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1 BMSC CM contained a heat-labile factor that increased BM
2 BMSC co-transplantation doubles the number of functional
3 BMSCs from six of the eight centers were tested for thei
4 BMSCs inoculated alone induced osteoblast suppression, a
5 BMSCs isolated from femur and tibia of Sprague-Dawley ra
6 BMSCs may reduce pathogen burden by inhibiting growth th
7 BMSCs secreted TGF-beta1 into the cerebrospinal fluid, a
8 BMSCs that migrated from the injection site survived at
9 antly lower levels in cultures of Bag-1(+/-) BMSCs supplemented with BMP-2, while genes with roles in
10 vel genetically engineered Tet-Off-SDF-1beta BMSCs, which over-express SDF-1beta under tight doxycycl
12 microRNA-9 (miR-9) modified BMSCs (pri-miR-9-BMSCs) can significantly reduce the pancreatic edema, in
17 y, our results show novel roles of BMSCs and BMSC-derived Dkk1 in the pathogenesis of multiple myelom
20 ses abnormalities in osteoblast function and BMSC differentiation and identify a previously unrecogni
21 ively interferes with CLL cell migration and BMSC-mediated drug resistance, and establishes a rationa
23 liferation rate of both commercial AMSCs and BMSCs as compared to primary culture AMSCs, suggesting p
24 osteoblastogenesis of MC3T3-E1 cultures and BMSCs induced towards the bone lineage by multi-layered
25 d demonstrates the requirement for NOTCH and BMSCs in fracture repair, irrespective of fracture stabi
26 the quantity formed was highly variable and BMSCs from only three centers supported hematopoiesis.
27 oreover, increased chondrogenesis of ank/ank BMSCs and increased chondrogenic transdifferentiation an
32 ritical role mediating the crosstalk between BMSCs and T(reg) in the bone marrow microenvironment.
36 osis factor alpha and M-CSF was increased by BMSCs cultured on both micro- and nanoscale titanium top
40 at long-lasting antihyperalgesia produced by BMSCs required their chemotactic factors such as CCL4 an
41 d a preconditioned group with PVE and CD133+ BMSC cotreatment (PVE+SC group, n = 11) and a group pret
43 lysis suggest that PVE, together with CD133+ BMSC pretreatment, could positively impact overall outco
44 shown that portal venous infusion of CD133+ BMSCs substantially increases hepatic proliferation, whe
45 demonstrated that bone marrow stromal cell (BMSC) and CB-BF pellet cultures make cartilage in vitro
48 To identify human bone marrow stromal cell (BMSC) subsets with enhanced ability to engraft/contribut
49 s (MSC) derived from bone marrow stem cells (BMSC) and adipose tissue stem cells (ASC) of humans and
50 c potential of human bone marrow stem cells (BMSC) with stem cells derived from human dental pulp (DP
51 dies suggest that bone marrow stromal cells (BMSC) can promote myeloma growth and survival and osteol
52 ells with patient bone marrow stromal cells (BMSC) showed similar beta1 integrin-specific enhancement
53 which stimulates bone marrow stromal cells (BMSC) to AML blast transfer of mitochondria through AML-
55 bone, while bone marrow stromal/stem cells (BMSCs) and other skeletal progenitors may also contribut
58 Bone marrow-derived mesenchymal stem cells (BMSCs) have the potential of repairing SAP, but the deta
59 bone marrow-derived mesenchymal stem cells (BMSCs) increases the production of T(reg) cells via a me
60 ntiation of cultured bone marrow stem cells (BMSCs) into the adipocyte lineage was suppressed by 17-b
61 d senescence of bone mesenchymal stem cells (BMSCs) isolated from a TDO patient, providing a molecula
62 of local bone marrow mesenchymal stem cells (BMSCs) on osteoarthritis (OA) of the temporomandibular j
