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1 lass in the management of diseases caused by Aspergillus.
2 affecting the quantity of asexual spores in Aspergillus.
3 ared with that for the Bruker library alone (Aspergillus, 93% versus 69%; Fusarium, 84% versus 42%; a
6 ere misidentified by MALDI-TOF MS (including Aspergillus amoenus [n = 2] and Aspergillus calidoustus
7 found to expand during antifungal treatment (Aspergillus amstelodami, Epicoccum nigrum, and Wallemia
8 he most representative pathogenic molds (154 Aspergillus and 136 non-Aspergillus isolates) with the B
9 o identify selected toxigenic species of the Aspergillus and Fusarium genera isolated from grapes and
10 xin-producing fungal species from the genera Aspergillus and Fusarium using solid-state voltammetry i
12 a natural carcinogenic mycotoxin produced by Aspergillus and Penicillium fungi and so it commonly app
15 d therapy had significantly higher levels of Aspergillus and total fungus in their bronchoalveolar la
17 ntifungal immunity (primarily to Candida and Aspergillus) and will also examine the emerging field of
18 f asthma symptoms suggests that Penicillium, Aspergillus, and Cladosporium species pose a respiratory
19 ed ceftriaxone administration and culturable Aspergillus, and demonstrated significantly increased in
20 The presence of Cladosporium, Alternaria, Aspergillus, and Penicillium species increased the exace
22 this study, we investigated whether certain Aspergillus antigens are more allergenic than others, as
25 th a pro-inflammatory TH17-like phenotype in Aspergillus-associated immunopathology, and identifies k
27 S (including Aspergillus amoenus [n = 2] and Aspergillus calidoustus [n = 1]) although 10 (3.1%) of t
28 ung or airways infection by endemic fungi or aspergillus can be diagnosed with respiratory sample cul
29 ciated with increased levels of Penicillium, Aspergillus, Cladosporium, and Alternaria species, altho
30 ndings reveal distinct killing mechanisms of Aspergillus conidia and hyphae by human neutrophils, lea
32 for effective host defense against Candida, Aspergillus, Cryptococcus, and others, with specific ele
35 hain reaction (PCR)-based detection of serum Aspergillus DNA for the early diagnosis and therapy of i
36 ed monitoring strategy based on serum GM and Aspergillus DNA was associated with an earlier diagnosis
37 (bmGT) are secondary metabolites produced by Aspergillus during invasive, hyphal growth and may prove
38 s relatively low (<10%), despite unavoidable Aspergillus exposure in patients with a potentially simi
39 two separate allergic stimuli (ovalbumin and Aspergillus extract), genetic removal of Muc5ac abolishe
44 over, treatment with voriconazole due to the Aspergillus flavus and meropenem due to the Pseudomonas
45 s, a qualitative analytical method to detect Aspergillus flavus in food samples, based on the identif
48 cies in all pulmonary syndromes, followed by Aspergillus flavus which is a common cause of allergic r
49 ol, and perillyl alcohol were tested against Aspergillus flavus, Aspergillus ochraceus, Fusarium oxys
50 xin contamination, caused by fungal pathogen Aspergillus flavus, is a major quality and health proble
53 hogens, including real-time visualization of Aspergillus fumigatus (5 d for culturing, 1-2 d for imag
54 HDM-exposed mothers, the magnitude of HDM or Aspergillus fumigatus (AF) extract-induced airway hyperr
56 relevant fungi such as Candida albicans and Aspergillus fumigatus also form biofilms during infectio
58 s, caused most commonly by Candida albicans, Aspergillus fumigatus and Cryptococcus neoformans, resul
59 ad of agriculturally derived azole-resistant Aspergillus fumigatus and emerging threats such as multi
60 onse to alpha-(1,3)-glucan polysaccharide of Aspergillus fumigatus and ensuing CD4+ T-cell polarizati
61 lungs of mice sensitized and challenged with Aspergillus fumigatus and evaluated ex vivo in tissue cu
62 cy of pH- and calcium-mediated signalling in Aspergillus fumigatus and found that calcium chelation s
63 d juvenile Scnn1b-Tg and wild-type mice with Aspergillus fumigatus and house dust mite allergen and c
64 ides produced by the opportunistic pathogens Aspergillus fumigatus and Pseudomonas aeruginosa, respec
