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1 or the treatment of ocular manifestations of Behcet's disease.
2 enic pathways in the spondyloarthritides and Behcet's disease.
3 on in the management of venous thrombosis in Behcet's disease.
4 fied 5 novel genetic susceptibility loci for Behcet's disease.
5 ve emerged as a successful approach to treat Behcet's disease.
6 venile idiopathic arthritis, sarcoidosis and Behcet's disease.
7 d the International Study Group criteria for Behcet's disease.
8 ss in patients with a familial occurrence of Behcet's disease.
9 her characteristic or diagnostic features of Behcet's disease.
10 95% confidence interval [CI], 0.19-0.74) and Behcet's disease (aHR, 0.10; 95% CI, 0.01-0.85) were ass
12 a number of inflammatory diseases, including Behcet's disease and age-related macular degeneration.
13 lvement of other autoimmune diseases such as behcet's disease and ankylosing spondylitis, and ocular
14 the genetic association between HLA-B*51 and Behcet's disease and exploring additional susceptibility
15 cause monogenic recessive diseases: MEFV in Behcet's disease and Henoch-Schonlein purpura, and A1AT
16 (GWAS) in vasculitis have been published, in Behcet's disease and Kawasaki disease; none of the genes
17 as demonstrated efficacy in the treatment of Behcet's disease and other forms of posterior uveitis.
18 udy is the first systematic genome screen in Behcet's disease and provides evidence of linkage to sev
19 ious uveitides (the ocular manifestations of Behcet's disease and sarcoidosis) based on multicolor fl
25 chemokines and its receptor in patients with Behcet's disease (BD) and their associations with diseas
26 ssociated with the autoinflammatory disorder Behcet's disease (BD) in epistasis with HLA-B*51, which
39 r uveitis cases included diagnosis with JIA, Behcet's disease, bilateral uveitis, history of cataract
41 with 311,459 SNPs in 1,215 individuals with Behcet's disease (cases) and 1,278 healthy controls from
43 correlated with disease activity in terms of Behcet's Disease Current Activity Form (BDCAF) (p < 0.00
44 und in the peripheral blood of patients with Behcet's disease during episodes of ocular inflammation.
46 anding knowledge on large vessel problems in Behcet's disease, highlighting recent contributions.
49 ed the genetic association between UBAC2 and Behcet's disease in 3 independent sets of patients and c
57 gest that the robust HLA-B*51 association in Behcet's disease is explained by a variant located betwe
62 ared to ankylosing spondylitis (p = 0.0001), behcet's disease (p = 0.0001), presumed latent tuberculo
63 pect to ankylosing spondylitis (p = 0.0001), behcet's disease, (p = 0.0001) presumed latent tuberculo
65 olving patients with ankylosing spondylitis, behcet's disease, presumed sarcoidosis, presumed latent
70 a patient who had a vasculopathy fitting the Behcet's disease type, but who lacked the other characte
74 ulitis occurs in a subgroup of patients with Behcet's disease who are at high risk for disease-relate
76 ave shown an increased rate of thrombosis in Behcet's disease, with a different clinical presentation
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