ライフサイエンスコーパス: BPI

1 BPI (bactericidal/permeability-increasing) is a potent a

2 BPI and LBP(N)-BPI(C) promote apparently CD14-independen

3 BPI binding to the bacterial surface rapidly triggers po

4 BPI could thus induce a p53-like response even in the pr

5 BPI gene transcript and protein were detected in airway

6 BPI has two domains with the same fold, but with little

7 BPI levels at 15 min of dialysis with CTA (10.91 +/- 3.6

8 BPI mRNA was detected in bovine retina; retinal pigment

9 BPI produced a dose-dependent increase (up to 3-fold) in

10 BPI release is probably mediated by non-complement facto

11 BPI was previously shown to retain aspects of its own fu

12 BPI, but not control protein thaumatin, activated extrac

13 BPI-CETP chimeras are inhibited by LPS but cannot be inh

14 BPI-derived peptide P2 rapidly halted oxygen consumption

15 BPI-enhanced delivery of the blebs to MDDC did not incre

16 BPI-inducible protein A (BipA) is a member of the family

17 BPI-reported pain reduction from baseline to 4 weeks aft

18 BPIs occurred in less than 1% of patients.

19 all imaging tractography study comparing 118 BPI patients and 86 healthy control individuals.

20 -LBP binding even at very low (1:40 to 1:20) BPI:LPS molar ratios.

21 a 1.02-point lower (95% CI, -1.58 to -0.47) BPI score at 12 months (3.57 vs 4.59).

22 itiation factor (eIF) 4E-binding protein (4E-BPI), sensitizes fibroblasts to apoptosis in a manner st

23 Proteins containing a BPI N-terminal domain had greater heparin binding capaci

24 domain of BPI, thus creating the notion of a BPI/PLUNC structural superfamily.

25 all-bowel pathogen that causes cholera, to a BPI-derived peptide, P2.

26 how disrupted Shh signaling protects against BPI could uncover variants in the Shh pathway that cause

27 Baseline FACT-G (P = .0016) and BPI (P = .0001) scores were significantly associated wit

28 Both, fluorescently labeled blebs and BPI were internalized by MDDC under these conditions.

29 in of LBP, COOH-terminal domain of BPI), and BPI(N)-LBP(C) with purified (3)H-LPS and, subsequently,

30 detailed clinical observations that EvC and BPI do not occur in the same individuals, led us to hypo

31 ctivities of combinations of cathepsin G and BPI were additive, as were those of combinations of cath

32 factors, along with elongation factor G and BPI-inducible protein A.

33 Thus, defensins, cathepsin G, and BPI are the major anti-H. capsulatum effector molecules

34 n the presence of Ca(2+) and Mg(2+), LBP and BPI each promote aggregation of LPS to protein-LPS aggre

35 human monocyte response to LPS, both LBP and BPI inhibited LPS-stimulated TNF-alpha production in mou

36 results in a significant increase in LBP and BPI levels.

37 conditions of in vitro culture, both LBP and BPI suppressed, in a dose-dependent manner, the ability

38 udy presented here, plasma levels of LBP and BPI were measured, predialysis, 15 min into dialysis and

39 6) Da), but only LPS associated with LBP and BPI(N)-LBP(C) is disaggregated in the presence of CD14.

40 ther, the association between LBP levels and BPI release during hemodialysis and clinical and laborat

41 nces, the physical properties of LBP-LPS and BPI-LPS complexes have been compared in this study by se

42 t structural differences between SPLUNC1 and BPI.

43 define how closely related proteins such as BPI and LBP can have opposing effects on the body's resp

44 status models (ASMs) were considered: ASM1 (BPI and schizoaffective disorder, BP cases (SABP) only),

45 with myeloid differentiation, such as AZU1, BPI, CTSG, LYZ and RNASE2 as well as of antiproliferativ

46 Overall, mean (SD) baseline BPI scores in the intervention and control groups were 5

47 The correlation between BPI levels at 15 min of dialysis with plasma LBP levels

48 Similarly, the correlation between BPI levels at 15 min of dialysis with the clinical and l

49 ier after Mg2+-induced displacement of bound BPI.

