ライフサイエンスコーパス: B

1 tment of the protein phosphatase 2A B' (PP2A B').

2 xamination and Trail Making Test Parts A and B.

3 brane, resulting in the release of Cathepsin B.

4 repression of the lysomal protease cathepsin B.

5 length is associated with familial risk for BD.

6 ased in the amygdala of subjects with SZ and BD.

7 oration of the mastectomy scar and radiation bed.

8 OL (B = 2.61; P < .001) and less depression (B = -0.78; P < .001) among patients who reported a termi

9 o provide spatial selectivity with arbitrary B 1 field distributions in non-conductors.

10 We demonstrate that Env-specific HLA-B*14-restricted activity is substantially more efficacio

11 ly more efficacious than the subdominant HLA-B*14-restricted Gag response.

12 8)F-labeling methodologies, specifically for B-(18)F, Si-(18)F, Al-(18)F, and iodine (III)-mediated r

13 of 65.7% (95% CI, 59.4%-72.1%) and in trial B, 192 of 255 (75.3%) vs 25 of 260 (9.6%; P < .001), for

14 cterial sepsis, we found that the numbers of B-1a cells in various anatomical locations were signific

15 ositive reframing was related to better QOL (B = 2.61; P < .001) and less depression (B = -0.78; P <

16 l duplication, and the smaller 1.5 Mb LCR22A-B 22q11.2 deletion carry inversions of LCR22B-D or LCR22

17 strains of bacteria: Lactobacillus rhamnosus B 442, Lactobacillus rhamnosus 1937, and Lactococcus lac

18 Given the approximate prevalence of HLA-B*53 carriage in African (20%) and Hispanic (6%) populat

19 the probability of all 4 patients being HLA-B*53 carriers, and 2 of 3 African patients being homozyg

20 3 African patients being homozygous for HLA-B*53:01, is approximately 0.00002.

21 rinated ethene removal was 87% in the CYP2E1 bed, 85% in the WT bed, and 34% in the unplanted bed in

22 oup A (98.2%) and 50 of 57 patients in group B (87.7%; P=0.06).

23 4 months of age was 43% in arm A, 52% in arm B (9%, 7-11) and 54% in arm C (11%, 9-13; overall p<0.00

24 e that ectopic expression of insulin epitope B:9-23 (InsB9-23) by thymic APCs is insufficient to indu

25 ression contributes to maintenance of murine B-ALL cells with compromised Ikaros function.

26 nic model, that over-expression of VEGF-A165 b alone is sufficient to rescue the increase in proteinu

27 enza haemagglutinin and botulinum neurotoxin B, along with 6,286 control sequences to probe contribut

28 fluorescence and excellent photostability, (b) an N-methyl group at each end of the squaraine core t

29 In this model, a loop defined by the SCI's B and C domains encircles the C-terminal segment of the

30 f E.coli, as well as effects of amphotericin-B and miconazole on S. cerevisiae through the device's t

31 m are controlled at low concentrations (both B and P are less than 1 ppm).

32 S-induced activation of nuclear factor kappa B and reduced the expression of proinflammatory proteins

33 ARTEMIS is essential for the development of B and T lymphocytes.

34 ated the possible involvement of the subunit b and the OSCP in the PTP by generating clonal cells, HA

35 tion into a set number of bins (quantization B) and an alternative quantization with bins of fixed wi

36 Schizophrenia (SCZ), bipolar disorder (BD) and recurrent major depressive disorder (rMDD) are c

37 (75%) had esophageal varices, 21% were Child-B, and 29% had at least 1 previous episode of liver deco

38 wer resolution) HLA typing for HLA-A and HLA-B, and allele-level for HLA-DRB1.

39 uding amphotericin B, liposomal amphotericin B, and flucytosine, need to be much more widely availabl

40 ound that rmIFN induces the activation of T, B, and NK cells and exhibits antiviral, antiproliferativ

41 arachidonoyl-fluorophosphate and palmostatin B, and partially prevented by JZL184 and WWL113.

