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1 ences the expression of virulence factors in Bacillus anthracis.
2 W rabbits exposed to aerosols of Ames strain Bacillus anthracis.
3 ion of spores of Bacillus species, including Bacillus anthracis.
4 not the same species as, Bacillus cereus and Bacillus anthracis.
5 thrax is caused by the sporulating bacterium Bacillus anthracis.
6 boxylic acid) (DPA), which is a biomarker of Bacillus anthracis.
7 assessment of potential therapeutics against Bacillus anthracis.
8 - and capsule-encoding virulence plasmids of Bacillus anthracis.
9 es, designated ltaS1 to -4, in the genome of Bacillus anthracis.
10 nases Bas2152 (PrkD) and Bas2037 (PrkG) from Bacillus anthracis.
11 om the human pathogen and bioterrorism agent Bacillus anthracis.
12 s expression of the major virulence genes of Bacillus anthracis.
13 x using recombinant protective Ag (rPA) from Bacillus anthracis.
14 a serine/threonine kinase (STK) expressed by Bacillus anthracis.
15 i, a spore forming nonpathogenic simulant of Bacillus anthracis.
16 sufficient to protect against infection with Bacillus anthracis.
17 ), serine/threonine kinase (BA-Stk1) pair in Bacillus anthracis.
18 by the Gram-positive spore-forming bacterium Bacillus anthracis.
19 or the virulence of the pathogenic bacterium Bacillus anthracis.
20 haired HRS/J mice are extremely resistant to Bacillus anthracis.
21 racteristics of recombinant DNA primase from Bacillus anthracis.
22 by the gram-positive spore-forming bacterium Bacillus anthracis.
23 sented by another deadly bacterial pathogen, Bacillus anthracis.
24 L11 exhibit antimicrobial activities against Bacillus anthracis.
25 at infections caused by the biodefense agent Bacillus anthracis.
26 ulator of plasmid-encoded virulence genes in Bacillus anthracis.
27 n or putative polysaccharide deacetylases of Bacillus anthracis.
28 positive pathogens Staphylococcus aureus and Bacillus anthracis.
29 y the spore-forming, gram-positive bacterium Bacillus anthracis.
30 athogenic bacilli Listeria monocytogenes and Bacillus anthracis.
31 ne and/or during disseminated infection with Bacillus anthracis.
32 ne responses of wild herbivore hosts against Bacillus anthracis.
33 agent of the disease anthrax is the spore of Bacillus anthracis.
34 i-phagocytic capsule conferring virulence on Bacillus anthracis.
35 conditions, similar to previous findings in Bacillus anthracis.
36 contribute to infections by bacteria such as Bacillus anthracis.
37 , including 5 strains of Yersinia pestis and Bacillus anthracis.
38 Wip1, a tectivirus that infects the pathogen Bacillus anthracis.
39 elements (29 nt), a fluoride riboswitch from Bacillus anthracis(48 nt), and a frame-shifting element
40 racellular vesicles from the supernatants of Bacillus anthracis, a Gram-positive bacillus that is a p
43 uring advanced stages of inhalation anthrax, Bacillus anthracis accumulates at high levels in the blo
44 We also provide evidence that the S-layer of Bacillus anthracis acts as a molecular sieve that is chi
46 rmining the median lethal dose (LD50) of the Bacillus anthracis Ames strain in guinea pigs and invest
48 y diverse panel of inbred mice and spores of Bacillus anthracis Ames, we investigated host susceptibi
49 ema factor, is the major virulence factor of Bacillus anthracis, an agent that causes high mortality
50 toxin (LT) is a critical virulence factor of Bacillus anthracis and an important means by which this
52 -C pathogens: Category A priority pathogens; Bacillus anthracis and Clostridium botulinum, and Catego
54 o exhibit epitope diversity, and epitopes of Bacillus anthracis and Clostridium tetani toxins, as the
58 efficient transfer of ICEBs1 into and out of Bacillus anthracis and that cwlT was needed for ICEBs1 t
59 M by analyzing the nine-strain pan-genome of Bacillus anthracis and up to 62 strains of Escherichia c
61 tivity against MRSA, Listeria monocytogenes, Bacillus anthracis, and a vancomycin-resistant Enterococ
63 genes is presented from sequenced B. cereus, Bacillus anthracis, and Bacillus thuringiensis strains.
