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1 /=290 pg/mL) serum NT-proBNP (N-terminal pro-B-type natriuretic peptide).
2 endothelin-1, creatinine, and N-terminal pro-B-type natriuretic peptide.
3 r elevated baseline levels of N-terminal pro B-type natriuretic peptide.
4 measurement of the ventricular stress marker B-type natriuretic peptide.
5 tration, and 0.76 (0.60-0.93) for changes in B-type natriuretic peptide.
6 other risk factors including N-terminal pro-B-type natriuretic peptide.
7 or clinical variables and amino-terminal pro-B-type natriuretic peptide.
8 ore, left ventricular ejection fraction, and B-type natriuretic peptide.
9 en for C-reactive protein and N-terminal pro B-type natriuretic peptide.
10 ed preoperative end-systolic volume index or B-type natriuretic peptide.
11 ted by further adjustment for N-terminal pro-B-type natriuretic peptide.
13 ection fraction (ejection fraction=63+/-15%; B-type natriuretic peptide=431+/-366 pg/mL) was conducte
14 Secondary endpoints were quality of life, B-type natriuretic peptide, 6-min walk distance, and EF.
15 Patients with AF had higher N-terminal pro-B-type natriuretic peptide, aldosterone, endothelin-1, t
16 gene expression (atrial natriuretic peptide, B-type natriuretic peptide, alpha-skeletal actin), and c
17 Levels of N-terminal fragment of prohormone B-type natriuretic peptide also improved risk prediction
18 concentration, hemoglobin concentration, and B-type natriuretic peptide also significantly increased
19 after further adjustment for N-terminal pro-B-type natriuretic peptide and after exclusion of partic
20 lded modest improvement over a model without B-type natriuretic peptide and ankle-brachial index (C s
21 ts were similar to those with N-terminal pro-B-type natriuretic peptide and better than those with th
22 Patients had echocardiograms and measures of B-type natriuretic peptide and C-reactive protein before
23 Although significantly higher in group 2, B-type natriuretic peptide and echocardiography cardiac
24 Further adjustment for amino-terminal pro-B-type natriuretic peptide and estimated renal function
25 ial infarction (AMI) by using N-terminal pro-B-type natriuretic peptide and Global Registry of Acute
26 composed of the 3 biomarkers in addition to B-type natriuretic peptide and high-sensitivity C-reacti
27 ncludes age, biomarkers (N-terminal fragment B-type natriuretic peptide and high-sensitivity cardiac
30 f either B-type natriuretic peptide (BNP) or B-type natriuretic peptide and its amino-terminal cleava
31 beyond what is obtained from N-terminal pro-B-type natriuretic peptide and peak stress left ventricu
32 istic=0.59); model 2: model 1+N-terminal pro-B-type natriuretic peptide and peak stress left ventricu
33 estimate the values of serum N-terminal pro-B-type natriuretic peptide and procalcitonin and the cha
34 rve 0.90 +/- 0.01), with no improvement when B-type natriuretic peptide and resting saturations were
35 ence of heterogeneity between N-terminal pro B-type natriuretic peptide and saxagliptin (P for intera
37 rdial wall stress and injury (N-terminal pro-B-type natriuretic peptide and troponin) versus enalapri
39 use of natriuretic peptides (N-terminal pro-B type natriuretic peptide) and rest/exercise echocardio
41 -terminal pro-atrial natriuretic peptide and B-type natriuretic peptide) and lower blood concentratio
42 change in primary (change in N-terminal pro B-type natriuretic peptide) and secondary (change in lef
44 nt for clinical risk factors, N-terminal pro-B-type natriuretic peptide, and a wide range of echocard
45 le combination of standard CVD risk factors, B-type natriuretic peptide, and ankle-brachial index (mo
46 line body mass index, height, N-terminal pro-B-type natriuretic peptide, and cystatin C, with longer
47 based on cardiac troponin I, N-terminal pro-B-type natriuretic peptide, and d-dimer levels at baseli
49 high-sensitivity troponin T, N-terminal pro-B-type natriuretic peptide, and high-sensitivity C-react
50 nsitivity C-reactive protein, N-terminal pro-B-type natriuretic peptide, and high-sensitivity cardiac
51 or conventional risk markers, N-terminal pro-B-type natriuretic peptide, and hs-TnT, hs-TnI levels in
52 rular filtration rate, higher N-terminal pro-B-type natriuretic peptide, and ischemic cause of heart
53 of angiotensin inhibition therapy, elevated B-type natriuretic peptide, and larger left ventricular
54 n, d-dimer, homoarginine, and N-terminal pro B-type natriuretic peptide, and lower levels of low-dens
55 rmonal activity (aldosterone-to-renin ratio, B-type natriuretic peptide, and N-terminal proatrial nat
56 s to the multivariable model were H/M, LVEF, B-type natriuretic peptide, and NYHA functional class.
