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1 ences the expression of virulence factors in Bacillus anthracis.
2 W rabbits exposed to aerosols of Ames strain Bacillus anthracis.
3 ion of spores of Bacillus species, including Bacillus anthracis.
4 not the same species as, Bacillus cereus and Bacillus anthracis.
5 thrax is caused by the sporulating bacterium Bacillus anthracis.
6 boxylic acid) (DPA), which is a biomarker of Bacillus anthracis.
7 assessment of potential therapeutics against Bacillus anthracis.
8 - and capsule-encoding virulence plasmids of Bacillus anthracis.
9 es, designated ltaS1 to -4, in the genome of Bacillus anthracis.
10 nases Bas2152 (PrkD) and Bas2037 (PrkG) from Bacillus anthracis.
11 om the human pathogen and bioterrorism agent Bacillus anthracis.
12 s expression of the major virulence genes of Bacillus anthracis.
13 x using recombinant protective Ag (rPA) from Bacillus anthracis.
14 a serine/threonine kinase (STK) expressed by Bacillus anthracis.
15 i, a spore forming nonpathogenic simulant of Bacillus anthracis.
16 sufficient to protect against infection with Bacillus anthracis.
17 ), serine/threonine kinase (BA-Stk1) pair in Bacillus anthracis.
18 by the Gram-positive spore-forming bacterium Bacillus anthracis.
19 or the virulence of the pathogenic bacterium Bacillus anthracis.
20 haired HRS/J mice are extremely resistant to Bacillus anthracis.
21 racteristics of recombinant DNA primase from Bacillus anthracis.
22 by the gram-positive spore-forming bacterium Bacillus anthracis.
23 sented by another deadly bacterial pathogen, Bacillus anthracis.
24 L11 exhibit antimicrobial activities against Bacillus anthracis.
25 at infections caused by the biodefense agent Bacillus anthracis.
26 ulator of plasmid-encoded virulence genes in Bacillus anthracis.
27 n or putative polysaccharide deacetylases of Bacillus anthracis.
28 positive pathogens Staphylococcus aureus and Bacillus anthracis.
29 y the spore-forming, gram-positive bacterium Bacillus anthracis.
30 athogenic bacilli Listeria monocytogenes and Bacillus anthracis.
31 ne and/or during disseminated infection with Bacillus anthracis.
32 ne responses of wild herbivore hosts against Bacillus anthracis.
33 agent of the disease anthrax is the spore of Bacillus anthracis.
34 i-phagocytic capsule conferring virulence on Bacillus anthracis.
35  conditions, similar to previous findings in Bacillus anthracis.
36 contribute to infections by bacteria such as Bacillus anthracis.
37 , including 5 strains of Yersinia pestis and Bacillus anthracis.
38 Wip1, a tectivirus that infects the pathogen Bacillus anthracis.
39 elements (29 nt), a fluoride riboswitch from Bacillus anthracis(48 nt), and a frame-shifting element
40 racellular vesicles from the supernatants of Bacillus anthracis, a Gram-positive bacillus that is a p
41                                              Bacillus anthracis, a Gram-positive pathogen, produces S
42                                              Bacillus anthracis, a spore-forming bacterium, is such a
43 uring advanced stages of inhalation anthrax, Bacillus anthracis accumulates at high levels in the blo
44 We also provide evidence that the S-layer of Bacillus anthracis acts as a molecular sieve that is chi
45          These plasmids are analogous to the Bacillus anthracis Ames plasmids pXO1 and pXO2 that enco
46 rmining the median lethal dose (LD50) of the Bacillus anthracis Ames strain in guinea pigs and invest
47 dentify key genetic features of the letters' Bacillus anthracis Ames strain.
48 y diverse panel of inbred mice and spores of Bacillus anthracis Ames, we investigated host susceptibi
49 ema factor, is the major virulence factor of Bacillus anthracis, an agent that causes high mortality
50 toxin (LT) is a critical virulence factor of Bacillus anthracis and an important means by which this
51 ed to pathogenic Bacillus species, including Bacillus anthracis and Bacillus thuringiensis.
52 -C pathogens: Category A priority pathogens; Bacillus anthracis and Clostridium botulinum, and Catego
53  community relevant spore-forming pathogens, Bacillus anthracis and Clostridium difficile.