42 val was 87% in the CYP2E1 bed, 85% in the WT bed, and 34% in the unplanted bed in 2012.

43 (27-2450 MHz), moisture content (4.2-19.6% w.b.) and temperature (20-90 degrees C).

44 thracis DNA into individual PCR mixtures and B. anthracis CFU into human blood.

45 and dynamic range were determined by spiking B. anthracis DNA into individual PCR mixtures and B. ant

46 Among the virulence factors of B. anthracis is the S-layer-associated protein BslA, whi

47 ter-based detection cartridge and tested for B. anthracis on a GeneXpert instrument.

48 s in growth characteristics among strains of B. anthracis, which is considered to be a clonal organis

49 id antibody, and anti-Ro/SS-A and anti-La/SS-B antibodies.

50 ng the cytokinin response, mechanism of type-B ARR activation, and basis by which cytokinin regulates

51 light on the physiological role of the type-B ARRs in regulating the cytokinin response, mechanism o

52 om the interface Si atoms to the neighboring B atoms.

53 prevalence and abundance of putatively anti-Bd bacteria to determine if Bd-inhibitory bacteria are d

54 re produced by laser vaporization of a mixed B/Bi target and characterized by photoelectron spectrosc

55 We also found that the RING B-box coiled-coil (RBCC) domain in KAP1 and the proximal

56 After passage of the B/Brisbane/60/2008 virus in the presence of 46B8, we iso

57 perature adaptation between B. pertussis and B. bronchiseptica may result from selective adaptation o

58 As one of the most notorious ectoparasites, bed bugs rely heavily on human or animal blood sources f

59 is is the first comprehensive map of TSSs in B. burgdorferi and characterization of previously un-ann

60 These results show that B. burgdorferi can transform a ubiquitous but normally n

61 rrelia burgdorferi Here, we investigated how B. burgdorferi exploits Fn to interact with endothelia u

62 brain differences have been associated with BD, but results from neuroimaging studies have been inco

63 Three independent observers (A, B, C) measured PVRs at two different time points during

64 ms based on the trnL gene were designed (A-, B-, C- and D-trnL systems).

65 c assay is a highly discriminatory assay for B. canis genotyping, and can serve as a useful molecular

66 the source of contamination in an event of a B. canis outbreak.

67 d MLVA methods are limited in discriminating B. canis strains.

68 arabinose residues, decreased the extent of B cell activation.

69 affinity-matured human NANP-reactive memory B cell antibodies elicited by natural Pf exposure that p

70 the treatment of B cell neoplasms, including B cell chronic lymphocytic leukemia (B-CLL).

71 Unlike B cell depletion, pan-T cell aplasia is prohibitively to

72 nduced mTOR activation impairs IL-7-mediated B cell development.

73 SIV-associated B cell dysfunction associated with the pathogenic SIV in

74 s an innate link between viral infection and B cell immunity.

75 e therefore expected to evolve only when the B cell lineage evolving breadth is consistently capturin

76 on and are associated with cancer, including B cell lymphomas.

77 0-positive cells did not display the typical B cell morphology, having in general a more dendritic ce

78 hallmark anti-CD20 mAb for the treatment of B cell neoplasms, including B cell chronic lymphocytic l

79 on Network Analysis (WGCNA) was enriched for B cell pathways, and shared seventeen genes with a mouse

80 ance/survival of the mature naive peripheral B cell population.

81 patient suffering from recurrent EBV-induced B cell proliferations including Hodgkin's lymphoma becau

82 evels of B-lineage transcription factors and B cell receptor (BCR)/pre-BCR-signaling genes.

83 e have demonstrated that the kinetics of the B cell response are similar for all four FMDV serotypes

84 nt resulted in suppression of virus-specific B cell responses by inhibiting proliferation of germinal

85 suppresses follicular helper T cell-mediated B cell responses in the germinal center reaction.

86 ory molecule playing a role in physiological B cell responses.

