ライフサイエンスコーパス: BAL fluid

1 or lung tissue, r = 0.887 and P < 0.0001 for BAL fluid).

2 sure increased numbers of macrophages in the BAL fluid.

3 Macrophages were isolated from BAL fluid.

4 uring both acute and latent infection in the BAL fluid.

5 flammatory cytokines to higher levels in the BAL fluid.

6 of albumin and lactate dehydrogenase in the BAL fluid.

7 L-33 induced IL-13-producing nuocytes in the BAL fluid.

8 constituting up to 50% of the total cells in BAL fluid.

9 f Pneumocystis colonization by nested PCR of BAL fluid.

10 trophil chemoattractant capacity seen in BOS BAL fluid.

11 manual method was more sensitive for GHE in BAL fluid.

12 correlated with the concentration of IL-5 in BAL fluid.

13 Aspergillus galactomannan antigen or DNA in BAL fluid.

14 ly with the number of neutrophils present in BAL fluid.

15 related against neutrophil concentrations in BAL fluid.

16 nuclear cells and/or > or =2% eosinophils in BAL fluid.

17 I(+) myeloid dendritic cells were present in BAL fluid.

18 induced tumor necrosis factor (TNF)-alpha in BAL fluid.

19 d transfusions, and the cell differential of BAL fluid.

20 as PGE(2) was not up-regulated in COX-2(-/-) BAL fluid.

21 lymphoid cells and basophils were scarce in BAL fluid.

22 frequency of dual-positive TH2/TH17 cells in BAL fluid.

23 in lung homogenates, and ELISA of Muc5ac in BAL fluid.

24 vated in BOS patient bronchoalveolar lavage (BAL) fluid.

25 levels of chemokines in the bronchoalveolar (BAL) fluid.

26 nts were measured in bronchoalveolar lavage (BAL) fluid.

27 atory cells found in bronchoalveolar lavage (BAL) fluid.

28 luated biomarkers in bronchoalveolar lavage (BAL) fluid.

29 hich showed greater sensitivity than qCx for BAL fluid (100% versus 67%; P = 0.04; n = 15).

30 increased levels of IL-4, IL-5, and IL-13 in BAL fluid after 11 challenges whereas IL-10 and TGF-beta

31 polyunsaturated acyl groups predominated in BAL fluid after bleomycin injury, with 22:5 and 22:6 spe

32 numbers of leukocytes and cytokine levels in BAL fluid after OVA sensitization and 7 airway challenge

33 of neutrophils and neutrophil chemokines in BAL fluid after s.i.n.

34 D metabolites were increased in concentrated BAL fluids after allergen compared to saline challenge.

35 (+) T lymphocytes in bronchoalveolar lavage (BAL) fluid after incubation with purified protein deriva

36 Cytokine (IL-5) levels in BAL fluid also increased markedly following 100 mug down

37 evidenced by neutrophilia or eosinophilia on BAL fluid analysis before CYC therapy and on repeat anal

38 Persistently abnormal results on BAL fluid analysis following CYC treatment is a common f

39 f PFTs and had abnormal findings on followup BAL fluid analysis.

40 e in sialylated glycans from eosinophils and BAL fluid analyzed by mass spectrometry.

41 ease in HIV that was detectable in acellular BAL fluid and a slower decrease in the HIV RNA and DNA l

42 ildren with STRA had significantly increased BAL fluid and biopsy specimen eosinophil counts compared

43 Airway inflammation, remodeling, and BAL fluid and biopsy specimen T(H)2 cytokines were quant

44 As a control group, BAL fluid and biopsy specimens from 22 patients with SCI

45 rofiles of antigen-specific CD4 T cells from BAL fluid and blood before and after the initiation of h

46 ge and correlated with eosinophil numbers in BAL fluid and blood.

47 the soluble form of IL-13Ralpha2 in serum or BAL fluid and did not affect IL-13-dependent signal tran

48 mined the effects of SIV infection on AMs in BAL fluid and IMs in lung tissue of rhesus macaques.