50 nd functional interference scale of the BPI (BPI-FI).

51 ses of E. coli to sublethal injury caused by BPI.

52 isolate sensitive to synergistic killing by BPI and defensins.

53 ort that a specific inhibitor of BCL6 called BPI can trigger a p53 response in DLBCL cells.

54 Compared with controls, BPI patients had significant reductions in mean generali

55 helium, sites where the previously described BPI family members are not expressed.

56 e-N-propoxy bacteriopurpurinimide (dipropoxy-BPI), which was readily oxidized in oxygen atmosphere yi

57 a proband affected with bipolar I disorder (BPI) and at least one other member affected with BPI or

58 n euthymic subjects with bipolar I disorder (BPI) and healthy control subjects.

59 most linkage studies of Bipolar I disorder (BPI) have used relatively small samples.

60 hrough a proband who had bipolar I disorder (BPI) were interviewed by a psychiatrist, assigned an all

61 studies of biomarkers of bipolar I disorder (BPI).

62 corresponding disulfide analogue (disulfide-BPI).

63 ell line was treated in vitro with disulfide-BPI, yielding a CC50 value of 0.05 +/- 0.005 muM.

64 ive bearing 3,4-ethylenedioxythiophene (EDOT-BPI) and its palladium complex (EDOT-PdBPI) were synthes

65 ns but produce profoundly different effects: BPI and a bioactive N-terminal fragment BPI-21 exert a s

66 Epithelial BPI was, in part, responsible for killing a commensal st

67 Neutrophil extract BPI content on post-BMT days +20, +30, and +100 (169+/-3

68 bactericidal/permeability-increasing factor (BPI), regulate delivery of LPS to CD14 antigen on effect

69 , the Cohen kappa coefficients were 0.99 for BPI, 0.99 for BPII, and 0.98 for recurrent unipolar diso

70 ier in blood and exudate cells than mRNA for BPI, consistent with release of progressively less matur

71 that GPC4 is a specific binding protein for BPI on RPE to mediate the activation of ERK1/2, Akt, and

72 Risk for BPI was more weakly predicted by deviations from the FCA

73 A possible role for BPI in clearance of cell-free endotoxin has also been su

74 0 using the Brief Pain Inventory-Short Form (BPI-SF) were randomly assigned to receive either O3-FAs

75 ales of the Brief Pain Inventory-short form (BPI-sf), length of Intensive Care Unit stay and postoper

76 ncluded the Brief Pain Inventory-Short Form (BPI-SF), Western Ontario and McMaster Universities Osteo

77 25 with the Brief Pain Inventory Short Form (BPI-SF).

78 pain score (Brief Pain Inventory-Short Form [BPI-SF] question 3 worst pain, score </=3 vs >/=4).

79 hree of the Brief Pain Inventory Short Form [BPI-SF] questionnaire) or mildly symptomatic (score of 2

80 cts: BPI and a bioactive N-terminal fragment BPI-21 exert a selective and potent antibacterial effect

81 Cationic peptides derived from BPI exhibit anti-inflammatory activity.

82 n, and a peptidomimetic (XMP.Z) derived from BPI were examined.

83 n even when over 40% of the sequence is from BPI.

84 ing EvC in the Amish confers protection from BPI.

85 is [(BPI)Cu(O2CPh)] (1-O2CPh), formed from [(BPI)Cu(PPh3)2], oxidant, and benzoic acid.