66 ere highly susceptible to aeroallergens from Aspergillus fumigatus and the house dust mite, resulting
67 onditions may result from the interaction of Aspergillus fumigatus and the immune system of its human
70 breakthrough infections with fungal pathogen Aspergillus fumigatus are associated with caspofungin pr
73 S (7-dimethylallyl tryptophan synthase) from Aspergillus fumigatus catalyze C(4)- and C(7)-prenylatio
74 The most common fungus in asthmatics was Aspergillus fumigatus complex and this taxon accounted f
77 T) formation, are involved in the killing of Aspergillus fumigatus conidia and hyphae, using neutroph
78 d cell death with apoptosis-like features in Aspergillus fumigatus conidia, the most prevalent human
81 Here we report a fluorogenic probe to image Aspergillus fumigatus directly in human pulmonary tissue
82 elevance of the SeptiFast assay in detecting Aspergillus fumigatus DNA in whole blood samples from 38
84 ermatitis, we examined the effect of topical Aspergillus fumigatus extract exposure in wild-type and
86 rdingly, beta-glucan surface exposure during Aspergillus fumigatus germination activates an Atg5-depe
89 ndida albicans, Cryptococcus neoformans, and Aspergillus fumigatus have transitioned from a rare curi
90 /-) mice have an impaired ability to inhibit Aspergillus fumigatus hyphal growth in vitro and in infe
92 mmune defense against the opportunistic mold Aspergillus fumigatus In this study, we investigated the
99 the protection against fungal infections by Aspergillus fumigatus is essential but not fully underst
105 illus species as well as azole resistance in Aspergillus fumigatus Its performance has been validated
106 cteria Pseudomonas aeruginosa and the fungus Aspergillus fumigatus M-CSF treatment during engraftment
107 -18 structures and a structural model of the Aspergillus fumigatus mtTyrRS showed that the overall to
109 , CD8, and natural killer (NK) cells against Aspergillus fumigatus over 5 time points and compared th
116 utcomes following repeated inhalation of dry Aspergillus fumigatus spores aerosolized at concentratio
118 man pathogenic fungus Aspergillus fumigatus, Aspergillus fumigatus tetramycovirus-1 (AfuTmV-1), which
122 as aeruginosa, Streptococcus pneumoniae, and Aspergillus fumigatus when mice were heavily engrafted w
124 exposed to spores of the environmental mould Aspergillus fumigatus, a major opportunistic pathogen.
128 gE or skin prick test response positivity to Aspergillus fumigatus, Alternaria alternata, or Cladospo
130 hreatening lung disease caused by the fungus Aspergillus fumigatus, and is a leading cause of invasiv
131 cluding Pseudomonas aeruginosa, Haemophilus, Aspergillus fumigatus, and nontuberculous mycobacteria.
132 acoccidioides brasiliensis, and occasionally Aspergillus fumigatus, are primary pulmonary pathogens o
133 us isolated from the human pathogenic fungus Aspergillus fumigatus, Aspergillus fumigatus tetramycovi
135 llosis, an infection caused predominantly by Aspergillus fumigatus, have increased due to the growing
137 ffectors against the widely distributed mold Aspergillus fumigatus, which is a major threat for immun
138 been shown to suppress house dust mite- and Aspergillus fumigatus-induced allergic inflammation in m
150 ma was spiked with various concentrations of Aspergillus genomic DNA before extraction following inte
151 s were spiked with various concentrations of Aspergillus genomic DNA for extraction, following intern
152 atus is the most virulent species within the Aspergillus genus and causes invasive infections with hi
154 minate, the extracellular destruction of the Aspergillus hyphae needs opsonization by Abs and involve
155 lacking both AIM2 and NLRP3 fail to confine Aspergillus hyphae to inflammatory foci, leading to wide
159 6944 or rs419598 was associated with reduced Aspergillus-induced interleukin 1beta and tumor necrosis
162 nd two occurred in the everolimus group (one aspergillus infection and one pneumonia aspiration).
164 riazoles are the mainstay of therapy against Aspergillus infections for treatment and prophylaxis.
165 This is illustrated by the prevalence of Aspergillus infections in patients with neutropenia or p
170 athogenic molds (154 Aspergillus and 136 non-Aspergillus isolates) with the BMD and Etest methods.
