ライフサイエンスコーパス: Bordetella pertussis

1 ainst the endemic human respiratory pathogen Bordetella pertussis.

2 e phenomenology of infection and immunity to Bordetella pertussis.

3 rotection against both B. bronchiseptica and Bordetella pertussis.

4 respiratory disease caused by the bacterium Bordetella pertussis.

5 rom an IncP-1b plasmid, pBP136 isolated from Bordetella pertussis.

6 ry disease of humans caused by the bacterium Bordetella pertussis.

7 is an important virulence factor produced by Bordetella pertussis.

8 n AB(5) toxin produced by the human pathogen Bordetella pertussis.

9 ssis toxin (PT) across the outer membrane of Bordetella pertussis.

10 in, an autotransporter passenger domain from Bordetella pertussis.

11 nserved in both Bordetella parapertussis and Bordetella pertussis.

12 ines may not protect against transmission of Bordetella pertussis.

13 isease caused by the obligate human pathogen Bordetella pertussis.

14 regulates expression of virulence factors in Bordetella pertussis.

15 chiseptica and the expression of vrg loci in Bordetella pertussis.

16 ence-associated filamentous hemagglutinin of Bordetella pertussis.

17 in homologues of the wlbA and wlbL genes of Bordetella pertussis.

18 ly related CyaC acyltransferase expressed by Bordetella pertussis.

19 recognized by mAb prepared against Fim2 from Bordetella pertussis.

20 ng E. coli and with pertactin precursor from Bordetella pertussis.

21 r the secretion of pertussis toxin (PT) from Bordetella pertussis.

22 bly and secretion of the holotoxin fusion by Bordetella pertussis.

23 irulence regulon of the whooping-cough agent Bordetella pertussis.

24 (CyaA) plays a key role in the virulence of Bordetella pertussis.

25 ory illness caused by the bacterial pathogen Bordetella pertussis.

26 on and virulence of the whooping cough agent Bordetella pertussis.

27 ory illness caused by the bacterial pathogen Bordetella pertussis.

28 ory illness caused by the bacterial pathogen Bordetella pertussis.

29 The limits of detection were as follows: for Bordetella pertussis, 2 CFU/ml; for Legionella pneumophi

30 Bordetella pertussis, a causative agent of whooping coug

31 We have used Bordetella pertussis, a common neonatal respiratory trac

32 tion and failure to induce direct killing of Bordetella pertussis, a synthetic scheme was devised for

33 Bordetella pertussis adenylate cyclase (AC) toxin belong

34 f YipB and the enzymatic active component of Bordetella pertussis adenylate cyclase (Cya) was translo

35 ESAT-6 or CFP10 with genetically detoxified Bordetella pertussis adenylate cyclase (CyaA) are recogn

36 Bordetella pertussis adenylate cyclase (CyaA) toxin has

37 rmational behavior of an RTX domain from the Bordetella pertussis adenylate cyclase consisting of nin

38 in-dependent adenylate cyclase domain of the Bordetella pertussis adenylate cyclase protein was trans

39 The catalytic domain of Bordetella pertussis adenylate cyclase toxin (ACT) trans

40 Epinephrine and Bordetella pertussis adenylate cyclase toxin (ACT), cAMP

41 The use of genetically detoxified Bordetella pertussis adenylate cyclase toxin (CyaA) as a

42 -residue receptor-binding domain (RD) of the Bordetella pertussis adenylate cyclase toxin CyaA fused

43 and T25 fragments of the catalytic domain of Bordetella pertussis adenylate cyclase were fused to CTA

44 sts involving C-terminal fusions with a Cya (Bordetella pertussis adenylate cyclase) reporter indicat

45 CR3 serves as the receptor for the Bordetella pertussis adhesin filamentous hemagglutinin (

46 (FHA) is a dominant cell surface-associated Bordetella pertussis adhesin.

47 s toxin (PT), a secreted virulence factor of Bordetella pertussis, ADP ribosylates mammalian G(i) pro

48 Antibody-dependent complement killing of Bordetella pertussis after immunization with a three-com

49 ells stimulated with LPS or heat-inactivated Bordetella pertussis Ag.

50 Temporal expression patterns of the Bordetella pertussis alcaligin, enterobactin and haem ir

51 Pathogenic microorganisms such as Bordetella pertussis also express proteins with lectin-l

52 Bordetella pertussis, an obligate human pathogen and the

53 The persistence of Bordetella pertussis and B. parapertussis within vaccina

54 Two highly infectious bordetellae, Bordetella pertussis and B. parapertussis, have emerged

55 be caused by two closely related pathogens, Bordetella pertussis and B. parapertussis.