49 In cells collected from BAL fluid and in PBMCs, the concentrations of interferon

50 s well as increased levels of adiponectin in BAL fluid and increased adiponectin expression by airway

51 ompared the assay to quantitative culture of BAL fluid and lung tissue in a rabbit model of experimen

52 specific in detecting A. fumigatus DNA from BAL fluid and lung tissue in experimental IPA.

53 protocol, DNA was extracted from samples of BAL fluid and lung tissues from noninfected and A. fumig

54 Mononuclear cells from BAL fluid and peripheral blood mononuclear cells (PBMCs)

55 dioidal antigen preparation, cells from both BAL fluid and peripheral blood obtained from patients wi

56 398 and 368 dysregulated metabolites in the BAL fluid and plasma of sensitized and challenged mice w

57 We analyzed BAL fluid and serum for a total of 244 biomolecules usin

58 lecules studied, 52 and 14 biomolecules from BAL fluid and serum, respectively, were significantly di

59 y Gram-positive cocci (GPC), but cultures of BAL fluid and subcarinal lymph node biopsy tissue were n

60 ted with eosinophil trafficking-only EDN (in BAL fluid and WBC) and MIP-1alpha (in WBC) levels were h

61 , T cell responses were detected only in the BAL fluid and were lower and more restricted than those

62 These BAL fluids and exogenous polyamines were able to induce

63 Bronchoalveolar lavage (BAL) fluid and blood were collected before initiation of

64 s were determined in bronchoalveolar lavage (BAL) fluid and blood.

65 rvival in both human bronchoalveolar lavage (BAL) fluid and fetal bovine serum (FBS), (ii) survival i

66 Aspergillus spp. in bronchoalveolar lavage (BAL) fluid and in tissue vary in sensitivity and specifi

67 Bronchoalveolar lavage (BAL) fluid and lung biopsy specimens were stained with h

68 g sporangiospores in bronchoalveolar lavage (BAL) fluid and lung tissue homogenates from rabbits was

69 r viral loads in the bronchoalveolar lavage (BAL) fluid and nasal and ocular swabs in aged animals, s

70 d cellularity in the bronchoalveolar lavage (BAL) fluid and notable lung eosinophilia not seen in con

71 g acute infection in bronchoalveolar lavage (BAL) fluid and peripheral blood.

72 rgic asthma, matched bronchoalveolar lavage (BAL) fluid and plasma were collected from three groups o

73 mmatory protein-2 in bronchoalveolar lavage (BAL) fluid and pulmonary PMNL recruitment.

74 as been validated on bronchoalveolar lavage (BAL) fluid and serum specimens, but recent evidence sugg

75 mmatory cytokines in bronchoalveolar lavage (BAL) fluid and the lungs.

76 and CD4(+) cells in bronchoalveolar lavage (BAL) fluid and was associated with significantly increas

77 to the number of CFU per gram in tissue and BAL fluid, and by superior detection of therapeutic resp

78 et correlated with the number of MSCs in the BAL fluid, and myofibroblasts present in the fibrotic le

79 6, IL-23, C3a, and serum amyloid A levels in BAL fluid, and these correlated with IL-1beta and C3a le

80 anced by the concentrated supernatant of rat BAL fluid, and this effect was abolished by the selectiv

81 less protein in the bronchoalveolar lavage (BAL) fluid, and a significant reduction in the number of

82 both in vivo, in the bronchoalveolar lavage (BAL) fluid, and in vitro, following Ag stimulation of pe

83 on, and lung tissue, bronchoalveolar lavage (BAL) fluid, and plasma were investigated.

84 ng, IL1RN protein in bronchoalveolar lavage (BAL) fluid, and trafficking of lymphocytes and neutrophi

85 Higher values of CXCL10 (IP-10) in BAL fluid are associated with ACR in LTRs, suggesting a

86 ice restored levels of IL-10 and TGF-beta in BAL fluid as well as suppressed AHR and inflammation.

87 HIT1) bioactivity in bronchoalveolar lavage (BAL) fluid, as well as CCL18, YKL-40 and CHIT1 mRNA leve

88 iferation of plasmacytoid dendritic cells in BAL fluid at 3 days postinfection in RMs infected with S

89 Free neutrophil elastase activity in BAL fluid at 3 months of age was associated with persist

90 ith IPS have elevated levels of MCP-1 in the BAL fluid at the time of diagnosis.