87 protective or deleterious effects on loss of B cell self-tolerance in vivo, we depleted neutrophils a

88 This dynamic change in B cell survival mechanisms is unique to virus-infected c

89 ), MRD detection method, phenotype/genotype (B cell, T cell, Philadelphia chromosome), and EFS and OS

90 ed to lethality in infected animals, whereas B cell-deficient mice showed CD4(+) T cell loss but reco

91 Myasthenia gravis (MG) is a prototypical B cell-mediated autoimmune disease affecting 20-50 peopl

92 ned a model of specific antibody deficiency, B cell-specific CD79a-Cre x XBP1 (X-box binding protein-

93 ty in the activation of the lineage germline B cell.

94 osit MYD88/p100 signaling as a regulator for B-cell activation.

95 phia chromosome (Ph)-positive or Ph-negative B-cell acute lymphoblastic leukaemia who were due to rec

96 tion compromises the activity of the pivotal B-cell antigen receptor (BCR)-proximal effector spleen t

97 c antigen receptor (CAR) that target CD19 in B-cell cancers, although data regarding B-cell lymphomas

98 innate lymphoid, myeloid, and dendritic, and B-cell fate alternatives are excluded by different mecha

99 Human CD40L(+) ILC3s provide innate B-cell help and are involved in an innate immunoregulato

100 mphomas (DELs) are subtypes of diffuse large B-cell lymphoma (DLBCL) associated with poor outcomes af

101 licular lymphoma (FL; n = 29), diffuse large B-cell lymphoma (DLBCL; n = 34), DLBCL arising from chro

102 pression of downstream genes, such as Bcl-2 (B-cell lymphoma 2), c-Myc, MMP7 (matrix metalloproteinas

103 ous NHL histologies, including diffuse large B-cell lymphoma, Richter's transformation, mantle cell l

104 ar lymphoma, and MYC/BCL2/BCL6 in high-grade B-cell lymphomas are essential for diagnosis.

105 9 in B-cell cancers, although data regarding B-cell lymphomas are limited.

106 ed by mutations in JAK2, CALR, or MPL In the B-cell lymphomas, detection of characteristic rearrangem

107 lymphoma, Burkitt's lymphoma, diffuse large B-cell lymphomas, nasopharyngeal carcinoma (NPC), and ly

108 ndamental differences in the pathogenesis of B-cell lymphoproliferative disorders.

109 ated to regulate signaling downstream of the B-cell receptor (BCR), Fc receptors (FcRs), and toll-lik

110 Delivery of neurotransmitters across the T-B-cell synapse may be advantageous in the face of infect

111 icantly increases CD20 levels in established B-cell tumor cell lines and primary malignant cells.

112 ibitory activity for allergen-IgE binding to B cells (IgE-facilitated allergen binding).

113 t higher proportions of IgM+CD21-/low memory B cells (P < .05), CD4+IFNgamma+ cells (P < .01), CD4+IL

114 ution to the gut, increases of the activated B cells and circulating tissue-like memory B cells, and

115 chromosomal rearrangements in primary murine B cells and discovered that RAG1/2 causes aberrant inser

116 on, we compared the fate of IgE-ICs in human B cells and in CD23-expressing monocyte-derived dendriti

117 B cells contribute to multiple aspects of autoimmune dis

118 f multiple sclerosis suggests that depleting B cells could be useful for treatment.

119 In contrast, TIM-4(+) B cells decreased B16-F10 metastasis and s.c. tumor grow

120 Before birth, B cells develop in the fetal liver (FL).

121 oss of naive B cells, loss of resting memory B cells due to their redistribution to the gut, increase

122 B cells expressing the T-bet transcription factor, a mar

123 r helper (pTfh) cells, which provide help to B cells for developing into Ab-secreting cells, were sim

124 B cells from hypersensitive patients, but not controls,

125 ranscriptomes and proteomes of primary mouse B cells from wild-type and STAT6-deficient mice cultured

126 The potential protective role of B cells in controlling cCMV-related disease and the clin

127 B cells influence the pathogenesis of multiple sclerosis

128 Flow cytometric analysis of peripheral blood B cells of 30 MC-negative HCV-infected patients and 15 h

129 icial substrates or live cells, we show that B cells primarily use force-dependent extraction and res

