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1 albeit with an especially high abundance of Bifidobacterium.
2 tobacillus + Lactococcus (6 x 10(10) CFU/g), Bifidobacterium (1 x 10(10)/g), Propionibacterium (3 x 1
4 ial markers: Bacteroidales HF183 (HF183) and Bifidobacterium adolescentis (BifAd); one viral marker:
5 e two most dominant Bifidobacterium species, Bifidobacterium adolescentis and Bifidobacterium pseudoc
6 ned with the GH5 enzyme stimulated growth of Bifidobacterium adolescentis but not of Lactobacillus br
7 an symbiont bacterial species, in particular Bifidobacterium adolescentis, that could, alone, induce
8 abundance of certain bacteria (for example, Bifidobacterium, Akkermansia and Faecalibacterium), high
10 ment of early microbiota and to increase the Bifidobacterium amounts as observed in human-milk-fed in
11 cteroides, Roseburia-Eubacterium rectale and Bifidobacterium and an increase of Enterobacteriaceae, D
14 that the relative abundance of Allobaculum, Bifidobacterium and Coriobacteriaceae taxa associated wi
15 ignificant increases in species of the genus Bifidobacterium and decreases in Bacteroides vulgatus wi
17 against select gut commensals (Bacteroides, Bifidobacterium and Lactobacillus species), and were non
18 support a relationship between abundances of Bifidobacterium and of putative pathogens or a significa
24 d milk product containing dairy starters and Bifidobacterium animalis potentiates colonic short chain
25 Escherichia coli, Lactobacillus acidophilus, Bifidobacterium animalis subsp lactis, Faecalibacterium
27 and EP-HN019, 1 mL of suspensions containing Bifidobacterium animalis subsp. lactis (B. lactis) HN019
28 ei subsp. paracasei (L. casei 01); QB - with Bifidobacterium animalis subsp. lactis (BB 12); and QC,
30 comprising, Lactobacillus acidophilus LA-5, Bifidobacterium animalis subsp. lactis BB-12 and Propion
31 assette (ABC) transporter from the probiotic Bifidobacterium animalis subsp. lactis Bl-04 binds alpha
32 consumed a fermented milk product containing Bifidobacterium animalis subsp. lactis DN-173 010 strain
33 ct of 4-week use of yogurt supplemented with Bifidobacterium animalis subsp. lactis DN-173010 versus
34 robiotic fermented milks were produced using Bifidobacterium animalis subsp. lactis HN019 in co-cultu
35 of the study was to investigate the role of Bifidobacterium animalis subsp. lactis in preventing nos
37 ysiological responses of two fully sequenced Bifidobacterium animalis subsp. lactis strains, BL-04 an
38 sei IMVB-7280, Bifidobacterium animalis VKL, Bifidobacterium animalis VKB) at a dose of 50 mg/kg (5 x
39 trains (2:1:1 Lactobacillus casei IMVB-7280, Bifidobacterium animalis VKL, Bifidobacterium animalis V
40 bacillus reuteri, Lactobacillus casei GG, or Bifidobacterium animalis) in the gastrointestinal tracts
41 acillus reuteri, Lactobacillus casei GG, and Bifidobacterium animalis) to colonize, infect, stimulate
42 quencing of the 16S ribosomal RNA identified Bifidobacterium as associated with the antitumor effects
43 er of Enterococcus, Prevotella, Bacteroides, Bifidobacterium, Bacteroides uniformis, Eggerthella lent
44 cillus acidophilus, Lactobacillus rhamnosus, Bifidobacterium bifidum and Streptococcus faecium) in ca
45 ether consuming Lactobacillus gasseri KS-13, Bifidobacterium bifidum G9-1, and B. longum MM-2 compare
46 n conducted until now, inferred pathways for Bifidobacterium bifidum include perchlorate reduction vi
48 f Bifidobacterium longum subsp. infantis and Bifidobacterium bifidum using individual HMO, and compar
49 tal of 10(7) colony-forming units (CFU)/g of Bifidobacterium bifidum, Bifidobacterium breve, Bifidoba
50 obacillus rhamnosum, Bifidobacterium longum, Bifidobacterium bifidum, Saccharomyces boulardi, Sacchar
51 rmentation of okara(ET) by a pure culture of Bifidobacterium bifidus was mainly represented by acetic
53 d to test the effectiveness of the probiotic Bifidobacterium breve BBG-001 to reduce necrotising ente
54 g fed with a diet containing scGOS/lcFOS and Bifidobacterium breve M-16V (GF/Bb) or a control diet.
