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1                                              BUN levels at 24 wk were reduced from 68.8 +/- 9.7 mg/dl
2                                              BUN levels were persistently higher among nonsurvivors t
3                                              BUN measurements confirmed renal protection at 48 hr in
4                                              BUN, serum creatinine concentration and urinalysis were
5 ](0.516) x [1.8/cystatin C (mg/L)](0.294)[30/BUN (mg/dl)](0.169)[1.099](male)[height (m)/1.4](0.188).
6 .15+/-0.3 vs. Group C = 2.10+/-0.06; Group A BUN = 27.0+/-6.0 vs. Group C = 31.1+/-6.4).
7 59+/-0.63 vs. Group C = 12.8+/-1.01; Group A BUN = 64.1+/-2.73 vs. Group C = 104.9+/-12.21)--however,
8                          Combining admission BUN and change in BUN at 24 hours produced an AUC of 0.9
9                        An elevated admission BUN/Cr identifies decompensated patients with heart fail
10 ably, in patients with an elevated admission BUN/Cr, the risk of death associated with RD (estimated
11                    However, higher admission BUN/Cr was also associated with post-discharge worsening
12                             Higher admission BUN/Cr was associated with in-hospital IRF (odds ratio,
13 However, in patients with a normal admission BUN/Cr, RD was not associated with increased mortality (
14 was used to evaluate the impact of admission BUN, change in BUN, admission Hgb, and change in Hgb on
15 tamin B12 (P=0.05), creatinine (P=0.03), and BUN (P=0.05), and blood cyclosporine trough level (C0, P
16 ly increased serum phosphate, 1,25(OH)2D and BUN, and decreased serum PTH and FGF23, relative to vehi
17 were demonstrated between breath ammonia and BUN (0.86 to 0.96), which demonstrates the possibility o
18  gave an R(2) of 0.95 for breath ammonia and BUN correlation and an R(2) of 0.83 for breath ammonia a
19 ion between breath ammonia concentration and BUN and creatinine.
20 ressure, serum creatinine concentration, and BUN were obtained at baseline, during treatment with aml
21                                  Peak Cr and BUN (mg%) were significantly greater in Group C vs. Grou
22          By contrast, in P-/- creatinine and BUN increased only moderately (fourfold over sham).
23 findings but did reduce serum creatinine and BUN, as observed in patients with CKD.
24  to investigate the associations of eGFR and BUN with risk of incident CHD in the prospective Dongfen
25                 In contrast, proteinuria and BUN levels were significantly reduced in mice treated wi
26 y (MDRD) formula that age, gender, race, and BUN account for creatinine production (CP).
27 ith CP independent of age, gender, race, and BUN.
28  poor sensitivity and specificity of SCr and BUN, we used rat toxicology studies to compare the diagn
29 etabolic control, which is the time-averaged BUN (BUNa) or steady-state BUN (BUNs) for intermittent h
30             ROC analysis identified baseline BUN (P = .026, area under ROC curve [AUC] = 0.818), cyst
31  primary outcome was the interaction between BUN- and HDLD-associated mortality.
32 ime-averaged BUN (BUNa) or steady-state BUN (BUNs) for intermittent hemodialysis (IHD) or continuous
33 ely attenuated renal failure, as measured by BUN levels in mice fed an adenine diet known to cause re
34  (BUN) level, creatinine (Cr) concentration, BUN:Cr ratio, and hepatic transaminase levels (P </= .05
35 ainment of intensive CRRT metabolic control (BUNs = 60 mg/dl), required urea clearance rates of appro
36 did not confer functional (serum creatinine, BUN and FITC-labeled inulin clearance) or histological p
37                            Serum creatinine, BUN, folate and vitamin B12, and blood cyclosporine trou
38  level and recipients age, serum creatinine, BUN, folate concentrations, and creatinine clearance.
39 ly, there was no change in serum creatinine, BUN, or mean arterial pressure values.
40 inuria (6.6 +/- 4.12 mg/24 hours), decreased BUN and serum creatinine levels (15.71 +/- 8.17 mg/dl an
41 ponse, increases estimated GFR and decreases BUN, serum phosphorus, and uric acid concentrations in p
42 a K (ml/h) versus patient weight for desired BUNs values of 60, 80, and 100 mg/dl were also generated
43 th significant reductions in cystic disease, BUN and serum creatinine levels.
44 educed survival in patients with an elevated BUN level and improved survival in patients with a norma
45 sient, and RD, in the setting of an elevated BUN/Cr, remains strongly associated with death.
