ライフサイエンスコーパス: B

1 B cells contribute to multiple aspects of autoimmune dis

2 B cells expressing the T-bet transcription factor, a mar

3 B cells from hypersensitive patients, but not controls,

4 B cells influence the pathogenesis of multiple sclerosis

5 B-cell-specific Moloney murine leukemia virus integratio

6 pression of downstream genes, such as Bcl-2 (B-cell lymphoma 2), c-Myc, MMP7 (matrix metalloproteinas

7 by recruitment of the protein phosphatase 2A B' (PP2A B').

8 In contrast, TIM-4(+) B cells decreased B16-F10 metastasis and s.c. tumor grow

9 the source of contamination in an event of a B. canis outbreak.

10 Three independent observers (A, B, C) measured PVRs at two different time points during

11 very strong electric field across the c-Si/a-B interface systems where the charge transfer occurs mai

12 ms based on the trnL gene were designed (A-, B-, C- and D-trnL systems).

13 ctor kappa-light-chain-enhancer of activated B cells.

14 ution to the gut, increases of the activated B cells and circulating tissue-like memory B cells, and

15 ion in adaptive immunological memory against B. pertussis.

16 ly, antifungal drugs, including amphotericin B, liposomal amphotericin B, and flucytosine, need to be

17 uding amphotericin B, liposomal amphotericin B, and flucytosine, need to be much more widely availabl

18 f E.coli, as well as effects of amphotericin-B and miconazole on S. cerevisiae through the device's t

19 2,000 confirmations for each of types A and B over the study period.

20 36-month prospective, randomized, FAME A and B trials.

21 xamination and Trail Making Test Parts A and B.

22 crete proportion of infiltrating T cells and B cells underwent proliferation within the pituitary par

23 mediating energy metabolism, cell cycle, and B cell receptor signaling.

24 innate lymphoid, myeloid, and dendritic, and B-cell fate alternatives are excluded by different mecha

25 trients, flavan-3-ols (i.e., epicatechin and B-type procyanidins) as also hydroxycinnamoyl-quinic aci

26 evels of B-lineage transcription factors and B cell receptor (BCR)/pre-BCR-signaling genes.

27 s an innate link between viral infection and B cell immunity.

28 Thus, the dichotomy between T-like and B-like cells and the presence of dedicated lymphopoietic

29 thracis DNA into individual PCR mixtures and B. anthracis CFU into human blood.

30 with less contribution from neutrophils and B cells.

31 ac natriuretic peptides (NPs), atrial NP and B-type NP, regulate fluid homeostasis and arterial BP th

32 perature adaptation between B. pertussis and B. bronchiseptica may result from selective adaptation o

33 s to sense alum and, in turn, activate T and B cells.

34 ibly including tissue macrophages) and T and B lymphocytes in the presence of detectable inflammatory

35 s, B. burgdorferi-infected nymphal ticks and B. mayonii-infected nymphal ticks by measuring metabolis

36 Apolipoprotein B mRNA-editing catalytic polypeptide (APOBEC) 3 proteins

37 ]: -1.03 [-1.17, -0.89]), and apolipoprotein B (change in SD units [95% confidence interval]: -0.98 [

38 s study, we demonstrated that apolipoprotein B mRNA-editing catalytic polypeptide 3A (A3A) and A3G ex

39 The apolipoprotein B mRNA editing enzyme catalytic polypeptide-like APOBEC3

40 o provide spatial selectivity with arbitrary B 1 field distributions in non-conductors.

41 4 months of age was 43% in arm A, 52% in arm B (9%, 7-11) and 54% in arm C (11%, 9-13; overall p<0.00

42 SIV-associated B cell dysfunction associated with the pathogenic SIV in

43 Central to this regulation is Aurora B kinase, which phosphorylates kinetochore substrates to

44 rences in low-temperature adaptation between B. pertussis and B. bronchiseptica may result from selec

45 The Bi-B double and triple bond strengths are calculated to be

46 The BiB2 O(-) and Bi2 B(-) clusters are produced by laser vaporization of a mi

47 Before birth, B cells develop in the fetal liver (FL).

48 Flow cytometric analysis of peripheral blood B cells of 30 MC-negative HCV-infected patients and 15 h

49 Here, the authors engineer BoNT/B to improve its affinity to human receptors and enhance

50 m are controlled at low concentrations (both B and P are less than 1 ppm).

