ライフサイエンスコーパス: B. subtilis

1 B. subtilis aconitase is a bifunctional protein; to dete

2 B. subtilis biofilm formation was triggered by certain p

3 B. subtilis coat proteins (CotY, CotE, CotV and CotW) ex

4 B. subtilis has a thicker layer of peptidoglycan and lac

5 B. subtilis has three MreB isologues with partially diff

6 B. subtilis joins an ever-expanding group of bacteria, i

7 B. subtilis QST713 produces the lipopeptides in a ratio

8 B. subtilis strains lacking SpoVAF or SpoVAEa and SpoVAF

9 B. subtilis, E. coli, and pga-deleted E. coli carrying t

10 We enriched the phosphatase activity from a B. subtilis cell extract and suppose that dephosphorylat

11 associated GR operon, and transcription of a B. subtilis D gene was controlled by RNA polymerase sigm

12 Here we explain how to produce a B. subtilis SSB probe that exhibits 9-fold fluorescence

13 By screening a B. subtilis knock-out library for deficiency in acetylat

14 ication of metabolic states between adjacent B. subtilis biofilms, providing a possible generalizable

15 ium, belonging to the species P. aeruginosa, B. subtilis, and S. aureus.

16 ichia coli cells from the root surface after B. subtilis colonization, suggesting a possible protecti

17 Xene membranes reaches more than 73% against B. subtilis and 67% against E. coli as compared with tha

18 hat unlike U34 at the wobble position of all B. subtilis tRNAs of known sequence, U34 in the mutant t

19 Although B. subtilis was no longer detected in the guts of fish e

20 ctions in the interior of the filament among B. subtilis, P. aeruginosa and Salmonella enterica.

21 xpression level and mutation phenotype among B. subtilis strains, suggesting interstrain variation in

22 cy natural transformation, but the ancestral B. subtilis strain NCIB3610 is poorly competent.

23 as a mixed biofilm of S. oneidensis MR-1 and B. subtilis 3610.

24 n the disinfection efficacies of E. coli and B. subtilis .

25 Using V. cholerae, E. coli and B. subtilis as models, we discuss how both environmental

26 slinking, we have stabilized the E. coli and B. subtilis MutL-beta complexes and have characterized t

27 Finally, in both E. coli and B. subtilis, (p)ppGpp inhibits replication elongation in

28 glycans in the septal PG of both E. coli and B. subtilis, organisms separated by 1 billion years of e

29 In model bacteria, such as E. coli and B. subtilis, regulation of cell-cycle progression and ce

30 ionary phase efficiently in both E. coli and B. subtilis.

31 ing to EF-P modification in B. subtilis, and B. subtilis encodes the first EF-P ortholog that retains

32 rized gene amj (alternate to MurJ; ydaH) and B. subtilis MurJ (murJBs; formerly ytgP) are a synthetic

33 se secreted by non-virulent bacteria such as B. subtilis, can shift the delicate procoagulant-anticoa

34 While enzymes from both organisms assembled B. subtilis Lipid II into glycan strands, only the B. su

35 ilis subspecies subtilis RO-NN-1 and AUSI98, B. subtilis subspecies spizizenii TU-B-10(T) and DV1-B-1

36 We find that the average B. subtilis cell assembles approximately 26 flagellar ba

37 dark toxicity to the Gram positive bacterium B. subtilis and good photothermal killing efficiency tow

38 graming in the model Gram-positive bacterium B. subtilis.

39 n in cells has not been investigated because B. subtilis Lipid II was not previously available.

40 c liquid cultures demonstrates that, in both B. subtilis and P. aeruginosa, a turbulent flow forms in

41 photothermal killing efficiency toward both B. subtilis and Gram negative E. coli, features that dem

42 crucial for Arabidopsis root colonization by B. subtilis and provide insights into how matrix synthes

43 nts, such as those presumably encountered by B. subtilis in the soil.

44 egulated in response to biofilm formation by B. subtilis.

45 as the major inhibitory molecule produced by B. subtilis GS67.