86 LBP, holoBPI, BPI-21, and LBP/BPI chimeras were compared for their abi

87 However, BPI can enhance both the sedimentation velocity and appa

88 ecombinant 21-kDa modified fragment of human BPI (rBPI21), containing the active antimicrobial and en

89 fragments and the crystal structure of human BPI have established that BPI consists of two functional

90 .4 angstroms, the crystal structure of human BPI shows a boomerang-shaped molecule formed by two simi

91 Further, while human BPI also suppressed LPS-dependent NO secretion in mouse

92 etic studies of BP disorder since bipolar I (BPI) and BPII disorders often cluster in the same famili

93 gation of high numbers of EvC and Bipolar I (BPI) cases in several large Amish families descending fr

94 reatment outcome in patients with bipolar I (BPI) disorder.

95 Risk for SZ and bipolar illness (BPI) are predicted by school achievement (SA) at age 16

96 results localize the site of LPS binding in BPI to a region no larger than the amino terminal 155 am

97 The percentage change in BPI levels between predialysis and 15 min was 1341 +/- 2

98 The changes in BPI levels from predialysis to 15 min and between pre- a

99 rical pain responder (> or = 30% decrease in BPI) than the 137 patients in the usual-care group (P <

100 The relative deficiency in BPI of newborns raised the possibility that supplementin

101 ved from newborn cord blood are deficient in BPI.

102 existence of an anatomic disconnectivity in BPI and further underscore a role for interhemispheric d

103 ain of BPI may fulfill a similar function in BPI-specific disposal pathways for Gram-negative bacteri

104 tervention group had greater improvements in BPI pain severity over the 12 months of the trial whethe

105 , and subcortical structures, are present in BPI subjects, even while euthymic.

106 Median time to progression in BPI-SF pain at its worst was 5.7 months (95% CI 5.6-5.7)

107 pathophysiological features of psychosis in BPI.

108 Secondary pain outcomes included BPI interference and severity, global pain improvement,

109 The bactericidal/permeability increasing (BPI) and lipopolysaccharide (LPS)-binding (LBP) proteins

110 er of the bacterial/permeability increasing (BPI) fold-containing protein family, which shares struct

111 er of the bacterial/permeability-increasing (BPI) family of antimicrobial proteins but was first iden

112 of the bactericidal/permeability-increasing (BPI) fold-containing (BPIF) protein family, sharing stru

113 own as bactericidal/permeability-increasing (BPI) fold-containing protein, family A, member 1 (BPIFA1

114 of the bactericidal/permeability-increasing (BPI) gene with pulmonary function in a cohort of patient

115 donor bactericidal/permeability-increasing (BPI) haplotypes were associated with a 2-fold to 3-fold

116 Bactericidal/permeability-increasing (BPI) protein has been shown to play an important role in

117 to the bactericidal/permeability-increasing (BPI) protein.

118 ne effects on the Behavioral Problems Index (BPI) and Home Observation Measurement of the Environment

119 index [BDI] versus bronchoprotection index [BPI]: 1.62 +/- 0.21 versus 2.02 +/- 0.40 [mean +/- SEM],

120 We defined 2 bone PET indices (BPIs) that incorporate anatomic information from CT and

121 ndelian disease loci in bipolar individuals (BPI and BPII, p = 0.06).

122 Moreover, heparitinase also inhibited BPI actions on VEGF and PDGF-B mRNA expression induced b

123 hly sensitive to the BCL6 peptide inhibitor, BPI.

124 nal 10% time on boosted protease inhibitors (BPIs) was associated with reduced KS incidence in the th

125 seroma, paresthesia, brachial plexus injury (BPI), and lymphedema was available for 821 patients.

126 otein-protein interactions similar to intact BPI.

127 thout significant depletion of intracellular BPI stores.

128 fter treatment, by the Brief Pain Inventory (BPI) and the M D Anderson Symptom Inventory (MDASI).

129 utcome measure was the Brief Pain Inventory (BPI) average pain severity score (range 0-10, where 0=no

130 bscale (NTX subscale), Brief Pain Inventory (BPI), and UNISCALE (UNI).

