ライフサイエンスコーパス: BCA

1 BCA attenuated stroke lesion volume as measured by 9.4T

2 BCA increased luciferase activity in cells that were tra

3 BCA levels were significantly increased in plasma and br

4 BCA removes Cu(I) from Atox1 in a three-step process inv

5 BCA significantly increased GOT mRNA and protein express

6 BCA was defined as elevated levels of insulin autoantibo

7 BCA-1 (CXCL13) was completely absent in control biopsy s

8 BCA-1 (CXCL13) was expressed within lymphoid aggregates

9 BCA-1/CXCL13, which mediates homing to B cell zones, eli

10 B cell-attracting chemokine 1 (BCA-1) responses correlate with CXC chemokine receptor (

11 expression of B cell-attracting chemokine 1 (BCA-1, CXCL13), a lymphoid chemokine involved in B-cell

12 for elevated B-cell-attracting chemokine 1 (BCA-1; fourth quartile vs first: odds ratio [OR], 2.7; 9

13 ysis of the model showed that the rates of a BCA colonising diseased and/or healthy plant tissues and

14 l of phytoestrogen isoflavones, biochanin A (BCA) was identified as the most potent inducer of GOT ge

15 the 26 competing oligomers (i.e., AAA, ABB, BCA).

16 Balanced chromosomal abnormalities (BCAs) represent a relatively untapped reservoir of singl

17 Vs), but balanced chromosomal abnormalities (BCAs) still require karyotyping for clinical detection.

18 cance of balanced chromosomal abnormalities (BCAs), their characterization has largely been restricte

19 : Nafion, base-hydrolyzed cellulose acetate (BCA), and fibronectin.

20 y using the Cu(I) chelator bicinchonic acid (BCA) as a metal acceptor.

21 fication to the standard bicinchoninic acid (BCA) assay protocol allows for an accurate, rapid, and e

22 d using Western blot and bicinchoninic acid (BCA) assay.

23 s were determined by the Bicinchoninic acid (BCA) method, Ara h 1 and Ara h 2 were evaluated by SDS-P

24 hich is analogous to the bicinchoninic acid (BCA) protein assay widely used in proteomics, will be ap

25 ma levels of the branched chain amino acids (BCAs) leucine/isoleucine and their deaminated metabolite

26 ssociation to form free catalytically active BCA along with CB[7]*1 and CB[7]*2.

27 lective method for boron conjugate addition (BCA) to alpha,beta-unsaturated carbonyls is presented.

28 enantioselective boryl conjugate additions (BCAs) that generate products containing boron-substitute

29 ics of foliar pathogen and biocontrol agent (BCA) populations in order to predict the likelihood of s

30 of developing bifunctional chelating agents (BCAs), this new synthetic strategy offers the possibilit

31 Using biological control agents (BCAs) is an essential component of integrated pest and d

32 is a gold NP-based biobarcode amplification (BCA) assay, which can detect HIV-1 p24 antigen at levels

33 rategy using DNA-bio-bar-code amplification (BCA) and Nb.BbvCI nicking enzyme-assisted strand cycle f

34 roscopy and biomolecular component analysis (BCA) for a comparative analysis of the molecular composi

35 ng gene data, Bayesian Concordance Analysis (BCA), as implemented in the BUCKy software, in which the

36 Addition of CB[7] to BCA*1 and BCA*2 results in the transient formation of the BCA*1*CB

37 e transient formation of the BCA*1*CB[7] and BCA*2*CB[7] ternary complexes that undergo rapid dissoci

38 Both BCA and BCA-Ac(18) are catalytically active, and circular dichro

39 da, also had no detectable levels of GCA and BCA.