54 o exhibit epitope diversity, and epitopes of Bacillus anthracis and Clostridium tetani toxins, as the
55               A bibliometric analysis of the Bacillus anthracis and Ebola virus archival literature w
56                  Therefore, the abilities of Bacillus anthracis and Escherichia coli gyrase and topoi
57               Interactions between spores of Bacillus anthracis and macrophages are critical for the
58 efficient transfer of ICEBs1 into and out of Bacillus anthracis and that cwlT was needed for ICEBs1 t
59 M by analyzing the nine-strain pan-genome of Bacillus anthracis and up to 62 strains of Escherichia c
60 of growth inhibition against F. tularensis , Bacillus anthracis , and Staphylococcus aureus .
61 tivity against MRSA, Listeria monocytogenes, Bacillus anthracis, and a vancomycin-resistant Enterococ
62 t oxidative stress in Staphylococcus aureus, Bacillus anthracis, and Bacillus subtilis.
63 genes is presented from sequenced B. cereus, Bacillus anthracis, and Bacillus thuringiensis strains.
64  from the culture medium of Bacillus cereus, Bacillus anthracis, and Bacillus thuringiensis when stra
65 res of the Bacillus cereus group (B. cereus, Bacillus anthracis, and Bacillus thuringiensis) are surr
66 ry sites of YpeB cleavage were identified in Bacillus anthracis, and it was shown that the stable pro
67 cterial species, including Escherichia coli, Bacillus anthracis, and Streptococcus pneumoniae, studie
68 tor, and edema factor, the protein toxins of Bacillus anthracis , are among its most important virule
69 e of the pathogens Clostridium difficile and Bacillus anthracis, are uniquely stable cell forms, high
70 tective antigen (PA83) of anthrax toxin from Bacillus anthracis as a foreign antigen and expressed PA
71  due to the pathogenic exotoxins produced by Bacillus anthracis as well as other virulence factors of
72            Rapid presymptomatic diagnosis of Bacillus anthracis at early stages of infection plays a
73 inations of polystyrene beads, gram-positive Bacillus anthracis, B. thuringiensis, and B. atrophaeus
74 simple method was developed for detection of Bacillus anthracis (BA) endospores and for differentiati
75 tency against Staphylococcus aureus (Sa) and Bacillus anthracis (Ba) helicases.
76 ally, primates infected with toxin-secreting Bacillus anthracis bacilli developed a rapid and marked
77  strains of the Bacillus cereus group, i.e., Bacillus anthracis, Bacillus cereus, Bacillus mycoides,
78 analogues, bind dihydrofolate reductase from Bacillus anthracis (BaDHFR) with lower affinity than is
79  protein product of one such gene, MccF from Bacillus anthracis (BaMccF), is able to cleave intact an
80                                 The P4H from Bacillus anthracis (BaP4H) has been postulated to act on
81           The lung is the terminal target of Bacillus anthracis before death, whatever the route of i
82 uctures of BLIP-II alone and in complex with Bacillus anthracis Bla1 beta-lactamase revealed no signi
83 PB and HPB carrying ARGs in the manures were Bacillus anthracis, Bordetella pertussis, and B. anthrac
84 uccessfully implemented for the detection of Bacillus anthracis, botulinum B, and tularemia in comple
85 btilis and other Bacillus species, including Bacillus anthracis, bound rabbit IgM through an unconven
86 tive antibody (Ab)-mediated immunity against Bacillus anthracis but has limited efficacy and duration
87 ins play a major role in the pathogenesis of Bacillus anthracis by subverting the host defenses.
88 ee binary bacterial toxins: anthrax toxin of Bacillus anthracis, C2 toxin of Clostridium botulinum, a
89 exes with different classes of inhibitors of Bacillus anthracis, Campylobacter jejuni, and Clostridiu
90                                              Bacillus anthracis can cause inhalational anthrax.
91 potential biological warfare agents, such as Bacillus anthracis, causal agent of anthrax in humans an
92              The endospore forming bacterium Bacillus anthracis causes lethal anthrax disease in huma
93 er, these methods rely on recovery of viable Bacillus anthracis cells from swabs of cutaneous lesions
94 nt compounds in vivo protecting mice against Bacillus anthracis challenge.
95 ystal structure at 2.10 A resolution for the Bacillus anthracis coenzyme A-disulfide reductase isofor
96     Germination is a key step for successful Bacillus anthracis colonization and systemic disseminati
97  toxin (LT) is an A-B type toxin secreted by Bacillus anthracis, consisting of the cellular binding m
98                   The Gram-positive pathogen Bacillus anthracis contains 24 genes whose products harb
99                                              Bacillus anthracis contains two megaplasmids, pXO1 and p
100           Insertional mutagenesis of exsM in Bacillus anthracis DeltaSterne resulted in a partial sec
101 ing, synthetic, and structural studies using Bacillus anthracis DHPS.
