ライフサイエンスコーパス: Brodmann

1 Brodmann area (BA) 3a of the primary somatosensory corte

2 Brodmann showed areas 26, 29, 30, 23, and 31 on the huma

3 Brodmann's 100-year-old summary map has been widely used

4 Brodmann's area 10 is one of the largest cytoarchitectur

5 Brodmann's area 44 delineates part of Broca's area withi

6 read with the prefrontal cortex (9 out of 10 Brodmann's areas in the left hemisphere) and temporal lo

7 hemisphere) and temporal lobe (10 out of 11 Brodmann's areas in the left hemisphere) while the pulvi

8 ntify regions of hypoperfusion/infarct of 16 Brodmann areas.

9 metimes bridged to other columns in layer 4 (Brodmann layer 4C).

10 (i) CUD with the largest rostroventral ACC [Brodmann Area (BA) 10, 11, implicated in default brain f

11 ivity of the anterior cingulate cortex (ACC; Brodmann's areas 24 and 32), whereas no ACC response to

12 ing syntactic processing, patients activated Brodmann areas 45/47 bilaterally and right middle tempor

13 he enzyme were compared for 18 samples of AD Brodmann area 9/10 frontal cortex with 11 age-matched co

14 ma-power time courses were estimated for all Brodmann areas by combing magnetoencephalographic and MR

15 The effects of age and Brodmann areas were analyzed with a nested multiple anal

16 at connects lateral orbitofrontal cortex and Brodmann area 10 with the anterior temporal lobes.

17 uantitative histology in the hippocampus and Brodmann area 9 in 72 clinically matched individuals wit

18 rder in the CA region of the hippocampus and Brodmann's area 9 of the prefrontal cortex.

19 h at the junction of the anterior insula and Brodmann area 47, in Brodmann area 37, and unilaterally

20 the medial surface of the temporal lobe and Brodmann's area 21 of the middle temporal gyrus.

21 ing the neocortex into regions approximating Brodmann areas (BAs).

22 ortex (mPFC) centered on the prelimbic area (Brodmann's area 32), at five different intervals after t

23 The ipsilateral premotor area (Brodmann area 6), bilateral posterior parietal areas (Br

24 nce for the crucial role of Wernicke's area (Brodmann's area 22) in word comprehension and indicate t

25 d 42) and auditory-visual association areas (Brodmann areas 20 and 37) but were rarely found in somat

26 misphere, and surrounding association areas (Brodmann's areas 10, 11, 12, and 32).

27 homotopic posterior parietal cortical areas (Brodmann areas 39 and surroundings) via the posterior an

28 area 6), bilateral posterior parietal areas (Brodmann area 7) and precuneus showed an increase in rCB

29 ried out a deep RNA-seq analysis of the BA4 (Brodmann area 4) motor cortex from seven human HD brains

30 posterior parietal cortex (PPC) bilaterally [Brodmann area (BA) 40, extending into BA 7] and dorsolat

31 is known about whether individual cingulate Brodmann areas show gender-specific patterns of age-rela

32 cts, an rCBF increase in subgenual cingulate Brodmann's area 25 and a decrease in right prefrontal co

33 iated with metabolism in anterior cingulate (Brodmann area 24/25) and orbitofrontal (Brodmann area 11

34 activity) in the rostral anterior cingulate (Brodmann's area 24/32).

35 Brodmann area 39), left posterior cingulate (Brodmann area 31) and left nucleus accumbens/caudate.

36 ients in the left dorsal anterior cingulate [Brodmann area (BA) 32] but decreased in the right fronta

37 Across species, Broca's area comprises Brodmann's areas 44 and 45.

38 ortex Brodmann area 36 and entorhinal cortex Brodmann area 28.

39 ity involved the medial orbitofrontal cortex Brodmann area 13, which is implicated in reward, and whi

40 nectivity of the medial orbitofrontal cortex Brodmann area 13.

41 Second, the lateral orbitofrontal cortex Brodmann area 47/12 had increased functional connectivit

42 ectivity of the lateral orbitofrontal cortex Brodmann area 47/12 is related to depression.