46 abF, markedly decreased biotin production by B. subtilis resting cells whereas a strain having a ceru

47 ed polyamine norspermidine is synthesized by B. subtilis using the equivalent of the Vibrio cholerae

48 data identify the genes and proteins used by B. subtilis to produce PNAG as a significant carbohydrat

49 lity of metabolically active cells (E. coli, B. subtilis, Enterococcus, P. aeruginosa and Salmonella

50 and Zn(II) as substrates and can complement B. subtilis strains defective in the endogenous export s

51 Surprisingly, after disruption of decoated B. subtilis spores with lysozyme and fractionation, appr

52 e report here that in a spermidine-deficient B. subtilis mutant, the structural analogue norspermidin

53 scriptomic analysis of a spermidine-depleted B. subtilis speD mutant uncovered a nitrogen-, methionin

54 nvolved in DPA(2,6) movement into developing B. subtilis spores.

55 important for successful competition during B. subtilis pellicle formation.

56 notion that the enzyme is tetrameric during B. subtilis sporulation.

57 Cloning the entire B. subtilis epsHIJK locus into pga-deleted E. coli, Kleb

58 restingly, when 29 protein p1 was expressed, B. subtilis cells were about 1.5-fold longer than contro

59 ntibiotics optimizes competitive fitness for B. subtilis.

60 The layer orders inferred for B. subtilis and B. megaterium are consistent with measur

61 r observation begins to answer, at least for B. subtilis, a long-standing question on the exonucleoly

62 ructural features of spermidine required for B. subtilis biofilm formation are unknown and so are the

63 ved in, but is not absolutely essential for, B. subtilis pellicle formation.

64 rmidine biosynthetic pathway are absent from B. subtilis, confirming that norspermidine is not physio

65 Data from B. subtilis 3610 and S. coelicolor A3(2) provided a mean

66 Chimeric proteins derived from B. subtilis AbrB and the Spx C-terminus showed that a 28

67 itive action was observed for fengycins from B. subtilis, as well as the detergent CHAPS, when combin

68 crystal structures of full-length GabR from B. subtilis: a 2.7-A structure of GabR with PLP bound an

69 Bacillus pumilus as well as a paralogue from B. subtilis called YweA.

70 is6-LonBs, ClpPBs, and ClpXBs proteases from B. subtilis was analyzed.

71 the same structure as the LanI protein from B. subtilis, SpaI, despite the lack of significant seque

72 Furthermore, B. subtilis EF-P is post-translationally modified with a

73 Furthermore, B. subtilis has been instrumental in the study of hetero

74 Furthermore, B. subtilis PBP1 catalyzed the exchange of both D-amino

75 porulation during growth in gastrointestinal B. subtilis isolates, presumably as a form of survival a

76 In B. subtilis, deletion of a D gene (B. subtilis gerKD [gerKDbs]) adjacent to the gerK operon

77 When we repaired the dtd gene, B. subtilis became resistant to the biofilm-inhibitory e

78 or Bacillus megaterium, although germinated B. subtilis spores were rapidly killed.

79 peron is constitutively expressed in growing B. subtilis cells independently from sdpC.

80 quantitative direct bioautography via HPTLC-B. subtilis was shown as a reliable tool for streamlined

81 h massively parallel sequencing, to identify B. subtilis chromosomal DNA fragments that bind CodY in

82 In B. subtilis and Streptococcus pneumoniae, condensin comp

83 In B. subtilis and T. brucei, ms2ct6A disappeared and remai

84 In B. subtilis, CodY controls dozens of genes, but the thre

85 In B. subtilis, deletion of a D gene (B. subtilis gerKD [ge

86 In B. subtilis, the RBMs flank the region of the chromosome

87 hat the mechanism for sigma(V) activation in B. subtilis is controlled by regulated intramembrane pro

88 sigma(B) activity in B. subtilis is tightly regulated via at least three dist

89 nocytogenes, to inhibit sigma(B) activity in B. subtilis through perturbation of signal transduction

90 stress known to induce sigma(B) activity in B. subtilis.

91 he idea of an important role for c-di-AMP in B. subtilis and suggest that the levels of the nucleotid

92 tivities of CcdA1 and CcdA2 were analyzed in B. subtilis, neither protein retained activity in cytoch

93 The disruption of autolysis in B. subtilis cultures by TiO2 NPs suggests the mechanisms

94 controls several multicellular behaviours in B. subtilis, including biofilm formation.

95 athway is a bona fide precursor of biotin in B. subtilis.

96 hologous proteins can substitute for BslA in B. subtilis and confer a degree of protection, whereas Y

97 inactive omega-epsilon-zeta TA cassettes in B. subtilis mutants that were defective for different pr

98 ur understanding of amino acid chemotaxis in B. subtilis and gain insight into how a single chemorece

99 aptation systems contribute to chemotaxis in B. subtilis and whether they interact with one another.