64 from the culture medium of Bacillus cereus, Bacillus anthracis, and Bacillus thuringiensis when stra
65 res of the Bacillus cereus group (B. cereus, Bacillus anthracis, and Bacillus thuringiensis) are surr
66 ry sites of YpeB cleavage were identified in Bacillus anthracis, and it was shown that the stable pro
67 cterial species, including Escherichia coli, Bacillus anthracis, and Streptococcus pneumoniae, studie
68 tor, and edema factor, the protein toxins of Bacillus anthracis , are among its most important virule
69 e of the pathogens Clostridium difficile and Bacillus anthracis, are uniquely stable cell forms, high
70 tective antigen (PA83) of anthrax toxin from Bacillus anthracis as a foreign antigen and expressed PA
71 due to the pathogenic exotoxins produced by Bacillus anthracis as well as other virulence factors of
73 inations of polystyrene beads, gram-positive Bacillus anthracis, B. thuringiensis, and B. atrophaeus
74 simple method was developed for detection of Bacillus anthracis (BA) endospores and for differentiati
76 ally, primates infected with toxin-secreting Bacillus anthracis bacilli developed a rapid and marked
77 strains of the Bacillus cereus group, i.e., Bacillus anthracis, Bacillus cereus, Bacillus mycoides,
78 analogues, bind dihydrofolate reductase from Bacillus anthracis (BaDHFR) with lower affinity than is
79 protein product of one such gene, MccF from Bacillus anthracis (BaMccF), is able to cleave intact an
82 uctures of BLIP-II alone and in complex with Bacillus anthracis Bla1 beta-lactamase revealed no signi
83 PB and HPB carrying ARGs in the manures were Bacillus anthracis, Bordetella pertussis, and B. anthrac
84 uccessfully implemented for the detection of Bacillus anthracis, botulinum B, and tularemia in comple
85 btilis and other Bacillus species, including Bacillus anthracis, bound rabbit IgM through an unconven
86 tive antibody (Ab)-mediated immunity against Bacillus anthracis but has limited efficacy and duration
88 ee binary bacterial toxins: anthrax toxin of Bacillus anthracis, C2 toxin of Clostridium botulinum, a
89 exes with different classes of inhibitors of Bacillus anthracis, Campylobacter jejuni, and Clostridiu
91 potential biological warfare agents, such as Bacillus anthracis, causal agent of anthrax in humans an
93 er, these methods rely on recovery of viable Bacillus anthracis cells from swabs of cutaneous lesions
95 ystal structure at 2.10 A resolution for the Bacillus anthracis coenzyme A-disulfide reductase isofor
96 Germination is a key step for successful Bacillus anthracis colonization and systemic disseminati
97 toxin (LT) is an A-B type toxin secreted by Bacillus anthracis, consisting of the cellular binding m
108 e active in vitro against bacterial forms of Bacillus anthracis encountered in vivo, as well as in vi
110 solution that the active form of DAPDC from Bacillus anthracis, Escherichia coli, Mycobacterium tube
112 hal concentrations of the anthrax bacterium, Bacillus anthracis, for grazing animals in a natural sys
114 or off-label broad-spectrum efficacy against Bacillus anthracis; Francisella tularensis; Coxiella bur
116 anthrax by facilitating the dissemination of Bacillus anthracis from the lung in early disease and pr
117 Anthrax toxin, a major virulence factor of Bacillus anthracis, gains entry into target cells by bin
118 e GSLEs that have been shown to be active in Bacillus anthracis germination: sleB, cwlJ1, and cwlJ2.
125 m the secondary cell wall polysaccharides of Bacillus anthracis, has been chemically synthesized.