91 vels were reduced in bronchoalveolar lavage (BAL) fluid, BAL cells, and primary alveolar macrophages

92 at baseline and study days 4 and 7 to obtain BAL fluid (BALF) for measurement of total protein, cerul

93 active TGF-beta1 in bronchoalveolar lavage (BAL) fluid (BALF).

94 ed that MV, found in bronchoalveolar lavage (BAL) fluids (BALF) of LTR at CLAD diagnosis, are elevate

95 aspirates (NPAs) and bronchoalveolar lavage (BAL) fluid before HCT.

96 T cell proliferation was higher in the BAL fluid but delayed and reduced in peripheral blood in

97 ion of IL-8 reduced migration of PMNs toward BAL fluid by approximately 50%, indicating that IL-8 is

98 Lung nitrates were measured in BAL fluid by use of the Greiss reaction, and cytokines w

99 -glucan detection in bronchoalveolar lavage (BAL) fluid by Fungitell assay aims to further improve up

100 subsets constituted secondary TH types, and BAL fluid CD8(+) T cells were almost exclusively IFN-gam

101 However, it is unknown whether the BAL fluid cell count obtained after CYC treatment of alv

102 ificantly differ from that in patients whose BAL fluid cell counts had normalized (P = 0.70 and P = 0

103 Of the 25 study patients, in only 6 did the BAL fluid cell counts normalize after CYC therapy.

104 Expression profiling of BAL fluid cells collected before and after challenge sho

105 Recognition was significantly absent from BAL fluid cells of patients with other lung diseases, in

106 cells and in reduced bronchoalveolar lavage (BAL) fluid cellularity and BAL fluid levels of tumor nec

107 /=2% eosinophilia in bronchoalveolar lavage (BAL) fluid, chronic lung allograft dysfunction (CLAD)-fr

108 detection of therapeutic response to AMB in BAL fluid compared to culture.

109 al cell counts and numbers of neutrophils in BAL fluid compared to the OVA-sensitized and ragweed-cha

110 creased IL-5 and IL-13 protein expression in BAL fluid compared to vehicle-treated mice.

111 ith IPF had double the burden of bacteria in BAL fluid compared with 44 control subjects.

112 llenge, and reduced IL-13 cytokine levels in BAL fluid compared with values seen in vehicle-treated a

113 ke morphology in the bronchoalveolar lavage (BAL) fluid, constituting up to 50% of the total cells in

114 secutive patients with ADA deficiency, 7 had BAL fluid containing periodic acid-Schiff-positive surfa

115 culture-based diagnostic methods applied to BAL fluid could facilitate accurate diagnosis and more-t

116 us antigen or DNA in bronchoalveolar lavage (BAL) fluid could facilitate earlier diagnosis, thereby g

117 and identified some specific limitations of BAL fluid culture in the diagnosis of pneumonia.

118 report the first correlation of quantitative BAL fluid culture results with culture-independent evide

119 on one of the seminal papers on quantitative BAL fluid culture, to explain why she believes that quan

120 o explain why she believes that quantitative BAL fluid cultures are the optimal strategy for VAP diag

121 -tocotrienol abated HDM-induced elevation of BAL fluid cytokine and chemokine levels, total reactive

122 exposing healthy alveolar macrophages to CF BAL fluid decreased bacterial killing, and this was reve

123 HBD2 is present in bronchoalveolar lavage (BAL) fluid during conditions associated with airway infl

124 F) levels in hamster bronchoalveolar lavage (BAL) fluid early after intranasal challenge.

125 imals showed reduced bronchoalveolar lavage (BAL) fluid eosinophil counts, reduced airway resistance

126 chemokine genes and bronchoalveolar lavage (BAL) fluid for the presence of chemokine proteins.