51 CD4(+) and CD8(+) T cells but no binding by B cells.

52 it remained unknown if, and how, calcineurin B-like calcium sensors (CBLs) and CBL-interacting protei

53 As carmaphycin B is toxic to mammalian cells, we synthesized a series o

54 Cathepsin B (CtsB) contributes to atherosclerosis and cancer progr

55 Co-expression of cathepsin B and cathepsin B-resistant mutant LFABP in McA-RH7777 cells resulted in

56 compared with cells co-expressing cathepsin B and wildtype LFABP.

57 Co-expression of cathepsin B and cathepsin B-resistant mutant LFABP in McA-RH7777 c

58 brane, resulting in the release of Cathepsin B.

59 repression of the lysomal protease cathepsin B.

60 t the block of CD74 degradation in CatS(-/-) B cells is incomplete, so that NTF levels are significan

61 keys, SGN-CD19B effectively depleted CD20(+) B lymphocytes in peripheral blood and lymphoid tissues c

62 ly composed of CD3+ T cells, scattered CD20+ B cells, and relatively few PD-1+ (programmed cell death

63 ious suggestions that APOE, CHRNA3/5, CDKN2A/B, SH2B3 and FOXO3A influence longevity.

64 pan-T cells, CD4(+) T cells, CD8(+) T cells, B cells, and NK cells, with 49 recombinant chemokines us

65 (75%) had esophageal varices, 21% were Child-B, and 29% had at least 1 previous episode of liver deco

66 lly, CpG increased the number of circulating B cells, which produced elevated amounts of tumor necros

67 GCGR) are members of the secretin-like class B family of G-protein-coupled receptors (GPCRs) and have

68 A separate longitudinal sample (cohort B) included 16 MDD patients who underwent MRI at baselin

69 sessed cerebral Abeta on Pittsburgh Compound B (PiB) positron emission tomography, gait speed over 4.

70 ivity was defined as 11C-Pittsburgh compound B target-to-cerebellar ratio above 1.5 within a composit

71 beta) and background particle concentration (B) to be independent for use in fate modeling.

72 tr-css operons and adjacent deletion of cssA/B/C and a part of csc, encoding the serogroup C capsule

73 ned a model of specific antibody deficiency, B cell-specific CD79a-Cre x XBP1 (X-box binding protein-

74 f multiple sclerosis suggests that depleting B cells could be useful for treatment.

75 OL (B = 2.61; P < .001) and less depression (B = -0.78; P < .001) among patients who reported a termi

76 OL (B = 3.50; P < .001) and less depression (B = -1.01; P < .001) among patients who acknowledged the

77 representative siderophore, desferrioxamine B (DFOB), iron (Fe) was released at higher rates and to

78 ood agreement with experimentally determined B factors and with fluctuations predicted by the Gaussia

79 d MLVA methods are limited in discriminating B. canis strains.

80 ac in the lower uterine segment and elevated B-Hcg levels, the possibility of scar ectopic pregnancy

81 ddition, we find that EmMBD2/3 and EmGATAD2A/B proteins form a coiled-coil interaction known to be cr

82 Transformations employ [B(pin)]2-methane as a conjunctive reagent, resulting in

83 e that ectopic expression of insulin epitope B:9-23 (InsB9-23) by thymic APCs is insufficient to indu

84 icantly increases CD20 levels in established B-cell tumor cell lines and primary malignant cells.

85 ted MEK1/2 in various tumor cells expressing B-Raf(V600E) or K-Ras(G12C/D) Intriguingly, coimmunoprec

86 c assay is a highly discriminatory assay for B. canis genotyping, and can serve as a useful molecular

87 on Network Analysis (WGCNA) was enriched for B cell pathways, and shared seventeen genes with a mouse

88 osit MYD88/p100 signaling as a regulator for B-cell activation.