172 at had PK or EK for failed TPK conducted for Aspergillus keratitis showed better outcomes in terms of
173 perienced repeated pulmonary infections with Aspergillus, leading to multiple hospitalizations and lo
176 nbreakthrough IMIs shows a shift towards non-Aspergillus molds with a significantly increased proport
178 crystal structures of C-terminally truncated Aspergillus nidulans and Coccidioides posadasii mtTyrRSs
179 terised all Fur proteins of the model fungus Aspergillus nidulans and discovered novel functions and
180 educe the secondary metabolite background in Aspergillus nidulans and minimize the rediscovery of com
187 the melanin pathway, we utilized an advanced Aspergillus nidulans heterologous system for the express
188 the model fungi Saccharomyces cerevisiae and Aspergillus nidulans However, the roles of myosins in th
189 ges during mitosis, which in vertebrates and Aspergillus nidulans involves movement of Nup2 from NPCs
190 ment (conidiation) in the filamentous fungus Aspergillus nidulans is governed by orchestrated gene ex
193 is work, we investigated the contribution of Aspergillus nidulans sphingolipid Delta8-desaturase (Sde
197 riptional response of the filamentous fungus Aspergillus nidulans to the presence of high and low glu
200 discovery of VezA, a vezatin-like protein in Aspergillus nidulans, as a factor critical for early end
201 of mitochondrial morphology and function in Aspergillus nidulans, systematic characterization was ca
202 cent protein fusions of four SAC proteins in Aspergillus nidulans, the homologs of Mad2, Mps1, Bub1/B
203 tracking MT +end-binding proteins (+TIPS) in Aspergillus nidulans, we find that MTs are regulated to
205 This investigation focuses on clathrin in Aspergillus nidulans, with the aim of understanding its
206 cence microscopy that KlpA-a kinesin-14 from Aspergillus nidulans-is a context-dependent bidirectiona
215 industry) and brandy distillery wastes with Aspergillus niger and Rhizopus oligosporus were investig
218 (Cu2 (CO3 )(OH)2 ) and bioaccumulated within Aspergillus niger colonies when grown on different inorg
219 nd purified alpha-casein (alphas-CN) with an Aspergillus niger derived prolyl endoproteinase (An-PEP)
220 sing Escherichia coli expressing recombinant Aspergillus niger epoxide hydrolase as the model enzyme
222 the emulsions assays were conducted against Aspergillus niger given its strong resistance and its re
224 gh resolution x-ray structures of a PME from Aspergillus niger in deglycosylated and Asn-linked N-ace
226 tnC from the kotanin biosynthetic pathway of Aspergillus niger was expressed in Saccharomyces cerevis
228 ausing parasite Plasmodium vivax, the fungus Aspergillus niger, and the TEM-family of beta-lactamase
229 successful history of citrate production in Aspergillus niger, the molecular mechanism of citrate ac
232 al resolution was used to collect spectra of Aspergillus ochraceus, a mold producer of ochratoxin A (
233 ohol were tested against Aspergillus flavus, Aspergillus ochraceus, Fusarium oxysporum, Saccharomyces
234 of life were not detected for patients with Aspergillus or other non-Fusarium species as the causati
235 ucoid P. aeruginosa (OR, 0.77; P = .013) and Aspergillus (OR, 0.47; P = .039), but not Staphylococcus
237 an NADP(H)-dependent reductive aminase from Aspergillus oryzae (AspRedAm, Uniprot code Q2TW47) that
238 through rational heterologous expression in Aspergillus oryzae coupled with isolation and detailed s
239 euromutilin cluster was reconstructed within Aspergillus oryzae giving production of pleuromutilin in
243 tarch aerogel and led to slower lethality of Aspergillus parasiticus cells inoculated on pistachio nu
244 tous fungus and an important plant pathogen, Aspergillus parasiticus, without inhibiting fungal growt
248 s (IA) have been established by the European Aspergillus PCR Initiative for the testing of whole bloo
249 plantation, respiratory virus infection, and Aspergillus PCR positivity were all significant risk fac
250 time PCR and, with clinical risk factors and Aspergillus PCR results, subjected to multilogistic regr
256 While some comparison of the performance of Aspergillus PCR when testing these different sample type
257 The current study focused on germination of Aspergillus penicillioides, a xerophile which is also ab
258 Spores from a variety of species, including Aspergillus penicillioides, Eurotium halophilicum, Xeroc
259 ustralian database for the identification of Aspergillus, Scedosporium, and Fusarium species (n = 28)
260 clinically important fungi (CIF), defined as Aspergillus, Scedosporium, and Trichosporon species and
261 entity of a set of 34 clinical isolates from Aspergillus section Circumdati from the United States an
266 nome sequences for ten novel, highly diverse Aspergillus species and compared these in detail to sist
267 st that has the ability to detect a range of Aspergillus species as well as azole resistance in Asper
270 The ability of neutrophils to interfere with Aspergillus species hyphal growth was impaired after HSC
273 ween invasive fungi (eg, Candida species and Aspergillus species) and pathogenic bacteria can be part
274 ve pathogens of these outbreaks were usually Aspergillus species, but Zygomycetes and other fungi wer
285 an 100 strains from the four main pathogenic Aspergillus spp. revealed minimal inhibitory concentrati
286 oides immitis/posadasii, Fusarium oxysporum, Aspergillus spp., and Bipolaris spp.) was also identifie
287 e molds (predominant types: order Mucorales, Aspergillus spp., and Fusarium spp.) significantly prolo
293 lator laeA, and deletion of mcrA homologs in Aspergillus terreus and Penicillum canescens alters the
296 errein production were studied in the fungus Aspergillus terreus to elucidate the contribution of ter
299 sentation of Candida spp. was reduced, while Aspergillus, Wallemia, and Epicoccum spp. were increased
300 in response to environmental Alternaria and Aspergillus, was elevated in children with a maternal hi
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