43 ectivity of the lateral orbitofrontal cortex Brodmann area 47/12 with these three brain areas was low

44 The lateral orbitofrontal cortex Brodmann area 47/12, involved in non-reward and punishin

45 BF) decreases in medial orbitofrontal cortex Brodmann's area 10/11, which were absent in the healthy

46 , especially involving the perirhinal cortex Brodmann area 36 and entorhinal cortex Brodmann area 28.

47 ocannabinoid system in the prefrontal cortex Brodmann's area 9 of 42 schizophrenia subjects and match

48 25 and a decrease in right prefrontal cortex Brodmann's area 9, were not present in the depressed gro

49 ngular gyrus, and the temporal visual cortex Brodmann area 21.

50 diminished in the visual association cortex (Brodmann area [BA] 18; -20.0% vs. control, F((2,22)) = 8

51 investigated in the primary auditory cortex (Brodmann area 41), a site of conserved pathology in Sz.

52 e observed in the anterior cingulate cortex (Brodmann area 24), and they persisted after recovery sle

53 d cingulate gyrus/anterior cingulate cortex (Brodmann area 24).

54 activation in the anterior cingulate cortex (Brodmann area 32) and the orbitofrontal cortex.

55 tex and bilateral anterior cingulate cortex (Brodmann area 32), and enhanced connectivity between DN

56 Brodmann's area 32) or the cingulate cortex (Brodmann's area 24).

57 i was low in the subgenual cingulate cortex (Brodmann's area 25) and high in the amygdala displayed t

58 In contrast, the anterior cingulate cortex (Brodmann's areas 24 and 32) was more active when respond

59 44) and the left anterior cingulate cortex (Brodmann's areas 24/32).

60 n area 38), the ventromedial frontal cortex (Brodmann area 11/32) bilaterally, and the amygdaloid com

61 of arteriolosclerosis in the frontal cortex (Brodmann area 9) was strongly associated with hippocampa

62 right superior dorsolateral frontal cortex (Brodmann's area 8), the right inferior frontal pole (Bro

63 receptors were found in the frontal cortex (Brodmann's areas 6, 7, 8, 9, 10, 11, 44, 45, 47), anteri

64 in the entorhinal area of the human cortex (Brodmann's area 28).

65 n the left parieto-temporo-occipital cortex (Brodmann area 37) in reading-epilepsy patients compared

66 eactivity were detected in occipital cortex (Brodmann's area 18) in either group, or in the hippocamp

67 erior flow deficits in the occipital cortex (Brodmann's areas 18 and 19), usually symmetric, and best

68 as demonstrated in the orbitofrontal cortex (Brodmann area 11) with a right-sided predominance.

69 sions); namely lateral orbitofrontal cortex (Brodmann area 47) and medial prefrontal cortex.

70 l motor cortex and inferior parietal cortex (Brodmann area 40), the lateral premotor cortex and bilat

71 teral prefrontal cortex and parietal cortex (Brodmann areas 9 and 40) was related to task performance

72 recorded from the anterior parietal cortex (Brodmann's areas 3a, 3b, 1, and 2) of monkeys performing

73 y in the anterior rostral prefrontal cortex (Brodmann area 10) and temporal poles (Brodmann area 20/3

74 stimulation to the medial prefrontal cortex (Brodmann area 10) and the dorsolateral prefrontal cortex

75 s within the ventromedial prefrontal cortex (Brodmann area 10) during punished reversal errors compar

76 eas in right dorsolateral prefrontal cortex (Brodmann area 10).

77 activation in left medial prefrontal cortex (Brodmann area 10, 32), right hippocampus, bilateral angu

78 ation in the dorsolateral prefrontal cortex (Brodmann area 46/9) in both the control and Parkinson's

79 =9) in the dorsal lateral prefrontal cortex (Brodmann Area 9) of sudden death medication-free individ

80 ruited right dorsolateral prefrontal cortex (Brodmann areas 45 and 46) to a significantly greater ext

81 d lesions in premotor and prefrontal cortex (Brodmann areas 6, 8, 9, and 46), and 100% with posterior

82 ected within dorsolateral prefrontal cortex (Brodmann areas 9 and 46) and amygdala.