89 8)F-labeling methodologies, specifically for B-(18)F, Si-(18)F, Al-(18)F, and iodine (III)-mediated r

90 ter-based detection cartridge and tested for B. anthracis on a GeneXpert instrument.

91 a common gammaherpes virus with tropism for B lymphocytes and infection in immunocompetent individua

92 s, mTORC1 activation is required for fueling B cells prior to DZ proliferation rather than for allowi

93 assenger allele system to assay, in mouse GC B cells, sequence-intrinsic SHM-targeting rates of nucle

94 biting proliferation of germinal center (GC) B cells.

95 lin light chain editing occurred, generating B cells with up-regulated Nod1, including follicular and

96 ), MRD detection method, phenotype/genotype (B cell, T cell, Philadelphia chromosome), and EFS and OS

97 ty in the activation of the lineage germline B cell.

98 onal Study Group on Pancreatic Fistula Grade B or C) and hospital-related inpatient costs for 90 days

99 ar lymphoma, and MYC/BCL2/BCL6 in high-grade B-cell lymphomas are essential for diagnosis.

100 eacetyl parasiticolide A, flufuran, gregatin B, hydroxysydonic acid, nicotinic acid, phomaligin A, sp

101 Group B coxsackieviruses are responsible for chronic cardiac i

102 oup A (98.2%) and 50 of 57 patients in group B (87.7%; P=0.06).

103 scription from a Gastroenterologist in group B were associated with eradication success.

104 mg/0.1 mL) 1-3 days before PPV, while Group B received IVB (2.5 mg/0.1 mL) 5-10 days before PPV.

105 to incorporate the biological cofactor heme-B for catalysis.

106 Hepatitis B virus (HBV) chronic infection affects up to 240 millio

107 Hepatitis B virus (HBV)-encoded X protein (HBx) plays a critical r

108 Hepatitis B viruses (HBVs), which are enveloped viruses with rever

109 of T cells engineered to express a hepatitis B virus-specific (HBV-specific) T cell receptor (TCR) ma

110 2334 RA patients who had available hepatitis B surface antigen (HBsAg) data, 123 patients positive fo

111 treatment in patients with chronic hepatitis B (CHB).

112 CC), often associated with chronic hepatitis B virus (HBV) infection.

113 with acute, resolved, and chronic hepatitis B virus (HBV)infection but might also signify occult HBV

114 herapeutic developments in chronic hepatitis B.

115 biomarkers toward better defining hepatitis B virus cure should occur in parallel with development o

116 Six patients had hepatitis B surface antibody (anti-HBs) titres above 10 mIU/mL at

117 We analyzed changes in hepatitis B virus and hepatitis delta virus (HDV) viral loads (VL)

118 ed with a similar modest change in hepatitis B virus core antigen polypeptide (HBcAg/p21) synthesis,

119 d missed opportunities to initiate hepatitis B vaccination.

120 three national serosurvey data of hepatitis B in China, we propose an age- and time-dependent discre

121 The serum gradient of hepatitis B surface antigen (HBsAg) varies over time after cessati

122 alyze in real time the assembly of Hepatitis B Virus (HBV) capsids below the pseudocritical concentra

123 The management of hepatitis B virus (HBV) e antigen-positive viremic patients with n

124 The study of hepatitis B virus and development of curative antivirals are hampe

125 For the discovery of hepatitis B virus integration sites from probe capture data, the v

126 ons by either hepatitis A virus or hepatitis B virus (HBV), or a noninfectious cause for their ALF.

127 HBV functional cure is sustained hepatitis B surface antigen loss and anti-HBs gain, with normaliza

128 to be HCV and antibody reacting to hepatitis B core antigen+, and less likely to have diabetes or hyp

129 usefulness of this new transgenic hepatitis B model.

130 sor surface and post modified with hepatitis B surface antigen.

