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1 curring triacylated lipoprotein agonist from Borrelia burgdorferi.
2 olonization for the Lyme disease spirochaete Borrelia burgdorferi.
3 us life cycle of the Lyme disease spirochete Borrelia burgdorferi.
4 in, thyroglobulin, LPS, influenza virus, and Borrelia burgdorferi.
5 uding the Lyme disease spirochete bacterium, Borrelia burgdorferi.
6 ritical components to the immune response to Borrelia burgdorferi.
7 mented genome of the Lyme disease spirochete Borrelia burgdorferi.
8 ops in C3H mice infected with the spirochete Borrelia burgdorferi.
9 P-producing protein (diguanylate cyclase) of Borrelia burgdorferi.
10 ) was studied in the Lyme disease spirochete Borrelia burgdorferi.
11 were infected with the causative bacterium, Borrelia burgdorferi.
12 ribed, including Torque teno virus (TTV) and Borrelia burgdorferi.
13 d infectivity in the Lyme disease spirochete Borrelia burgdorferi.
14 ease caused by infection with the spirochete Borrelia burgdorferi.
15 t defense against the extracellular pathogen Borrelia burgdorferi.
16 s on serum iron levels in mice infected with Borrelia burgdorferi.
17 rt control over the inflammation elicited by Borrelia burgdorferi.
18 to infection with the Lyme disease pathogen Borrelia burgdorferi.
19 ogens, including the Lyme disease spirochete Borrelia burgdorferi.
20 rthritis in mice infected with the bacterium Borrelia burgdorferi.
21 acterized the hypothetical protein BB0794 in Borrelia burgdorferi.
22 has an essential role in the eradication of Borrelia burgdorferi.
23 reliosis model of infection of C3H mice with Borrelia burgdorferi.
24 ly or indirectly modulate gene expression of Borrelia burgdorferi.
25 three phases of disease after infection with Borrelia burgdorferi.
26 is serologic detection of antibodies against Borrelia burgdorferi.
27 bacterial enhancer binding protein (bEBP) in Borrelia burgdorferi.
28 one of the major outer membrane proteins of Borrelia burgdorferi.
31 quenced genomes and reveal a bistability for Borrelia burgdorferi, a genome with pronounced replicati
33 to-borne virus producing ephemeral nidi, and Borrelia burgdorferi, a slowly amplifying chronic pathog
35 e neuroborreliosis, caused by the spirochete Borrelia burgdorferi, affects both peripheral and centra
36 The persistence of dormant, noncultivable Borrelia burgdorferi after ceftriaxone treatment was exa
43 5 cells/ml for B. afzelii and 8 cells/ml for Borrelia burgdorferi and Borrelia garinii Clinical sampl
44 sal agents of Lyme disease in North America, Borrelia burgdorferi and Borrelia mayonii, are transmitt
45 orrelia burgdorferi bound to AMP, GluRS from Borrelia burgdorferi and Burkholderia thailandensis boun
47 acteria, that is the Lyme disease spirochete Borrelia burgdorferi and the rickettsial agents Anaplasm
50 ion liposomes, His-tagged antigen rOspA from Borrelia burgdorferi, and lipophilic analogue norAbuMDP-
51 model of Lyme borreliosis, we observed that Borrelia burgdorferi antigens, but not infectious spiroc
52 determination of the serologic responses to Borrelia burgdorferi antigens, with the exception of the
54 s studies showed that the immune response to Borrelia burgdorferi appears to lack robust T-dependent
55 pression quantitative trait locus underlying Borrelia burgdorferi arthritis-associated locus 1 (Bbaa1
57 rime example is the Lyme disease spirochete, Borrelia burgdorferi (B. burgdorferi), which mechanicall
68 f the RpoS regulon in mammalian host-adapted Borrelia burgdorferi, bb0728 (cdr) was found to be duall
71 se is caused by the three spirochete species Borrelia burgdorferi, Borrelia afzelii, and Borrelia gar
72 structures were determined for a CysRS from Borrelia burgdorferi bound to AMP, GluRS from Borrelia b