126 of Staphylococcus aureus and petrobactin of Bacillus anthracis hold considerable potential as a sing
127 nce identity with anthrax lethal factor from Bacillus anthracis; however, we have shown that the toxi
128 have determined three crystal structures of Bacillus anthracis IMPDH, in a phosphate ion-bound (term
129 T cells that recognize the protective Ag of Bacillus anthracis in both anthrax vaccine-adsorbed vacc
131 B. subtilis vesicles, but also vesicles from Bacillus anthracis, indicating a mechanism that crossed
132 identified several compounds that protected Bacillus anthracis infected macrophages from cell death.
133 termine if Nod1/Nod2 are involved in sensing Bacillus anthracis infection and eliciting protective im
134 t observations derived from animal models of Bacillus anthracis infection are inconsistent with aspec
135 ly shown to have increased susceptibility to Bacillus anthracis infection relative to wild-type anima
136 pears to be important in the pathogenesis of Bacillus anthracis infection, but its causes are unclear
137 estigated the effect of alpha-GalCer against Bacillus anthracis infection, the agent of anthrax.
138 he most prevalent form of naturally acquired Bacillus anthracis infection, which is associated with e
146 The lethal factor (LF) enzyme secreted by Bacillus anthracis is a zinc hydrolase that is chiefly r
152 r illustrated by the demonstration that once Bacillus anthracis is engineered to express high levels
160 ne of the two essential virulence factors of Bacillus anthracis is the poly-gamma-D-glutamic acid (ga
162 ction of cytokine responses and induction of Bacillus anthracis lethal factor (LF)-specific adaptive
165 inflammasome was identified as the sensor of Bacillus anthracis lethal toxin (LT) in mouse macrophage
166 protective antigen and lethal factor of the Bacillus anthracis lethal toxin using semiconductor quan
167 ation of NLRP1 by various stimuli, including Bacillus anthracis lethal toxin, Toxoplasma gondii, mura
169 immune response to other virulence factors (Bacillus anthracis LF and EF) than HLA-homozygous subjec
170 ral important rod-shaped pathogens including Bacillus anthracis, Listeria monocytogenes, and Clostrid
172 Here we report the crystal structures of Bacillus anthracis NadD in complex with three NadD inhib
173 m-negative, including Staphylococcus aureus, Bacillus anthracis, Neisseria gonorrhoeae, and Neisseria
174 of the manganese-tyrosyl radical cofactor of Bacillus anthracis NrdF and the redox properties of B. a
179 x stems from the shielding properties of the Bacillus anthracis poly-gamma-d-glutamic acid capsule.
181 The etiologic agent of inhalational anthrax, Bacillus anthracis, produces virulence toxins that are i
183 ted that a linear determinant in domain 2 of Bacillus anthracis protective Ag (PA) is a potentially i
184 have shown that intranasal coapplication of Bacillus anthracis protective Ag (PA) together with a B.
186 tion between the human CMG2 receptor and the Bacillus anthracis protective antigen (PA) is essential
187 y using Lactobacillus acidophilus to deliver Bacillus anthracis protective antigen (PA) via specific
188 idually disease enhancing or neutralizing to Bacillus anthracis protective antigen (PA), a component
190 sent study, using a plasmid that encodes the Bacillus anthracis protective antigen (PA63) gene fragme
191 diverse GC responses to two complex antigens-Bacillus anthracis protective antigen and influenza hema
194 we generated IgG2a and IgG2b variants of the Bacillus anthracis protective antigen-binding IgG1 monoc
195 in (Atx), a key virulence factor secreted by Bacillus anthracis, provides a robust biophysical model
196 , or 3-mercaptopyruvate sulfurtransferase in Bacillus anthracis, Pseudomonas aeruginosa, Staphylococc
197 mini-pXO1 plasmid containing a replicon from Bacillus anthracis pXO1 (181.6 kb) was identified by mak
200 toxins produced by Clostridium botulinum and Bacillus anthracis represents a particularly challenging
206 ctrochemical genosensor for the detection of Bacillus anthracis, specific towards the regulatory gene
212 omponent of complement, and a portion of the Bacillus anthracis spore surface protein BclA, all of wh