102 ng glycans on spores, whereas others such as Bacillus anthracis do not.
103 is a tripartite virulence factor produced by Bacillus anthracis during infection.
104                  The production of cAMP from Bacillus anthracis edema toxin (ET) activates gene expre
105                                              Bacillus anthracis edema toxin (ET) consists of protecti
106                                              Bacillus anthracis elaborates a poly-gamma-d-glutamic ac
107                              The envelope of Bacillus anthracis encompasses a proteinaceous S-layer w
108 e active in vitro against bacterial forms of Bacillus anthracis encountered in vivo, as well as in vi
109                                          The Bacillus anthracis endospore loses resistance properties
110  solution that the active form of DAPDC from Bacillus anthracis, Escherichia coli, Mycobacterium tube
111                                              Bacillus anthracis exhibits a rapid growth rate during s
112 hal concentrations of the anthrax bacterium, Bacillus anthracis, for grazing animals in a natural sys
113                                              Bacillus anthracis forms metabolically dormant endospore
114 or off-label broad-spectrum efficacy against Bacillus anthracis; Francisella tularensis; Coxiella bur
115 d adenosine synthesis also enabled escape of Bacillus anthracis from phagocytic clearance.
116 anthrax by facilitating the dissemination of Bacillus anthracis from the lung in early disease and pr
117   Anthrax toxin, a major virulence factor of Bacillus anthracis, gains entry into target cells by bin
118 e GSLEs that have been shown to be active in Bacillus anthracis germination: sleB, cwlJ1, and cwlJ2.
119          Here we demonstrate by NMR that the Bacillus anthracis glmS riboswitch selectively binds the
120                                              Bacillus anthracis grows in chains of rod-shaped cells,
121                   The Gram-positive pathogen Bacillus anthracis grows in characteristic chains of ind
122                                              Bacillus anthracis has four GSLEs: CwlJ1, CwlJ2, SleB, a
123                                              Bacillus anthracis has four putative GSLEs, based upon s
124                   These results suggest that Bacillus anthracis has the ability to evade the host's i
125 m the secondary cell wall polysaccharides of Bacillus anthracis, has been chemically synthesized.
126  of Staphylococcus aureus and petrobactin of Bacillus anthracis hold considerable potential as a sing
127 nce identity with anthrax lethal factor from Bacillus anthracis; however, we have shown that the toxi
128  have determined three crystal structures of Bacillus anthracis IMPDH, in a phosphate ion-bound (term
129  T cells that recognize the protective Ag of Bacillus anthracis in both anthrax vaccine-adsorbed vacc
130            The lung is the site of entry for Bacillus anthracis in inhalation anthrax, the deadliest
131 B. subtilis vesicles, but also vesicles from Bacillus anthracis, indicating a mechanism that crossed
132  identified several compounds that protected Bacillus anthracis infected macrophages from cell death.
133 termine if Nod1/Nod2 are involved in sensing Bacillus anthracis infection and eliciting protective im
134 t observations derived from animal models of Bacillus anthracis infection are inconsistent with aspec
135 ly shown to have increased susceptibility to Bacillus anthracis infection relative to wild-type anima
136 pears to be important in the pathogenesis of Bacillus anthracis infection, but its causes are unclear
137 estigated the effect of alpha-GalCer against Bacillus anthracis infection, the agent of anthrax.
138 he most prevalent form of naturally acquired Bacillus anthracis infection, which is associated with e
139 mortality rates associated with inhalational Bacillus anthracis infection.
140                                              Bacillus anthracis is a bioterrorism agent classified by
141                                              Bacillus anthracis is a Gram-positive spore-forming bact
142                The antiphagocytic capsule of Bacillus anthracis is a major virulence factor.
143                                              Bacillus anthracis is a spore-forming, Gram-positive pat
144                                              Bacillus anthracis is a sporulating Gram-positive bacter
145                                              Bacillus anthracis is a tier 1 select agent with the pot
146    The lethal factor (LF) enzyme secreted by Bacillus anthracis is a zinc hydrolase that is chiefly r
147               Inhalational anthrax caused by Bacillus anthracis is associated with high mortality pri
148                              Pathogenesis of Bacillus anthracis is associated with the production of
149              The anthrax edema toxin (ET) of Bacillus anthracis is composed of the receptor-binding c
150                            The exosporium of Bacillus anthracis is comprised of two distinct layers:
151                             The virulence of Bacillus anthracis is critically dependent on the cytoto
152 r illustrated by the demonstration that once Bacillus anthracis is engineered to express high levels
153                            The arginase from Bacillus anthracis is not well characterized; therefore,
154  poly gamma-D-glutamic acid (gammaDPGA) from Bacillus anthracis is presented.