46  Of the 6 routine laboratory tests examined, BUN yielded the highest area under the concentration-tim
47 rry-Ig-treated mice developed renal failure (BUN > 50 mg/dl) compared with 18 of 38 (47.4%) mice in c
48 ictors for RHC placement: sepsis, PaO2/FIO2, BUN, and Pa.
49 sis, we also sampled the patient's blood for BUN and creatinine.
50 = 11) developed severe LPS-induced ARF (24-h BUN 44.0 +/- 4.1 vs 112.1 +/- 20.0 mg/dl).
51 completely resistant to LPS-induced ARF (6-h BUN of 32.3 +/- 1.1 vs 61.7 +/- 5.6 mg/dl).
52 ad significantly more albuminuria and higher BUN levels than MRL-lpr controls.
53 id doses, current and prior pain, and higher BUN levels were post-transplantation risk factors for gr
54                              After 18 hours, BUN levels and renal cortical FC/CE content were determi
55  group E=3.99 +/- 1.5 mg%, and group B 72 hr BUN=241.3 +/- 32.8 mg% vs. group E=52.6 +/- 22.5 mg%).
56        Combining admission BUN and change in BUN at 24 hours produced an AUC of 0.91 for mortality.
57 luate the impact of admission BUN, change in BUN, admission Hgb, and change in Hgb on mortality.
58 tudies revealed no significant difference in BUN between diabetic SPARC-null mice and diabetic WT mic
59 8 +/- 0.01 mg/min per 100 g) and increase in BUN (Intact, 20.3 +/- 2.1; OVX, 32.6 +/- 5.1; OVX+E(2),
60                For every 5-mg/dl increase in BUN during the first 24 hours, the age- and gender-adjus
61 ine the relationship between early trends in BUN and Hgb with respect to mortality.
62             The model (BALI), which included BUN >or=25 mg/dL, Age >or=65 years, LDH >or=300 IU/L, an
63 Mice injected with 15 mg/kg CP had increased BUN and serum creatinine and CP caused remarkable pathol
64 lockers, digitalis use, blood urea nitrogen (BUN) >25, body mass index (BMI) > or =30 kg/m2, and New
65 .1 mg/dL; P = .008) and blood urea nitrogen (BUN) (27 vs 21 mg/dL; P = .006) decreased significantly.
66 o correlating well with blood urea nitrogen (BUN) (r = 0.61, p < 0.01, n = 96).
67 stoperative increase in blood urea nitrogen (BUN) and creatinine compared with those who did not (17-
68 by measurement of serum blood urea nitrogen (BUN) and creatinine, chemistries (sodium, potassium, chl
69  that were analyzed for blood urea nitrogen (BUN) and creatinine, which are the accepted standards in
70 h later for analysis of blood urea nitrogen (BUN) and kidney tissue (n = 8 per group).
71 ked (100%) reduction in blood urea nitrogen (BUN) and serum creatinine and a highly significant reduc
72 mg/24 hours), increased blood urea nitrogen (BUN) and serum creatinine levels (39.86 +/- 13.45 mg/dl
73 nd significantly higher blood urea nitrogen (BUN) and total and direct bilirubin and alkaline phospha
74 um creatinine (SCr) and blood urea nitrogen (BUN) are the primary options for monitoring kidney dysfu
75 um creatinine (SCr) and blood urea nitrogen (BUN) daily for 3 days, and neutrophil infiltration deter
76 reater than 15 seconds, blood urea nitrogen (BUN) greater than 50 mg/dL, intubation, performance stat
77 eart disease (CHD), and blood urea nitrogen (BUN) has been shown to be a strong predictor of mortalit
78 rum creatinine (SCr) or blood urea nitrogen (BUN) in monitoring generalized renal function after expo
79 rt rate, cardiac index, blood urea nitrogen (BUN) level, creatinine (Cr) concentration, BUN:Cr ratio,
80 irty h after injection, blood urea nitrogen (BUN) levels were 30.3 +/- 4.4 and 114.8 +/- 23.5 mg/dl f
81 um creatinine (SCr) and blood urea nitrogen (BUN) levels were evaluated postischemia.
82 poalbuminemia, elevated blood urea nitrogen (BUN) levels, and evidence of severe nephritis.
83 lkaline phosphatase and blood urea nitrogen (BUN) levels.
84 was not associated with blood urea nitrogen (BUN) or the BUN to creatinine ratio.