131 The Brief Pain Inventory (BPI), Functional Assessment of Cancer Therapy-Hepatobili

132 al (FACT-G) scale, the Brief Pain Inventory (BPI), the Medical Outcomes Study (MOS) questionnaire, an

133 d by question 3 of the Brief Pain Inventory (BPI).

134 st moderate intensity (Brief Pain Inventory [BPI] score >/=5).

135 , cancer-related pain (Brief Pain Inventory [BPI] worst pain score > or = 6), or both.

136 scale) and sustained (Brief Pain Inventory [BPI], 0- to 10-point scale) change in pain.

137 that the resting state of the catalyst is [(BPI)Cu(O2CPh)] (1-O2CPh), formed from [(BPI)Cu(PPh3)2],

138 The tridentate bipyridylimino isoindoline (BPI) ligands of these complexes were designed to enable

139 ptors, we have compared interactions of LBP, BPI, LBP(N)-BPI(C) (NH(2)-terminal domain of LBP, COOH-t

140 LBP, holoBPI, BPI-21, and LBP/BPI chimeras were compared for their ability to promote

141 Postdialysis LBP:BPI ratios were 50 +/- 6%, 18 +/- 4%, and 22 +/- 6% of p

142 during HD and consequent lowering of the LBP:BPI ratio could potentially afford some protection again

143 Lower KS incidence was observed with longer BPI use, after accounting for potential IRIS and other f

144 primary end point was the difference in mean BPI-SF worst pain scores at 6 weeks, which was lower for

145 et of N-substituted benzoperylene monoimide (BPI) fluorophores was synthesized and characterized stru

146 udy examines the feasibility of using murine BPI (mBPI) expressed on halophilic Archaeal gas vesicle

147 ve compared interactions of LBP, BPI, LBP(N)-BPI(C) (NH(2)-terminal domain of LBP, COOH-terminal doma

148 BPI and LBP(N)-BPI(C) promote apparently CD14-independent LPS associati

149 respectively) was similar to the neutrophil BPI content of normal controls (163+/-35 ng per 106 neut

150 studies led to the identification of nickel BPI complexes that could undergo stable charge-discharge

151 Compared with baseline, the mean observed BPI-SF score decreased by 1.74 points at 12 weeks and 2.

152 played synergy with CAP18p in the absence of BPI.

153 roup analyses revealed similar activation of BPI and control subjects during passive viewing but sign

154 Analysis of BPI fragments and the crystal structure of human BPI hav

155 The 972 kindreds include 2284 cases of BPI disorder, 498 individuals with bipolar II disorder (

156 uthors have extended the characterization of BPI interaction with membrane proteins from bovine RPE.

157 rophil extracts were analyzed for content of BPI by Western blotting and ELISA and for defensin pepti

158 ery of LPS to CD14, the C-terminal domain of BPI may fulfill a similar function in BPI-specific dispo

159 minal domain of LBP, COOH-terminal domain of BPI), and BPI(N)-LBP(C) with purified (3)H-LPS and, subs

160 omain, corresponding to the active domain of BPI, thus creating the notion of a BPI/PLUNC structural

161 The N-terminal domains of BPI and LBP bound lipid A with their characteristic appa

162 ability of E. coli to tolerate low doses of BPI and escape the progression of sublethal to lethal da

163 esicles ("blebs"), we examined the effect of BPI on interactions of metabolically labeled ([(14)C]-ac

164 racterized the previously unknown effects of BPI in the eye and the molecular mechanisms involved in

165 Early (<15 min) reversible effects of BPI on bacterial colony-forming ability and outer membra

166 The constitutive expression of BPI was restricted to cell-bound protein and unaffected

167 inant 21-kDa modified N-terminal fragment of BPI (rBPI(21)), which has the biological properties of t

168 t 21 kDa modified amino-terminal fragment of BPI (rBPI21) reduced H2O2-induced apoptosis in RPE and i

169 n a recombinant 21-kDa bioactive fragment of BPI, rBPI21, and the gram-positive pathogen Streptococcu

170 Because the interaction of BPI with cell-free endotoxin, during infection, occurs m

171 possibility that supplementing the levels of BPI in plasma might enhance newborn antibacterial defens

172 nome-wide significant evidence of linkage of BPI to a candidate region of approximately 10 cM in 5q31

173 we examined the subcellular localization of BPI and p15 in PMNs.