155  perspective, the glmS ribozyme derived from Bacillus anthracis is the best characterized.
156                                              Bacillus anthracis is the causative agent of anthrax in
157                                              Bacillus anthracis is the causative agent of anthrax, an
158                                              Bacillus anthracis is the causative agent of anthrax, wh
159                                              Bacillus anthracis is the causative agent of anthrax.
160 ne of the two essential virulence factors of Bacillus anthracis is the poly-gamma-D-glutamic acid (ga
161 ver, the role of lipoprotein biosynthesis in Bacillus anthracis is unknown.
162 ction of cytokine responses and induction of Bacillus anthracis lethal factor (LF)-specific adaptive
163 bed to initiate the inflammasome response to Bacillus anthracis lethal factor.
164                                          The Bacillus anthracis lethal toxin (LT) has been shown to a
165 inflammasome was identified as the sensor of Bacillus anthracis lethal toxin (LT) in mouse macrophage
166  protective antigen and lethal factor of the Bacillus anthracis lethal toxin using semiconductor quan
167 ation of NLRP1 by various stimuli, including Bacillus anthracis lethal toxin, Toxoplasma gondii, mura
168 an alveolar epithelial cells are a target of Bacillus anthracis lethal toxin.
169  immune response to other virulence factors (Bacillus anthracis LF and EF) than HLA-homozygous subjec
170 ral important rod-shaped pathogens including Bacillus anthracis, Listeria monocytogenes, and Clostrid
171                           Interestingly, the Bacillus anthracis lrgAB mutant displayed decreased stat
172     Here we report the crystal structures of Bacillus anthracis NadD in complex with three NadD inhib
173 m-negative, including Staphylococcus aureus, Bacillus anthracis, Neisseria gonorrhoeae, and Neisseria
174 of the manganese-tyrosyl radical cofactor of Bacillus anthracis NrdF and the redox properties of B. a
175 Escherichia coli, Staphylococcus aureus, and Bacillus anthracis particles.
176       Spore germination is the first step to Bacillus anthracis pathogenicity.
177            Here, we describe the capacity of Bacillus anthracis peptidoglycan (BaPGN) to trigger an a
178                   The lethal toxin (LeTx) of Bacillus anthracis plays a central role in the pathogene
179 x stems from the shielding properties of the Bacillus anthracis poly-gamma-d-glutamic acid capsule.
180                                              Bacillus anthracis produces virulence toxins required fo
181 The etiologic agent of inhalational anthrax, Bacillus anthracis, produces virulence toxins that are i
182                                              Bacillus anthracis proliferates to high levels within ve
183 ted that a linear determinant in domain 2 of Bacillus anthracis protective Ag (PA) is a potentially i
184  have shown that intranasal coapplication of Bacillus anthracis protective Ag (PA) together with a B.
185  with GalXM conjugated to a protein carrier, Bacillus anthracis protective Ag.
186 tion between the human CMG2 receptor and the Bacillus anthracis protective antigen (PA) is essential
187 y using Lactobacillus acidophilus to deliver Bacillus anthracis protective antigen (PA) via specific
188 idually disease enhancing or neutralizing to Bacillus anthracis protective antigen (PA), a component
189  large polypeptide segments derived from the Bacillus anthracis protective antigen (PA).
190 sent study, using a plasmid that encodes the Bacillus anthracis protective antigen (PA63) gene fragme
191 diverse GC responses to two complex antigens-Bacillus anthracis protective antigen and influenza hema
192                          Using ovalbumin and Bacillus anthracis protective antigen protein as model a
193                           Single channels of Bacillus anthracis protective antigen, PA(63), were reco
194 we generated IgG2a and IgG2b variants of the Bacillus anthracis protective antigen-binding IgG1 monoc
195 in (Atx), a key virulence factor secreted by Bacillus anthracis, provides a robust biophysical model
196 , or 3-mercaptopyruvate sulfurtransferase in Bacillus anthracis, Pseudomonas aeruginosa, Staphylococc
197 mini-pXO1 plasmid containing a replicon from Bacillus anthracis pXO1 (181.6 kb) was identified by mak
198                                              Bacillus anthracis pXO1 minireplicon (MR) plasmid consis
199                                              Bacillus anthracis remains a serious bioterrorism concer
200 toxins produced by Clostridium botulinum and Bacillus anthracis represents a particularly challenging
201                              Pathogenesis by Bacillus anthracis requires coordination between two dis
202                                              Bacillus anthracis secretes two bipartite toxins, edema
203                                              Bacillus anthracis secretes two virulence factors: a tri
204                                          The Bacillus anthracis secretome includes protective antigen
205                                              Bacillus anthracis shares many regulatory loci with the
206 ctrochemical genosensor for the detection of Bacillus anthracis, specific towards the regulatory gene
207                   The outermost layer of the Bacillus anthracis spore consists of an exosporium compr
208                   The outermost layer of the Bacillus anthracis spore consists of an exosporium compr
209 :1 ratio to toxin and protected mice against Bacillus anthracis spore infection.