83 ostmortem tissue from the prefrontal cortex (Brodmann's area 46) of 14 matched triads of subjects wit

84 of the right dorsolateral prefrontal cortex (Brodmann's area 46/9) in the context of normal task-depe

85 eas damage to the lateral prefrontal cortex (Brodmann's area 9) in monkeys causes a loss of inhibitor

86 volumes were found in the prefrontal cortex (Brodmann's area 9) of PTSD patients than in comparison s

87 92--287%) in dorsolateral prefrontal cortex (Brodmann's area 9) of schizophrenic and bipolar subjects

88 on, the left dorsolateral prefrontal cortex (Brodmann's area 9) was more active for color naming than

89 n the thalamus and medial prefrontal cortex (Brodmann's area 9).

90 ter in the left and right prefrontal cortex (Brodmann's areas 11, 10, 8, and 44) and the left anterio

91 mPFC lesions of prelimbic cortex (Brodmann's Area 32) retarded EB conditioning in the trac

92 , involving rostral ventral premotor cortex (Brodmann area 44) and intraparietal sulcus.

93 The right dorsal premotor cortex (Brodmann area 6) and the right precuneus (Brodmann area

94 n responses in the left sensorimotor cortex (Brodmann area [BA] 4), bilaterally in the supplementary

95 ects activated the superior temporal cortex (Brodmann area [BA] 22) bilaterally, the precentral gyrus

96 We investigated primary visual cortex (Brodmann area 17) from the Stanley Neuropathology Consor

97 ity of neurons in the primary visual cortex (Brodmann's area 17, BA17).

98 , cerebellum, prefrontal association cortex [Brodmann's area 9 (BA9)] and motor cortex [Brodmann's ar

99 ea 6, 10] and the anterior cingulate cortex [Brodmann's area 32]).

100 ipsilesional posterior primary motor cortex [Brodmann area (BA) 4p], contralesional anterior primary

101 [Brodmann's area 9 (BA9)] and motor cortex [Brodmann's area 4 (BA4)].

102 nclude right dorsolateral prefrontal cortex [Brodmann's area (BA 9)], bilateral parietal (BA 40/7), a

103 the right medial orbital prefrontal cortex [Brodmann's area (BA) 25 and medial BA 11], where methylp

104 n expression in the human prefrontal cortex [Brodmann's area 11 (BA11) and BA47].

105 the left posterior inferior temporal cortex [Brodmann area (BA) 37, or Wernicke's Wortschatz], left c

106 al cortical regions (viz., ectorhinal cortex=Brodmann's area 36, perirhinal cortex=area 35, lateral e

107 vinar nuclei of the thalamus and 39 cortical Brodmann's areas.

108 in auditory and speech association cortices (Brodmann areas 22, 39, and 42) and auditory-visual assoc

109 10 and 24) and posterior cingulate cortices (Brodmann's area 31), and post hoc analyses indicated tha

110 emotor, and the anterior cingulate cortices (Brodmann's areas 4, 6, and 32).

111 and right ventrolateral prefrontal cortices (Brodmann's area [BA] 47) and the right amygdala, a prior

112 in the mesial and lateral premotor cortices (Brodmann area 6).

113 dmann's areas 10 and 46), temporal cortices (Brodmann's area 22), hippocampi, caudate nuclei, and cer

114 alone recruited "core" regions of deduction [Brodmann area (BA) 10p and 8m], whereas linguistic infer

115 ded cytoarchitectonically into four distinct Brodmann areas (3a, 3b, 1, and 2), but these areas have

116 Tissue samples containing the DLPFC (Brodmann area 46) were Golgi-stained, and basilar dendri

117 ging results suggests that posterior-dorsal (Brodmann area, BA, 44) and anterior-ventral parts (BA 45

118 d (individually and by averages of estimated Brodmann's areas and brain regions) using linear regress

119 motor (Brodmann area 46) and verbal fluency (Brodmann area 45) tasks.