100 Using conditional alleles of condensin in B. subtilis, we demonstrate that depletion of its activi

101 Examination of the genes neighboring cotH in B. subtilis led us to identify two spore coat proteins,

102 erved for RppH-dependent mRNA degradation in B. subtilis cells.

103 ugO, is necessary for biofilm development in B. subtilis, and that overexpression of mstX induces bio

104 r unknown RNA binding protein might exist in B. subtilis that can promote antitoxin/toxin RNA interac

105 hich is one of four known 3' exonucleases in B. subtilis.

106 When expressed in B. subtilis, FrvA increases resistance to iron both in w

107 ability to induce biofilm gene expression in B. subtilis.

108 Seven mutant flagellar filaments in B. subtilis and two in P. aeruginosa capture two differe

109 Finally, spatial control of flagella in B. subtilis seems more relevant to the inheritance of fl

110 nalysis showed that the biofilm formation in B. subtilis negates suppression of MAMPs-activated defen

111 as novel regulators of biofilm formation in B. subtilis.

112 We found in B. subtilis that the rapid localization of RecA to repai

113 replicative DNA polymerase PolC functions in B. subtilis, we applied photobleaching-assisted microsco

114 replacement of the wild-type spoVAD gene in B. subtilis with any of these spoVAD gene variants effec

115 essed the transcription of selected genes in B. subtilis.

116 production in the absence of cell growth in B. subtilis.

117 Iron and manganese homeostasis in B. subtilis are closely intertwined: a pfeT mutant is ir

118 des the primary route of magnesium import in B. subtilis and that the other putative transport protei

119 induction imposes severe iron limitation in B. subtilis resulting in derepression of both Fur- and P

120 ugh the emergence of redundant mechanisms in B. subtilis and related organisms.

121 the pathway leading to EF-P modification in B. subtilis, and B. subtilis encodes the first EF-P orth

122 regulation of cell chaining and motility in B. subtilis.

123 tion of a dominant negative gerD mutation in B. subtilis.

124 ore, we postulate that adaptive mutations in B. subtilis can be generated through a novel mechanism m

125 evious work, a deletion of the pks operon in B. subtilis was found to induce prodiginine production b

126 act dynamically to individualize origins in B. subtilis and, when loaded along eukaryotic chromosome

127 The retention of the indirect pathway in B. subtilis and B. halodurans likely reflects the ancien

128 ry step of the initiator assembly pathway in B. subtilis, in contrast to the prevailing model of bact

129 of penicillin-binding protein 2B (PBP2B) in B. subtilis cells did not affect the subcellular localiz

130 of ribonuclease E, a protein not present in B. subtilis.

131 nsor that can be used as a helicase probe in B. subtilis and closely related gram positive bacteria.

132 When Alp7A-GFP is produced in B. subtilis along with untagged Alp7R, Alp7A-GFP also co

133 1.1 and delta, an RNAP-associated protein in B. subtilis, bearing implications for the so-far unknown

134 corporation of D-amino acids into protein in B. subtilis.

135 We found that loss of RecD2 in B. subtilis sensitized cells to several DNA-damaging age

136 fect, indicating a new function for RecD2 in B. subtilis.

137 s one of the prominent c-di-AMP receptors in B. subtilis.

138 biosynthesis is differentially regulated in B. subtilis from classically studied Gram-negative flage

139 ity control function of IleRS is required in B. subtilis for efficient sporulation and suggests that

140 We show that loss of RER in B. subtilis causes strand- and sequence-context-dependen

141 least for the establishment of cell shape in B. subtilis.

142 ts that govern the entry into sporulation in B. subtilis and discuss how the use of regulated cell de

143 although this inhibition is much stronger in B. subtilis.

144 of a functional c-di-GMP signaling system in B. subtilis that directly inhibits motility and directly

145 function of the three adaptation systems in B. subtilis.

146 6 TF regulons with previously known TFBSs in B. subtilis and projected them to other Bacillales genom

147 We observe that in B. subtilis, a relA mutant grows exclusively as unchaine

148 Taken together, these data show that, in B. subtilis, a previously uncharacterized posttranslatio

149 The inferred TRN in B. subtilis comprises regulons for 129 TFs and 24 regula

150 Unlike in B. subtilis, SpoIIQ of Clostridium difficile has intact

151 Solar radiation alone did not inactivate B. subtilis spores under the conditions investigated.

152 We purified the AR9 nvRNAP from infected B. subtilis cells and characterized its transcription ac

153 s bound to its natural substrate, the intact B. subtilis peptidoglycan.

