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1 olation from protoplasts of Petunia and oat (Avena sativa).
2 tro with recombinant phytochrome A from oat (Avena sativa).
3 he light-oxygen-voltage 2 (LOV2) domain from Avena sativa.
4 additions to the haploid complement of oat (Avena sativa, 2n = 6x = 42) among F(1) plants generated
6 us victoriae causes Victoria blight of oats (Avena sativa) and is pathogenic due to its production of
8 sion profiles for multiple cultivars of oat (Avena sativa) and wheat with and without cold treatment.
9 ey (Hordeum vulgare), maize (Zea mays), oat (Avena sativa), and wheat (Triticum aestivum); but the di
10 ulgare), wheat (Triticum aestivum), and oat (Avena sativa) are anchored by a set of curated correspon
11 mydomonas reinhardtii and the LOV2 domain of Avena sativa, both before and after the photoreaction, t
14 sis heat shock protein 21 (HSP21) mRNA, oat (Avena sativa) globulin, wheat (Triticum aestivum) germin
16 of T1 plants of the cultivated hexaploid oat Avena sativa L. cotransformed by microprojectile bombard
17 ynthase from etioplasts from dark-grown oat (Avena sativa L. cv Garry) seedlings using traditional co
18 We solubilized 90% of the FCBP from oat (Avena sativa L. cv Victory) root PM in an active form wi
20 ro in plasma membrane preparations from oat (Avena sativa L.) aleurone and from leaves and stems of w
23 somes present in plants with a complete oat (Avena sativa L.) chromosome complement provides a unique
24 and in vivo protein phosphorylations in oat (Avena sativa L.) coleoptile segments were analyzed by so
25 a mays L.) chromosome addition lines of oat (Avena sativa L.) from oat x maize crosses enables us to
26 enome was investigated in 13 transgenic oat (Avena sativa L.) lines produced using microprojectile bo
27 nsgene loci in two unrelated transgenic oat (Avena sativa L.) lines transformed using microprojectile
28 cut from the peduncular-1 internode of oat (Avena sativa L.) shoots so as to contain the gravirespon
29 partitioning method from two different oat (Avena sativa L.) tissues, the root and coleoptile, was c
30 etiolated wheat (Triticum aestivum L.), oat (Avena sativa L.), barley (Hordeum vulgare L.), tobacco (
32 e been recovered via embryo rescue from oat (Avena sativa L., 2n = 6x = 42) x maize (Zea mays L., 2n
33 addition lines of hexaploid cultivated oat (Avena sativa L., 2n = 6x = 42), where maize chromosomes
34 ed the effects of more than 100 mutations in Avena sativa light-oxygen-voltage domain 2, a model prot
35 he inhibitory domains to the light-sensitive Avena sativa light-oxygen-voltage-sensing (LOV) 2-photot
39 mutagenized population of LOV2 derived from Avena sativa (oat) phot1 were screened for variants that
41 hetic interaction between the LOV2 domain of Avena sativa phototropin 1 (AsLOV2) and an engineered PD
43 n the naturally photoactive LOV2 domain from Avena sativa phototropin 1 and the Escherichia coli trp
44 t a conserved glutamine residue [Q513 in the Avena sativa phototropin 1 LOV2 (AsLOV2) domain] switche
46 combinant C450A mutant of the LOV2 domain of Avena sativa phototropin was reconstituted with universa
47 ting myosin VI by fusing the light-sensitive Avena sativa phototropin1 LOV2 domain to a peptide from
48 eletion and alanine-scanning mutants of oat (Avena sativa) phyA in transgenic tobacco (Nicotiana taba
49 ochemical and functional characterization of Avena sativa phytochrome A (AsphyA) as a potential prote
50 stivum), barley (Hordeum vulgare), and oats (Avena sativa), predominate in the northern temperate zon
52 c tissues from rye (Secale cereale) and oat (Avena sativa) were studied in an isothermal calorimeter
53 tions, we singly inoculated and coinoculated Avena sativa with two virus species, barley yellow dwarf
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