131 wer resolution) HLA typing for HLA-A and HLA-B, and allele-level for HLA-DRB1.

132 the probability of all 4 patients being HLA-B*53 carriers, and 2 of 3 African patients being homozyg

133 tricted response targets Gag (DRYFKTLRA, HLA-B*14-DA9).

134 3 African patients being homozygous for HLA-B*53:01, is approximately 0.00002.

135 xic NK potential on the basis of KIR3DL1/HLA-B subtype combinations in vitro and evaluated their impa

136 Given the approximate prevalence of HLA-B*53 carriage in African (20%) and Hispanic (6%) populat

137 We demonstrate that Env-specific HLA-B*14-restricted activity is substantially more efficacio

138 ly more efficacious than the subdominant HLA-B*14-restricted Gag response.

139 A subdominant HLA-B*14-restricted response targets Gag (DRYFKTLRA, HLA-B*1

140 determinant of differential HLA-A versus HLA-B downregulation activity.

141 planets are known to transit the even hotter B-type stars.

142 rrelia burgdorferi Here, we investigated how B. burgdorferi exploits Fn to interact with endothelia u

143 In both mouse and human B cells, BCR ligands that deliver a TLR9 agonist induce

144 immune murine B cell subsets and CD27- human B cells.

145 on, we compared the fate of IgE-ICs in human B cells and in CD23-expressing monocyte-derived dendriti

146 ric form of the toxin, suggesting that human B cells recognize epitopes on the dimerized form of LukA

147 ference between groups in the change in IBDQ-B scores between the start and nadir was not significant

148 esorption, we show that an acute loss of IFT-B through cilia decapitation precedes resorption.

149 lenate uptake by 100% in S. elata and 40% in B. juncea.

150 The disruption of autolysis in B. subtilis cultures by TiO2 NPs suggests the mechanisms

151 c antigen receptor (CAR) that target CD19 in B-cell cancers, although data regarding B-cell lymphomas

152 This dynamic change in B cell survival mechanisms is unique to virus-infected c

153 at Hh-mediated transcription is increased in B-lineage cells from Gli3-deficient FL and showed that t

154 We show that loss of RER in B. subtilis causes strand- and sequence-context-dependen

155 is is the first comprehensive map of TSSs in B. burgdorferi and characterization of previously un-ann

156 on and are associated with cancer, including B cell lymphomas.

157 the treatment of B cell neoplasms, including B cell chronic lymphocytic leukemia (B-CLL).

158 patient suffering from recurrent EBV-induced B cell proliferations including Hodgkin's lymphoma becau

159 enza A/H1N1, influenza A/H3N2, and influenza B strains.

160 Human CD40L(+) ILC3s provide innate B-cell help and are involved in an innate immunoregulato

161 Justicidin B is structurally similar to more potent vacuolar-type H

162 the rate of release of nuclear factor kappa B (NFkappaB) from DNA target sites in a process we have

163 S-induced activation of nuclear factor kappa B and reduced the expression of proinflammatory proteins

164 in kinase A and inhibition of Protein kinase B (AKT).

165 ulated kinase, phosphorylated protein kinase B, phosphorylated mammalian target of rapamycin, phospho

166 at an interaction between Xist RNA and Lamin B receptor (LBR) is necessary and sufficient for Xist sp

167 mphomas (DELs) are subtypes of diffuse large B-cell lymphoma (DLBCL) associated with poor outcomes af

168 licular lymphoma (FL; n = 29), diffuse large B-cell lymphoma (DLBCL; n = 34), DLBCL arising from chro

169 ous NHL histologies, including diffuse large B-cell lymphoma, Richter's transformation, mantle cell l

170 lymphoma, Burkitt's lymphoma, diffuse large B-cell lymphomas, nasopharyngeal carcinoma (NPC), and ly

171 l duplication, and the smaller 1.5 Mb LCR22A-B 22q11.2 deletion carry inversions of LCR22B-D or LCR22

172 cluding B cell chronic lymphocytic leukemia (B-CLL).