57 mass index, electrocardiographic parameters, B-type natriuretic peptide, and other biochemical and he
58 ctional class or Ross class), N-terminal pro-B-type natriuretic peptide, and quality of life (QOL) we
60 cle ergometry, measurement of N-terminal pro-B-type natriuretic peptide, and subjective health assess
61 Ultrasensitive troponin I, N-terminal pro-B-type natriuretic peptide, and the interleukin family m
63 re significantly older, had higher levels of B-type natriuretic peptide, and were predominantly hyper
64 sensitivity troponin and N-terminal fragment B-type natriuretic peptide], and clinical history of pri
66 oncentrations, hemoglobin concentration, and B-type natriuretic peptide are reliable alternatives to
67 tic resonance imaging and BNP/N-terminal pro-B-type natriuretic peptide, are emerging as promising in
68 or predicting 90-day mortality compared with B-type natriuretic peptide (area under the curve: 0.674
69 rement of soluble ST2 and amino-terminal pro-B-type natriuretic peptide at 12 hours added value to Ca
72 natriuretic peptides, such as N-terminal pro-B-type natriuretic peptide/B-type natriuretic peptide, t
73 sponse to cardiac stress, the heart secretes B-type natriuretic peptide (BNP(1-32)) and amino-termina
74 cted to define the association between serum B-type natriuretic peptide (BNP) activation and survival
75 safety and efficacy of 8 weeks of chronic SC B-type natriuretic peptide (BNP) administration in human
76 rial, we determined whether plasma levels of B-type natriuretic peptide (BNP) and cardiac troponin I
77 quantified cardiac hemodynamic stress using B-type natriuretic peptide (BNP) and cardiomyocyte damag
78 tracellular cGMP production is stimulated by B-type natriuretic peptide (BNP) and degraded by phospho
79 las Heart Study underwent measurements of 1) B-type natriuretic peptide (BNP) and N-terminal pro-B-ty
80 dy was to determine the relationship between B-type natriuretic peptide (BNP) and survival in patient
82 ective evaluation of the interaction between B-type natriuretic peptide (BNP) and the effect of ranol
83 designed to monitor daily concentrations of B-type natriuretic peptide (BNP) and to determine how th
87 h-sensitivity C-reactive protein (hsCRP) and B-type natriuretic peptide (BNP) concentrations at 72 h
90 mpanied by anorexia and increased release of B-type natriuretic peptide (BNP) from ventricular cardio
93 eptides atrial natriuretic peptide (ANP) and B-type natriuretic peptide (BNP) in heart tissue may als
94 sought to determine the prognostic value of B-type natriuretic peptide (BNP) in patients with heart
95 secrete atrial natriuretic peptide (ANP) and B-type natriuretic peptide (BNP) in response to mechanic
99 Numerous experimental studies suggest that B-type natriuretic peptide (BNP) is cardioprotective; ho
100 correlation between TMAO concentrations and B-type natriuretic peptide (BNP) levels (r = 0.23; p < 0
101 herefore sought to examine whether admission B-type natriuretic peptide (BNP) levels predict the deve
103 ble reduction in the concentration of either B-type natriuretic peptide (BNP) or B-type natriuretic p
105 nators in serum by LR were troponin I (TnI), B-type natriuretic peptide (BNP), and creatine kinase-MB
106 ardiograph, a decreasing or normalization of B-type natriuretic peptide (BNP), and hemodynamics with
109 (NPs), atrial natriuretic peptide (ANP) and B-type natriuretic peptide (BNP), have central roles in
113 n and fibrinogen), neurohormonal activation (B-type natriuretic peptide [BNP] and N-terminal proatria
116 for mortality in addition to N-terminal pro-B-type natriuretic peptide (C-statistic: 0.59 versus 0.6
117 in T, creatine kinase, myoglobin, N-terminal B-type natriuretic peptide, C-reactive protein, and leuk
118 n, lower expression of cardiac stress genes (B-type natriuretic peptide, collagen gene expression), l
119 (48 versus 40), higher median N-terminal pro-B-type natriuretic peptide concentration (403 versus 320
120 (-5.1 to -0.8, p=0.0070), and N-terminal pro B-type natriuretic peptide concentration in plasma -970
121 as associated with NT-proBNP (N-terminal pro-B-type natriuretic peptide) concentration (0.57 on a log
122 the kallikrein B locus were associated with B-type natriuretic peptide concentrations in blacks.