64 s study, rat bone marrow stromal stem cells (BMSCs) were tracked after IV administration to rats with
66 d we now find that bone marrow stroma cells (BMSCs) are severely and permanently damaged by the pre-c
67 of primary mouse bone marrow stromal cells (BMSCs) and 3T3-L1 pre-adipocytes via interaction with Pp
69 eloma adhesion to bone marrow stromal cells (BMSCs) and enhancing myeloma colony formation in semisol
71 clear that human bone marrow stromal cells (BMSCs) can be immunosuppressive and escape cytotoxic lym
72 e metabolomics of bone marrow stromal cells (BMSCs) derived from hyperglycaemic (type 2 diabetes mell
74 neath a confluent layer of BM stromal cells (BMSCs) due to interference with the CXCL12 gradient esta
75 imary FBLP-1 null bone marrow stromal cells (BMSCs) exhibited significantly reduced extracellular mat
76 .t.) injection of bone marrow stromal cells (BMSCs) following lumbar puncture alleviates early- and l
77 undifferentiated bone marrow stromal cells (BMSCs) from KO mice express higher levels of PPAR-gamma
78 rrow-derived mesenchymal stem/stromal cells (BMSCs) hold great potential for cell-based therapy, yet
79 -1(+) multipotent bone marrow stromal cells (BMSCs) in mice, as well as on well-characterized, clinic
81 implant-adherent bone marrow stromal cells (BMSCs) in osteoclastogenesis is influenced by surface to
84 and modulate CLL bone marrow stromal cells (BMSCs) known to provide a "homing and nurturing" environ
85 ifferentiation of bone marrow stromal cells (BMSCs) of Bag-1(+/-) (heterozygous) female mice was decr
86 hypothesized that bone marrow stromal cells (BMSCs) participate in this process, and that osterix (Os
88 odel in which the bone marrow stromal cells (BMSCs) remained unresponsive to OB differentiation signa
89 e, but can induce bone marrow stromal cells (BMSCs) to form bone in a mouse muscle pouch model, exhib
90 ne marrow derived mesenchymal stromal cells (BMSCs) were used to CBTs for ConA-induced ALF and Fah-de
91 ulations of mouse bone marrow stromal cells (BMSCs), a common problem being contamination with hemato
92 temic infusion of bone marrow stromal cells (BMSCs), a major type of multipotent stromal cells, produ
94 ing human primary bone marrow stromal cells (BMSCs), lymphoblasts, and skin fibroblasts, we show that
95 h can derive from bone marrow stromal cells (BMSCs), takes a role in modulating osteoblast and osteoc
102 tipotent, bone marrow-derived stromal cells (BMSCs, also known as mesenchymal stem cells [MSCs]), are
105 /hypoxia-inducible factor-1alpha axis in CLL-BMSCs with production of vascular endothelial growth fac
106 ial and primary culture AMSCs and commercial BMSCs demonstrated no statistically significant differen
107 BMSC-supported osteoclastogenesis, confluent BMSCs were cultured with parathyroid hormone (PTH), 1,25
108 ression in freshly isolated BMSCs or control BMSCs cultured in parallel but in nondifferentiating med
109 necrosis factor alpha, and M-CSF in cultured BMSCs at different time points were measured by real-tim
110 ransplantation of primary, but not cultured, BMSCs quantitatively reconstitutes stroma function in vi
113 rast, triglyceride content in differentiated BMSCs or 3T3-L1 cells was suppressed as a result of memb
114 versely, deficiency of XBP1 in healthy donor BMSCs displayed a range of effects on BMSCs that were op
115 the intrinsic properties of OB/BMSCs (i.e., BMSC-extracellular matrix adhesion and migration, cell g
116 gnificantly protected genetically engineered BMSCs from H2O2-induced cell death through increasing au
117 vator of NF-kappaB ligand (RANKL), enhancing BMSC support of MM cell growth and osteoclast formation