173 In the retrospective cohort, luminal B prostate cancers exhibited the poorest clinical progno

174 ukaemia (CLL) is a clonal disorder of mature B cells.

175 nduced mTOR activation impairs IL-7-mediated B cell development.

176 suppresses follicular helper T cell-mediated B cell responses in the germinal center reaction.

177 d B cells and circulating tissue-like memory B cells, and expansion of the B regulatory cells (Bregs)

178 t higher proportions of IgM+CD21-/low memory B cells (P < .05), CD4+IFNgamma+ cells (P < .01), CD4+IL

179 ystems of infected hosts to recall of memory B cells that recognized the lateral patch, the principal

180 affinity-matured human NANP-reactive memory B cell antibodies elicited by natural Pf exposure that p

181 oss of naive B cells, loss of resting memory B cells due to their redistribution to the gut, increase

182 ed that frequencies of class-switched memory B cells were increased in the patients, whereas frequenc

183 t carriage of Nef variants with enhanced MHC-B downregulation ability is associated with reduced brea

184 ed with reduced breadth and magnitude of MHC-B-restricted cellular immune responses in HIV-infected i

185 t Nef position 9 that contributes to the MHC-B downregulation function in HIV-1 subtype C and show th

186 re produced by laser vaporization of a mixed B/Bi target and characterized by photoelectron spectrosc

187 ranscriptomes and proteomes of primary mouse B cells from wild-type and STAT6-deficient mice cultured

188 ression contributes to maintenance of murine B-ALL cells with compromised Ikaros function.

189 ptosis and is shared by all preimmune murine B cell subsets and CD27- human B cells.

190 chromosomal rearrangements in primary murine B cells and discovered that RAG1/2 causes aberrant inser

191 nctions from CH12F3 cells and primary murine B cells, respectively.

192 erimental stroke prevents loss of splenic MZ B cells, preserves IgM levels, and reduces bacterial bur

193 l)iminomethane (BIM) complex [Pt(kappa(2) -N,B-(Cy2) BIM)(CNAr(Dipp2) )] can effect the oxidative ins

194 quencing method can use as few as 1000 naive B cells.

195 rom human immature dendritic cells and naive B cells to assess the expression of CD40-downstream gene

196 hereas frequencies of transitional and naive B cells were decreased.

197 infection is characterized by loss of naive B cells, loss of resting memory B cells due to their red

198 phia chromosome (Ph)-positive or Ph-negative B-cell acute lymphoblastic leukaemia who were due to rec

199 om the interface Si atoms to the neighboring B atoms.

200 e show here that a single marker, Neuromedin B mRNA (Nmb), identifies RTN neurons in rodents.

201 enza haemagglutinin and botulinum neurotoxin B, along with 6,286 control sequences to probe contribut

202 cing and RNA sequencing, we identify a novel B-lymphoid program for transcriptional repression of glu

203 tica may result from selective adaptation of B. pertussis to the human host.

204 anode nanojunction architecture, composed of B-doped carbon nitride nanolayer and bulk carbon nitride

205 PRIL system is best known for its control of B cell homeostasis, and it is a target of therapeutic in

206 ARTEMIS is essential for the development of B and T lymphocytes.

207 arabinose residues, decreased the extent of B cell activation.

208 Among the virulence factors of B. anthracis is the S-layer-associated protein BslA, whi

209 We demonstrate here that the formation of B-N Lewis pairs at the periphery of anthracene leads to

210 omotes chronic gammaherpesvirus infection of B cells.IMPORTANCE Gammaherpesviruses infect a majority

211 that these cells expressed reduced levels of B-lineage transcription factors and B cell receptor (BCR

212 protective or deleterious effects on loss of B cell self-tolerance in vivo, we depleted neutrophils a

213 cterial sepsis, we found that the numbers of B-1a cells in various anatomical locations were signific

214 ndamental differences in the pathogenesis of B-cell lymphoproliferative disorders.