40 ed the effects of bovine carbonic anhydrase (BCA) and CO2-rich gas streams on the carbonation rate of

41 two polypeptides--bovine carbonic anhydrase (BCA) and peracetylated BCA (BCA-Ac(18))--having the same

42 nity of unlabeled bovine carbonic anhydrase (BCA) for a variety of ligands (most of which are benzene

43 se) A, myoglobin, bovine carbonic anhydrase (BCA) II, hemoglobin (Hb), and the hemoglobin-haptoglobin

44 e active sites of bovine carbonic anhydrase (BCA) or acetylcholinesterase (AChE) and inhibit their ca

45 h carbon cloth anode (CCA) or biochar anode (BCA) were inserted into raw water saturated soils contai

46 ndothelium-removed bovine coronary arteries (BCAs) precontracted with 30 mmol/L KCl to the NO donor S

47 , lesion area at the brachiocephalic artery (BCA) was reduced dramatically by approximately 85% in fr

48 Moreover, detection at BCA-coated CFMs can be tuned by altering hydrolysis time

49 bonic anhydrase (BCA) and peracetylated BCA (BCA-Ac(18))--having the same sequence of amino acids but

50 Both BCA and BCA-Ac(18) are catalytically active, and circula

51 GOx) and urease, measurements of proteins by BCA assay, analysis of pH, and concentration of antioxid

52 Remarkably, Cu(I) uptake from solution by BCA is much slower than the transfer from holo-Atox1, pr

53 the principal features of the NHC-catalyzed BCA and SCA processes are detailed.

54 he expression of the constitutive chemokines BCA-1 (CXCL13) and SDF-1 (CXCL12) in salivary glands fro

55 Our findings suggest that circulating BCA-1, sTNFR2, and sVEGFR2 are associated with NHL risk

56 of a second BCA to generate apo-Atox1 and Cu-BCA(2).

57 bimolecular formation of an initial Atox1-Cu-BCA complex followed by dissociation of Atox1 and the bi

58 isplacement of the protein from the Atox1-Cu-BCA intermediate by the second BCA.

59 ollicular B cell-attracting chemokine CXCL13/BCA-1, suggesting a novel follicular B-helper-like T cel

60 on vetch), which contains beta-cyanoalanine (BCA) and gamma-glutamyl beta-cyanoalanine (gammaGBCA) is

61 SE and CSE inhibited with beta-cyanoalanine (BCA) in both isolated CSE enzymes (p < 0.005) and in C6

62 tic acid (AOAA, 100 mum), beta-cyanoalanine (BCA, 500 mum) and hydroxylamine (HA, 100 mum), altered t

63 kers found in common vetch: ss-cyanoalanine (BCA) and gamma-glutamyl-ss-cyanoalanine (GCA).

64 Biliary cystadenoma (BCA) and biliary cystadenocarcinoma (BCAC) are rare tumo

65 The DNA-BCA system contains a locked nucleic acid (LNA) modified

66 r mass, suggesting that vascular endothelial BCA-1 expression may be consequent to transcytosis.

67 The recoveriess were determined as 95.8% for BCA and 97.4% for gammaGBCA.

68 ned associated with NHL after adjustment for BCA-1, sTNFR2, and sVEGFR2.

69 specimens containing PCNSL were positive for BCA-1.

70 sitively for CXCR5, the primary receptor for BCA-1.

71 ilies of persons with IDDM were screened for BCA.

72 e effects, non-fungal BCA and strategies for BCA application, although with a cost in model tractabil

73 vs SCE for (PCA)Co(2) and 0.39 V vs SCE for (BCA)Co(2).

74 constants of several unlabeled ligands from BCA, using plots of levitation height versus time.

75 heterogeneity, fungicide effects, non-fungal BCA and strategies for BCA application, although with a

76 nd, although it is most applicable to fungal BCA systems, does not address a specific biocontrol syst

77 features that are remarkably similar to GCs, BCA-1 (CXCL13) and SDF-1 (CXCL12) may contribute to the

78 ysine-epsilon-NHCOCH(3) groups and generates BCA-Ac(18).

79 %) recurrences; 2 patients who initially had BCA recurred with BCAC.