174 hat localization of a synergistic partner of BPI (p15s) in more readily released secondary granules a

175 ngrafted marrow contain normal quantities of BPI and defensins.

176 we postulated that the C-terminal region of BPI may serve a similar delivery function but to distinc

177 structure of PSP and known active regions of BPI.

178 The release of BPI during HD and consequent lowering of the LBP:BPI rat

179 To evaluate the therapeutic role of BPI in ischemic retinopathies, the antiangiogenic activi

180 These findings suggest a novel role of BPI in the interaction of bacterial outer membrane vesic

181 were found in the SA and IQs of siblings of BPI vs matched control probands.

182 electron microscopy confirmed the storage of BPI in primary granules and showed that p15s are stored

183 n 524 subjects in a genetic linkage study of BPI disorder.

184 We assessed the utility as such of BPI fold-containing family A member 2 (BPIFA2), also kno

185 terminus of CETP and the carboxy terminus of BPI did not retain any observable CETP function.

186 rmeability, the mechanistic understanding of BPI's action may provide novel therapeutic opportunities

187 36 (IRR = 0.14; 95% CI, .02-1.00) months on BPIs compared with <6 months.

188 Months on BPIs was associated with lower KS incidence (P = .02).

189 (LOD) 3.4 for rs722642 at 78 cM) using only BPI and schizoaffective (SA), BP cases.

190 were those of combinations of cathepsin G or BPI with HNP-1, HNP-2, and HNP-3.

191 phorylation was not reduced by either LBP or BPI under conditions that result in selective TNF-alpha

192 o LPS was also not modified by either LBP or BPI.

193 han that of CAP18p ( approximately 20 nM) or BPI ( approximately 50 nM).

194 Diagnoses of SZ or BPI in the Swedish Hospital Discharge Register and the S

195 Secondary outcomes were improvement in other BPI subscales and at other time points, FACT-Hep and EOR

196 rences occurred over time in sustained pain (BPI mean pain, 0.07 point [CI, -0.23 to 0.37 points]; BP

197 content of the antimicrobial (poly)peptides BPI and defensins.

198 pain, 0.07 point [CI, -0.23 to 0.37 points]; BPI worst pain, -0.14 point [CI, -0.59 to 0.31 points]),

199 les allows the neutrophil to mobilize potent BPI-dependent antibacterial activity extracellularly wit

200 re no significant differences in predialysis BPI levels between the three dialyzers (P = 0.21).

201 s itself related to the host defense protein BPI (bactericidal/permeability-increasing protein).

202 actericidal-permeability-increasing protein (BPI) also mediated fungistasis against H. capsulatum in

203 actericidal permeability-increasing protein (BPI) and lipopolysaccharide (LPS)-binding protein, have

204 actericidal/permeability-increasing protein (BPI) and lipopolysaccharide-binding protein (LBP), which

205 actericidal permeability increasing protein (BPI) differs from LBP by inhibiting LPS-induced human mo

206 actericidal/permeability-increasing protein (BPI) fold-containing family B, member 3 (BPIFB3) is an e

207 actericidal/permeability-increasing protein (BPI) fold-containing family B, member 6 (BPIFB6), whose

208 actericidal/permeability-increasing protein (BPI) had been shown to possess anti-inflammatory and end

209 actericidal/permeability-increasing protein (BPI) has been shown to induce various effects in retinal