154 Interestingly, B. subtilis has two 6S RNAs, 6S-1 and 6S-2, but only 6S-

155 ty by the antitoxin EcMazE diverges from its B. subtilis homolog.

156 uorescent D-amino carboxamide probe to label B. subtilis PG in vivo and found that this probe labels

157 Surfactin not only lysed B. subtilis vesicles, but also vesicles from Bacillus an

158 flagella and the absence of a periplasm make B. subtilis a premier organism for the study of the earl

159 Moreover, B. subtilis suppression of MAMPs-activated root defense

160 accumulation of 29 DNA was higher in mutant B. subtilis cells with increased length.

161 -aminopentanol moiety attached to Lys(32) of B. subtilis EF-P that is required for swarming motility.

162 of the SSB C terminus impairs the ability of B. subtilis to form repair centers in response to damage

163 etion of prkC or prpC altered the ability of B. subtilis to grow under gluconeogenic conditions.

164 atly stimulates the endonuclease activity of B. subtilis MutL and supports this activity even in the

165 We suggest that administration of B. subtilis EPS can be used to broadly inhibit T cell ac

166 criptional regulation during anaerobiosis of B. subtilis.

167 Experimental and SNP analyses of B. subtilis genomes show mutational footprints consisten

168 , we have carried out functional analysis of B. subtilis thiI and the adjacent gene, nifZ, encoding f

169 ults show a distinct chemotactic behavior of B. subtilis toward a particular root segment, which we i

170 Biochemical characterization of B. subtilis RecD2 showed that it is a 5'-3' helicase and

171 The severe germination defect of B. subtilis cwlJ sleB or cwlJ sleB ypeB spores was compl

172 The dispersal of B. subtilis was very limited, particularly under protect

173 ctional residues in the N-terminal domain of B. subtilis MutL that are critical for mismatch repair i

174 Here, we report that biofilm formation of B. subtilis in LB medium is triggered by a combination o

175 Although the genome of B. subtilis encodes three c-di-AMP-producing diadenlyate

176 is required for the efficient germination of B. subtilis spores.

177 ge amounts of c-di-AMP impairs the growth of B. subtilis and results in the formation of aberrant cur

178 that c-di-AMP is essential for the growth of B. subtilis and shows that an excess of the molecule is

179 These two compounds impede the growth of B. subtilis under oxidative stress, and crystal structur

180 solutions markedly enhanced inactivation of B. subtilis spores in 10 mM phosphate buffer; increasing

181 s, respectively, whereas the inactivation of B. subtilis spores was slightly enhanced.

182 we report that a gastrointestinal isolate of B. subtilis sporulates with high efficiency during growt

183 or and even to a greater extent than loss of B. subtilis itself.

184 y of understanding the immunity mechanism of B. subtilis in particular and of other lantibiotic produ

185 Specifically, a fengycin-defective mutant of B. subtilis GS67 lost inhibitory activity against pathog

186 exopolysaccharide (EPS)-deficient mutant of B. subtilis was used, suggesting that EPS are the protec

187 Using mutants of B. subtilis that prevent flagellum rotation, they measur

188 ancy in the bacitracin resistance network of B. subtilis is a general principle to be found in many b

189 ose, designed from a detailed observation of B. subtilis levansucrase (SacB) acceptor structural requ

190 Thus, the rplJL operon of B. subtilis is regulated by transcription attenuation an

191 is, therefore, whether the dd-peptidases of B. subtilis are separately specific to carboxylate or ca

192 The peptidoglycan of B. subtilis in the vegetative stage, however, has the N-

193 S rRNA gene amplicons showed the presence of B. subtilis in the gut during the seven days of probioti

194 g to determine the transcription profiles of B. subtilis strains expressing mutant CodY proteins with

195 ines and genome-wide mutational profiling of B. subtilis lacking RNase HII, the enzyme that incises a

196 tant difference in biochemical properties of B. subtilis and E. coli RNA polymerases, specifically in

197 portant for the anti-infective properties of B. subtilis and its relatives.

198 tivity, it appears to prevent the release of B. subtilis sigma(B) from its anti-sigma factor RsbW.

199 set of the intrinsic antibiotic resistome of B. subtilis.

200 Finally, the secretome of B. subtilis might be used for the green synthesis of Ag-

201 rong, fungicidal activity and selectivity of B. subtilis QST713 lipopeptides.