210                                          The Bacillus anthracis spore is the causative agent of the d
211                    The sdAb-A5 binds BclA, a Bacillus anthracis spore protein, with high affinity (K(
212 omponent of complement, and a portion of the Bacillus anthracis spore surface protein BclA, all of wh
213                   The outermost layer of the Bacillus anthracis spore, the exosporium, is composed of
214                                              Bacillus anthracis spores are enclosed by an exosporium
215                                              Bacillus anthracis spores are the etiologic agent of ant
216                                 Ingestion of Bacillus anthracis spores causes gastrointestinal (GI) a
217                        Pulmonary exposure to Bacillus anthracis spores initiates inhalational anthrax
218 the interaction between macrophage cells and Bacillus anthracis spores is of significant importance w
219            Nutrient-dependent germination of Bacillus anthracis spores is stimulated when receptors l
220                                              Bacillus anthracis spores, the etiological agents of ant
221 etrocyclin-1 protects mice from infection by Bacillus anthracis spores.
222 alational anthrax is caused by inhalation of Bacillus anthracis spores.
223 , we demonstrate that PHB deficiency impairs Bacillus anthracis sporulation through diminishing the e
224 ied as a novel low-molecular weight thiol in Bacillus anthracis, Staphylococcus aureus, and several o
225 tors of several bacterial toxins produced by Bacillus anthracis, Staphylococcus aureus, Clostridium p
226                                  Colonies of Bacillus anthracis Sterne allow the growth of papillatio
227 and T cell-mediated immune responses against Bacillus anthracis Sterne challenge.
228  for adhesion of the anthrax vaccine strain, Bacillus anthracis Sterne, to host cells.
229                                           In Bacillus anthracis str. Sterne, resistance to oxidative
230                   The susceptibility of most Bacillus anthracis strains to beta-lactam antibiotics is
231                         Transcription of the Bacillus anthracis structural genes for the anthrax toxi
232          During high-impact events involving Bacillus anthracis, such as the Amerithrax incident of 2
233 harbors S-layer genes, including homologs of Bacillus anthracis surface array protein (Sap), extracta
234 (ET) is one of two binary toxins produced by Bacillus anthracis that contributes to the virulence of
235 for the development of improved vaccines for Bacillus anthracis that increase not only neutralizing A
236              Anthrax is caused by strains of Bacillus anthracis that produce two key virulence factor
237  the events associated with the emergence of Bacillus anthracis the causative agent of anthrax-a leth
238                                           In Bacillus anthracis the siderophore petrobactin is vital
239                                              Bacillus anthracis, the anthrax agent, is a member of th
240 irus, the etiological agent of smallpox, and Bacillus anthracis, the bacterial pathogen responsible f
241 e culture, shows significant activity toward Bacillus anthracis, the bacterial pathogen responsible f
242                                              Bacillus anthracis, the causative agent of anthrax and a
243                                              Bacillus anthracis, the causative agent of anthrax disea
244                                    Spores of Bacillus anthracis, the causative agent of anthrax, are
245                               Infection with Bacillus anthracis, the causative agent of anthrax, can
246 we discover that the gram-positive bacterium Bacillus anthracis, the causative agent of anthrax, does
247                                              Bacillus anthracis, the causative agent of anthrax, form
248                                              Bacillus anthracis, the causative agent of anthrax, is a
249                                              Bacillus anthracis, the causative agent of anthrax, is a
250                                              Bacillus anthracis, the causative agent of anthrax, is p
251                                              Bacillus anthracis, the causative agent of anthrax, reli
252                                              Bacillus anthracis, the causative agent of anthrax, repl
253                                              Bacillus anthracis, the causative agent of anthrax, requ
254                         A surface adhesin of Bacillus anthracis, the causative agent of anthrax, requ
255                                           In Bacillus anthracis, the causative agent of anthrax, tran
256                                              Bacillus anthracis, the causative agent of anthrax, util
257 We report the 1.40 A structure of a P4H from Bacillus anthracis, the causative agent of anthrax, whos
258 derophore, is required for full virulence of Bacillus anthracis, the causative agent of anthrax.