85 uate accuracy of serial blood urea nitrogen (BUN) versus serial hemoglobin (Hgb) measurement for pred
86 urohormonal activation, blood urea nitrogen (BUN), could identify patients destined to experience adv
87 psis, APACHE III score, blood urea nitrogen (BUN), creatinine, net fluid balance, and positive end-ex
88 atively associated with blood urea nitrogen (BUN), supernatant IL-4, serum IL-6, monoclonal immunoglo
89 ar changes were seen in blood urea nitrogen (BUN).
90 um creatinine (SCr) and blood urea nitrogen (BUN).
91 sus 0.77 +/- 0.2 mg/dl; blood urea nitrogen (BUN): 20.1 +/- 14.1 versus 10.3 +/- 3.28 mg/dl] than tho
92 72 hr plasma creatinine (CR); urea nitrogen (BUN); thromboxane B2 (TXB2) and 6-keto prostaglandin F(1
93 nificantly higher serum blood-urea-nitrogen (BUN) and creatinine levels.
94 R) patients (defined by blood urea nitrogen [BUN] >or=50 mg/dl and/or serum creatinine >or=2.5 mg/dl)
95 by 20 h of reperfusion (blood urea nitrogen [BUN] values, 46.6 +/- 6.9 and 68.4 +/- 7.9 mg/dl in C3 -
96  improved survival in patients with a normal BUN level.
97 ble logistic regression compared accuracy of BUN, Hgb, and additional routine laboratory tests.
98 olimus showed side effects with elevation of BUN, cholesterol, triglycerides, and ALT after 120 days.
99 -severe decline in eGFR or a raised level of BUN might be associated with increased risk of incident
100 e injury and TFF3 augmented the potential of BUN and SCr to detect kidney damage.
101 ed with individuals in the lowest tertile of BUN, those in the highest tertile were at significantly
102 iated with HDLD use is strongly dependent on BUN concentrations with reduced survival in patients wit
103  with cold perception deficit, creatinine or BUN in the females.
104           Albumin outperformed either SCr or BUN for detecting kidney tubule injury and TFF3 augmente
105                        Pre- and postdialysis BUN levels were reported in all children.
106 calculated from predialysis and postdialysis BUN measurements in patients receiving intermittent dial
107 ed for calculating PCRn from the predialysis BUN and Kt/V.
108 t/NLL)) + 0.168, where Co is the predialysis BUN in mg/dL.
109     Based on measurements of blood pressure, BUN, creatinine, albuminuria, genotyping and immunoblott
110 linear relationship, was utilized to produce BUN versus time curves by the direct quantification meth
111 in 43 gene (connexin 43+/-) had proteinuria, BUN, and serum creatinine levels significantly lower tha
112                           Age, gender, race, BUN, and serum creatinine (Scr) were used to calculate M
113 ission blood urea nitrogen/creatinine ratio (BUN/Cr) could identify decompensated heart failure patie
114                       Kim-1 outperforms SCr, BUN and urinary NAG in multiple rat models of kidney inj
115 tal-based cohort study, we identified serial BUN measurement as the most valuable single routine labo
116 nge of serum bilirubin, creatinine and serum BUN levels before and after the first treatment with ADV
117 igher levels of total kidney collagen, serum BUN, and urinary protein than Mrc2-sufficient Col4alpha3
118 on, decreased body mass, and increased serum BUN/creatinine ratio.
119 the time-averaged BUN (BUNa) or steady-state BUN (BUNs) for intermittent hemodialysis (IHD) or contin
120                                 Steady-state BUN versus time profiles for the same simulated patient
121 emic vascular resistance, and changes in the BUN:Cr ratio(P </= .01).
122 ciated with blood urea nitrogen (BUN) or the BUN to creatinine ratio.
123 ata were available in real time, whereas the BUN and creatinine data were available generally 24 h la
124 e diagnostic performance of urinary Kim-1 to BUN, SCr and urinary N-acetyl-beta-D-glucosaminidase (NA
125  C3 deposition was inversely proportional to BUN values (r = -0.63; P < 0.001), which presumably indi
126 Four of five transgene-negative animals with BUN levels of > 100 mg/dl were anuric; the remaining ani
127                    Serum AGE correlated with BUN (r = 0.6, P < 0.002 for CML; r = 0.4, P < 0.05 for M
128 ell C3 staining was strongly correlated with BUN values (r = 0.83, P < 0.001), as was C9 staining (r
129                             In patients with BUN levels below the median, there was no associated ris
130  both of which correlated significantly with BUN values (P < 0.001).
131                             In subjects with BUN levels above the median (21.0 mg/dl), both the unadj
132 ciation functional class >II, age >70 years, BUN >26 mg/dl, QRS duration >0.12 s, and atrial fibrilla

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