202 the high-quality Sanger genome sequences of B. subtilis subspecies subtilis RO-NN-1 and AUSI98, B. s

203 wing cells, dormant and germinated spores of B. subtilis, and dormant spores of several other Bacillu

204 e no killing or rupture of dormant spores of B. subtilis, Bacillus cereus or Bacillus megaterium, alt

205 he spo0B gene in a delta-knock-out strain of B. subtilis compared with the wild-type.

206 inally, we show that domesticated strains of B. subtilis carry a mutation in sigH, which influences t

207 goal in this study was to isolate strains of B. subtilis that exhibit high levels of biocontrol effic

208 In contrast to domesticated lab strains of B. subtilis which form smooth, essentially featureless c

209 We found that in wild strains of B. subtilis, surfactin disrupted vesicles while in labor

210 We present the crystal structure of B. subtilis PhoD.

211 3 are sufficient to disrupt the structure of B. subtilis spores resulting in decreased viability.

212 The crystal structure of B. subtilis SpoVAD has been determined recently, and a s

213 ve established a model system for studies of B. subtilis-tomato plant interactions in protection agai

214 We conclude that the susceptibility of B. subtilis to the biofilm-inhibitory effects of D-amino

215 Here we report the synthesis of B. subtilis Lipid II and its use by purified B. subtilis

216 tant tRNA binds strongly to the AUA codon on B. subtilis ribosomes but only weakly to AUG.

217 analysis was used to map TiO2 deposition on B. subtilis cell walls and released enzymes, supporting

218 allate (TF3) exhibited inhibitory effects on B. subtilis c-di-AMP synthase, DisA.

219 Studies on B. subtilis emphasized the genetics and biochemistry of

220 subtilis' or HSBS) was compared to that onto B. subtilis biomass with a low concentration of sulfhydr

221 on of natural products in the model organism B. subtilis and paves the way to the development of futu

222 the class 2 mutations in both Gram-positive B. subtilis and Gram-negative Escherichia coli.

223 Like the Gram-positive B. subtilis SpoIIIJ, the conserved arginine was required

224 ) exogenous norspermidine at 25 muM prevents B. subtilis biofilm formation, (3) endogenous norspermid

225 pendent adhesive properties of the probiotic B. subtilis natto (Bsn).

226 increased the population of matrix-producing B. subtilis cells and that this activity could be abolis

227 Purified B. subtilis aconitase bound to the citZ 5' leader RNA in

228 B. subtilis Lipid II and its use by purified B. subtilis PBP1 and E. coli PBP1A.

229 Here we report that purified B. subtilis RppH requires at least two unpaired nucleoti

230 rast, the phylogenetic range of recognizable B. subtilis RppH orthologs appears to be restricted to t

231 reconstituted in vitro by mixing recombinant B. subtilis PxpA, PxpB, and PxpC proteins.

232 ined following immunization with recombinant B. subtilis spores were able to reduce the adhesion of C

233 In addition to regulating B. subtilis biofilm formation, we found that RapP regula

234 Consistent with this regulatory scheme, B. subtilis FtsE mutants that are unable to bind or hydr

235 Second, B. subtilis synthesizes its own siderophore bacillibacti

236 ore, we monitor the motility state of single B. subtilis cells across multiple generations by the exp

237 Thus, the mesophilic species B. subtilis and E. coli share the same sigma1.1 fold, wh

238 richia coli (E. coli) and Bacillus subtilis (B. subtilis) by bacterial growth on the membrane surface

239 rium (S. typhimurium) and Bacillus subtilis (B. subtilis) were examined and observed.

240 richia coli (E. coli) and Bacillus subtilis (B. subtilis).

241 These results suggest B. subtilis senses restriction of flagellum rotation as

242 s, including those with smaller genomes than B. subtilis.