130 s, mTORC1 activation is required for fueling B cells prior to DZ proliferation rather than for allowi

131 ric form of the toxin, suggesting that human B cells recognize epitopes on the dimerized form of LukA

132 ystems of infected hosts to recall of memory B cells that recognized the lateral patch, the principal

133 rom human immature dendritic cells and naive B cells to assess the expression of CD40-downstream gene

134 Thus, upregulation of EBI2 drives the B cells toward the extrafollicular area, whereas downreg

135 crete proportion of infiltrating T cells and B cells underwent proliferation within the pituitary par

136 hereas frequencies of transitional and naive B cells were decreased.

137 ed that frequencies of class-switched memory B cells were increased in the patients, whereas frequenc

138 mic or splenic DC, whereas thymic or splenic B cells were poor donors.

139 Nod1, including follicular and marginal zone B cells with natural autoreactivity.

140 lin light chain editing occurred, generating B cells with up-regulated Nod1, including follicular and

141 d B cells and circulating tissue-like memory B cells, and expansion of the B regulatory cells (Bregs)

142 pan-T cells, CD4(+) T cells, CD8(+) T cells, B cells, and NK cells, with 49 recombinant chemokines us

143 ly composed of CD3+ T cells, scattered CD20+ B cells, and relatively few PD-1+ (programmed cell death

144 In both mouse and human B cells, BCR ligands that deliver a TLR9 agonist induce

145 infection is characterized by loss of naive B cells, loss of resting memory B cells due to their red

146 erimental stroke prevents loss of splenic MZ B cells, preserves IgM levels, and reduces bacterial bur

147 assenger allele system to assay, in mouse GC B cells, sequence-intrinsic SHM-targeting rates of nucle

148 lly, CpG increased the number of circulating B cells, which produced elevated amounts of tumor necros

149 quencing method can use as few as 1000 naive B cells.

150 biting proliferation of germinal center (GC) B cells.

151 with less contribution from neutrophils and B cells.

152 o showed superior activation of Env-specific B cells.

153 s to sense alum and, in turn, activate T and B cells.

154 h cells than other lineages of more specific B cells.

155 (VL) sequences of individual and Ag-specific B cells.

156 ctor kappa-light-chain-enhancer of activated B cells.

157 CD4(+) and CD8(+) T cells but no binding by B cells.

158 omotes chronic gammaherpesvirus infection of B cells.IMPORTANCE Gammaherpesviruses infect a majority

159 used a significant increase in percentage of B-cells and significantly decreased percentage of myeloi

160 ]: -1.03 [-1.17, -0.89]), and apolipoprotein B (change in SD units [95% confidence interval]: -0.98 [

161 treatment in patients with chronic hepatitis B (CHB).

162 matoes with this coating and inoculated with B. cinerea showed a significant decrease in fungal growt

163 ne CRPs, the PCP-Bs (for pollen coat protein B-class) that are related to embryo surrounding factor (

164 In contrast to antenatal BS, classic BS manifests with highly variable phenotypes

165 cluding B cell chronic lymphocytic leukemia (B-CLL).

166 The BiB2 O(-) and Bi2 B(-) clusters are produced by laser vaporization of a mi

167 g option for industrial packed and fluidized bed CO2 capture systems due to large particles with a di

168 to be HCV and antibody reacting to hepatitis B core antigen+, and less likely to have diabetes or hyp

169 Group B coxsackieviruses are responsible for chronic cardiac i

170 age-matched TD controls (<18 years old) and (b) decreases in the amygdala as people with ASD age into

171 representative siderophore, desferrioxamine B (DFOB), iron (Fe) was released at higher rates and to

172 two major QTLs identified in undomesticated B. distachyon colocalize with VERNALIZATION1/PHYTOCHROME

173 We conjugated unimNPs with the cholera toxin B domain (CTB) for RGC-targeting and with Cy5.5 for unim

174 anode nanojunction architecture, composed of B-doped carbon nitride nanolayer and bulk carbon nitride

175 The Bi-B double and triple bond strengths are calculated to be

176 t carriage of Nef variants with enhanced MHC-B downregulation ability is associated with reduced brea

177 determinant of differential HLA-A versus HLA-B downregulation activity.