215 used a significant increase in percentage of B-cells and significantly decreased percentage of myeloi

216 ly integrate the transcriptional response of B-ALL to GCs with a next-generation short hairpin RNA sc

217 The potential protective role of B cells in controlling cCMV-related disease and the clin

218 well as genes that affect the sensitivity of B-ALL cells to dex.

219 s in growth characteristics among strains of B. anthracis, which is considered to be a clonal organis

220 hallmark anti-CD20 mAb for the treatment of B cell neoplasms, including B cell chronic lymphocytic l

221 te (ADC) being investigated for treatment of B-cell malignancies, which has improved potency compared

222 the Artisan Myopia or Artisan Toric (Ophtec B.V., Groningen, The Netherlands) iris-fixated pIOL for

223 these data and mutations identified in other B-cell malignancies, 1716 genes were sequenced in 113 FL

224 onstrate their use for the introduction of P=B units into organic systems.

225 arachidonoyl-fluorophosphate and palmostatin B, and partially prevented by JZL184 and WWL113.

226 ance/survival of the mature naive peripheral B cell population.

227 ory molecule playing a role in physiological B cell responses.

228 tion compromises the activity of the pivotal B-cell antigen receptor (BCR)-proximal effector spleen t

229 Multiple myeloma (MM) is a plasma B-cell hematologic cancer that causes significant skelet

230 tment of the protein phosphatase 2A B' (PP2A B').

231 progesterone via PR-A, but not the longer PR-B isoform, with increased progesterone sensitivity when

232 d with an hCD22 ligand were shown to prevent B cell activation, increase cell death, and induce toler

233 ronolactone and usual care on N-terminal pro-B-type natriuretic peptide (NT-proBNP) levels compared w

234 (48 versus 40), higher median N-terminal pro-B-type natriuretic peptide concentration (403 versus 320

235 mL]; P<0.0001), despite lower N-terminal pro-B-type natriuretic peptide levels.

236 V, RVESRI, and log NT-proBNP (N-Terminal Pro-B-Type Natriuretic Peptide) were retained (chi(2), 62.2;

237 Hedgehog (Hh) signaling promotes B lymphopoiesis in a non-cell-autonomous fashion in vitr

238 ne CRPs, the PCP-Bs (for pollen coat protein B-class) that are related to embryo surrounding factor (

239 Myasthenia gravis (MG) is a prototypical B cell-mediated autoimmune disease affecting 20-50 peopl

240 ositive reframing was related to better QOL (B = 2.61; P < .001) and less depression (B = -0.78; P <

241 ctive coping was associated with better QOL (B = 3.50; P < .001) and less depression (B = -1.01; P <

242 tion into a set number of bins (quantization B) and an alternative quantization with bins of fixed wi

243 GCs) in lymphoid tissues where self-reactive B cells expand and differentiate.

244 otential of SGN-CD19B in relapsed/refractory B-NHL.

245 9 in B-cell cancers, although data regarding B-cell lymphomas are limited.

246 of the campaign in the SLSJ region (relative B-IMD risk: 0.22; P = .04).

247 strains of bacteria: Lactobacillus rhamnosus B 442, Lactobacillus rhamnosus 1937, and Lactococcus lac

248 We also found that the RING B-box coiled-coil (RBCC) domain in KAP1 and the proximal

249 In this model, a loop defined by the SCI's B and C domains encircles the C-terminal segment of the

250 e strong support for Branigan & Pickering's (B&P's) model, largely because the priming effects are mo

251 mmunity effects with meningococcal serogroup B vaccines and the need for a booster dose to sustain in

252 (VL) sequences of individual and Ag-specific B cells.

253 o showed superior activation of Env-specific B cells.

254 h cells than other lineages of more specific B cells.

255 Interestingly, PLA-specific B cells showed increased CCR5 expression after high-dose

256 nt resulted in suppression of virus-specific B cell responses by inhibiting proliferation of germinal

257 and dynamic range were determined by spiking B. anthracis DNA into individual PCR mixtures and B. ant

258 mic or splenic DC, whereas thymic or splenic B cells were poor donors.

259 are significantly lower than in SPPL2a(-/-) B cells.

