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1 BMR cells proliferated actively for 7-20 population doub
2 BMR increased gradually throughout pregnancy at a mean (
3 BMR is distinguished by its adaptations to life undergro
4 BMR, SMR, 24-h EE, respiratory quotient, heart rate, and
6 t evidence exists to support higher absolute BMR/RMR and TDEE in pubertal than in prepubertal adolesc
7 dies supported significantly higher absolute BMR/RMR during puberty (SMD: 1.10-5.93), and all of the
11 etween circulating leptin concentrations and BMR occurred only because of inadequate control for the
12 .3%) in antagonistically selected lines, and BMR was slightly, but not significantly, lower (4.2%).
13 tive genetic covariance between VO(2)max and BMR was positive and statistically different from zero.
15 2%) in lines selected for increased MMR, and BMR was slightly, but not significantly, higher (2.5%).
16 brain size to maternal investment times and BMR, we provide a direct quantitative comparison of brai
18 the following: (a) complex formation between BMR and NADPH; (b) reduction of FAD with formation of th
20 f weight gain were not greater in the bottom BMR group (0.3 +/- 1.0 kg/y) than in the top BMR group (
21 defined as energy intake <130% of calculated BMR or WW intake <40 g . d(-1) was seen in 71% and 82% o
26 M(b) exceeded the slope for the equation for BMR vs. M(b), whereas slopes for the equations of PMR(C)
27 els, narrow-sense heritability (h(2)(N)) for BMR was <0.1, but estimates of h(2)(N) for VO(2)max were
29 t gain more weight than did adults with high BMRs, implying that habitual differences in food intake
30 efinition, the BMR was different in the high-BMR group (2001 +/- 317 kcal/d; n = 86) than in the low-
33 6 mo without experiencing a large decline in BMR, BMD, or select components of energy expenditure.
36 and thermodynamic control of the flavins in BMR is significantly different from that in microsomal P
37 ate gestational weight gain and increases in BMR, which are not totally offset by reductions in AEE.
39 lyses support our predictions that shifts in BMR and thermal conductance confer important adaptations
42 ake or activity counterbalance variations in BMR as a risk factor for weight gain in a typical Wester
46 2001 +/- 317 kcal/d; n = 86) than in the low-BMR group (1510 +/- 222 kcal/d; n = 77), but they were c
47 s breeding in temperate habitats had a lower BMR than did temperate residents, suggesting that these
49 aily energy need was determined by measuring BMR and assessing physical activity with 8-d self-report
51 irements were energy absorption of >/=81% of BMR and WW absorption of >/=21 g . d(-1), which were equ
53 c independent contrasts, and a comparison of BMR of 13 phylogenetically matched pairs, one species fr
56 r this resistance, we examined the growth of BMR fibroblasts in vitro of the species Spalax judaei an
57 dition of 2 electron equivalents per mole of BMR results in the reduction of the high potential flavi
61 ur results suggest that cancer resistance of BMR is conferred by massive necrotic response to overpro
63 ironments and will enable the utilization of BMR models for biomedical research in the fight against
69 ic correlation between basal metabolic rate (BMR) and maximal (VO(2)max) rate of oxygen consumption i
72 investment traits and basal metabolic rate (BMR) correlate with relative brain size, but current hyp
73 y composition, and the basal metabolic rate (BMR) in obese female patients during the initial 3 y aft
74 onlactating (NPNL), 2) basal metabolic rate (BMR) increases in pregnancy and the increase is positive
75 e indicated that a low basal metabolic rate (BMR) is an independent predictor of future weight gain,
78 r diet-only program on basal metabolic rate (BMR), bone mineral density (BMD), energy expended during
80 rgy expenditure (TEE), basal metabolic rate (BMR), sleeping metabolic rate (SMR), and minimal SMR (MS
84 ed data for basal or resting metabolic rate (BMR/RMR), total daily energy expenditure (TDEE), and/or
86 ych showed that tropical birds had a reduced BMR, compelling evidence for a connection between the li
88 (E) were positively correlated with residual BMR, whereas residual PMR(C) and residual BMR were not c
109 e capacity also increased with NPP (and with BMR and food intake), because species of high-NPP enviro
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