74 domains, interacted with Leptospira spp. and Borrelia burgdorferi, but not with Escherichia coli or S
75 shown that MyD88 is important for uptake of Borrelia burgdorferi by bone marrow derived macrophages
76 To further characterize the function of the Borrelia burgdorferi C-terminal protease CtpA, we used s
81 The genome of the Lyme disease spirochete Borrelia burgdorferi contains multiple homologues of che
82 ne (OM) of the pathogenic diderm spirochete, Borrelia burgdorferi, contains integral beta-barrel oute
85 We recently showed that a homolog of CsrA in Borrelia burgdorferi (CsrA(Bb), BB0184) was upregulated
87 ecific DNA-binding protein was purified from Borrelia burgdorferi cytoplasmic extracts, and determine
88 ouse model of Lyme disease has revealed that Borrelia burgdorferi differentially expresses numerous o
89 s, we found that the Lyme disease spirochete Borrelia burgdorferi displays a complex pattern of growt
91 jor lipoproteins expressed on the surface of Borrelia burgdorferi during tick feeding and the early p
106 egy adopted by the Lyme disease spirochaete, Borrelia burgdorferi, for adaptation and survival in the
107 hemoreceptors of the Lyme disease spirochete Borrelia burgdorferi form long, thin arrays near both ce
113 merging for differential pathogenicity among Borrelia burgdorferi genotypes in the United States.
115 ligate pathogen, the Lyme disease spirochete Borrelia burgdorferi has a streamlined genome that encod
116 cytosis of the Lyme disease-causing pathogen Borrelia burgdorferi has been shown to be important for
120 interactions of the Lyme disease spirochete Borrelia burgdorferi Here, we investigated how B. burgdo
121 rray of human and animal pathogens including Borrelia burgdorferi; however, how tick immune component
123 014, in whom routine serologic screening for Borrelia burgdorferi (IgG as determined by enzyme-linked
125 od culture based on microscopic detection of Borrelia burgdorferi in Barbour-Stoenner-Kelly medium af
128 spC genotypes of the Lyme disease spirochete Borrelia burgdorferi in the tick vector by in vitro cult
129 was previously implicated in development of Borrelia burgdorferi-induced arthritis in C3H mice, and
132 ion between neuroborreliosis (nervous system Borrelia burgdorferi infection) and Lyme encephalopathy
139 major vector of the Lyme disease spirochete Borrelia burgdorferi, influence spirochete colonization
147 ary, we have found substantial evidence that Borrelia burgdorferi is capable of forming biofilm in vi
150 ost bacterial pathogens, the guaAB operon of Borrelia burgdorferi is present on plasmid cp26, which a
161 The aetiological agent of Lyme disease, Borrelia burgdorferi, is transmitted via infected Ixodes
163 proposed that round morphologic variants of Borrelia burgdorferi, known variably as "cyst forms" and
166 ected with the Lyme disease-causing bacteria Borrelia burgdorferi, leading to induction of antimicrob
172 ious studies, we showed that localization of Borrelia burgdorferi monomeric surface lipoprotein OspA
173 Lyme arthritis-susceptible C3H/HeJ mice with Borrelia burgdorferi, mRNA expression of 5-LO and 5-LO-a
175 nlike the genomes of most organisms, that of Borrelia burgdorferi notably encodes a single HtrA gene
180 brane (OM), we generated constructs in which Borrelia burgdorferi outer surface lipoprotein A (OspA)
182 DbpA) are thought to play important roles in Borrelia burgdorferi pathogenesis by serving as adhesins
190 This study describes the selection of two Borrelia burgdorferi recombinant proteins and evaluation
192 , and serologic evaluation for antibodies to Borrelia burgdorferi remains the recommended modality fo
196 o 34 degrees C, leading to a hypothesis that Borrelia burgdorferi senses environmental temperature to
