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1 The veterinarian was coinfected with Bartonella henselae.
2 chia chaffeensis, Rickettsia rickettsii, and Bartonella henselae.
3 ilar to the VirB type IV secretion system of Bartonella henselae.
4 been identified upstream of the htrA gene of Bartonella henselae.
6 e to the B. bacilliformis gene is present in Bartonella henselae, a bacterium that is closely related
7 at-scratch disease is an infection caused by Bartonella henselae, a fastidious gram-negative bacillus
10 ter membrane protein family of proteins from Bartonella henselae and Bartonella quintana, respectivel
11 ichia, and Rickettsia species, as well as on Bartonella henselae and Escherichia coli, and the assay
12 etect and differentiate Bartonella quintana, Bartonella henselae, and Coxiella burnetii from surgical
13 ogens Brucella abortus, Brucella melitensis, Bartonella henselae, and Legionella pneumophila, which a
16 ific-pathogen-free cats were inoculated with Bartonella henselae- and/or Bartonella clarridgeiae-infe
17 rom animals known to be infected with either Bartonella henselae, B. clarridgeiae, or B. vinsonii sub
18 athogenic Bartonella species and subspecies: Bartonella henselae, B. quintana, B. washoensis, and B.
19 m the veterinarian were not reactive against Bartonella henselae, B. quintana, or B. elizabethae anti
21 patient's blood and serum were negative for Bartonella henselae, Bartonella quintana, and B. bacilli
22 erium avium complex isolates and one each of Bartonella henselae, Bartonella quintana, Shigella flexn
25 and it facilitated the primary isolation of Bartonella henselae from blood and aqueous fluid of natu
27 rovide molecular microbiological evidence of Bartonella henselae genotype San Antonio 2 (SA2) infecti
29 aciens, Bordetella pertussis, Brucella spp., Bartonella henselae, Helicobacter pylori and Legionella
30 mechanism contributes to the persistence of Bartonella henselae in the chronically infected vascular
31 = 64, 24 (62%) also had titers > or = 64 to Bartonella henselae, indicating serologic cross-reactivi
32 f bacillary peliosis hepatis due to systemic Bartonella henselae infection in a patient after kidney
46 cal and epidemiological studies suggest that Bartonella henselae is the etiological agent of cat scra
47 with the facultative intracellular bacterium Bartonella henselae, is characterized by angiogenic lesi
49 ophils with intracellular pathogens, such as Bartonella henselae, Listeria monocytogenes, Legionella
50 rium tumefaciens and similar pathogens (e.g. Bartonella henselae) may also be able to transform human
51 oronary artery disease and seropositivity to Bartonella henselae (odds ratio [OR], 0.852; 95% confide
52 ng was conducted, eight were seroreactive to Bartonella henselae, one to E. chaffeensis, and one to R
55 egative pathogens were identified, including Bartonella henselae Pap31, Brucella Omp31, Agrobacterium
56 hat the closest homologs to HbpA include the Bartonella henselae phage-associated membrane protein, P
57 ector protein A (BepA) of vasculotumorigenic Bartonella henselae protects the infected human endothel
58 human pathogens have emerged recently (e.g., Bartonella henselae, Rickettsia felis), and their mechan
60 rtonella clarridgeiae and several strains of Bartonella henselae, the agent of cat scratch disease, w
61 in North America are Bartonella quintana and Bartonella henselae, the agents of trench fever, bacilla
62 Sera from patients who had a high titer for Bartonella henselae, the causative agent of bacillary an
64 ittens born to antibody-positive queens with Bartonella henselae to determine the contribution of ant
67 ood culture- and serum antibody-negative for Bartonella henselae, were randomly allocated to groups a
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