120 een the mirrored representations in the four Brodmann areas, as predicted from electrophysiology meas

121 les were evaluated for the anterior frontal (Brodmann area [BA] 10), posterior frontal (BA 6), pariet

122 nvergent evidence indicates that frontopolar Brodmann area 10, and more generally the anterior prefro

123 retrieval and phonological processing (e.g., Brodmann's areas 37 and 39) were less likely to show tre

124 right hippocampus, bilateral angular gyrus (Brodmann area 39), left posterior cingulate (Brodmann ar

125 erved in the right anterior cingulate gyrus (Brodmann area 24), in the intraparietal sulcus of right

126 In the inferior frontal gyrus [Brodmann's area 44]), receptor up-regulation was correla

127 [Brodmann's area 9], inferior frontal gyrus [Brodmann's area 47], medial frontal gyrus [Brodmann's ar

128 [Brodmann's area 47], medial frontal gyrus [Brodmann's area 6, 10] and the anterior cingulate cortex

129 t prefrontal cortex (right precentral gyrus [Brodmann's area 9], inferior frontal gyrus [Brodmann's a

130 ctic processing co-activated left hemisphere Brodmann areas 45/47 and posterior middle temporal gyrus

131 In Brodmann area 9, cognitively active individuals had sign

132 In Brodmann areas 25, 32, and 32', neuronal cell bodies are

133 to the dorsal anterior cingulate area 32 in Brodmann's human brain map, is anterior and dorsal to th

134 hich corresponds to the prelimbic area 32 in Brodmann's monkey brain map, caudal and ventral to the g

135 the anterior insula and Brodmann area 47, in Brodmann area 37, and unilaterally in the left middle te

136 owed the most rapid increases in activity in Brodmann area 10 and the right dorsolateral prefrontal c

137 measured by semiquantitative Western blot in Brodmann's area 21 (middle temporal gyrus) of postmortem

138 We describe an area centered in Brodmann's area 13m of the medial OFC (mOFC) where taste

139 udies were performed in prefrontal cortex in Brodmann area 9 and hippocampus obtained in 27 suicide s

140 mic acid decarboxylase (GAD) is decreased in Brodmann area 9 (BA9) of the dorsolateral prefrontal cor

141 yer II and III pyramidal neuron dendrites in Brodmann area 46 dorsolateral prefrontal cortex using th

142 delta1, and gamma1 isozymes were examined in Brodmann's areas 8 and 9 of postmortem brains obtained f

143 detected at alpha frequencies (10-14 Hz) in Brodmann area 6, but did not covary with the number of r

144 d stimulation coordinates were identified in Brodmann area 46.

145 ) changes in thickness of cortical layers in Brodmann areas 11, 10, 24a and 4 differed; and (iii) dif

146 e Peg Test correlated with GM volume loss in Brodmann area 44 (Broca area; P = .02).

147 1) of the left prefrontal cortex (maximal in Brodmann's area 10) was seen.

148 Counts of reelin mRNA-positive neurons in Brodmann's area 10 of either nonpsychiatric subjects or

149 up-regulated, we studied both parameters in Brodmann's area (BA) 9 from the McLean 66 Cohort Collect

150 reduced grey-matter volumes, particularly in Brodmann's area 48 on the medial surface of the temporal

151 These studies were performed in Brodmann area (BA) 9 and hippocampus obtained from 26 su

152 f 10722 neuronal and 19913 glial profiles in Brodmann areas 9 and 17.

153 pared in the DLPFC, as well as separately in Brodmann areas 9 and 46.

154 ht anterior cingulate gyrus, specifically in Brodmann's area 24'.

155 These included Brodmann areas 19/37, the inferior (Brodmann Area 39), a

156 ere network of correlated activity including Brodmann areas 45/47 and posterior middle temporal gyrus

157 activation in both visual cortex, including Brodmann's areas 18 and 19 and the fusiform gyrus, and s

158 A cortical analysis by individual Brodmann's areas was performed.

159 included Brodmann areas 19/37, the inferior (Brodmann Area 39), and superior parietal lobule (Brodman

160 ndividual posterotemporal and inferoparietal Brodmann's areas (21, 22 and 39, 40, respectively) the c

161 apes, we found that neurons in posterior IT (Brodmann's areas TEO and posterior TE) integrate informa

162 eft posterior inferior temporal gyrus (iTG) (Brodmann area 37/19).

163 the dorsolateral prefrontal cortex (lateral Brodmann 9) while participants rested in the MRI scanner

164 ight dorsolateral prefrontal cortex and left Brodmann area 10 at the time of this deception.

165 d to the right intraparietal lobule and left Brodmann area 9 (BA9).