56 in two closely related respiratory pathogens Bordetella pertussis and Bordetella bronchiseptica Altho

57 pproximately 10% of the annotated genomes of Bordetella pertussis and Bordetella bronchiseptica and c

58 Bordetella pertussis and Bordetella bronchiseptica are c

59 Bordetella pertussis and Bordetella bronchiseptica are t

60 Bordetella pertussis and Bordetella bronchiseptica estab

61 ium at pH values ranging from pH 6.0 to 7.6, Bordetella pertussis and Bordetella bronchiseptica FtrAB

62 tin siderophore by the respiratory pathogens Bordetella pertussis and Bordetella bronchiseptica is de

63 We investigated Bordetella pertussis and Bordetella bronchiseptica LPS-d

64 Bordetella pertussis and Bordetella bronchiseptica rely

65 omparison of murine respiratory infection by Bordetella pertussis and Bordetella bronchiseptica strai

66 Bordetella pertussis and Bordetella bronchiseptica, gram

67 Bordetella pertussis and Bordetella bronchiseptica, whic

68 erophore biosynthesis and transport genes in Bordetella pertussis and Bordetella bronchiseptica.

69 Bordetella pertussis and Bordetella parapertussis are cl

70 Of the three Bordetella species, Bordetella pertussis and Bordetella parapertussis are no

71 dy confirmed that Bordetella bronchiseptica, Bordetella pertussis and Bordetella parapertussis have t

72 PCR assay for detecting and differentiating Bordetella pertussis and Bordetella parapertussis in nas

73 fections, we challenged IL-1R(-/-) mice with Bordetella pertussis and Bordetella parapertussis, the c

74 ipA homologues are expressed in Bvg(i) phase Bordetella pertussis and Bordetella parapertussis.

75 ates during 2008-2010 were tested by PCR for Bordetella pertussis and Bordetella parapertussis.

76 the activities of adenylyl cyclase toxin of Bordetella pertussis and edema toxin of Bacillus anthrac

77 ci is identical to the tracheal cytotoxin of Bordetella pertussis and has been shown to kill ciliated

78 ne-containing compounds to support growth of Bordetella pertussis and influence pertussis toxin trans

79 ce protection from mucosal pathogens such as Bordetella pertussis and influenza virus.

80 We introduced these mutations into Bordetella pertussis and monitored pertussis toxin produ

81 tes the expression of the virulence genes of Bordetella pertussis and negatively regulates a second s

82 nostic PCR target for discriminating between Bordetella pertussis and other Bordetella species that a

83 factor homologous to adenylyl cyclases from Bordetella pertussis and Pseudomonas aeruginosa, is high

84 PTx) is a major virulence factor produced by Bordetella pertussis and, in its detoxified form PTd, is

85 within the inactivated adenylate cyclase of Bordetella pertussis), and purified protein derivative o

86 romoter region prevent batB transcription in Bordetella pertussis, and although expressed, the batB g

87 ARGs in the manures were Bacillus anthracis, Bordetella pertussis, and B. anthracis (sulfonamide resi

88 mologous to the filamentous hemagglutinin of Bordetella pertussis, and MchB has homology to other Tps

89 Intestinal parasites, Bordetella pertussis, and other infectious disease agent

90 ogenic species, such as Helicobacter pylori, Bordetella pertussis, and Pseudomonas aeruginosa.

91 e of pertactin, an AT virulence protein from Bordetella pertussis, and show that OM secretion of the

92 BvgS sensor kinase in its phosphorylation in Bordetella pertussis, and the effect of the T194M mutati

93 from infection of the respiratory tract with Bordetella pertussis, and the secreted adenylate cyclase

94 tain bacteria (e.g., Haemophilus influenzae, Bordetella pertussis, and Vibrio cholerae) due to substi

95 , Shigella flexneri, Pseudomonas aeruginosa, Bordetella pertussis, and Yersinia pestis), has prompted

96 sms, performs haem ligation to a coexpressed Bordetella pertussis apocytochrome c in an Escherichia c

97 Bordetella parapertussis and Bordetella pertussis are closely related species that ca

98 Current acellular vaccines against Bordetella pertussis are effective in preventing severe

99 Lipopolysaccharides (LPS) of Bordetella pertussis are important modulators of the imm

100 ases of pertussis (whooping cough) caused by Bordetella pertussis are on the rise, and understanding

101 ssis toxin (Ptx) expression and secretion in Bordetella pertussis are regulated by a two-component si

102 s of antimicrobial susceptibility testing of Bordetella pertussis are time consuming and require spec

103 usely adherent Escherichia coli, and BrkA of Bordetella pertussis, are localized to the bacterial pol

104 Environmental factors, such as Bordetella pertussis, are thought to sensitize central e

105 of IS481, a frequent target for diagnosis of Bordetella pertussis, as approximately 5%.