123 After extensive adjustments, N-terminal pro-B-type natriuretic peptide concentrations predicted ICU
124 cretoneurin measurements, and N-terminal pro-B-type natriuretic peptide did not improve patient class
125 ty of disease as indicated by N-terminal pro B-type natriuretic peptide, E/E', and left atrial volume
126 ent after adjustment for age, N-terminal pro-B-type natriuretic peptide, ejection fraction, E/E', and
127 rotein, IL-6, troponin T, and N-terminal pro-B-type natriuretic peptide, elevated soluble gp130 (fift
129 he initial studies showing the usefulness of B-type natriuretic peptide for aiding in heart failure d
130 /- 130 m; P<0.001), and decreased N-terminal B-type natriuretic peptide (from 2322 +/- 1234 pg/mL to
131 6.2% to 27.1+/-6.6%; P=0.23), N-terminal pro B-type natriuretic peptide (from 3322+/-3411 to 3672+/-5
132 ; P=0.001), and a decrease in N-terminal pro B-type natriuretic peptide (from 3672+/-5165 to 1488+/-1
134 le ST2, >/= 500 ng/mL and amino-terminal pro-B-type natriuretic peptide, >/= 4,500 ng/L) had higher 3
136 n time, left atrium dimension, E/e', and pro B-type natriuretic peptide (hazard ratio, 1.05; 95% conf
137 In backward elimination analyses, higher log-B-type natriuretic peptide (hazard ratio, 1.39 per 1-SD
138 0.84 to 0.99; p = 0.027) and N-terminal pro-B-type natriuretic peptide (hazard ratio: 0.98; 95% conf
139 on functional class [WHO-FC], N-terminal-pro-B-type natriuretic peptide, hemodynamics) and lung-trans
140 djustment for clinical prognostic variables, B-type natriuretic peptide, high-sensitivity C-reactive
141 that included clinical prognostic variables, B-type natriuretic peptide, high-sensitivity C-reactive
142 ECG, coronary artery calcium, N-terminal pro B-type natriuretic peptide, high-sensitivity cardiac tro
143 ECG, coronary artery calcium, N-terminal pro B-type natriuretic peptide, high-sensitivity cardiac tro
145 sponse to LCZ696 for lowering N-terminal pro B-type natriuretic peptide; however, left atrial volume
146 after adjustment for clinical variables and B-type natriuretic peptide (HR: 1.23; 95% CI: 1.04 to 1.
148 luding clinical variables and N-terminal pro-B-type natriuretic peptide) improved discrimination (C-s
149 ivity C-reactive protein, and N-terminal pro-B-type natriuretic peptide in 9698 participants aged 54
150 ssment (11 risk factors) plus N-terminal pro-B-type natriuretic peptide in terms of discrimination, c
152 11% vs. 55 +/- 10%; p < 0.001; n = 259), and B-type natriuretic peptide increased (median [interquart
153 eight loss, reduction in amino terminal, pro B-type natriuretic peptide, increased plasma renin activ
154 ical variables (including log N-terminal pro-B-type natriuretic peptide) indicated that increased LV
155 , matrix metalloproteinase-3, N-terminal pro-B-type natriuretic peptide, interleukin-6, soluble CD40
156 , matrix metalloproteinase-3, N-terminal pro-B-type natriuretic peptide, interleukin-6, soluble CD40
158 Cardiovascular parameters (N-terminal pro-B-type natriuretic peptide, left and right ventricular f
159 eraged proportional change in N-terminal pro-B-type natriuretic peptide level from baseline to 180 da
160 thoracic echocardiography and measurement of B-type natriuretic peptide level from venous blood sampl
161 se (CVD), a 10-year risk of CVD < 20%, and a B-type natriuretic peptide level greater than their gend
162 of 25% [IQR, 19%-33%]; median N-terminal pro-B-type natriuretic peptide level of 2049 pg/mL [IQR, 105
163 variable HR for a 1-SD higher N-terminal pro-B-type natriuretic peptide level, 1.15; 95% CI, 1.04-1.2
164 ge, ejection fraction, plasma N-terminal pro-B-type natriuretic peptide level, and prior hospitalizat
165 level, troponin T level, log N-terminal pro-B-type natriuretic peptide level, fibroblast growth fact
166 water indexed for ideal body weight, plasma B-type natriuretic peptide level, hemoglobin, and plasma
170 p > 0.001), and higher median N-terminal pro-B-type natriuretic peptide levels (3,495 vs. 1,730 ng/dl
171 CI, 1.55-2.44), and abnormal N-terminal pro-B-type natriuretic peptide levels (defined as >400 pg/mL