178 t Nef position 9 that contributes to the MHC-B downregulation function in HIV-1 subtype C and show th

179 GCGR) are members of the secretin-like class B family of G-protein-coupled receptors (GPCRs) and have

180 to incorporate the biological cofactor heme-B for catalysis.

181 bidopsis thaliana pollen-borne CRPs, the PCP-Bs (for pollen coat protein B-class) that are related to

182 lysis of a prototypical Vbeta8.1(+) TCR-H-2D(b)-GAP5040-48 ternary complex revealed that germline-enc

183 tes the efficacy of non-viral TUS-based hSef-b gene delivery approach for the treatment of prostate c

184 lification of a 148 bp fragment from the cyt b gene with a universal primer pair in HRM analyses.

185 d not previously undergone prostate biopsy, (b) had prior negative biopsy findings with increased pro

186 ac in the lower uterine segment and elevated B-Hcg levels, the possibility of scar ectopic pregnancy

187 eacetyl parasiticolide A, flufuran, gregatin B, hydroxysydonic acid, nicotinic acid, phomaligin A, sp

188 caudate and putamen of 16 RC BD-I, 34 non-RC BD-I and 44 healthy controls were assessed.

189 e cortex (ACC), caudate and putamen of 16 RC BD-I, 34 non-RC BD-I and 44 healthy controls were assess

190 of the campaign in the SLSJ region (relative B-IMD risk: 0.22; P = .04).

191 three national serosurvey data of hepatitis B in China, we propose an age- and time-dependent discre

192 cursor of the active gibberellin, GA1, by UV-B in this zone, which is regulated, at least in part, by

193 85% in the WT bed, and 34% in the unplanted bed in 2012.

194 A separate longitudinal sample (cohort B) included 16 MDD patients who underwent MRI at baselin

195 In addition, UV-B increased leaf quercetin content and total antioxidant

196 tive synthetic route to hexahydropyrrolo[2,3-b]indoles via Lewis acid-catalyzed SN2-type ring opening

197 so suggest that prior exposure to H1 or type B influenza may differentially affect cross-reactivity o

198 om human postvaccination sera with only type B influenza preexposure consistently showed good correla

199 e derived from sera with neither H1 nor type B influenza preexposure.

200 putatively anti-Bd bacteria to determine if Bd-inhibitory bacteria are dominant microbiome members.

201 very strong electric field across the c-Si/a-B interface systems where the charge transfer occurs mai

202 rons in the amygdala of patients with SZ and BD, interpreted here as decreased SST expression, may di

203 orophylls into chlorophyll a and chlorophyll b is necessary for advanced monitoring of plant growth.

204 Justicidin B is structurally similar to more potent vacuolar-type H

205 As carmaphycin B is toxic to mammalian cells, we synthesized a series o

206 lenate uptake by 100% in S. elata and 40% in B. juncea.

207 Central to this regulation is Aurora B kinase, which phosphorylates kinetochore substrates to

208 nical outcomes when used as carriers for the B-KPro, with no significant differences in device retent

209 it remained unknown if, and how, calcineurin B-like calcium sensors (CBLs) and CBL-interacting protei

210 Thus, the dichotomy between T-like and B-like cells and the presence of dedicated lymphopoietic

211 at Hh-mediated transcription is increased in B-lineage cells from Gli3-deficient FL and showed that t

212 that these cells expressed reduced levels of B-lineage transcription factors and B cell receptor (BCR

213 ly, antifungal drugs, including amphotericin B, liposomal amphotericin B, and flucytosine, need to be

214 a common gammaherpes virus with tropism for B lymphocytes and infection in immunocompetent individua

215 keys, SGN-CD19B effectively depleted CD20(+) B lymphocytes in peripheral blood and lymphoid tissues c