260 id antibody, and anti-Ro/SS-A and anti-La/SS-B antibodies.

261 yo-electron microscopy structures of subtype B B41 SOSIP Env trimers in complex with CD4 and antibody

262 liciting local mucosal immunity and systemic B cell- and T cell-mediated memory responses.

263 ound that rmIFN induces the activation of T, B, and NK cells and exhibits antiviral, antiproliferativ

264 Delivery of neurotransmitters across the T-B-cell synapse may be advantageous in the face of infect

265 We found that B. microti reaches high parasitemia levels during the fi

266 icial substrates or live cells, we show that B cells primarily use force-dependent extraction and res

267 These results show that B. burgdorferi can transform a ubiquitous but normally n

268 Thus, upregulation of EBI2 drives the B cells toward the extrafollicular area, whereas downreg

269 nical outcomes when used as carriers for the B-KPro, with no significant differences in device retent

270 ed by mutations in JAK2, CALR, or MPL In the B-cell lymphomas, detection of characteristic rearrangem

271 e have demonstrated that the kinetics of the B cell response are similar for all four FMDV serotypes

272 ue-like memory B cells, and expansion of the B regulatory cells (Bregs).

273 ated to regulate signaling downstream of the B-cell receptor (BCR), Fc receptors (FcRs), and toll-lik

274 After passage of the B/Brisbane/60/2008 virus in the presence of 46B8, we iso

275 e therefore expected to evolve only when the B cell lineage evolving breadth is consistently capturin

276 mong uninfected I. scapularis nymphal ticks, B. burgdorferi-infected nymphal ticks and B. mayonii-inf

277 ibitory activity for allergen-IgE binding to B cells (IgE-facilitated allergen binding).

278 r helper (pTfh) cells, which provide help to B cells for developing into Ab-secreting cells, were sim

279 We conjugated unimNPs with the cholera toxin B domain (CTB) for RGC-targeting and with Cy5.5 for unim

280 of 65.7% (95% CI, 59.4%-72.1%) and in trial B, 192 of 255 (75.3%) vs 25 of 260 (9.6%; P < .001), for

281 Circulating RBP4, TTR, B-type natriuretic peptide (BNP), and troponin I (TnI) c

282 Aortic dissection, commonly type B, occurs in an appreciable proportion of Marfan patient

283 e derived from sera with neither H1 nor type B influenza preexposure.

284 om human postvaccination sera with only type B influenza preexposure consistently showed good correla

285 so suggest that prior exposure to H1 or type B influenza may differentially affect cross-reactivity o

286 ng the cytokinin response, mechanism of type-B ARR activation, and basis by which cytokinin regulates

287 light on the physiological role of the type-B ARRs in regulating the cytokinin response, mechanism o

288 0-positive cells did not display the typical B cell morphology, having in general a more dendritic ce

289 two major QTLs identified in undomesticated B. distachyon colocalize with VERNALIZATION1/PHYTOCHROME

290 Unlike B cell depletion, pan-T cell aplasia is prohibitively to

291 In addition, UV-B increased leaf quercetin content and total antioxidant

292 cursor of the active gibberellin, GA1, by UV-B in this zone, which is regulated, at least in part, by

293 Our results indicate that UV-B supplementation may provide a method to manipulate the

294 Vitamin B-6 deficiency was defined as PLP <20 nmol/L, and insuff

295 ed to lethality in infected animals, whereas B cell-deficient mice showed CD4(+) T cell loss but reco

296 matoes with this coating and inoculated with B. cinerea showed a significant decrease in fungal growt

297 ible interactions of arsenic metabolism with B vitamins and AS3MT variants on diabetes risk.

298 ng of MRN in cancer, we engineered mice with B lymphocytes lacking MRN, or harboring MRN in which MRE

299 cells from the lungs of mice reinfected with B. pertussis produced significantly more IL-17 than gamm

300 ID), a 600 nt sequence encompassing the Xist B-repeat element.

301 Nod1, including follicular and marginal zone B cells with natural autoreactivity.