172 currence rates in addition to an increase in B-type natriuretic peptide levels (P=0.01), C-reactive p
180 hospitalized with acute coronary syndromes, B-type natriuretic peptide levels measured at admission
181 ad NT-proBNP levels of 1000 pg/mL or more or B-type natriuretic peptide levels of 250 pg/mL or more,
182 both), in contrast to plasma N-terminal pro-B-type natriuretic peptide levels that were not (area un
184 ung ultrasound, echocardiography, and plasma B-type natriuretic peptide levels were determined before
189 worse quality of life, higher N-terminal pro-B-type natriuretic peptide levels, and a poorer prognosi
190 ntricular function, decreased N-terminal pro B-type natriuretic peptide levels, and better exercise c
191 minute walk distances, higher N-terminal pro-B-type natriuretic peptide levels, and the presence of r
193 ricular reverse remodeling, MR severity, and B-type natriuretic peptide levels, compared with CABG al
194 with worse intracardiac hemodynamics, higher B-type natriuretic peptide levels, lower exercise capaci
195 k factors (N-terminal fragment of prohormone B-type natriuretic peptide levels, von Willebrand factor
199 umin had significantly higher N-terminal pro-B-type natriuretic peptide levels; in addition, early ri
200 uble receptors and NT-proBNP (N-Terminal Pro-B-Type Natriuretic Peptide) levels were important across
202 ss, mitral regurgitation, and N-terminal pro-B-type natriuretic peptide, lower TAPSE, and assignment
205 d glomerular filtration rate, N-terminal pro-B-type natriuretic peptide, mineralocorticoid receptor a
206 s strong as that obtained for N-terminal pro-B-type natriuretic peptide (multivariable HR for a 1-SD
207 oplasmin, nitrotyrosine-bound ceruloplasmin, B-type natriuretic peptide, norepinephrine, and high-sen
209 c peptide (BNP(1-32)) and amino-terminal pro-B-type natriuretic peptide (NT-proBNP(1-76)) after intra
210 After further adjusting for N-terminal pro-B-type natriuretic peptide (NT-proBNP) and C-reactive pr
212 ce of elevated troponin I and N-terminal pro-B-type natriuretic peptide (NT-proBNP) and their associa
213 nship with N-terminal fragment of prohormone B-type natriuretic peptide (NT-proBNP) are unknown.
215 he prognostic value of plasma N-terminal pro-B-type natriuretic peptide (NT-proBNP) for death and car
217 was to determine the value of N-terminal pro-B-type natriuretic peptide (NT-proBNP) in adults with co
218 whether serial measurement of N-terminal pro-B-type natriuretic peptide (NT-proBNP) in community-dwel
219 ssess the prognostic value of N-terminal pro-B-type natriuretic peptide (NT-proBNP) in patients with
222 assess whether a reduction in N-terminal pro-B-type natriuretic peptide (NT-proBNP) is associated wit
224 ided by concentrations of amino-terminal pro-B-type natriuretic peptide (NT-proBNP) is superior to st
225 ular mortality over and above N-terminal pro-B-type natriuretic peptide (NT-proBNP) level in older ad
226 s, and percent reduction in serum N terminal B-type natriuretic peptide (NT-proBNP) level-and relief
227 on (P < .001); and had higher N-terminal pro-B-type natriuretic peptide (NT-proBNP) levels (P < .001)
228 ronolactone and usual care on N-terminal pro-B-type natriuretic peptide (NT-proBNP) levels compared w
231 -minute walk distance, plasma N-terminal pro-B-type natriuretic peptide (NT-proBNP) levels, and healt
233 oponin T (cTnT), and elevated N-terminal pro-B-type natriuretic peptide (NT-proBNP) on cardiovascular
234 tivity troponin T (hsTnT) and N-terminal pro-B-type natriuretic peptide (NT-proBNP) strongly predict
235 We measured plasma GDF-15, N-terminal pro-B-type natriuretic peptide (NT-proBNP), and C-reactive p
236 nsitive troponin T (cTnT) and N-terminal pro-B-type natriuretic peptide (NT-proBNP), and the subseque
237 ity C-reactive protein (CRP), N-terminal pro-B-type natriuretic peptide (NT-proBNP), growth different
238 We studied the association of N-terminal pro-B-type natriuretic peptide (NT-proBNP), high-sensitive t
239 ostic value of the biomarkers N-terminal pro-B-type natriuretic peptide (NT-proBNP), high-sensitivity