216 ibly including tissue macrophages) and T and B lymphocytes in the presence of detectable inflammatory

217 ng of MRN in cancer, we engineered mice with B lymphocytes lacking MRN, or harboring MRN in which MRE

218 cing and RNA sequencing, we identify a novel B-lymphoid program for transcriptional repression of glu

219 Hedgehog (Hh) signaling promotes B lymphopoiesis in a non-cell-autonomous fashion in vitr

220 In contrast to antenatal BS, classic BS manifests with highly variable phenotypes.

221 We found that B. microti reaches high parasitemia levels during the fi

222 usefulness of this new transgenic hepatitis B model.

223 e show here that a single marker, Neuromedin B mRNA (Nmb), identifies RTN neurons in rodents.

224 The apolipoprotein B mRNA editing enzyme catalytic polypeptide-like APOBEC3

225 Apolipoprotein B mRNA-editing catalytic polypeptide (APOBEC) 3 proteins

226 s study, we demonstrated that apolipoprotein B mRNA-editing catalytic polypeptide 3A (A3A) and A3G ex

227 We demonstrate here that the formation of B-N Lewis pairs at the periphery of anthracene leads to

228 the rate of release of nuclear factor kappa B (NFkappaB) from DNA target sites in a process we have

229 otential of SGN-CD19B in relapsed/refractory B-NHL.

230 ation, or a more direct topography involving bed nucleus vs central nucleus divisions; (2) CRF conten

231 Aortic dissection, commonly type B, occurs in an appreciable proportion of Marfan patient

232 system relative to both control subjects and BD offspring.

233 onal Study Group on Pancreatic Fistula Grade B or C) and hospital-related inpatient costs for 90 days

234 OSCP, lacking the membrane domain of subunit b or the OSCP, respectively, in which the corresponding

235 2,000 confirmations for each of types A and B over the study period.

236 oli that identified high-affinity cytochrome bd oxidase as an essential bacterial gene product for mo

237 e strong support for Branigan & Pickering's (B&P's) model, largely because the priming effects are mo

238 rences in low-temperature adaptation between B. pertussis and B. bronchiseptica may result from selec

239 cells from the lungs of mice reinfected with B. pertussis produced significantly more IL-17 than gamm

240 tica may result from selective adaptation of B. pertussis to the human host.

241 ion in adaptive immunological memory against B. pertussis.

242 ulated kinase, phosphorylated protein kinase B, phosphorylated mammalian target of rapamycin, phospho

243 sessed cerebral Abeta on Pittsburgh Compound B (PiB) positron emission tomography, gait speed over 4.

244 Transformations employ [B(pin)]2-methane as a conjunctive reagent, resulting in

245 by recruitment of the protein phosphatase 2A B' (PP2A B').

246 In the retrospective cohort, luminal B prostate cancers exhibited the poorest clinical progno

247 ddition, we find that EmMBD2/3 and EmGATAD2A/B proteins form a coiled-coil interaction known to be cr

248 ted MEK1/2 in various tumor cells expressing B-Raf(V600E) or K-Ras(G12C/D) Intriguingly, coimmunoprec

249 Considering variations in chlorophyll a:b ratio with leaf age and physiological stress, a furthe

250 mg/0.1 mL) 1-3 days before PPV, while Group B received IVB (2.5 mg/0.1 mL) 5-10 days before PPV.

251 at an interaction between Xist RNA and Lamin B receptor (LBR) is necessary and sufficient for Xist sp

252 ue-like memory B cells, and expansion of the B regulatory cells (Bregs).

253 s and unrestricted branch-site-based models (BS-REL, BUSTED and RELAX)), our results consistently sho

254 ID), a 600 nt sequence encompassing the Xist B-repeat element.

255 ed with reduced breadth and magnitude of MHC-B-restricted cellular immune responses in HIV-infected i

256 ious suggestions that APOE, CHRNA3/5, CDKN2A/B, SH2B3 and FOXO3A influence longevity.