218 the interaction between macrophage cells and Bacillus anthracis spores is of significant importance w
223 , we demonstrate that PHB deficiency impairs Bacillus anthracis sporulation through diminishing the e
224 ied as a novel low-molecular weight thiol in Bacillus anthracis, Staphylococcus aureus, and several o
225 tors of several bacterial toxins produced by Bacillus anthracis, Staphylococcus aureus, Clostridium p
233 harbors S-layer genes, including homologs of Bacillus anthracis surface array protein (Sap), extracta
234 (ET) is one of two binary toxins produced by Bacillus anthracis that contributes to the virulence of
235 for the development of improved vaccines for Bacillus anthracis that increase not only neutralizing A
237 the events associated with the emergence of Bacillus anthracis the causative agent of anthrax-a leth
240 irus, the etiological agent of smallpox, and Bacillus anthracis, the bacterial pathogen responsible f
241 e culture, shows significant activity toward Bacillus anthracis, the bacterial pathogen responsible f
246 we discover that the gram-positive bacterium Bacillus anthracis, the causative agent of anthrax, does
257 We report the 1.40 A structure of a P4H from Bacillus anthracis, the causative agent of anthrax, whos
258 derophore, is required for full virulence of Bacillus anthracis, the causative agent of anthrax.
259 ria that has hitherto not been identified in Bacillus anthracis, the causative agent of anthrax.
260 activity against the Gram-positive bacterium Bacillus anthracis, the causative agent of anthrax.
261 ific bactericidal activity toward strains of Bacillus anthracis, the causative agent of anthrax.
263 operties of metallo-beta-lactamase Bla2 from Bacillus anthracis, the enzyme was overexpressed, purifi
264 sterol-dependent cytolysin (CDC) secreted by Bacillus anthracis, the etiologic agent for anthrax.
266 ysis of a systemic bacterial infection using Bacillus anthracis, the etiological agent of anthrax dis
267 Here, we report that the peptidoglycan of Bacillus anthracis, the etiological agent of anthrax, is
270 In Bacillus cereus and its close relative Bacillus anthracis, the major pilin protein BcpA is clea
274 se anthrax lethal factor (LF) is secreted by Bacillus anthracis to promote disease virulence through
275 , GrlA(E85A), GrlA(S81F/E85A) and GrlA(S81F) Bacillus anthracis topoisomerase IV, their sensitivity t
276 macrophages, the lethal factor component of Bacillus anthracis toxin binds to a carrier protein (PA)
277 dies (MAbs) have been reported for the other Bacillus anthracis toxin components, but relatively few
278 that HNP-1 to HNP-3 inhibit lethal toxin of Bacillus anthracis, toxin B of Clostridium difficile, di
280 bs) are potential therapeutic agents against Bacillus anthracis toxins, since there is no current tre
282 h under iron limitation, Bacillus cereus and Bacillus anthracis, two human pathogens from the Bacillu
286 es were considered the primary target of the Bacillus anthracis virulence factor lethal toxin because
289 s of its target, the protective antigen from Bacillus anthracis We show how rational design based on
290 Using virulent and nonvirulent strains of Bacillus anthracis, we have shown that secretion of ATP
291 a potential role for NK cells in immunity to Bacillus anthracis, we utilized primary human and murine
292 the CcpA homologues of Bacillus subtilis and Bacillus anthracis were not affected by the Stk1 ortholo
293 bioterrorism agents like Yersinia pestis and Bacillus anthracis which feature on the Center for Disea
294 is initiated by endospores of the bacterium Bacillus anthracis, which are introduced into the lung.
295 threatening disease caused by infection with Bacillus anthracis, which expresses lethal factor and th
296 premature termination of hasA translation in Bacillus anthracis, which is known to escape phagocytic
297 known to confer ciprofloxacin resistance in Bacillus anthracis, Yersinia pestis, and Francisella tul
299 er water or nucleic acids from BT organisms (Bacillus anthracis, Yersinia pestis, Francisella tularen
300 elease of bacterial biothreat agents such as Bacillus anthracis, Yersinia pestis, or Burkholderia pse
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