166 ght the functional relationship between left Brodmann area 45 and the left posterior middle temporal

167 egrity and neural activity-primarily in left Brodmann area 45 and posterior middle temporal gyrus-wer

168 d performance; poor tissue integrity in left Brodmann area 45 was associated with reduced functional

169 es showed that only tissue integrity in left Brodmann areas 45/47 was correlated with activity and pe

170 d network of regions including parts of left Brodmann areas 37 and 40 is necessary for reading and sp

171 alysis demonstrated that dysfunction of left Brodmann areas 40 (supramarginal gyrus) and 37 (posterio

172 motor adaptation task demonstrated that left Brodmann area 44 (BA44) played a key role in adaptation,

173 by MEG in the right superior parietal lobe (Brodmann's Area 7).

174 so involved the inferolateral temporal lobe (Brodmann area 20/21) and fusiform gyrus.

175 bilateral superior anterior temporal lobes (Brodmann's area 38) are selectively activated when parti

176 mann Area 39), and superior parietal lobule (Brodmann Area 7).

177 ssive, controls) x anteroposterior location (Brodmann areas 25, 24, 31, 29) x hemisphere (right, left

178 nterior cingulate cortex (ACC) and/or medial Brodmann area (BA) 9 were, in some cases, impaired on vo

179 ions were done for sulci, gyri, and modified Brodmann areas to link macroscopic anatomical and micros

180 sting condition, in two regions of the MPFC (Brodmann Areas 10/32 and 24/25).

181 d circuitry, medial prefrontal cortex (mPFC; Brodmann area 9/10/32), to reward outcome (p(corrected)

182 nterior left inferior prefrontal cortex near Brodmann's Area (BA) 45/47 and more posterior and dorsal

183 y was carried out in Patas monkey neocortex (Brodmann's areas 2, 3, 4, 6, 9, 17, 18, and 24).

184 t sections of the medial temporal neocortex (Brodmann's area 22) of 5 male AD patients aged 60-88 yea

185 rodmann's areas 7B and 39 and left occipital Brodmann's area 19.

186 a centered just posteriorly on the border of Brodmann areas 4a and 6, which we distinguished from a m

187 teral prefrontal cortex (DLPFC, comprised of Brodmann areas 9 and 46) from 19 individuals with a prem

188 osteromedial cortex (PMC), which consists of Brodmann areas 23, 29, 30, 31, and 7m.

189 me in both membrane and cytosol fractions of Brodmann's areas 8 and 9 combined (prefrontal cortex).

190 al cortex (ILPFC)-that is, rodent homolog of Brodmann area 25 (BA25), and the lateral habenula (LHb)

191 tremely large, confirming the observation of Brodmann, who found large somata for these neurons in ca

192 rtical region, the planum temporale (part of Brodmann's area 22), has a cytoarchitectural homolog, ar

193 und that the volume of the subgenual part of Brodmann's area 24 (sg24) is reduced in familial forms o

194 eft inferior frontal gyrus, in the region of Brodmann's area 45.

195 ons into distal digit tip representations of Brodmann area 3b in the squirrel monkey.

196 transcranial magnetic stimulation (rTMS) of Brodmann Area (BA) nine of the left dorsolateral prefron

197 as primary visual cortex, striate cortex, or Brodmann's area 17) was defined in each subject by using

198 ate (Brodmann area 24/25) and orbitofrontal (Brodmann area 11) and prefrontal (Brodmann area 9/10) co

199 significantly higher metabolism in parietal Brodmann's areas 7B and 39 and left occipital Brodmann's

200 ilaterally in the visual cortex in patients [Brodmann area (BA) 17].

201 ng an error predicted left dorsolateral PFC (Brodmann area 8/9) activation 472 milliseconds after com

202 activated a region in the right rostral PFC (Brodmann area 10).