106 e the incidence and clinical presentation of Bordetella pertussis-associated hospitalization in perin

107 riaceae (SPATEs) and the pertactin family of Bordetella pertussis autotransporters is released from t

108 ular pertussis vaccines and the evolution of Bordetella pertussis away from vaccine-mediated immunity

109 A comparative analysis of the Bordetella pertussis, B. bronchiseptica, and B. parapert

110 Bordetella pertussis, B. parapertussis, and B. bronchise

111 T-PCR) assay for the rapid identification of Bordetella pertussis, B. parapertussis, and B. holmesii

112 vant, and treated simultaneously with killed Bordetella pertussis bacteria, as indicated.

113 trophil phagocytosis in host defense against Bordetella pertussis, bacteria were labeled extrinsicall

114 the fimbrial subunit genes fim2 and fim3 of Bordetella pertussis behave differently from each other

115 FhaC is an outer membrane transporter from Bordetella pertussis belonging to the two-partner secret

116 arison of real-time PCR positivity rates for Bordetella pertussis between specimens collected with ra

117 he discovery that the adenylate cyclase from Bordetella pertussis binds to the CD11b/CD18 integrin, w

118 nt diagnostic tool for the identification of Bordetella pertussis, Bordetella holmesii, and Bordetell

119 Bordetella pertussis, Bordetella parapertussis and Borde

120 Bordetella pertussis, Bordetella parapertussis, and Bord

121 or nasal administration of highly attenuated Bordetella pertussis BPZE1 provides effective and sustai

122 The Bordetella pertussis BrkA protein protects against the b

123 pathogens such as Agrobacterium tumefaciens, Bordetella pertussis, Brucella spp., Bartonella henselae

124 n established role in protective immunity to Bordetella pertussis, but this evidence is based largely

125 Bordetella pertussis BvgA is a global response regulator

126 Pertussis toxin (PT) is secreted from Bordetella pertussis by a type IV secretion system, know

127 died of respiratory illness were tested for Bordetella pertussis by conventional and real-time PCR a

128 either promote or antagonize phagocytosis of Bordetella pertussis by human neutrophils depending on w

129 d for the ability to promote phagocytosis of Bordetella pertussis by human neutrophils.

130 is transported across the outer membrane of Bordetella pertussis by the type IV secretion system kno

131 In infants, Bordetella pertussis can cause severe disease, manifeste

132 Bordetella pertussis causes a profound inflammatory resp

133 Bordetella pertussis causes severe respiratory illness a

134 Bordetella pertussis causes whooping cough, an endemic r

135 T), a monomer of DAP-type peptidoglycan from Bordetella pertussis, causes cytopathology in the respir

136 thogenic bacteria Pseudomonas aeruginosa and Bordetella pertussis contain in their outer membranes th

137 Bordetella pertussis contributed to a modest proportion

138 s toxin (PT), a virulence factor secreted by Bordetella pertussis, contributes to respiratory tract i

139 ependent adenylate cyclase gene derived from Bordetella pertussis (cyaA').

140 endent adenylate cyclase domain (Cya) of the Bordetella pertussis cyclolysin was used as a reporter p

141 f vaccinated adults are being diagnosed with Bordetella pertussis disease.

142 ite widespread childhood vaccination against Bordetella pertussis, disease remains prevalent.

143 office tested positive for low quantities of Bordetella pertussis DNA, we suspected prelaboratory con

144 The adenylate cyclase toxin (ACT) of Bordetella pertussis does not require a receptor to gene

145 rtussis toxin, we identified and mutated the Bordetella pertussis dsbA, dsbB, and dsbC homologues.