127 BAL fluid from 40% of patients with RA was positive for

128 Overall greater expression of cytokines in BAL fluid from African American patients as compared wit

129 cells were present at a higher frequency in BAL fluid from asthmatic patients compared with numbers

130 BAL fluid from asthmatic patients had increased TSLP but

131 BAL fluid from BMT mice at day 21 after infection contai

132 At day 5 after infection, the BAL fluid from C-EX (but not A-EX) mice had less IL-6, i

133 rominent neutrophil chemoattractant found in BAL fluid from individuals at the time of diagnosis of B

134 In vitro, BAL fluid from KGF-treated subjects inhibited pulmonary

135 d quantitative cultures in experiments using BAL fluid from neutropenic rabbits with experimentally i

136 These cells were not seen in the BAL fluid from normal animals or from Pneumocystis-infec

137 BAL fluid from patients living in a coccidioidomycosis-e

138 The sFasL in BAL fluid from patients with ALI was bioactive and prese

139 The concentration of L-ficolin in BAL fluid from patients with fungal infection was signif

140 -1-infected subjects, compared with those in BAL fluid from the HIV-1-uninfected control subjects (me

141 of the beta -chemokine RANTES were found in BAL fluid from the human immunodeficiency virus (HIV)-1-

142 ages during infection, after incubation with BAL fluids from Pneumocystis-infected animals, or after

143 rformed on acellular bronchoalveolar lavage (BAL) fluid from 30 subjects infected with HIV with advan

144 We obtained bronchoalveolar lavage (BAL) fluid from 45 Malawian adults, to measure the conce

145 ve TH2/TH17 cells in bronchoalveolar lavage (BAL) fluid from asthmatic patients, examine their respon

146 olin was measured in bronchoalveolar lavage (BAL) fluid from lung transplant recipients by enzyme-lin

147 vity is increased in bronchoalveolar lavage (BAL) fluid from patients with cystic fibrosis (CF).

148 ) E(2) levels in the bronchoalveolar lavage (BAL) fluid from wild-type and COX-1(-/-) mice were signi

149 We screened bronchoalveolar lavage (BAL) fluids from COPD-E (chronic obstructive pulmonary d

150 presence of virus in bronchoalveolar lavage (BAL) fluids from patients during the first year after su

151 Bronchoalveolar lavage (BAL) fluids from Pneumocystis-infected animals were foun

152 cific PCRs were comparable in sensitivity to BAL fluid galactomannan (GM) assay.

153 nd SLPI (R2=0.29 and P=.001) correlated with BAL fluid HIV-1 load but not with plasma HIV-1 load.

154 Culture of BAL fluid identified clinically unsuspected infection in

155 asthma had neutrophilic asthma and increased BAL fluid IL-1alpha, IL-6, IL-8, granulocyte colony-stim

156 No changes were observed in the EAR, BAL fluid IL-4 levels, or serum total and Ag-specific Ig

157 There was no increase in BAL fluid IL-4, IL-5, or IL-13 levels in patients with S

158 BAL fluid ILC2s from asthmatic patients with increased T

159 Unlike blood ILC2s, bronchoalveolar lavage (BAL) fluid ILC2s from asthmatic patients were resistant

160 Platelia EIA index cut-off > or =1.0 in the BAL fluid in a lung transplant recipient with a compatib

161 Neutrophil elastase activity in BAL fluid in early life was associated with early bronch

162 than culture in detection of A. fumigatus in BAL fluid in experimentally induced IPA (P+/-0.04).

163 l characteristics of bronchoalveolar lavage (BAL) fluid in acute pulmonary coccidioidomycosis have no

164 to culture-negative bronchoalveolar lavage (BAL) fluid in order to identify septate hyphae noted by

165 nnan (GM) testing of bronchoalveolar lavage (BAL) fluid in the diagnosis of invasive pulmonary asperg

166 IL-7 concentrations in BAL fluid increased significantly 48 h after segmental a

167 Field Effect genes were higher in cell-free BAL fluid, indicating they may be secreted by the TAMs.

168 y CD4(+) T lymphocytes in response to PPD in BAL fluid is a promising new diagnostic test for active

169 summary, we found that bacterial culture of BAL fluid is largely effective in discriminating acute i

170 cells as measured in bronchoalveolar lavage (BAL) fluid is more moderate and similar to the depletion

171 We examined bronchoalveolar lavage (BAL) fluid leukocytes, cytokines, mediators, and epithel

172 oalveolar lavage (BAL) fluid cellularity and BAL fluid levels of tumor necrosis factor-alpha (TNF-alp

173 injury increases the bronchoalveolar lavage (BAL) fluid levels of ATX protein 17-fold.

