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1 aging the DNA inside the capsid structure of B19 virus.
2 llas, respectively), HBoV (73% and 36%), and B19 virus (8% and 27%) were recorded for apes, while OWM
3 patocellular carcinoma cell line Hep G2 with B19 virus and assayed for apoptosis by using annexin V s
4 mmunoassay that detects the human parvovirus B19 virus (B19V) immunoglobulin M (IgM) or IgG in the se
5 ncubation with anticapsid antibodies against B19 virus, but not anticapsid antibodies against AAV, in
7 leukemia cell line which can be infected by B19 virus following erythroid differentiation with eryth
15 cute fulminant liver failure associated with B19 virus infection was characterized by hepatocellular
17 Analysis of caspase involvement showed that B19 virus-inoculated cultures had a significant increase
22 e and circulation of parvoviruses related to B19 virus, PARV4, and HBoV in nonhuman primates, plasma
23 were screened for antibodies to recombinant B19 virus, PARV4, and HBoV VP2 antigens by enzyme-linked
26 ome replication, internal polyadenylation of B19 virus RNAs at (pA)p is favored in cells which are bo
28 Blockage of the production of full-length B19 virus transcripts at the internal polyadenylation si
30 ary human erythroid cells by the recombinant B19 virus vector was significantly higher than that by t
31 sis and immunofluorescence demonstrated that B19 virus was able to infect the cells and produce its n
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