146 y of recombinant adenylate cyclase (CyaA) of Bordetella pertussis (EC ) by conductimetric determinati

147 ry acylation catalyzed by LpxL proteins from Bordetella pertussis, Escherichia coli, and Haemophilus

148 iverse microbes, including Candida glabrata, Bordetella pertussis, Escherichia coli, and L. pneumophi

149 Bordetella pertussis establishes infection by attaching

150 better-characterized TPS systems specify the Bordetella pertussis FHA and Haemophilus influenzae HMW1

151 Comparison of HMW1-PP and the Bordetella pertussis FHA secretion domain (Fha30) reveal

152 t homology with the N-terminal region of the Bordetella pertussis filamentous hemagglutinin (FHA), wh

153 ter-membrane, beta-barrel transporter of the Bordetella pertussis filamentous hemagglutinin adhesin.

154 A prominent feature of the promoters of Bordetella pertussis fimbrial subunit genes fim2, fim3 a

155 of recombinant adenylate cyclase toxin from Bordetella pertussis for the development of protective a

156 respiratory disease caused by the bacterium Bordetella pertussis, for which humans are the only know

157 Bordetella bronchiseptica and Bordetella pertussis form biofilms on abiotic surfaces a

158 hat we isolated Bordetella holmesii, but not Bordetella pertussis, from patients with pertussis-like

159 ells in immunity to the respiratory pathogen Bordetella pertussis gammadelta T cells, predominantly V

160 )-neutral (Escherichia coli) and (G+C)-rich (Bordetella pertussis) genomes.

161 n in vitro reactions, or in crude lysates of Bordetella pertussis grown under varying laboratory cond

162 n of growth phase-related gene regulation in Bordetella pertussis has been reported previously.

163 Infection of mice and pigs with Bordetella pertussis has many features of the infection

164 eBABE-conjugated BvgA to the fha promoter of Bordetella pertussis has revealed that three dimers, for

165 ter membrane receptor proteins, and although Bordetella pertussis has the bfrD and bfrE genes, the ro

166 reatment failures and in vitro resistance of Bordetella pertussis have been reported on several occas

167 nts and mammals - Agrobacterium tumefaciens, Bordetella pertussis, Helicobacter pylori and Legionella

168 The Bordetella pertussis heme utilization gene cluster hurIR

169 Bordetella pertussis identification was common among you

170 ory techniques are changing the landscape of Bordetella pertussis illness diagnosis.

171 rom living asymptomatic infected mice and of Bordetella pertussis in 1 microl of nasal aspirate from

172 is a paucity of data regarding the burden of Bordetella pertussis in African women and young infants,

173 thromycin resistance was first recognized in Bordetella pertussis in Arizona in 1994, and since then,

174 disease similar to whooping cough caused by Bordetella pertussis in children.

175 aterials performed similarly for recovery of Bordetella pertussis in culture.

176 nsertion sequence IS481 for the detection of Bordetella pertussis in nasopharyngeal swab specimens.

177 a provide evidence of ongoing circulation of Bordetella pertussis in the region.

178 is the most common method used for detecting Bordetella pertussis in the United States, most laborato

179 Diagnosis of Bordetella pertussis infection has been difficult due to

180 ulted in higher childhood mortality than did Bordetella pertussis infection in 2003-2004.

181 Bordetella pertussis infection may be a more common caus

182 Bordetella pertussis infection of the airways causes the

183 A previous study used a murine model of Bordetella pertussis infection to demonstrate that treat

184 human monocytes to mediate the clearance of Bordetella pertussis infection was examined.

185 Bordetella pertussis infections and illnesses were prosp

186 tions for the use of PCR in the diagnosis of Bordetella pertussis infections have been proposed, and

187 reased the ability of laboratories to detect Bordetella pertussis infections, it has also been associ

188 The BrkA protein of Bordetella pertussis inhibits killing by the antibody-de

189 such as the secretion of pertussis toxin by Bordetella pertussis into human cells and the delivery o

190 The adenylate cyclase toxin (ACT) of Bordetella pertussis intoxicates target cells by generat

191 Bordetella pertussis is a human pathogen that can infect

192 The filamentous hemagglutinin/FhaC pair of Bordetella pertussis is a model two-partner secretion sy

193 Bordetella pertussis is a strictly human pathogen.

194 Here, it is shown that Bordetella pertussis is an excellent bacterial model for

195 Bordetella pertussis is an important cause of infection

196 ial and sufficient to prevent infection with Bordetella pertussis is derived from data from animal an

197 Bordetella pertussis is highly infectious among humans b

198 Pertussis toxin of Bordetella pertussis is secreted by a type IV secretion

199 Bordetella pertussis is the causative agent of whooping

200 The Gram-negative bacterium Bordetella pertussis is the causative agent of whooping

201 While the human pathogen Bordetella pertussis is the etiological agent of the acu

202 Natural infection with Bordetella pertussis is thought to result in 4-20 years

203 cough, caused by the obligate human pathogen Bordetella pertussis is undergoing a worldwide resurgenc

