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1 vinar nuclei of the thalamus and 39 cortical Brodmann's areas.
5 's area 8), the right inferior frontal pole (Brodmann's area 10), and the right lateral orbitofrontal
6 BF) decreases in medial orbitofrontal cortex Brodmann's area 10/11, which were absent in the healthy
7 rons in four cortical regions (frontal pole [Brodmann's area 10], primary motor [area 4], primary som
8 sis were detected in the ventral prefrontal (Brodmann's areas 10 and 24) and posterior cingulate cort
12 ter in the left and right prefrontal cortex (Brodmann's areas 11, 10, 8, and 44) and the left anterio
14 half of the occipital pole (corresponding to Brodmann's area 17 and serving as control) to examine th
15 as primary visual cortex, striate cortex, or Brodmann's area 17) was defined in each subject by using
17 eactivity were detected in occipital cortex (Brodmann's area 18) in either group, or in the hippocamp
18 activation in both visual cortex, including Brodmann's areas 18 and 19 and the fusiform gyrus, and s
19 erior flow deficits in the occipital cortex (Brodmann's areas 18 and 19), usually symmetric, and best
22 measured by semiquantitative Western blot in Brodmann's area 21 (middle temporal gyrus) of postmortem
24 ndividual posterotemporal and inferoparietal Brodmann's areas (21, 22 and 39, 40, respectively) the c
25 nce for the crucial role of Wernicke's area (Brodmann's area 22) in word comprehension and indicate t
26 t sections of the medial temporal neocortex (Brodmann's area 22) of 5 male AD patients aged 60-88 yea
27 rtical region, the planum temporale (part of Brodmann's area 22), has a cytoarchitectural homolog, ar
28 dmann's areas 10 and 46), temporal cortices (Brodmann's area 22), hippocampi, caudate nuclei, and cer
29 nificantly lower metabolic activity in right Brodmann's areas 22 and 21 of the superior and middle te
30 und that the volume of the subgenual part of Brodmann's area 24 (sg24) is reduced in familial forms o
34 In contrast, the anterior cingulate cortex (Brodmann's areas 24 and 32) was more active when respond
35 ivity of the anterior cingulate cortex (ACC; Brodmann's areas 24 and 32), whereas no ACC response to
37 cts, an rCBF increase in subgenual cingulate Brodmann's area 25 and a decrease in right prefrontal co
38 i was low in the subgenual cingulate cortex (Brodmann's area 25) and high in the amygdala displayed t
40 10 and 24) and posterior cingulate cortices (Brodmann's area 31), and post hoc analyses indicated tha
41 n the mPFC centered on the prelimbic region (Brodmann's area 32) or the cingulate cortex (Brodmann's
43 ortex (mPFC) centered on the prelimbic area (Brodmann's area 32), at five different intervals after t
46 al cortical regions (viz., ectorhinal cortex=Brodmann's area 36, perirhinal cortex=area 35, lateral e
47 retrieval and phonological processing (e.g., Brodmann's areas 37 and 39) were less likely to show tre
48 bilateral superior anterior temporal lobes (Brodmann's area 38) are selectively activated when parti
49 recorded from the anterior parietal cortex (Brodmann's areas 3a, 3b, 1, and 2) of monkeys performing
52 the right auditory cortex, corresponding to Brodmann's areas 42 and 22, as well as in area 41 (prima
57 nding recurrent excitation of neurons within Brodmann's Area 46 of the dorsolateral prefrontal cortex
58 ostmortem tissue from the prefrontal cortex (Brodmann's area 46) of 14 matched triads of subjects wit
59 of the right dorsolateral prefrontal cortex (Brodmann's area 46/9) in the context of normal task-depe
61 [Brodmann's area 9], inferior frontal gyrus [Brodmann's area 47], medial frontal gyrus [Brodmann's ar
62 reduced grey-matter volumes, particularly in Brodmann's area 48 on the medial surface of the temporal
63 [Brodmann's area 47], medial frontal gyrus [Brodmann's area 6, 10] and the anterior cingulate cortex
64 receptors were found in the frontal cortex (Brodmann's areas 6, 7, 8, 9, 10, 11, 44, 45, 47), anteri
66 significantly higher metabolism in parietal Brodmann's areas 7B and 39 and left occipital Brodmann's
67 right superior dorsolateral frontal cortex (Brodmann's area 8), the right inferior frontal pole (Bro
68 creased rCBF in two mesial prefrontal zones (Brodmann's areas 8 and 10), inferior orbital frontal lob
69 me in both membrane and cytosol fractions of Brodmann's areas 8 and 9 combined (prefrontal cortex).
70 delta1, and gamma1 isozymes were examined in Brodmann's areas 8 and 9 of postmortem brains obtained f
72 , cerebellum, prefrontal association cortex [Brodmann's area 9 (BA9)] and motor cortex [Brodmann's ar
73 ocannabinoid system in the prefrontal cortex Brodmann's area 9 of 42 schizophrenia subjects and match
75 eas damage to the lateral prefrontal cortex (Brodmann's area 9) in monkeys causes a loss of inhibitor
76 volumes were found in the prefrontal cortex (Brodmann's area 9) of PTSD patients than in comparison s
77 92--287%) in dorsolateral prefrontal cortex (Brodmann's area 9) of schizophrenic and bipolar subjects
78 on, the left dorsolateral prefrontal cortex (Brodmann's area 9) was more active for color naming than
80 25 and a decrease in right prefrontal cortex Brodmann's area 9, were not present in the depressed gro
81 t prefrontal cortex (right precentral gyrus [Brodmann's area 9], inferior frontal gyrus [Brodmann's a
82 ivation of bilateral frontostriatal regions (Brodmann's areas 9/46, 45/46; lenticular nucleus; and th
83 d (individually and by averages of estimated Brodmann's areas and brain regions) using linear regress
84 nclude right dorsolateral prefrontal cortex [Brodmann's area (BA 9)], bilateral parietal (BA 40/7), a
85 the right medial orbital prefrontal cortex [Brodmann's area (BA) 25 and medial BA 11], where methylp
86 refrontal cortex, including anterior-ventral Brodmann's Area (BA) 45/47 and more dorsal BA 44, increa
87 nterior left inferior prefrontal cortex near Brodmann's Area (BA) 45/47 and more posterior and dorsal
88 up-regulated, we studied both parameters in Brodmann's area (BA) 9 from the McLean 66 Cohort Collect
90 and right ventrolateral prefrontal cortices (Brodmann's area [BA] 47) and the right amygdala, a prior
91 using the degree of hypoperfusion in various Brodmann's areas--BA 22 (including Wernicke's area), BA
93 read with the prefrontal cortex (9 out of 10 Brodmann's areas in the left hemisphere) and temporal lo
94 hemisphere) and temporal lobe (10 out of 11 Brodmann's areas in the left hemisphere) while the pulvi
95 apes, we found that neurons in posterior IT (Brodmann's areas TEO and posterior TE) integrate informa
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