257 Only data acquired at a bed station that included the head were used for this st

258 enza A/H1N1, influenza A/H3N2, and influenza B strains.

259 We show that loss of RER in B. subtilis causes strand- and sequence-context-dependen

260 The disruption of autolysis in B. subtilis cultures by TiO2 NPs suggests the mechanisms

261 xic NK potential on the basis of KIR3DL1/HLA-B subtype combinations in vitro and evaluated their impa

262 Six patients had hepatitis B surface antibody (anti-HBs) titres above 10 mIU/mL at

263 2334 RA patients who had available hepatitis B surface antigen (HBsAg) data, 123 patients positive fo

264 The serum gradient of hepatitis B surface antigen (HBsAg) varies over time after cessati

265 HBV functional cure is sustained hepatitis B surface antigen loss and anti-HBs gain, with normaliza

266 sor surface and post modified with hepatitis B surface antigen.

267 ivity was defined as 11C-Pittsburgh compound B target-to-cerebellar ratio above 1.5 within a composit

268 Here, the authors engineer BoNT/B to improve its affinity to human receptors and enhance

269 beta) and background particle concentration (B) to be independent for use in fate modeling.

270 36-month prospective, randomized, FAME A and B trials.

271 e demonstrated that MsrQ is able to bind two b-type hemes through the histidine residues conserved be

272 ronolactone and usual care on N-terminal pro-B-type natriuretic peptide (NT-proBNP) levels compared w

273 (48 versus 40), higher median N-terminal pro-B-type natriuretic peptide concentration (403 versus 320

274 mL]; P<0.0001), despite lower N-terminal pro-B-type natriuretic peptide levels.

275 V, RVESRI, and log NT-proBNP (N-Terminal Pro-B-Type Natriuretic Peptide) were retained (chi(2), 62.2;

276 ac natriuretic peptides (NPs), atrial NP and B-type NP, regulate fluid homeostasis and arterial BP th

277 trients, flavan-3-ols (i.e., epicatechin and B-type procyanidins) as also hydroxycinnamoyl-quinic aci

278 planets are known to transit the even hotter B-type stars.

279 onstrate their use for the introduction of P=B units into organic systems.

280 he correct splice sites (SS) and branchsite (BS) used during splicing.

281 d missed opportunities to initiate hepatitis B vaccination.

282 mmunity effects with meningococcal serogroup B vaccines and the need for a booster dose to sustain in

283 Hepatitis B virus (HBV) chronic infection affects up to 240 millio

284 The management of hepatitis B virus (HBV) e antigen-positive viremic patients with n

285 CC), often associated with chronic hepatitis B virus (HBV) infection.

286 ons by either hepatitis A virus or hepatitis B virus (HBV), or a noninfectious cause for their ALF.

287 Hepatitis B virus (HBV)-encoded X protein (HBx) plays a critical r

288 with acute, resolved, and chronic hepatitis B virus (HBV)infection but might also signify occult HBV

289 The study of hepatitis B virus and development of curative antivirals are hampe

290 We analyzed changes in hepatitis B virus and hepatitis delta virus (HDV) viral loads (VL)

291 ed with a similar modest change in hepatitis B virus core antigen polypeptide (HBcAg/p21) synthesis,

292 biomarkers toward better defining hepatitis B virus cure should occur in parallel with development o

293 For the discovery of hepatitis B virus integration sites from probe capture data, the v

294 of T cells engineered to express a hepatitis B virus-specific (HBV-specific) T cell receptor (TCR) ma

295 Hepatitis B viruses (HBVs), which are enveloped viruses with rever

296 ible interactions of arsenic metabolism with B vitamins and AS3MT variants on diabetes risk.

297 use childhood blindness, in which the a- and b-wave responses of electroretinogram (ERG) are abolishe

298 scription from a Gastroenterologist in group B were associated with eradication success.

299 genuine effect of Trpc3 on macrophages, and (b) whether the reduced necrosis and macrophage apoptosis

300 ty cycle but generates MS/MS spectra rich in b/y-type and c/z(*)-type product ions.