80 If BCAs are treated with DPI under 21% O(2) and then expose

81 inhibitors to bovine carbonic anhydrase II (BCA II), the observed thermodynamic binding signature de

82 n complexes of bovine carbonic anhydrase II (BCA) and para-substituted benzenesulfonamide ligands wit

83 association of bovine carbonic anhydrase II (BCA) and para-substituted benzenesulfonamides with chain

84 NH3+ groups of bovine carbonic anhydrase II (BCA).

85 etalloprotein--bovine carbonic anhydrase II (BCA, EC 4.2.1.1)--on unfolding upon treatment with sodiu

86 to incubation for 30 min at 37 degrees C in BCA standard working reagent.

87 Data demonstrates that carbonation in BCA-amended reactors remained limited by CO2 supply, imp

88 gth-dependent manner, causing an increase in BCA mobility.

89 Activation of sGC by SNAP in BCA homogenate was also eliminated when vessels were pre

90 were not greatly affected by the introduced BCA.

91 lyses of beta-cyanoalanine and (13)C labeled BCA is used as an internal standard in gammaGBCA analysi

92 munostaining on selected specimens localized BCA-1 to malignant B lymphocytes and vascular endotheliu

93 BCA increased, HA inhibited) and/or LY83583 (BCA increased, AOAA and HA inhibited).

94 sGC and inhibiting NO-elicited cGMP-mediated BCA relaxation.

95 n samples and compared with the Pierce Micro BCA Protein Assay Kit.

96 nitude more rapidly than the acetyl-modified BCA.

97 bic interactions cause the hexanoyl-modified BCA to denature nearly three orders of magnitude more ra

98 Acetylation of BCA with acetic anhydride converts all 18 lysine-epsilon

99 n-off switching of the catalytic activity of BCA is delineated by the combined inference of UV/vis ca

100 nd increases the hydrophobic surface area of BCA.

101 tive site was probed by observing binding of BCA to a charged arylsulfonamide using affinity capillar

102 Eighteen cases of BCA were detected; 153 unrelated autoantibody-negative c

103 Therefore the determination of BCA and gammaGBCA analytes in accurate and reliable mann

104 After excision of BCA, long-term outcomes were good; however, patients wit

105 The ectopic expression of BCA-1 (CXCL13) on endothelial cells and within GC-like s

106 In PCNSL, expression of BCA-1 by malignant lymphocytes and vascular endothelium

107 itu hybridization verified the expression of BCA-1 mRNA by malignant B cells, but not vascular endoth

108 ed that the number of exchanged hydrogens of BCA was higher when the protein was bound to a ligand wi

109 he stability of a rung of a charge ladder of BCA to denaturation with SDS.

110 High levels of BCA-1 (CXCL13) were also found on endothelial cells in s

111 If another small molecule ligand of BCA is dissolved in the solution, the rate of protein ef

112 r the differences in selectivity profiles of BCA and SCA processes.

113 patients who underwent surgical resection of BCA or BCAC were identified.

114 glycine chain destabilizes the structure of BCA in a length-dependent manner, causing an increase in

115 idea that charged residues on the surface of BCA do not guide protein folding and raises the broader

116 interact hydrophobically with the surface of BCA.

117 ty that was cryptic to karyotyping in 21% of BCAs, highlighting the limitations of conventional cytog

118 At least 33.9% of BCAs resulted in gene disruption that likely contributed

119 We explored the landscape of BCAs at nucleotide resolution in 273 subjects with a spe

120 cortisone produced an elevation in levels of BCAs and their metabolites which were lowered by insulin

121 The direct modulation of BCAs by glucocorticoids and insulin may provide the basi

122 d by SNAP were eliminated by pretreatment of BCAs with DPI under 21% O(2) but not under N(2).