259 ria that has hitherto not been identified in Bacillus anthracis, the causative agent of anthrax.
260 activity against the Gram-positive bacterium Bacillus anthracis, the causative agent of anthrax.
261 ific bactericidal activity toward strains of Bacillus anthracis, the causative agent of anthrax.
262                                              Bacillus anthracis, the causative agent of the disease a
263 operties of metallo-beta-lactamase Bla2 from Bacillus anthracis, the enzyme was overexpressed, purifi
264 sterol-dependent cytolysin (CDC) secreted by Bacillus anthracis, the etiologic agent for anthrax.
265                                              Bacillus anthracis, the etiological agent of anthrax dis
266 ysis of a systemic bacterial infection using Bacillus anthracis, the etiological agent of anthrax dis
267    Here, we report that the peptidoglycan of Bacillus anthracis, the etiological agent of anthrax, is
268                                              Bacillus anthracis, the etiological agent of anthrax, is
269                                              Bacillus anthracis, the etiological agent of anthrax, is
270    In Bacillus cereus and its close relative Bacillus anthracis, the major pilin protein BcpA is clea
271         In common with Bacillus subtilis and Bacillus anthracis, the presence of anhydromuropeptides
272                                           In Bacillus anthracis, the siderophore petrobactin is requi
273                   The key genes required for Bacillus anthracis to cause anthrax have been acquired r
274 se anthrax lethal factor (LF) is secreted by Bacillus anthracis to promote disease virulence through
275 , GrlA(E85A), GrlA(S81F/E85A) and GrlA(S81F) Bacillus anthracis topoisomerase IV, their sensitivity t
276  macrophages, the lethal factor component of Bacillus anthracis toxin binds to a carrier protein (PA)
277 dies (MAbs) have been reported for the other Bacillus anthracis toxin components, but relatively few
278  that HNP-1 to HNP-3 inhibit lethal toxin of Bacillus anthracis, toxin B of Clostridium difficile, di
279          The protective antigen component of Bacillus anthracis toxins can interact with at least thr
280 bs) are potential therapeutic agents against Bacillus anthracis toxins, since there is no current tre
281                               We sampled the Bacillus anthracis transcriptome under a variety of grow
282 h under iron limitation, Bacillus cereus and Bacillus anthracis, two human pathogens from the Bacillu
283          In order to better characterize the Bacillus anthracis typing phage AP50c, we designed a gen
284                                 The pathogen Bacillus anthracis uses the Sortase A (SrtA) enzyme to a
285                                  The current Bacillus anthracis vaccine consists largely of protectiv
286 es were considered the primary target of the Bacillus anthracis virulence factor lethal toxin because
287         Edema toxin (ET) is one of the major Bacillus anthracis virulence factors and consists of the
288                                          The Bacillus anthracis virulence regulator AtxA controls tra
289 s of its target, the protective antigen from Bacillus anthracis We show how rational design based on
290    Using virulent and nonvirulent strains of Bacillus anthracis, we have shown that secretion of ATP
291 a potential role for NK cells in immunity to Bacillus anthracis, we utilized primary human and murine
292 the CcpA homologues of Bacillus subtilis and Bacillus anthracis were not affected by the Stk1 ortholo
293 bioterrorism agents like Yersinia pestis and Bacillus anthracis which feature on the Center for Disea
294  is initiated by endospores of the bacterium Bacillus anthracis, which are introduced into the lung.
295 threatening disease caused by infection with Bacillus anthracis, which expresses lethal factor and th
296 premature termination of hasA translation in Bacillus anthracis, which is known to escape phagocytic
297  known to confer ciprofloxacin resistance in Bacillus anthracis, Yersinia pestis, and Francisella tul
298                Forty-six distinct strains of Bacillus anthracis, Yersinia pestis, Francisella tularen
299 er water or nucleic acids from BT organisms (Bacillus anthracis, Yersinia pestis, Francisella tularen
300 elease of bacterial biothreat agents such as Bacillus anthracis, Yersinia pestis, or Burkholderia pse

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