204 to resist the shear force of the blood flow.Borrelia burgdorferi sensu lato spirochetes, the causati
205 plasmataceae, six genospecies in the complex Borrelia burgdorferi sensu lato, 11 species of Babesia,
206 infected tick, the Lyme disease spirochete, Borrelia burgdorferi sensu lato, colonizes the mammalian
207 ory potential of the 3 pathogenic species of Borrelia burgdorferi sensu lato, we stimulated monocyte-
211 presence of well-established populations of Borrelia burgdorferi sensu stricto, Borrelia bissettii,
214 In each group, Treg cells in SF dampened Borrelia burgdorferi-specific proliferative responses, a
218 nts with Lyme arthritis, and supernatants of Borrelia burgdorferi-stimulated peripheral blood mononuc
225 al serious diseases, including Lyme disease (Borrelia burgdorferi), syphilis (Treponema pallidum) and
226 borne illness in North America, is caused by Borrelia burgdorferi The long-term survival of B. burgdo
227 ck Ixodes scapularis limits proliferation of Borrelia burgdorferi, the aetiologic agent of Lyme disea
244 an important lipoprotein from the bacterium Borrelia burgdorferi, the causative agent of Lyme diseas
245 onses modulate disease during infection with Borrelia burgdorferi, the causative agent of Lyme diseas
249 esis for many infectious bacteria, including Borrelia burgdorferi, the causative agent of Lyme diseas
250 HGA), shares the same enzootic life cycle as Borrelia burgdorferi, the causative agent of Lyme diseas
253 ) respond to CD1d-presented glycolipids from Borrelia burgdorferi, the causative agent of Lyme diseas
255 and ordered (raft) membrane domains exist in Borrelia burgdorferi, the causative agent of Lyme diseas
262 ing protein expressed on the cell surface of Borrelia burgdorferi, the causative agent of Lyme diseas
270 lipoprotein profiles is a key strategy that Borrelia burgdorferi, the Lyme disease pathogen, has evo
278 Treponema pallidum and Lyme disease pathogen Borrelia burgdorferi, the pertinent glycan structures an
279 orrelate with protection from infection with Borrelia burgdorferi, the primary cause of Lyme disease
281 of gene regulation in the enzootic cycle of Borrelia burgdorferi, the spirochete that causes Lyme di
282 Persistent infection of a mammalian host by Borrelia burgdorferi, the spirochete that causes Lyme di
285 nfections with tick-transmitted Borreliella (Borrelia) burgdorferi, the cause of Lyme disease, repres
286 er elucidate the role of this protein in the Borrelia burgdorferi tick-mammal cycle, we conducted a t
287 tron tomography and image analysis of intact Borrelia burgdorferi to produce a three-dimensional (3-D
288 logy utilizing peptide antigens derived from Borrelia burgdorferi to stimulate interferon-gamma (IFN-
289 mammals forces the Lyme disease spirochete, Borrelia burgdorferi, to adapt to different host milieus
290 gands or the Lyme disease causing bacterium, Borrelia burgdorferi, to LAMP-1 lysosomal compartments.
291 y a positive IgG or IgM immunoblot assay for Borrelia burgdorferi--to receive a 12-week oral course o
293 derstanding of the molecular mechanisms that Borrelia burgdorferi uses to survive during mammalian in
294 HP) with 36 residues, a surface protein from Borrelia burgdorferi (VlsE) with 341 residues, and two p
295 In this report, the Lyme disease spirochete Borrelia burgdorferi was used as a genetic model to inve
296 , the only PilZ domain-containing protein in Borrelia burgdorferi, was reported to bind c-di-GMP, nei
297 ns with a positive skin or blood culture for Borrelia burgdorferi were enrolled in a prospective stud
299 c ligand for TLR2/1 heterodimers, as well as Borrelia burgdorferi, which is a strong activator of TLR
300 gainst the Lyme disease causing spirochaete, Borrelia burgdorferi, which uniquely expresses three hom
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