204 Adenylate cyclase (AC) toxin from Bordetella pertussis is unusual in that, unlike most oth

205 del toxin, the adenylate cyclase (CyaA) from Bordetella pertussis, is able to invade eukaryotic cells

206 xin virulence factor produced exclusively by Bordetella pertussis, is important for colonization of t

207 Pertussis (whooping cough), caused by Bordetella pertussis, is resurging in the United States

208 nomic organization of 14 clinical strains of Bordetella pertussis isolated over an 18-month period in

209 A total of 194 Bordetella pertussis isolates collected from 2008 throug

210 Bordetella pertussis isolates collected in Cincinnati fr

211 Bordetella pertussis isolates from 9 school cases and fr

212 Bordetella pertussis isolation by culture has low detect

213 Bordetella pertussis lacks O antigen and is sensitive to

214 Other pathogens, including Bordetella pertussis, Legionella pneumophila, Brucellasp

215 Bordetella pertussis LPS has a branched core structure a

216 )), Bordetella parapertussis (lpxA(Pa)), and Bordetella pertussis (lpxA(Pe)) was cloned and expressed

217 tigenic divergence of circulating strains of Bordetella pertussis might have on the efficacy of pertu

218 We used the murine Bordetella pertussis model to test the hypothesis that t

219 effect of adenylate cyclase (AC) toxin from Bordetella pertussis on host cells has been attributed t

220 tion between human neutrophils and wild-type Bordetella pertussis or mutants expressing altered lipop

221 ordetella bronchiseptica, Bordetella hinzii, Bordetella pertussis, or Bordetella parapertussis.

222 lase (AC) toxin is present on the surface of Bordetella pertussis organisms and their addition to euk

223 say on the BD Max system versus our in-house Bordetella pertussis PCR.

224 oding for two important virulence factors of Bordetella pertussis, pertactin and pertussis toxin, and

225 own ADP-ribosyltransferases (ADPRTs) such as Bordetella pertussis pertussis toxin and Mycoplasma pneu

226 (e.g., Bacillus anthracis, Yersinia pestis, Bordetella pertussis, Plasmodium falciparum, and Mycobac

227 T), an exotoxin virulence factor produced by Bordetella pertussis, plays an important early role in c

228 al swabs and induced sputum (cases only) for Bordetella pertussis polymerase chain reaction.

229 Our results indicate that the Bordetella pertussis population causing this epidemic wa

230 virulence factor of the whooping cough agent Bordetella pertussis, preferentially binds an inactive f

231 Bordetella pertussis produces a 73-kDa protein, BrkA (Bo

232 Bordetella pertussis produces multiple virulence factors

233 in pertactin and pertussis toxin, which are Bordetella pertussis proteins important for pathogenesis

234 ertussis and the identification of antigenic Bordetella pertussis proteins support the hypothesis tha

235 ussis vaccines containing 1 or more purified Bordetella pertussis proteins.

236 an pathogens as G. lamblia, Yersinia pestis, Bordetella pertussis, Pseudomonas aeruginosa, Vibrio cho

237 umefaciens VirB/VirD4, E. coli R388 Trw, and Bordetella pertussis Ptl systems support conjugative DNA

238 ssion of the RNA polymerase alpha subunit in Bordetella pertussis reduces expression of the virulence

239 The whooping cough agent Bordetella pertussis regulates the production of its vir

240 es of research and vaccination, infection by Bordetella pertussis remains a serious disease with no s

241 pathway for c-type cytochrome biogenesis in Bordetella pertussis requires at least four biogenesis p

242 utilization by Bordetella bronchiseptica and Bordetella pertussis requires the BfeA outer membrane re

243 tbreaks with documented cases of B. holmesii/Bordetella pertussis respiratory coinfection; whether th

244 mortality in non-vaccinated young children, Bordetella pertussis results in milder and prolonged cou

245 n complexes made with RNA polymerase and the Bordetella pertussis RR, BvgA, in its nonphosphorylated

246 ce of the whooping cough causative bacterium Bordetella pertussis Secreted as soluble protein, it tar

247 Legionella pneumophila, Legionella micdadei, Bordetella pertussis, Staphylococcus aureus, and Strepto

248 Bordetella pertussis strains lacking expression of perta

249 lities of the entire PFGE profiles for three Bordetella pertussis strains.

