ライフサイエンスコーパス: B7RP-1

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1 B7RP-1-Fc also increased the ability of the cells in the

2 B7RP-1-Fc also increased the levels of the chemokines CC

3 B7RP-1-Fc also increased the number of cells in lymph no

4 B7RP-1-Fc increased the number of cells in lymph nodes d

5 B7RP-1-Fc stimulated contact hypersensitivity (CH) given

6 B7RP-1-Fc stimulated the production of anti-keyhole limp

7 B7RP-1-Fc stimulates both cellular and humoral immune re

8 (ICOS) and its ligand B7-related protein 1 (B7RP-1) in the experimental murine schistosome infection

9 that blockade of ICOS-B7-related protein 1 (B7RP-1) in vivo dramatically reduces germinal center for

10 ostimulator (ICOS) and B7-related protein-1 (B7RP-1) constitute a receptor-ligand pair involved in T

11 olecules and is called B7-related protein-1 (B7RP-1), is expressed on B cells and macrophages.

12 ICOS), and its ligand, B7-related protein-1 (B7RP-1), were recently identified.

13 Transgenic mice expressing a B7RP-1-Fc fusion protein show lymphoid hyperplasia in th

14 ICOS and B7RP-1 define a new and distinct receptor-ligand pair th

15 ct with proteins in the CD28-B7 pathway, and B7RP-1 co-stimulates T cells in vitro independently of C

16 Treatment with an anti-B7RP-1 Ab ameliorates disease manifestations and leads t

17 Its ligand B7H/B7RP-1 is expressed on B cells and in non-immune tissues

18 R5(+) or CXCR5(-) T cells show that blocking B7RP-1 does not directly impact the production of IgG by

19 biotic or Lactobacillus casei treatment, but B7RP-1 showed increased expression on dendritic cells st

20 n of murine B7RP-1 fused with human IgG1 Fc (B7RP-1-Fc).

21 Finally, B7RP-1-Fc increased the presence of eosinophils in the b

22 In addition, ICOS-B7RP-1 costimulation was required for the development of

23 can be analyzed, to assess the role of ICOS-B7RP-1 in T cell help for B cell responses.

24 ve important implications for targeting ICOS-B7RP-1 therapeutically.

25 These findings suggest that the ICOS-B7RP-1 costimulatory pathway serves primarily to control

26 ation, survival, and migration, and the ICOS-B7RP-1 interaction could be involved in any or all of th

27 ese data demonstrate a key role for the ICOS-B7RP-1 pathway in acute and chronic rejection and highli

28 g was essential during priming, whereas ICOS-B7RP-1 regulated TH effector responses, and the up-regul

29 Furthermore, while ICOS-B7RP-1 interactions have no effect on the migration of T

30 murine ICOS, ICOS-Ig, was used to block ICOS/B7RP-1 interactions in a Th2 model of allergic airway di

31 In addition, blockade of ICOS/B7RP-1 interactions during ex vivo restimulation of lung

32 are dependent on the maintenance of the ICOS/B7RP-1 pathway.

33 ted with a tumor expressing the ICOS ligand, B7RP-1.

34 ruct with the extracellular domain of murine B7RP-1 fused with human IgG1 Fc (B7RP-1-Fc).

35 In this study, the stimulatory effects of B7RP-1 on cellular and humoral immune responses were inv

36 Further, the effects of B7RP-1-Fc were not dependent on immunization.

37 3, and IL-1beta, GM-CSF and up-regulation of B7RP-1, but low levels of Th1-associated cytokines (IL-1

38 Neither B7-DC or B7RP-1 was augmented after low-dose probiotic or Lactoba

39 8/CTLA4 family that binds a B7-like protein, B7RP-1.

40 Therefore, B7RP-1 exhibits co-stimulatory activities in vitro and i

41 When given near challenge time, B7RP-1-Fc stimulated CH more than a construct containing

42 ound that augmenting ICOS costimulation with B7RP-1-Fc increased the accumulation of T and B cells in

43 Presensitized mice treated with B7RP-1-Fc during antigen challenge show enhanced hyperse

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