55 ism by which scGOS/lcFOS in combination with Bifidobacterium breve M-16V protects against acute aller
56 supplementation for 8 wk with human-derived Bifidobacterium breve strains on fat distribution and co
58 5 transposon mutant library of the commensal Bifidobacterium breve UCC2003 that was further character
59 ng units (CFU)/g of Bifidobacterium bifidum, Bifidobacterium breve, Bifidobacterium longum, B. longum
60 otics had a significantly lower abundance of Bifidobacterium (by 55%; 95% CI, 43% to 87%; P = .006; a
61 nt, probiotic, Lactobacillus, Saccharomyces, Bifidobacterium, Candida, gastrointestinal- system, vagi
62 Veillonellaceae sp. HOT 155 (p < 0.01), and Bifidobacterium Cluster 1 (p = 0.11), and by qPCR, Strep
63 antitative perspective of the early-life gut Bifidobacterium colonization and shows how factors such
64 myces odontolyticus, Actinomyces meyeri, and Bifidobacterium dentium were all positive for so-called
65 Relative abundance of lactose-fermenting Bifidobacterium, Faecalibacterium, and Lactobacillus wer
67 association of a functional LCT SNP with the Bifidobacterium genus (P = 3.45 x 10(-8)) and provide ev
68 enetic variability of (pro)phages within the Bifidobacterium genus, a dominant bacterial group of the
70 s, Enterobacteriaceae, Escherichia coli, and Bifidobacterium in apple cultured faeces tended to resem
72 utes, increased Bacteroidetes, and decreased Bifidobacterium in the microbiome of AD participants.
75 ulating material for two probiotic bacteria: Bifidobacterium infantis and Lactobacillus plantarum by
76 e changes are prevented by administration of Bifidobacterium infantis, a probiotic known to decrease
77 ed the aggregation of a rod-shaped bacteria, Bifidobacterium infantis, during capillary electrophores
79 linical significance of 15 A. omnicolens and Bifidobacterium isolates identified by 16S rRNA gene seq
80 movements/wk), and this was significant for Bifidobacterium lactis (WMD: 1.5 bowel movements/wk; 95%
81 x 10(7) colony-forming units (CFU)/g each of Bifidobacterium lactis and Streptococcus thermophilus, f
82 (Lactobacillus rhamnosus strain GG [LGG] and Bifidobacterium lactis Bb12 [Bb12]), mimicking gut comme
83 with Lactobacillus paracasei CNCM I-2116 or Bifidobacterium lactis CNCM I-3446 had a treatment effec
84 er all 28 days, the cheese supplemented with Bifidobacterium lactis in its isolated form showed the h
85 probiotics, Lactococcus lactis NCC 2287 and Bifidobacterium lactis NCC 2818, were tested in a murine
86 biota co-metabolism were further modified by Bifidobacterium lactis NCC2818 supplementation, although
87 nd that, when orally administered to humans, Bifidobacterium longum AH1206 stably persists in the gut
88 e we investigate how two commensal bacteria, Bifidobacterium longum and Bacteroides fragilis, represe
89 ning Bifidobacterium pseudocatenulatum G4 or Bifidobacterium longum BB536 on plasma lipids, lipid per
90 receive (1) Lactobacillus rhamnosus LPR and Bifidobacterium longum BL999 (LPR+BL999), (2) L paracase
91 eloped to quantify the consumption of FOS by Bifidobacterium longum bv. infantis using a calibration
92 te genome sequence of an intestinal isolate, Bifidobacterium longum DJO10A that was minimally culture
94 actobacillus delbrueckii ssp. bulgaricus and Bifidobacterium longum in soymilk supplemented with tea
96 prospective study to evaluate the effects of Bifidobacterium longum NCC3001 (BL) on anxiety and depre
97 In this study, the mechanisms through which Bifidobacterium longum strain BB68 affects the longevity
100 d the utility of a beta-1,3-galactosidase in Bifidobacterium longum subsp. infantis ATCC 15697 (B. in
101 Accordingly, the complete genome sequence of Bifidobacterium longum subsp. infantis ATCC15697 reflect
103 llel glycoprofiling documented that numerous Bifidobacterium longum subsp. infantis strains preferent
104 icular infant-associated commensals, such as Bifidobacterium longum subsp. infantis, consume neutral
105 In our hospital, we documented 2 cases of Bifidobacterium longum subspecies infantis bacteremia in
107 idobacterium bifidum, Bifidobacterium breve, Bifidobacterium longum, B. longum subspecies infantis (i
108 illus acidophillus, Lactobacillus rhamnosum, Bifidobacterium longum, Bifidobacterium bifidum, Sacchar
111 iota is often colonized by two subspecies of Bifidobacterium longum: subsp. infantis (B. infantis) an
112 t role in caries initiation and that a novel Bifidobacterium may be a major pathogen in deep caries.
