not Staphylococcus
236                   The reductive aminase from Aspergillus oryzae (AspRedAm) was combined with a single
237  an NADP(H)-dependent reductive aminase from Aspergillus oryzae (AspRedAm, Uniprot code Q2TW47) that
238  through rational heterologous expression in Aspergillus oryzae coupled with isolation and detailed s
239 euromutilin cluster was reconstructed within Aspergillus oryzae giving production of pleuromutilin in
240                                              Aspergillus oryzae showed stability at all pH values stu
241 y flour fermented with Monascus purpureus or Aspergillus oryzae.
242 Bacillus circulans, Kluyveromyces lactis and Aspergillus oryzae.
243 tarch aerogel and led to slower lethality of Aspergillus parasiticus cells inoculated on pistachio nu
244 tous fungus and an important plant pathogen, Aspergillus parasiticus, without inhibiting fungal growt
245 anding mycelium of a popular plant pathogen, Aspergillus parasiticus.
246 , fungal culture, galactomannan antigen, and aspergillus PCR are useful tests.
247                 With the proposal to include Aspergillus PCR in the revised European Organization for
248 s (IA) have been established by the European Aspergillus PCR Initiative for the testing of whole bloo
249 plantation, respiratory virus infection, and Aspergillus PCR positivity were all significant risk fac
250 time PCR and, with clinical risk factors and Aspergillus PCR results, subjected to multilogistic regr
251                     This study confirms that Aspergillus PCR testing of plasma provides robust perfor
252                                              Aspergillus PCR testing of serum provides technical simp
253                                 Standardized Aspergillus PCR was performed on 423 whole-blood pellets
254                                 Standardized Aspergillus PCR was performed on plasma and serum sample
255          The sensitivity and specificity for Aspergillus PCR when testing serum were 68.4% and 76.2%,
256  While some comparison of the performance of Aspergillus PCR when testing these different sample type
257  The current study focused on germination of Aspergillus penicillioides, a xerophile which is also ab
258  Spores from a variety of species, including Aspergillus penicillioides, Eurotium halophilicum, Xeroc
259 ustralian database for the identification of Aspergillus, Scedosporium, and Fusarium species (n = 28)
260 clinically important fungi (CIF), defined as Aspergillus, Scedosporium, and Trichosporon species and
261 entity of a set of 34 clinical isolates from Aspergillus section Circumdati from the United States an
262 claudin-18 null and wild-type mice following aspergillus sensitization.
263 d airway responsiveness following intranasal aspergillus sensitization.
264  able to accurately identify 133/144 (93.6%) Aspergillus sp. isolates to the species level.
265              The dominant fungal genera were Aspergillus sp., Rhizopus sp., Penicillium sp. and Saroc
266 nome sequences for ten novel, highly diverse Aspergillus species and compared these in detail to sist
267 st that has the ability to detect a range of Aspergillus species as well as azole resistance in Asper
268                         Doxycycline-exposed, Aspergillus species extract-challenged CC10-IL-15 bitran
269        Resistance mechanisms for Candida and Aspergillus species have been extensively described and
270 The ability of neutrophils to interfere with Aspergillus species hyphal growth was impaired after HSC
271           Azole resistance among Candida and Aspergillus species is one of the greatest challenges to
272                                 Fusarium and Aspergillus species were the two predominant molds accou
273 ween invasive fungi (eg, Candida species and Aspergillus species) and pathogenic bacteria can be part
274 ve pathogens of these outbreaks were usually Aspergillus species, but Zygomycetes and other fungi wer
275 rol and bioremediation technologies based on Aspergillus species.
276 s the second transesterification reaction in Aspergillus species.
277 dole alkaloids isolated from Penicillium and Aspergillus species.
278 ndida species and fungistatic action against Aspergillus species.
279                                           An Aspergillus-specific lateral-flow device (LFD), quantita
280 , drug interactions, and azole resistance in Aspergillus spp limit therapy.
281 posttransplantation airway colonization with Aspergillus spp.
282                                              Aspergillus spp. (17%) and Staphylococcus aureus (10.7%)
283 Etest MIC values were consistently lower for Aspergillus spp. (EAs of <90%).
284               When wounds with Mucorales and Aspergillus spp. growth were compared, there was no sign
285 an 100 strains from the four main pathogenic Aspergillus spp. revealed minimal inhibitory concentrati
286 oides immitis/posadasii, Fusarium oxysporum, Aspergillus spp., and Bipolaris spp.) was also identifie
287 e molds (predominant types: order Mucorales, Aspergillus spp., and Fusarium spp.) significantly prolo
288 uses include Candida spp., Fusarium spp. and Aspergillus spp..
289       This is the first study introducing an Aspergillus strain for benzene removal and these results
290 MIC) values against non-albicans Candida and Aspergillus strains.
291  study was to improve the thermostability of Aspergillus sulphureus acidic xylanase.
292 hunamides A-G, were isolated from the fungus Aspergillus taichungensis (IBT 19404).
293 lator laeA, and deletion of mcrA homologs in Aspergillus terreus and Penicillum canescens alters the
294                                              Aspergillus terreus is an airborne human fungal pathogen
295                              A fungus called Aspergillus terreus produces a secondary metabolite in r
296 errein production were studied in the fungus Aspergillus terreus to elucidate the contribution of ter
297 entation of sugars by the filamentous fungus Aspergillus terreus.
298 roliferative cyclic tripeptide isolated from Aspergillus versicolor ZLN-60, is reported herein.
299 sentation of Candida spp. was reduced, while Aspergillus, Wallemia, and Epicoccum spp. were increased
300  in response to environmental Alternaria and Aspergillus, was elevated in children with a maternal hi

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