210 actericidal/permeability-increasing protein (BPI) is a natural protein, stored within the neutrophil

211 actericidal/permeability-increasing protein (BPI) is found in the primary granules of adult neutrophi

212 actericidal/permeability-increasing protein (BPI) is thought to play an important role in killing and

213 actericidal/permeability-increasing protein (BPI) of neutrophils is a lipopolysaccharide (LPS)-bindin

214 actericidal/permeability-increasing protein (BPI) on LPS-dependent activation of mouse thioglycolate-

215 actericidal/permeability-increasing protein (BPI) released from polymorphonuclear leukocytes (PMNs) i

216 actericidal/permeability-increasing protein (BPI) to 1.7 A.

217 actericidal/permeability-increasing protein (BPI) was originally identified as a lipopolysaccharide (

218 actericidal/permeability-increasing protein (BPI), a member of a genomically conserved lipid-interact

219 actericidal/permeability-increasing protein (BPI), an antibacterial and lipopolysaccharide-neutralizi

220 actericidal/permeability-increasing protein (BPI), which neutralizes LPS.

221 actericidal/permeability-increasing protein (BPI)-derived anti-microbial peptide.

222 actericidal/permeability-increasing protein (BPI)-derived peptide P2 and the murine alpha-defensin cr

223 actericidal/permeability-increasing protein (BPI).

224 actericidal/permeability increasing protein (BPI).

225 actericidal/permeability-increasing protein (BPI).

226 actericidal/permeability-increasing protein (BPI).

227 actericidal/permeability-increasing protein (BPI).

228 acteridical/permeability-increasing protein (BPI).

229 bactericidal/permeability inducing protein (BPI), and lipopolysaccharide binding protein (LBP) are m

230 the related plasma lipid transfer proteins, BPI illuminates a mechanism of lipid transfer for this p

231 otein glypican 4 (GPC4) serves as a putative BPI-binding protein.

232 These artificial fusions retain BPI functions such as lipopolysaccharide (LPS) binding a

233 RI-BPI showed superior duration of tissue penetration and c

234 Finally, RI-BPI could kill primary human DLBCL cells but had no effe

235 ide, retroinverso BCL6 peptide inhibitor (RI-BPI) selectively killed BCR rather than OxPhos-type DLBC

236 The RI-BPI could recapitulate the failure to form germinal cent

237 In this multicenter sample, BPI patients had reduced WM integrity in interhemispheri

238 t (ICE) and Brief Pain Inventory pain score (BPI-PS), is associated with patient perception of benefi

239 Mean (SE) BPI neuropathic pain severity score declined from 6.2 (0

240 Since BPI has unusual dual properties of promoting RPC and RPE

241 n though the chimera was able to retain some BPI-like properties.

242 Peak and steady state BPI concentrations were comparable with pharmacokinetic

243 Confirming earlier published studies, BPI inhibited, and LBP enhanced, the ability of LPS to s

244 olipase C, and anti-GPC4 antibody suppressed BPI-induced ERK and Akt phosphorylation in bovine RPE.

245 The synthesized BPIs exhibit positive solvachromatic emission (lambda(em

246 come was associated with improved short-term BPI scores (per $1,000, beta = -0.47; P = 0.01) and medi

247 nts were associated with improved short-term BPI scores (per $1,000, beta = -0.57; P = 0.04).

248 of addition of recombinant 21-kDa N-terminal BPI fragment (rBPI(21)) on the growth and tumor necrosis

249 structure of human BPI have established that BPI consists of two functionally distinct domains: a pot

250 The authors recently reported that BPI can induce ERK1/2 and Akt activity and that it incre

251 Scanning electron microscopy revealed that BPI treatment produced greater membrane perturbations in

252 he results of the present study suggest that BPI is tightly regulated and functionally expressed by e

253 These data suggest that BPI-derived compounds may have unique therapeutic potent

254 The BPI, Western Ontario and McMaster Universities Osteoarth

255 sis of the two indices indicated that as the BPI increased, the BDI reached a plateau.

