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1  burgdorferi, Anaplasma phagocytophilum, and Babesia microti.
2 and molecular tests for the causative agent, Babesia microti.
3 gnosed by peripheral red blood cell smear as Babesia microti.
4 tic Ehrlichia sp., Borrelia burgdorferi, and Babesia microti.
5 ere coinfected with Borrelia burgdorferi and Babesia microti.
6 ic cycle, including Borrelia burgdorferi and Babesia microti.
7 nfections with Borrelia burgdorferi (33.6%), Babesia microti (8.4%), Anaplasma phagocytophila (1.9%),
8  this parasite to be most closely related to Babesia microti (97.9% sequence similarity); sera from i
9 the United States is caused predominantly by Babesia microti, a tick-transmitted blood parasite.
10                                              Babesia microti, a tick-transmitted, intraerythrocytic p
11                                              Babesia microti, a tickborne intraerythrocytic parasite
12                                              Babesia microti, a zoonotic intraerythrocytic parasite,
13                        Borellia burgdorferi, Babesia microti and Anaplasma phagocytophilum rely almos
14 ly caused by the intraerythrocytic parasite, Babesia microti and transmitted by the same tick as Lyme
15 agent of human monocytic ehrlichiosis (HME), Babesia microti, and Borrelia burgdorferi.
16  burgdorferi, babesiosis, which is caused by Babesia microti, and human granulocytic ehrlichiosis (HG
17  in indirect fluorescent-antibody tests with Babesia microti antigen, however, suggesting that they r
18 ansplant recipient who survived infection by Babesia microti contracted through blood transfusion.
19 s and fatalities of babesiosis are caused by Babesia microti Current treatment for human babesiosis c
20            Serologic evidence of exposure to Babesia microti did not significantly impact the clinica
21 arasites could be seen on microscopy, and no Babesia microti DNA was detected in the blood of any sub
22 sitive, and accurate method for detection of Babesia microti in patient specimens.
23 testing were used to confirm the presence of Babesia microti in the donor's blood and to establish th
24 aneous infection of Borrelia burgdorferi and Babesia microti in the northeastern and northern midwest
25 of coinfection with Borrelia burgdorferi and Babesia microti in tick vectors, reservoir hosts, and pa
26 y who were suspected of local acquisition of Babesia microti infection.
27                                              Babesia microti is a tick-borne red blood cell parasite
28                   Human babesiosis caused by Babesia microti is an emerging tick-borne zoonosis of in
29  long-term carriers of the zoonotic parasite Babesia microti is evidenced by numerous reported cases
30          Although infection by the protozoan Babesia microti is rarely symptomatic in immunocompetent
31 babesiosis, which is caused by the piroplasm Babesia microti, is made by microscopic identification o
32 besiosis, a zoonosis caused by the protozoan Babesia microti, is usually not treated when the symptom
33                              A new strain of Babesia microti (KR-1) was isolated from a Connecticut r
34                    Tickborne transmission of Babesia microti mainly occurs in 7 states in the Northea
35 rently infected with Borrelia burgdorferi or Babesia microti (or both).
36 gainst a panel of 24 organisms consisting of Babesia microti, other Babesia species, Plasmodium speci
37 atment of babesiosis, including clearance of Babesia microti parasitemia.
38 abesia divergens-like/MO-1 organisms and not Babesia microti, the common United States species.
39 ime PCR assay targeting the 18S rRNA gene of Babesia microti, the dominant babesiosis pathogen in the
40 ted States have been tickborne and caused by Babesia microti, the etiologic agent of all previously d
41           The intraerythrocytic apicomplexan Babesia microti, the primary causative agent of human ba
42 acted from whole-blood specimens and detects Babesia microti with a limit of detection of approximate

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