ライフサイエンスコーパス: BOOP

1 BOOP has been recognized as a complication of lung and b

2 BOOP lesions are characterized by fibrous extensions int

3 BOOP was initially described as an idiopathic disease pr

4 ranasally inoculated with saline, 1 x 10(6) (BOOP), or 1 x 10(7) (ARDS) PFU reovirus 1/L, and evaluat

5 Although BOOP can be associated with a number of documented pulmo

6 stages of both reovirus 1/L-induced ARDS and BOOP.

7 pulation were similar to idiopathic BOOP and BOOP occurring in other disease settings.

8 For BOS and BOOP in particular, therapy has been based upon a paucit

9 in the backbone of therapy for IPS, BOS, and BOOP, TNF inhibition may augment management of IPS and p

10 findings of ACR, lymphocytic bronchiolitis, BOOP, and interstitial pneumonitis were directly associa

11 , including lymphoplasmacytic bronchiolitis, BOOP, and death.

12 lesions to characteristic, fibrotic cellular BOOP lesions over a 3-week time course.

13 with reovirus serotype 1/strain Lang develop BOOP lesions.

14 hiolitis with intraluminal purulent exudate, BOOP, and pulmonary edema.

15 l features and analyzes the risk factors for BOOP following HSC transplantation.

16 this report, we describe an animal model for BOOP in which CBA/J mice infected with reovirus serotype

17 Therapy for BOOP is based upon minimal clinical evidence.

18 This study shows that histologic BOOP following HSC transplantation has clinical features

19 control study of 49 patients with histologic BOOP and 161 control subjects matched by age and year of

20 and intraalveolar fibrosis similar to human BOOP.

21 elp elucidate the pathogenesis of idiopathic BOOP.

22 s clinical features that resemble idiopathic BOOP and is strongly associated with prior acute and chr

23 n this population were similar to idiopathic BOOP and BOOP occurring in other disease settings.

24 c lesion development in reovirus 1/L-induced BOOP and ARDS.

25 mmation and fibrosis in reovirus 1/L-induced BOOP and ARDS.

26 s in the development of reovirus 1/L-induced BOOP fibrotic lesions in CBA/J mice and suggests that T(

27 lesions associated with reovirus 1/L-induced BOOP, but still develop inflammation and fibrotic lesion

28 We report a case of BOOP after OLT to highlight the risk in all transplant p

29 ase (GVHD) and the subsequent development of BOOP (odds ratios, 3.8 [95% CI, 1.2 to 12.3] and 3.1 [95

30 Clinical features of BOOP in this population were similar to idiopathic BOOP

31 mechanisms responsible for the generation of BOOP in humans.

32 model that closely mimics the generation of BOOP lesions has been an impediment to basic studies of

33 We have developed an animal model of BOOP where CBA/J mice infected with 1 x 10(6) plaque-for

34 nce 1985, an increasing number of reports of BOOP have appeared in the clinical literature, and it is

35 Odds ratios, estimating the relative risk of BOOP in allogeneic HSC recipients, were calculated by co

36 ate features of organizing pneumonia (DAD or BOOP).

37 onchiolitis obliterans organizing pneumonia (BOOP) and acute respiratory distress syndrome (ARDS) are

38 onchiolitis obliterans organizing pneumonia (BOOP) and interstitial pneumonitis occurred at 4 to 6 we

39 onchiolitis obliterans organizing pneumonia (BOOP) has been reported following hematopoietic stem cel

40 onchiolitis obliterans organizing pneumonia (BOOP) in 1, and usual interstitial pneumonia (UIP) in 1.

41 onchiolitis obliterans organizing pneumonia (BOOP) is a clinical syndrome characterized by perivascul

42 onchiolitis obliterans organizing pneumonia (BOOP) is a term that was first applied in 1985 to descri

43 olitis obliterans with organizing pneumonia (BOOP) was the most common inflammatory disorder and fung

44 onchiolitis obliterans organizing pneumonia (BOOP), and others.

45 onchiolitis obliterans organizing pneumonia (BOOP).

46 onchiolitis obliterans organizing pneumonia (BOOP).

47 olitis obliterans with organizing pneumonia (BOOP).

48 ical literature, and it is now accepted that BOOP is a significant pulmonary syndrome.

49 mmation and fibrotic lesions associated with BOOP and ARDS.

50 ent of intraluminal fibrosis associated with BOOP.

51 Patients with BOOP were more likely to have acute GVHD involving the s