243 These findings led us to discover that B. subtilis cells that overproduce KinA can bypass the s

244 ve bacterium Bacillus subtilis We found that B. subtilis sigma1.1 is highly compact because of additi

245 Altogether our data indicate that B. subtilis blocks entry into sporulation in high-salini

246 Here we report that B. subtilis NusG makes sequence-specific contacts with a

247 Here we report that B. subtilis produces an additional biofilm-disassembly f

248 In addition, it reveals that B. subtilis can synthesize thin, Gram-negative-like PG l

249 We show that B. subtilis can export Ply, suggesting that the export p

250 We show that B. subtilis GS67 persists in the C. elegans intestine an

251 obable substrates for Mini-III and show that B. subtilis Mini-III is also involved in intron regulati

252 Here, we show that B. subtilis requires only the MotA/MotB stator during sw

253 We showed that B. subtilis cells lacking (p)ppGpp, due to either deleti

254 l status of conditioned media suggested that B. subtilis cells lacking 6S-1 RNA reduce the nutrient c

255 Moreover, modeling studies suggested that B. subtilis sigma1.1 requires minimal conformational cha

256 o protect mice from disease, suggesting that B. subtilis-mediated protection requires functional flag

257 The B. subtilis replisome is eukaryotic-like in that it reli

258 ted to the electrical signal released by the B. subtilis biofilm.

259 his, met, and leu revertants produced by the B. subtilis YB955 parental strain.

260 us and for the first time, characterized the B. subtilis SSB's DNA binding mode switching and stoichi

261 Here, we describe the B. subtilis PfeT protein (formerly YkvW/ZosA) as a P1B4

262 nucleotide is particularly important for the B. subtilis RNA polymerase to use RNA templates.

263 well characterized type I TA system from the B. subtilis chromosome, bsrG/SR4, reveals similarities b

264 sigma(G) activation, thus departing from the B. subtilis model.

265 of differentiation of null mutants from the B. subtilis ordered knockout collection.

266 S-adenosyl-methionine-I riboswitch from the B. subtilis yitJ gene encoding methionine synthase, can

267 Here we identify the B. subtilis flagellum as a mechanosensor that activates

268 Our findings offer novel insight into the B. subtilis phosphate starvation response and implicate

269 suggesting it participates in defence of the B. subtilis biofilm against Gram-positive bacteria as we

270 Site-directed mutagenesis of the B. subtilis CTV suggests that electrostatic forces are a

271 ase-dependent intracellular signaling of the B. subtilis DDR is achieved via production of L-malic ac

272 Cloning of the B. subtilis epsH-K genes into Escherichia coli with in-f

273 Examination of the B. subtilis genome sequence showed that these EF-P-depen

274 this study, we characterize features of the B. subtilis lysC leader RNA responsible for lys specific

275 Here, we describe the use of the B. subtilis model system to study the adaptation of thes

276 on this information, a homology model of the B. subtilis tau3-delta-delta' complex was constructed, w

277 ant protein or to TcdA26-39 expressed on the B. subtilis spore surface, cross-react with a number of

278 tilis Lipid II into glycan strands, only the B. subtilis enzyme cross-linked the strands.

279 The materials to produce the B. subtilis SSB probe are commercially available, so the

280 This cleavage is independent of PrsW, the B. subtilis site 1 protease, which cleaves the anti-sigm

281 s supported FtsZ assembly, but replacing the B. subtilis FtsZ linker with a 249-residue linker from A

282 ences in mutation rates of genes require the B. subtilis Y-family polymerase, PolY1 (yqjH).

283 the first nucleotide of its RNA targets, the B. subtilis enzyme has a binding pocket that prefers gua

284 on scattering spectra, we confirmed that the B. subtilis cell membrane is lamellar and determined tha

285 show that 29 protein p1 colocalizes with the B. subtilis cell division protein FtsZ and provide evide

286 Thus, B. subtilis and probably most bacteria use two distinct

287 Thus, B. subtilis switches from a unicellular to a multicellul

288 The behavioral differences in the two B. subtilis RNAs clearly demonstrate that they act indep

289 nd that norspermidine is absent in wild-type B. subtilis biofilms at all stages, and higher concentra

290 nt in both pellicle and planktonic wild-type B. subtilis cells and in strains with deletions in the e

291 Here, colonies of wild-type B. subtilis formed a spreading population that induced p

292 Mice that received wild-type B. subtilis prior to enteric infection were protected fr

293 Viable B. subtilis cells were identified and DNAs of two bacter

294 A fluorescent in vivo B. subtilis reporter system identified peptide motifs wh

295 Here, we show that, when B. subtilis colonizes Arabidopsis thaliana roots it form

296 To investigate whether B. subtilis encodes yet additional classes of transport

297 The results explain why B. subtilis with its Asn synthetase genes knocked out is

298 escribes an investigation of this issue with B. subtilis PBP4a.

299 implies that it shares more orthologues with B. subtilis subsp. subtilis NCIB 3610(T) (ANIm values, 8

300 Dynamic tests with B. subtilis and E. coli showed high antibacterial effici