203 ex (ACC) and medial prefrontal cortex (PFC) (Brodmann area 10/32) 80 milliseconds after committing an

204 Postmortem prefrontal cortices (PFC) (Brodmann's areas 10 and 46), temporal cortices (Brodmann

205 e contralateral dorsal premotor cortex [PMd, Brodmann area (BA) 6] in multiple sclerosis patients tha

206 's area 8), the right inferior frontal pole (Brodmann's area 10), and the right lateral orbitofrontal

207 ns of neuronal loss, the left temporal pole (Brodmann area 38) was the most significantly and consist

208 In addition, the right temporal pole (Brodmann area 38), the ventromedial frontal cortex (Brod

209 rons in four cortical regions (frontal pole [Brodmann's area 10], primary motor [area 4], primary som

210 ortex (Brodmann area 10) and temporal poles (Brodmann area 20/38) relative to offenders with ASPD-P a

211 tex (SI) and posterior parietal cortex (PPC; Brodmann areas 7/40).

212 x (Brodmann area 6) and the right precuneus (Brodmann area 7) showed a linear increase of rCBF as seq

213 tofrontal (Brodmann area 11) and prefrontal (Brodmann area 9/10) cortices, and with personality score

214 sis were detected in the ventral prefrontal (Brodmann's areas 10 and 24) and posterior cingulate cort

215 eft dorsolateral prefrontal cortical region (Brodmann areas 44, 45, and 46), where the average global

216 and the right lateral orbitofrontal region (Brodmann's area 47).

217 n the mPFC centered on the prelimbic region (Brodmann's area 32) or the cingulate cortex (Brodmann's

218 predominantly left medial prefrontal region [Brodmann area (BA) 8/9/10] was more active during the ta

219 found in somatosensory association regions (Brodmann area 21).

220 Twelve cortical regions (Brodmann's areas 8, 10, 44, 46, 23/31, 24/32, 20, 21, 22

221 ivation of bilateral frontostriatal regions (Brodmann's areas 9/46, 45/46; lenticular nucleus; and th

222 activity in early visual processing regions (Brodmann area (BA)17, BA18).

223 d gene expression data of two other regions: Brodmann area 19 (occipital cortex) and cerebellar corte

224 nificantly lower metabolic activity in right Brodmann's areas 22 and 21 of the superior and middle te

225 eral prefrontal cortex for the sensorimotor (Brodmann area 46) and verbal fluency (Brodmann area 45)

226 subgenual anterior cingulate cortex (sgACC; Brodmann area 25) predicts outcome in CT for depression,

227 and nonhuman area 32 has been impaired since Brodmann said he could not homologize with certainty hum

228 ctivity of the non-reward/punishment system (Brodmann area 47/12) with the precuneus (involved in the

229 lear evidence that the PPC site we targeted (Brodmann areas 7/40) contributes to tactile direction pe

230 sted that our findings support the view that Brodmann area 45 is involved in verbal response generati

231 The Brodmann area-based approach also facilitates comparison

232 in the lingual and fusiform gyri and in the Brodmann areas 22 and 38 in superior temporal sulcus (ST

233 ed pathological changes were observed in the Brodmann areas 44 (Broca's area) and 45.

234 somatotopic maps defining the extent of the Brodmann areas could be directly observed on the cortica

235 Thus, Brodmann's map understates the rostral extent of retrosp

236 The tissue types were assigned to Brodmann's areas by using the Perry postmortem histologi

237 half of the occipital pole (corresponding to Brodmann's area 17 and serving as control) to examine th

238 the right auditory cortex, corresponding to Brodmann's areas 42 and 22, as well as in area 41 (prima

239 were, with only a few exceptions, limited to Brodmann area 9, suggesting regional specificity of path

240 to the D3 and D4 receptors, and localized to Brodmann area 11 (orbitofrontal cortex).

241 gnant left frontal meningioma impinging upon Brodmann area 45, presented a 'pure' dynamic aphasia.

242 using the degree of hypoperfusion in various Brodmann's areas--BA 22 (including Wernicke's area), BA

243 refrontal cortex, including anterior-ventral Brodmann's Area (BA) 45/47 and more dorsal BA 44, increa

244 Results showed that bilateral ventral [Brodmann's areas (BA) 44, 45, and 47] and right dorsal (

245 Finally, in agreement with Brodmann, gigantopyramidal neurons in both the morpholog

246 Within Brodmann's area 32, a glucose metabolism deficit in the

247 nding recurrent excitation of neurons within Brodmann's Area 46 of the dorsolateral prefrontal cortex

248 creased rCBF in two mesial prefrontal zones (Brodmann's areas 8 and 10), inferior orbital frontal lob