ライフサイエンスコーパス: BFB

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1 BFB can be observed in progress using cytogenetic techni

2 as microarrays or sequencing collected after BFB has ceased.

3 zed cytogenetically may be more complex, and BFB cycles may play an important role in generating term

4 en the complexity of some cancer genomes and BFB's ability to generate a wide range of rearrangement

5 for whole chromosome loss and the associated BFB-mediated instability in tumorigenesis and may shed l

6 Syncope in patients with bifascicular block (BFB) is a common event whose causes might be difficult t

7 Breakage-fusion-bridge (BFB) cycle is a series of chromosome breaks and duplicat

8 notably seen in the breakage-fusion-bridge (BFB) cycle.

9 ication involves the breakage-fusion-bridge (BFB) cycle.

10 Chromosome breakage and break-fusion-bridge (BFB) cycles can also be observed during early embryo sac

11 that have undergone breakage-fusion-bridge (BFB) cycles leading to gene amplification.

12 ndergoes a series of breakage-fusion-bridge (BFB) cycles.

13 Breakage-fusion-bridge (BFB) is a mechanism of genomic instability characterized

14 t to arise through a breakage-fusion-bridge (BFB) mechanism.

15 indicating a series of breaks and fusions by BFB cycles.

16 fy a chromosomal region likely rearranged by BFB cycles, demonstrating the practicality of our approa

17 rsions are sufficient evidence for detecting BFB.

18 lts elucidate nucleotide-level events during BFB cycles and end processing for naturally occurring mi

19 ms to aid the interpretation of evidence for BFB.

20 th fold-back inversions to develop tests for BFB.

21 biosensors in slices of the basal forebrain (BFB) to study both depolarization-evoked adenosine relea

22 using cytogenetic techniques, but generally BFB must be inferred from data such as microarrays or se

23 nd of a broken chromosome) further indicated BFB cycles as underlying processes.

24 Breakpoints initiating BFB cycles were determined from recent array data from 1

25 To elucidate the molecular basis of BFB-mediated DNA amplification, we cloned 1q31 fragile s

26 inverted repeat structure characteristic of BFB; instead PDE9A was fused to intergenic regions of ch

27 tic tumors and confirm a previous finding of BFB as well as identify a chromosomal region likely rear

28 A critical step of BFB cycles leading to gene amplification is a palindromi

29 he germ line and passes through at least one BFB cycle to produce gametes with terminal deletions ass

30 Evoked and basal adenosine release in the BFB in vitro exhibited three key features: the magnitude

31 ristics of adenosine release recorded in the BFB in vitro reflect those that have been linked in vivo

32 g, preceded or accompanied initiation of the BFB cycle.

33 We first pose the BFB count-vector problem: given a chromosome segmentatio

34 ted with mdr1 gene amplification through the BFB mechanism.

35 ion and segment copy numbers, decide whether BFB can yield a chromosome with the given segment counts

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