174 iated with increased bronchoalveolar lavage (BAL) fluid levels of TNF-alpha, elevated numbers of dono

175 Following extraction from BAL fluids, levels of 25-hydroxy-vitamin D (25(OH)D) and

176 F-beta derived from parenchymal cells in the BAL fluid, lower nitrite levels, and reduced apoptosis,

177 Bronchoalveolar lavage (BAL) fluid, lung tissues, and whole blood were collected

178 sponses in blood and bronchoalveolar lavage (BAL) fluid lymphocytes compared with all other adjuvants

179 nged elevation of these chemokines in serial BAL fluid measurements predicted the development of CLAD

180 L fluid neutrophils strongly correlated with BAL fluid myeloperoxidase, IL-8, IL-1alpha, IL-6, granul

181 r CYC therapy for active lung disease alters BAL fluid neutrophil and eosinophil counts and whether t

182 , bronchial basement membrane thickness, and BAL fluid neutrophil and lymphocyte counts but not eosin

183 BAL fluid neutrophil counts negatively and positively co

184 irteen of these 52 molecules correlated with BAL fluid neutrophil counts.

185 Bronchoalveolar lavage (BAL) fluid neutrophilia after O3 exposure was significan

186 BAL fluid neutrophils strongly correlated with BAL fluid

187 RVs were found in 61% (41% in BAL fluid/NPAs and 20% in NPAs only).

188 T cells derived from bronchoalveolar lavage (BAL) fluid obtained during diagnostic bronchoscopy.

189 advocate the use of bronchoalveolar lavage (BAL) fluid obtained either bronchoscopically or by the u

190 leukin-5, and tumor necrosis factor alpha in BAL fluid occurred but were attenuated in PCP-infected S

191 0.008), free neutrophil elastase activity in BAL fluid (odds ratio, 3.02; 95% CI, 1.70 to 5.35; P<0.0

192 cognition of ESAT-6 and KatG in T cells from BAL fluid of 32/44 sarcoidosis subjects, compared to 1/2

193 and release of IL-5, IL-9, and GM-CSF in the BAL fluid of mice, indicating an involvement of pro-eosi

194 observed in the peripheral blood, MLNs, and BAL fluid of SIV-infected RMs.

195 When polyamines were depleted from the BAL fluids of infected animals, the ability of these BAL

196 t SP-D levels increased at least fourfold in BAL fluids of patients with P. carinii pneumonia.

197 , these markers remained elevated within the BAL fluids of treated animals, which was consistent with

198 ) were determined in bronchoalveolar lavage (BAL) fluid of 10 asthmatics and 15 normal controls taken

199 (MLNs), jejunum, and bronchoalveolar lavage (BAL) fluid of healthy and SIV-infected rhesus macaques (

200 be isolated from the bronchoalveolar lavage (BAL) fluid of human lung transplant recipients.

201 The bronchoalveolar lavage (BAL) fluid of infected BHS also contained higher levels

202 is detectable in the bronchoalveolar lavage (BAL) fluid of patients with ALI, but the mechanisms cont

203 ting its presence in bronchoalveolar lavage (BAL) fluid of subjects with atopic asthma before and aft

204 s collected from the bronchoalveolar lavage (BAL) fluid of the animals.

205 f neutrophils in the bronchoalveolar lavage (BAL) fluid of the lungs.

206 s of lung tissue and bronchoalveolar lavage (BAL) fluids of humans with active P. carinii pneumonia.

207 Bronchoalveolar lavage (BAL) fluids of iNOS(-/-) and WT mice showed similar leve

208 creased TNF-alpha in bronchoalveolar lavage (BAL) fluid (p < .001).