118 SDF-1 in the therapeutic effect of exogenous BMSCs was examined by both in vitro and in vivo studies.
120 -Fas antibody did not result in the expected BMSC apoptosis, regardless of concentration, suggesting
122 pathway is a critical maintenance factor for BMSCs during skeletal development, although the precise
126 OA cartilage and the rescuing effect of GFP-BMSC injections were impaired by the inhibitors of C-X-C
127 green fluorescent protein-labeled BMSCs (GFP-BMSCs) were weekly injected into the TMJ region for 4, 8
128 The reparative effects of exogenous GFP-BMSCs were investigated by morphological observation and
131 on synergistically in the recruitment of GFP-BMSCs towards degraded cartilage in mice OA of the TMJ.
134 hat, at 7 d post-transplant, the EphB2(high) BMSCs engrafted in the ISC region at levels of 2.1 +/- 0
136 hing, we suggest that cultured, banked human BMSCs may be effective in treating sepsis in high-risk p
141 e (GFP group) - were incorporated with human BMSCs into a solution of photocrosslinkable gelatin, whi
142 ed induced mouse asthma model, we studied if BMSCs could be beneficial in an allergic, Th2-dominant e
145 sed and decreased TCF-luciferase activity in BMSC from 18-kDa TgFGF2 and FGF2(lmw)(-/-) mice, respect
147 ationship between apoptosis and autophagy in BMSC homeostasis is complex and not well understood.
150 cell death and reduced clonogenic growth in BMSC-adherent myeloma cell lines, aldehyde dehydrogenase
154 hermore, increased expression of miR-146a in BMSCs correlated with inhibition of PGE synthase-2 and i
159 e detected a high level of Fas expression in BMSCs, stimulation of Fas with anti-Fas antibody did not
162 ious findings that overexpression of GILZ in BMSCs antagonizes TNF-alpha-elicited inflammatory respon
163 microRNA-188 (miR-188) is markedly higher in BMSCs from aged compared with young mice and humans.
166 o metabolites are substantially regulated in BMSCs from T2D mice, with the tricarboxylic acid (TCA) c
167 etermined that removal of NOTCH signaling in BMSCs and subsequent depletion of this population result
168 hereby a nutrient-responsive gene influences BMSCs differentiation, adipogenesis, and ultimately body
169 Consistent with its role in influencing BMSCs allocation, Noc(-/-) mice have reduced bone marrow
172 regulation caused migration of i.t.-injected BMSCs to DRGs through the CXCL12 receptor CXCR4, which w
173 with miR-146a expression in freshly isolated BMSCs or control BMSCs cultured in parallel but in nondi
174 have shown previously that freshly isolated BMSCs when induced to express neuronal stem cell markers
176 e this hypothesis, we tracked double-labeled BMSCs in implantation sites created in nude mice transpl
177 ity of radiolabeled or fluorescently labeled BMSCs traveled to and remained in peripheral organs (lun
178 Exogenous green fluorescent protein-labeled BMSCs (GFP-BMSCs) were weekly injected into the TMJ regi
184 , upon implantation into nude mice, mandible BMSCs formed 70% larger bone nodules containing three-fo
185 potential and augmented capacity of mandible BMSCs to induce bone formation in vitro and in vivo.
