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1 t some clinical overlap, Cowden syndrome and Bannayan-Riley-Ruvalcaba syndrome.
2 sia syndromes, including Cowden syndrome and Bannayan-Riley-Ruvalcaba syndrome.
3 EN have been shown to cause Cowden syndrome, Bannayan-Riley-Ruvalcaba syndrome and Proteus syndrome.
5 TEN mutations cause Cowden syndrome (CS) and Bannayan-Riley-Ruvalcaba syndrome (BRR), two hamartoma-t
6 TEN mutations cause Cowden syndrome (CS) and Bannayan-Riley-Ruvalcaba syndrome (BRR), two hamartoma-t
8 r hamartomatous polyposis syndromes, such as Bannayan-Riley-Ruvalcaba syndrome (BRRS) and Cowden dise
9 other inherited hamartoma syndromes, such as Bannayan-Riley-Ruvalcaba syndrome (BRRS) and Cowden synd
12 sk of breast and thyroid cancers, and 65% of Bannayan-Riley-Ruvalcaba syndrome (BRRS), characterized
13 of individuals with Cowden syndrome (CS) and Bannayan-Riley-Ruvalcaba syndrome (BRRS), respectively.
18 The three hamartomatous polyposis syndromes, Bannayan-Riley-Ruvalcaba syndrome, juvenile polyposis co
20 how that cell lines from Cowden syndrome and Bannayan-Riley-Ruvalcaba syndrome patients with germ lin
21 rlapping clinical features: Cowden syndrome, Bannayan-Riley-Ruvalcaba syndrome, Proteus syndrome and
22 yndrome" (PHTS) and include Cowden syndrome, Bannayan-Riley-Ruvalcaba syndrome, Proteus syndrome, and
23 reast, thyroid, and endometrial cancers, and Bannayan-Riley-Ruvalcaba syndrome, which is characterize
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