209 mpared with those found in control subjects (BAL fluid, P < .001; biopsy specimen, P < .01); within t

210 BAL fluid pan-Aspergillus and species-specific PCRs were

211 Confirming previous results, BAL fluid PGD(2) levels were highest in patients with se

212 yte phenotyping was performed, and acellular BAL fluid, plasma HIV RNA load, and BAL cell and periphe

213 eases in HGF concentrations were detected in BAL fluid, plasma, and fibroblast culture medium from Ca

214 testing of serum and bronchoalveolar lavage (BAL) fluid pools from positive and negative patient spec

215 generated from neutrophil myeloperoxidase in BAL fluid promoted aggregation of sFasL in vitro and in

216 Bronchoalveolar lavage (BAL) fluid prostaglandin D(2)(PGD(2)) levels are increas

217 These results show that pre-HCT BAL fluid RV positivity was a predictor for allo-LSs.

218 ad determined with the end-organ sample, the BAL fluid sample, was higher than the corresponding vira

219 Treatment of both the crude BAL fluid samples and P. carinii lysate and the 200 mM m

220 ncanavalin A-agarose beads, the inhibitor in BAL fluid samples and P. carinii lysate could be eluted

221 Alveolar macrophages treated with these BAL fluid samples became defective in phagocytosis but r

222 We tested archived BAL fluid samples from 132 HCT recipients with CMV pneum

223 he galactomannan (GM) test were used with 32 BAL fluid samples from 32 patients at risk of IPA.

224 A total of 78 BAL fluid samples from 78 patients at risk for IPA (74 s

225 BAL fluid samples from consecutive lung transplant recip

226 Parallel blood and BAL fluid samples from each subject were incubated with

227 test, and an Aspergillus PCR assay by using BAL fluid samples from immunocompromised patients.

228 32/33) of the serum and 78.8% (26/33) of the BAL fluid samples from infected animals were in agreemen

229 ytosis but remained normal when treated with BAL fluid samples from noninfected or Toxoplasma gondii-

230 The suppressive activity of the BAL fluid samples from P. carinii-infected rats on phago

231 , CTSS activity was detectable in 100% of CF BAL fluid samples from patients without Pseudomonas aeru

232 Lipid fractions of these BAL fluid samples had no suppressive activity on phagocy

233 Also, the CMV viral load in BAL fluid samples increased along with immunoreactivity

234 The suppressive activity of these BAL fluid samples was also lost when they were incubated

235 re size of 0.45 microm but was lost when the BAL fluid samples were digested with proteases such as t

236 Calcofluor staining and culture of all BAL fluid samples were negative for fungal infection.

237 d rats on phagocytosis was retained when the BAL fluid samples were passed through a filter with a po

238 Among 23 BAL fluid samples, all were positive by qPCR, while 22 w

239 gene expression using microarray analysis of BAL fluid samples.

240 tests are useful for the diagnosis of IPA in BAL fluid samples.

241 methods for diagnosing the disease by using BAL fluid samples.

242 omannan detection in bronchoalveolar lavage (BAL) fluid samples (GM test) is currently considered the

243 tection of Pc DNA in bronchoalveolar lavage (BAL) fluid samples by use of PCR.

244 were incubated with bronchoalveolar lavage (BAL) fluid samples from P. carinii-infected rats.

245 by hybrid capture in bronchoalveolar lavage (BAL) fluid samples from patients who had undergone lung

246 nalysis of acellular bronchoalveolar lavage (BAL) fluid samples on an observational prospective cohor

247 Bronchoalveolar lavage (BAL) fluid samples were analyzed using cytokine array te

248 Serial plasma and bronchoalveolar lavage (BAL) fluid samples were examined for changes in antibody

249 eosinophilia (TPE), bronchoalveolar lavage (BAL) fluid, serum, and supernatants from pulmonary and b

250 each batch were prepared by spiking a pooled BAL fluid specimen with a known dilution of Coccidioides

251 se, 72 paired NP and bronchoalveolar lavage (BAL) fluid specimen sets, mostly from transplant recipie

252 Bacteria were identified in 44 of 46 (95.7%) BAL fluid specimens by culture-independent sequencing, s

253 In 19 sets (26.4%), the NP and BAL fluid specimens were both positive for an RV; in 3 s

254 In a retrospective case series, 202 BAL fluid specimens were collected at the Johns Hopkins

255 .2%), the NP specimens were positive but the BAL fluid specimens were negative; and in 3 other sets,

256 sets, the NP specimens were negative but the BAL fluid specimens were positive.