250 ella bronchiseptica) and human-adapted (e.g. Bordetella pertussis) strains produce a surface-exposed

251 onent of the two-partner secretion system in Bordetella pertussis, suggests that the BamA beta-barrel

252 ious studies have reported that phagocytosed Bordetella pertussis survives in human neutrophils.

253 ate cyclase from the whooping cough pathogen Bordetella pertussis, synthesizes the second messenger b

254 , S2, and/or S4 subunits of PT in strains of Bordetella pertussis that either did or did not produce

255 nsidered the "master virulence regulator" in Bordetella pertussis, the causal agent of pertussis, and

256 Bordetella pertussis, the causative agent of human whoop

257 Bordetella pertussis, the causative agent of the acute c

258 The source of the muramyl peptide is Bordetella pertussis, the causative agent of the respira

259 been observed in other organisms, including Bordetella pertussis, the causative agent of whooping co

260 Bordetella pertussis, the causative agent of whooping co

261 interaction with wild-type and mutant LPS of Bordetella pertussis, the causative agent of whooping co

262 Bordetella pertussis, the causative agent of whooping co

263 Bordetella pertussis, the causative agent of whooping co

264 Bordetella pertussis, the causative agent of whooping co

265 Bordetella pertussis, the causative agent of whooping co

266 owed that strain BP338 of the human pathogen Bordetella pertussis, the causative agent of whooping co

267 e of the major virulence factors produced by Bordetella pertussis, the causative agent of whooping co

268 AB5-type exotoxin produced by the bacterium Bordetella pertussis, the causative agent of whooping co

269 Bordetella pertussis, the causative agent of whooping co

270 In the human respiratory pathogen Bordetella pertussis, the causative agent of whooping co

271 Bordetella pertussis, the etiologic agent of whooping co

272 CyaA is crucial for colonization by Bordetella pertussis, the etiologic agent of whooping co

273 T cell response relative to that induced by Bordetella pertussis, the more common cause of pertussis

274 Bordetella pertussis, the pathogenic bacteria responsibl

275 in, one of the virulence factors secreted by Bordetella pertussis, the pathogenic bacteria responsibl

276 es to prevent nasopharyngeal colonization by Bordetella pertussis, the principal causative agent of w

277 These interventions result in elevated anti-Bordetella pertussis titers, but there have been no stud

278 of infant morbidity and mortality caused by Bordetella pertussis To better inform such interventions

279 (AC) toxin is an essential toxin that allows Bordetella pertussis to invade eukaryotic cells, where i

280 roteins encoded by the bipA genes present in Bordetella pertussis Tohama I and Bordetella bronchisept

281 nd that dendritic cell-activating adjuvants [Bordetella pertussis toxin (PT) or CpG ODN or a squalene

282 In vivo intoxication with Bordetella pertussis toxin (PTX) elicits a variety of ph

283 Bordetella pertussis toxin (PTX) promotes insulin secret

284 e role of VWF and/or Weibel-Palade bodies in Bordetella pertussis toxin-induced hypersensitivity to h

285 ur observations with HMW1B, examination of a Bordetella pertussis TpsB protein called FhaC revealed t

286 zed syndromic case definition and tested for Bordetella pertussis using real-time polymerase chain re

287 that are required for efficient export from Bordetella pertussis via the Ptl system, a member of the

288 e role of pertussis toxin (PT), an important Bordetella pertussis virulence factor, in lung transcrip

289 Bordetella pertussis was detected in 2.1% (n = 26/1257)

290 Bordetella pertussis was detected in 53 of 4200 (1.3%) c

291 Bordetella pertussis was diagnosed in a human immunodefi

292 d mortality after established infection with Bordetella pertussis was explored.

293 A PCR test for Bordetella pertussis was primarily used by a private lab

294 Transposon mutagenesis of Bordetella pertussis was used to discover mutations in t

295 fundamental nature of protective immunity to Bordetella pertussis, we studied intranasal immunization

296 tested with multitarget PCR; B. holmesii and Bordetella pertussis were exclusively detected among 48

297 a 188-kDa adenylyl cyclase toxin secreted by Bordetella pertussis, which is the etiologic agent for w

298 Pertussis toxin is secreted from Bordetella pertussis with the assistance of the Ptl tran

299 n has recently been demonstrated to occur in Bordetella pertussis, with many transcripts, including k

300 A recent increase in Bordetella pertussis without the pertactin protein, an a