123 These results demonstrate that the HIV-1 p24 BCA assay offers 100-150-fold enhancement in the detecti

124 e carbonic anhydrase (BCA) and peracetylated BCA (BCA-Ac(18))--having the same sequence of amino acid

125 (HA, 100 mum), altered the NPV to PGF2alpha (BCA increased, HA inhibited) and/or LY83583 (BCA increas

126 hest dose of hydrocortisone increased plasma BCAs in both insulin groups but increased free fatty aci

127 hydrated CO2 species, apparently preventing BCA from catalyzing the dehydration reaction.

128 oryl and silyl conjugate addition reactions (BCA and SCA, respectively), which proceed without the ne

129 Remarkably, BCA breakpoints in eight subjects altered a single TAD e

130 ciation of Atox1 and the binding of a second BCA to generate apo-Atox1 and Cu-BCA(2).

131 the Atox1-Cu-BCA intermediate by the second BCA.

132 We sequenced BCAs in patients with autism or related NDDs, revealing

133 Moreover, submicroscopic BCAs and subarray threshold CNVs are intractable, or cry

134 nt the first experimental demonstration that BCA is not the rigid, static globulin that has been typi

135 We find that BCA is relatively fouling-resistant.

136 The BCA assay is a colorimetric method for estimating protei

137 the aortic root (P < 0.01) and by 50% at the BCA (P < 0.05) in fractalkine-deficient females at 16 we

138 ctalkine-deficient mice appears to be at the BCA rather than the aortic root.

139 thus, their fluorescence is quenched by the BCA chromophore (lambda(max) 562 nm).

140 Furthermore, the BCA assay detected HIV-1 infection 3 days earlier than d

141 uses europium NPs to replace gold NPs in the BCA assay to further simplify the detection method and d

142 ally unfolded intermediate stabilized in the BCA II folding/unfolding reaction, and it could be used

143 The reduction in the BCA population size was relatively small within 8 days;

144 *2 results in the transient formation of the BCA*1*CB[7] and BCA*2*CB[7] ternary complexes that under

145 ydrogens with increasing chain length of the BCA-bound ligand, across the series.

146 o overcome this limitation, we performed the BCA assays in opaque, white 384-well plates.

147 healthy plant tissues and the time that the BCA remains active are two of the more important factors

148 cy, it is necessary to frequently apply this BCA with the application interval depending on the growt

149 ccessfully adapted to the synthesis of three BCAs with great interest especially for biomedical appli

150 Addition of CB[7] to BCA*1 and BCA*2 results in the transient formation of th

151 rity of the immobilized ligands are bound to BCA.

152 ully detected the binding of brinzolamide to BCA II, a non-two-state folding protein.

153 l zone and peritoneal B1 B cells, respond to BCA-1, implying that responsiveness to this follicular c

154 Taken together, BCA represents a natural phytoestrogen that mitigates st

155 tal fate of Bacillus subtilis, a widely used BCA, focusing on its dispersal aspect in open field and

156 rison to controls, brucite carbonation using BCA was accelerated by up to 240%.

157 on for future physical-organic studies using BCA as a model to examine interactions between proteins

158 factors did not reliably predict BCAC versus BCA (sensitivity, 81%; specificity, 21%).

159 rm outcomes were associated with BCAC versus BCA, as well as the presence of spindle cell/ovarian str

160 On pathology most lesions were BCA (89.1%), whereas 27 (10.9%) were BCAC.

161 Whereas BCA-1 was not detected in normal human brain, all 24 bra

162 Alkaline brucite slurries were amended with BCA and supplied with 10% CO2 gas while aqueous chemistr

163 other dietary protein is not associated with BCA.

164 Children with BCA were breast-fed for a slightly longer duration than

165 The beads are incubated with BCA and allowed to reach an equilibrium state in which t

166 /6 mice were intraperitoneally injected with BCA (5 and 10 mg/kg) for 4 wk and subjected to ischemic

167 the inhibitory effects of DPI compared with BCAs that were not treated with dithionite.