256 ain from bone metastases, as measured by the BPI, increased from 39 of 149 (26%) before SBRT to 55 of

257 with placebo also significantly improved the BPI average pain interference score, the Fibromyalgia Im

258 d a significantly greater improvement in the BPI average pain severity score (P=0.015; estimated diff

259 These results suggest mutations in the BPI gene significantly influence the risk of developing

260 nically significant change at 1 month in the BPI subscale of symptom on average in the past 24 hours.

261 30) and functional interference scale of the BPI (BPI-FI).

262 a combination of systematic variation of the BPI ligand and the metal center along with mechanistic i

263 ents had superior scores on all items of the BPI-FI (ie, general activity, mood, walking ability, nor

264 nically meaningful (mean 3.4 [SD 2.9] on the BPI pain-at-its-worst item at baseline, 2.1 [2.4] at 4 w

265 s remains elusive, our data suggest that the BPI peptide sensitivity of OmpU-deficient V. cholerae is

266 for response using the ICE and 605 using the BPI-PS.

267 Similar results were obtained using the BPI-PS; observed improvements were typically of lesser m

268 Primary outcome was the BPI total score, which ranges from 0 ("no pain") to 10 (

269 Whereas the BPI family are predicted to share very closely similar t

270 larity searches have shown homology with the BPI-like family of innate immune genes, particularly wit

271 Pain was assessed with the BPI-SF questionnaire, and health-related quality of life

272 Thirty BPI and 26 control subjects underwent functional magneti

273 nal portion that imparts opsonic activity to BPI.

274 related LPS-binding protein, in contrast to BPI, did not increase association of the blebs with MDDC

275 suggest that the fate of E. coli exposed to BPI depends on both OmpR-independent mechanisms engaged

276 y depends on the high molar ratio of p15s to BPI in the extracellular fluid (approximately 50:1), whi

277 = 0.29; P < .001), and negatively related to BPI (r = -0.41; P < .0001).

278 Two BPI-derived compounds, rBPI(21) and XMP.Z, significantly

279 A structural alignment between the two BPI domains was used to compare the 3D-1D environments o

280 ction was significantly stronger (BDI versus BPI: 3.40 +/- 0.43 versus 6.98 +/- 1.42, p = 0.02).

281 al properties of LPS complexed to LBP versus BPI.

282 oarthritis, and taxane use, adjusted 12-week BPI-SF scores did not differ by arm (P = .58).

283 es (RPC), and endothelial cells (REC); while BPI protein was measured in human vitreous and plasma.

284 ts with BP (13 males and 26 females; 28 with BPI and 11 with BPII) and 35 healthy controls were inves

285 and at least one other member affected with BPI or bipolar II disorder (BPII), we identified four re

286 rees limited to relative pairs affected with BPI.

287 res used in a genetic study of families with BPI disorder.

288 to estimate the associations of income with BPI and HOME scores.

289 A sample of 66 patients with BPI disorder completed a self-report measure of lifetime

290 rum symptoms in this sample of patients with BPI disorder was associated with greater levels of depre

291 ment strategies are needed for patients with BPI disorder who endorse lifetime panic spectrum feature

292 Patients with BPI disorder who reported high lifetime panic-agoraphobi

293 o EvC individual has ever been reported with BPI.

294 family, sharing structural similarities with BPI-like proteins.

295 y, which shares structural similarities with BPI-like proteins.

296 ne the association of EITC payment size with BPI and HOME scores; in the second, we used EITC payment

297 ring emotion downregulation in subjects with BPI.

298 bilateral vlPFC compared with subjects with BPI.

299 oCAP18, exhibited antibacterial synergy with BPI, and the p15s also displayed synergy with CAP18p in

300 On the basis of its homology with BPIs and restricted expression of SPLUNC1 in serous cell