257 In BAL fluid specimens with no culture growth, the amount o

258 iratory pathogens in bronchoalveolar lavage (BAL) fluid specimens from subjects with suspected respir

259 clinically obtained bronchoalveolar lavage (BAL) fluid specimens from symptomatic and asymptomatic l

260 d a blinded study of bronchoalveolar lavage (BAL) fluid specimens from well-characterized models of I

261 sites, including bronchial alveolar lavage (BAL) fluid; sputum and lung tissue samples; and pleural

262 elated with the percentage of neutrophils in BAL fluid, suggesting that low SP-A levels were associat

263 Levels of neutrophil chemokines in BAL fluid supernatants were markedly elevated in the sen

264 = 15) of children with STRA with detectable BAL fluid T(H)2 cytokines had significantly lower lung f

265 r lung function than those with undetectable BAL fluid T(H)2 cytokines.

266 On the basis of the dominance of BAL fluid TH2 or TH2/TH17 cells, we identified 3 subgrou

267 esponse, with EDN levels being higher in the BAL fluid than in the serum (P < 0.02).

268 ds of infected animals, the ability of these BAL fluids to induce apoptosis was lost.

269 Increased BAL fluid total protein after O3 exposure was lowered by

270 ralizing activity in vitro and inhibition of BAL fluid total, eosinophil, and neutrophil counts in HD

271 BAL fluid TSLP levels correlated (r = 0.74) with steroid

272 nses to mycobacterial antigens in cells from BAL fluid used for sarcoidosis diagnosis suggests a stro

273 Memory CCR5(+) TH1 cells were enriched in BAL fluid versus blood, and pathogenic respiratory virus

274 ifungal therapy to the time of collection of BAL fluid was 6 days.

275 RV positivity in BAL fluid was a predictor for allo-LSs (hazard ratio, 3.

276 earch for bacteria, mycobacteia and fungi in BAL fluid was negative.

277 Eight hours after the s.i.n., BAL fluid was obtained.

278 Fiberoptic bronchoscopy with collection of BAL fluid was performed on seven patients with GC-sensit

279 inducing activity of the polyamine-depleted BAL fluids was restored when polyamines were added back.

280 Bronchoalveolar lavage (BAL) fluid was assessed for total and differential cell

281 A Gram stain of bronchoalveolar lavage (BAL) fluid was notable for many Gram-positive cocci (GPC

282 ARP results, and all microbiologic data from BAL fluid were collected.

283 /antiproteinase, and endotoxin levels in the BAL fluid were compared.

284 Epithelial cells and BAL fluid were evaluated for HPGDS (quantitative real-ti

285 cells was elevated and inflammatory cells in BAL fluid were increased.

286 -Aspergillus and species-specific qPCRs with BAL fluid were more sensitive than culture for detection

287 The fungal burdens in BAL fluid were more variable and not indicative of a pro

288 phils and levels of neutrophil chemokines in BAL fluid were significantly lower.

289 rplasia, and IL-4, IL-5, and IL-13 levels in BAL fluid were significantly reduced in fB-/- mice compa

290 t survival rates in yeast media, saline, and BAL fluid were similar to those of the wild type; howeve

291 BAL fluids were cultured for bacteria, mycobacteria, and

292 hough the absolute numbers of eosinophils in BAL fluids were similar between the strains, very few CD

293 culation), blood and bronchoalveolar lavage (BAL) fluid were collected from infected and uninfected a

294 kocytes in blood and bronchoalveolar lavage (BAL) fluid were compared for expression of FoxP3 by flow

295 factor alpha in the bronchoalveolar lavage (BAL) fluid were lower in C-EX and A-EX than in NON-EX mi

296 pearance of proinflammatory cytokines in the BAL fluid, whereas the inflammatory and cytokine respons

297 100-fold increase in protein content in lung BAL fluid, which included the CXC chemokines cytokine-in

298 evels of transforming growth factor beta1 in BAL fluid, while simultaneously reducing the number of i

299 and R. oryzae GHE in bronchoalveolar lavage (BAL) fluid with either extraction method was 1 GHE/ml.

300 A. niger, culture and PCR amplification from BAL fluid yielded similar sensitivities for untreated an