188 strate here that microRNA-9 (miR-9) modified BMSCs (pri-miR-9-BMSCs) can significantly reduce the pan
190 potential reason for the low yield of mouse BMSCs is the flushing of the marrow used to remove singl
191 reover, the expression of FAP by multipotent BMSCs may point toward the cellular origins of tumor str
195 reduction (a TBD-associated gene) in normal BMSCs by small interfering TERC-RNA (siTERC-RNA) recapit
201 rentiation) and the communication between OB/BMSCs and BMMs (i.e., RANKL expression) that controls os
202 dulating both the intrinsic properties of OB/BMSCs (i.e., BMSC-extracellular matrix adhesion and migr
209 tch between osteogenesis and adipogenesis of BMSCs and may represent a potential therapeutic target f
210 on observed after systemic administration of BMSCs to MCAO rats is likely due to the cellular changes
211 an in vitro system, and the contributions of BMSCs to myeloma pathogenesis in an intact, immune compe
213 do1 suppresses osteogenic differentiation of BMSCs, through a potential mechanism which involves in W
216 idomide abrogated this stimulatory effect of BMSCs and significantly decreased the percentage of SP c
221 show that miR-9 is a key paracrine factor of BMSCs attenuating SAP targeting the NF-kappaB1/p50 gene
223 ver, studies have shown that the majority of BMSCs are trapped in the lungs immediately after intrave
227 ed investigation of hepatogenic potential of BMSCs v/s DMSCs (DPSC, SCAP & DFSC) along-with secretome
230 ture, which may adversely affect recovery of BMSCs physically associated with the abluminal surface o
231 ta-estradiol (E2) enhanced responsiveness of BMSCs of wild-type and Bag-1(+/-) mice to BMP-2, and pro
232 ollectively, our results show novel roles of BMSCs and BMSC-derived Dkk1 in the pathogenesis of multi
234 and culturing an expandable subpopulation of BMSCs with enhanced intestinal homing and contribution t
235 ficantly impaired the growth and survival of BMSCs in vitro and decreased the number of osteoblast (O
237 on of connexin 43 (Cx43) via transduction of BMSCs with a lentivirus overcame this problem, enhancing
240 We found that SDF-1beta had no effect on BMSC proliferation; however, SDF-1beta significantly pro
242 donor BMSCs displayed a range of effects on BMSCs that were opposite to those cells with overexpress
245 Here, we evaluated whether NOTCH and/or BMSCs are required for fracture repair by performing non
248 SSCs) within the BM stromal cell population (BMSCs, also known as BM-derived mesenchymal stem cells),
250 2 levels in MC-4 before OBs or naive primary BMSCs, and Gfi1 induction was blocked by anti-TNF-alpha
254 J subchondral BMSCs enhanced by UAC promoted BMSCs to increase Cxcl12 and Rankl expression, in which
256 isoform, plays a critical role in regulating BMSC survival under oxidative stress through increasing
259 Microarray profiles of control and siTERC-BMSCs showed decreased hematopoietic factors at the mess
261 that Wnt5a/Ror2 signaling in TMJ subchondral BMSCs enhanced by UAC promoted BMSCs to increase Cxcl12
263 support a paracrine mechanism by which i.t. BMSCs target CXCL12-producing DRGs to elicit neuroprotec
264 TERC-RNA (siTERC-RNA) recapitulated the TBD-BMSC phenotype by reducing proliferation and secondary c
266 Upon in vivo transplantation into mice, TBD-BMSCs failed to form bone or support hematopoiesis, unli
269 ng an aptamer delivery system, we found that BMSC-specific overexpression of miR-188 in mice reduced
271 mmunohistochemical results demonstrated that BMSCs can recruit from peripheral circulation and partic
275 s the first evidence, to our knowledge, that BMSCs protect against ALI by restituting alveolar bioene
277 Emerging preclinical studies suggest that BMSCs may protect against infectious challenge either by
279 strated a suppression of the capacity of the BMSC cell line to promote Wnt5a-dependent migration (inc
282 d/or IL-13 activate the STAT6 pathway in the BMSCs resulting in an increase of their TGF-beta product
287 in beta7/alphaE-mediated myeloma adhesion to BMSCs, and -vascular endothelial growth factor-induced b
288 o Furthermore, upon in vivo transplantation, BMSC pellets remodelled into miniature bone/marrow organ
292 that XBP1s is a pathogenic factor underlying BMSC support of MM cell growth and osteoclast formation
293 explore the mechanism of the effect we used BMSCs isolated from a variety of knockout mice, performe
296 rences in manufacturing resulted in variable BMSC characteristics including their ability to form bon
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