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1                                              BLS results from mutations in genes that regulate the ex
2 lity of CPR performance were evaluated in 20 BLS-certified rescuers randomized to perform CPR for 5 m
3 tectable even in peripheral blood cells of a BLS patient within a few days.
4 ystem to restore normal immune function to a BLS patient for which no other therapeutic option curren
5 on that what most influences survival in any BLS effort is circulation, not ventilation.
6 milarity of the chemistry catalyzed by AS-B, BLS, and CPS, our work highlights the difficulty of pred
7 roke, showed no survival differences between BLS and ALS for respiratory failure, and showed better s
8  the standard European Resuscitation Council BLS course for laypersons.
9               Neurologic functioning favored BLS for all diagnoses.
10 ible for the phenotype of the putative fifth BLS complementation group.
11                            Three of the four BLS genetic complementation groups have defects in the a
12                  Telephone dispatcher-guided BLS cardiopulmonary resuscitation (CPR) has likewise sho
13 efect from a complementation group A type II BLS patient is a 24 aa deletion in the MHC class II tran
14                    Similar to the 2010 ILCOR BLS treatment recommendations, the importance of high qu
15 suring high-quality CPR with other important BLS interventions, such as ventilation and defibrillatio
16 ular mechanism of the stability of DQ0602 in BLS-1.
17 02/DQB1*0604 were generated and expressed in BLS-1.
18  to date on the most important international BLS interventions, diagnostics, and prognostic factors f
19 s in the bare lymphocyte syndrome cell line, BLS-1, revealed that HLA-DQA1*0102/DQB1*0602 is SDS stab
20 DS stable in the HLA-DM-deficient cell line, BLS-1.
21             Of 2592 patients (47.1%) who met BLS criteria for termination of resuscitation efforts, o
22        Taking into account the advantages of BLS and the potential capacity of the B subunit of Stx2
23 teins from complementation groups B and D of BLS, respectively, cannot support this interaction.
24 eview continued to focus on the interface of BLS sequencing and ensuring high-quality CPR with other
25 asizes that the critical lifesaving steps of BLS are (1) prevention, (2) immediate recognition and ac
26 ance of these determinants on the surface of BLS cells.
27 he B subunit of Stx2 at the amino termini of BLS.
28 arches were significantly lower than that of BLS (p<0.05), while significant decreases in the setback
29 Bronze" CPR, in which chest compression-only BLS is taught, was compared with the standard European R
30            RFX-B complements the predominant BLS complementation group (group B) and was found to be
31 d in our in-situ Brillouin light scattering (BLS) experiments.
32  high-resolution Brillouin light scattering (BLS).
33    Based on this reasoning, a new simplified BLS method has been proposed.
34 enzyme lumazine synthase from Brucella spp. (BLS) is a highly stable dimer of pentamers and a scaffol
35 when compared with telephone-guided standard BLS CPR.
36 tarch assay kit) of raw brown lentil starch (BLS) increased significantly by the additions of lipids/
37 h chest compression-only basic life support (BLS) efforts.
38  superiority of ALS over basic life support (BLS) is limited, but some studies suggest ALS may harm p
39                          Basic life support (BLS) protocols have been simplified.
40 wenty female pigs and 20 Basic Life Support (BLS)-certified rescuers.
41       AIDS and the bare lymphocyte syndrome (BLS) are severe combined immunodeficiencies.
42                    Bare lymphocyte syndrome (BLS) is an autosomal recessive severe-combined immunodef
43                The bare lymphocyte syndrome (BLS) is characterized by the absence of MHC class II tra
44 deficiency type II bare lymphocyte syndrome (BLS) lacks class II MHC gene transcription.
45             In the bare lymphocyte syndrome (BLS), which is a human severe combined immunodeficiency,
46 mmunodeficiency or bare lymphocyte syndrome (BLS).
47  cognate residues in beta-lactam synthetase (BLS) and carbapenem synthetase (CPS) (Tyr-348 and Tyr-34
48 y for several acute medical emergencies than BLS.
49                                          The BLS rule had a specificity of 0.987 (95% confidence inte
50                In this validation study, the BLS and ALS termination-of-resuscitation rules performed
51 s found to be mutant in cell lines from this BLS group.
52 ly teach laypersons a simplified approach to BLS, which requires only chest compressions and not mout
53 oke, and respiratory failure was higher with BLS than ALS (6.1 percentage points [95% CI, 5.4 to 6.8
54 nts showed superior unadjusted outcomes with BLS despite being older and having more comorbidities.
55 owed better survival at all time points with BLS than ALS for patients with AMI.

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