114 istration of probiotic bacteria of the genus Bifidobacterium on experimental periodontitis (EP) in ra
115 igation was to develop baking products using Bifidobacterium pseudocatenulatum ATCC27919, a phytase p
116 he effect of a yoghurt supplement containing Bifidobacterium pseudocatenulatum G4 or Bifidobacterium
117 um species, Bifidobacterium adolescentis and Bifidobacterium pseudocatenulatum, and an increased abun
118 sing cell numbers, Actinomyces gerencseriae, Bifidobacterium, S. mutans, Veillonella, S. salivarius,
119 trials (RCTs) of probiotics (Lactobacillus, Bifidobacterium, Saccharomyces, Streptococcus, Enterococ
120 four species: A. omnicolens (five isolates), Bifidobacterium scardovii (four isolates), B. longum (tw
121 inal bacterial genera such as Lactobacillus, Bifidobacterium, Snodgrassella, and Gilliamella were det
123 ry, the quantity of Lactic Acid Bacteria and Bifidobacterium sp. increased concurrently during the co
124 acillus casei, L. reuteri, L. acidophilus, a Bifidobacterium sp., Lactococcus lactis, or a Bacillus s
125 arriage of eight signature infant-associated Bifidobacterium species (B. longum, B. breve, B. bifidum
128 mentation of GOS selectively increased fecal Bifidobacterium species abundance, but this did not prod
129 al sources associated with A. omnicolens and Bifidobacterium species and addresses identification pro
131 but not placebo, increased the abundance of Bifidobacterium species in feces by 5-fold (P = .009; q
132 Breast milk enhances the predominance of Bifidobacterium species in the infant gut, probably due
133 his, we have determined the ability of three Bifidobacterium species isolated from the faeces of newb
134 omparative genome sequence analysis of three Bifidobacterium species provided a core genome set of 1,
135 udy highlights two major strategies found in Bifidobacterium species to process HMO, and presents det
136 placebo (192 +/- 93) (P = .721) Abundance of Bifidobacterium species was lower in fecal samples from
137 addition, a dearth of the two most dominant Bifidobacterium species, Bifidobacterium adolescentis an
138 e multistrain probiotic increased numbers of Bifidobacterium species, compared with placebo, and migh
139 es involved in immunity development, such as Bifidobacterium species, Sutturella wadsworthia, and Clo
143 rectale (cluster XIVab), Bacteroidetes, and Bifidobacterium spp, but decreased segmented filamentous
144 s a significant time-by-group interaction on Bifidobacterium spp. (P = 0.008) and Lactobacillus/Pedio
146 in a transient increase in the abundance of Bifidobacterium spp. and Clostridium spp. in fecal sampl
147 ere was a nonsignificant trend toward higher Bifidobacterium spp. in the Fe+GOS group (P = 0.099).
148 in reaction showed a significant increase in Bifidobacterium spp. in the OI group compared with contr
150 ase (p < 0.01) in the Lactobacillus spp. and Bifidobacterium spp. populations was observed during the
152 sal bacterial communities (Bacteroides spp., Bifidobacterium spp., Clostridium leptum group, Enteroba
153 omoted growth of beneficial bacteria such as Bifidobacterium spp., Lactobacillus spp., Bacteroides ac
155 he-shelf probiotic preparations that include Bifidobacterium strains also drove intestinal Th17 cell
156 obacillus and mutans streptococci (MS), most Bifidobacterium strains and non-mutans streptococci (non
159 Here, genome analyses of 47 representative Bifidobacterium (sub)species revealed the genes predicte
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