240 edian percent change in serum N-terminal pro-B-type natriuretic peptide (NT-proBNP), high-sensitivity
241 ost important biomarkers were N-terminal pro-B-type natriuretic peptide (NT-proBNP), high-sensitivity
242 or age, sex, body mass index, N-terminal pro-B-type natriuretic peptide (NT-proBNP), renal function,
244 ined whether repeated measurements of NT-pro-B-type natriuretic peptide (NT-proBNP), troponin T (Trop
245 igate the association between N-terminal pro-B-type natriuretic peptide (NT-proBNP), which is a marke
246 To determine whether an amino-terminal pro-B-type natriuretic peptide (NT-proBNP)-guided treatment
253 natriuretic peptide (BNP) and N-terminal pro-B-type natriuretic peptide (NT-proBNP); and 2) body fat
254 ysfunction: C-reactive protein (CRP); NT-pro-B-type natriuretic peptide (NT-proBNP); troponin I; matr
255 tion [LVEF] 26 +/- 6%, median N-terminal pro-B-type natriuretic peptide [NT-proBNP] 1,158 pg/ml), the
256 Risk Evaluation (EuroSCORE), N-terminal pro-B-type natriuretic peptide (NTproBNP), and high-sensitiv
259 porated LA volume, NT-proBNP (N-terminal pro-B-type natriuretic peptide), or left ventricular E/e' (a
261 es showed excellent discriminative power for B-type natriuretic peptide (p = 0.001; 95% CI, 0.99-1.00
262 tein cholesterol (p = 0.005), N-terminal pro-B-type natriuretic peptide (p = 0.004), and left ventric
263 ion improved Minnesota score (p = 0.019) and B-type natriuretic peptide (p = 0.045) and showed nonsig
265 xcursion) (P=0.002), (2) high N-terminal pro-B-type natriuretic peptide (P=0.085), (3) systemic (P=0.
267 nd men, as well as in the total cohort, were B-type natriuretic peptide, peak oxygen consumption by c
268 le risk model assessing 4 clinical variables-B-type natriuretic peptide, pkVO2, NYHA, and angiotensin
271 his study was to investigate circulating pro-B-type natriuretic peptide (proBNP(1-108)) in the genera
273 sults for soluble ST2 and amino-terminal pro-B-type natriuretic peptide provides early risk assessmen
276 ute kidney injury increased in parallel with B-type natriuretic peptide quartiles (5%, 9%, 15%, and 2
277 ly with hs-TnT (r = 0.44) and N-terminal pro-B-type natriuretic peptide (r = 0.39) but only weakly wi
278 h higher myeloperoxidase (r=0.42, P<0.0001), B-type natriuretic peptide (r=0.25, P=0.001), and asymme
279 systolic volume index, MR volume, and plasma B-type natriuretic peptide reduction of 22.2 mL/m(2), 28
280 phy, cardiomyocyte death, and N-terminal pro B-type natriuretic peptide release; all are classical ha
281 0; P<0.0001), log-transformed N-terminal pro B-type natriuretic peptides (RR per SD 1.27; P<0.0001),
282 nation of soluble ST2 and amino-terminal pro-B-type natriuretic peptide showed excellent discriminati
283 itivity C-reactive protein, myeloperoxidase, B-type natriuretic peptide, soluble fms-like tyrosine ki
284 ficantly from "no sepsis" for N-terminal pro-B-type natriuretic peptide, systemic vascular resistance
286 Patients with NF/LG had significantly lower B-type natriuretic peptide than those with LF/HG and LF/
287 culating levels of NT-proBNP (N-terminal pro-B-type natriuretic peptide) than HF patients without AF.
288 x, (4) circulating NT-proBNP (N-terminal pro-B-type natriuretic peptide), TNF-alpha, IL-6, IL-12, IL-
289 rement of soluble ST2 and amino-terminal pro-B-type natriuretic peptide to clinical parameters for ri
290 F included the combination of N-terminal pro-B-type natriuretic peptide, troponin-T, and urinary albu
291 ces were found between any 2 time points for B-type natriuretic peptide, tumor necrosis factor-alpha,
293 related with torsion, strain, N-terminal pro-B-type natriuretic peptide, vascular endothelial growth
296 s; in addition, early rise in N-terminal pro-B-type natriuretic peptide was associated with a better
299 ular function, end-systolic volume index and B-type natriuretic peptide were most strongly associated
300 V, RVESRI, and log NT-proBNP (N-Terminal Pro-B-Type Natriuretic Peptide) were retained (chi(2), 62.2;
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