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1 of a metalloprotease from the filarial worm Brugia malayi.
2 ss in the important human parasitic nematode Brugia malayi.
3 ainst the Wolbachia-dependent model filaria, Brugia malayi.
4 e tropical parasites Onchocerca volvulus and Brugia malayi.
5 r chronic exposure to the nematode parasite, Brugia malayi.
6 en developed for the human filarial parasite Brugia malayi.
7 a nematode model with the filarial parasite, Brugia malayi.
8 parasitic nematodes Wuchereria bancrofti and Brugia malayi.
9 gy to identify candidate vaccine antigens of Brugia malayi.
10 quisition in the parasitic filarial nematode Brugia malayi.
11 C. elegans and the human-parasitic nematode Brugia malayi.
12 rial nematodes, including Brugia pahangi and Brugia malayi.
13 nematode parasites Wuchereria bancrofti and Brugia malayi.
14 interleukin-4 (IL-4) in host defense against Brugia malayi.
15 ability to combat infection by the nematode Brugia malayi.
16 nematode parasites Wuchereria bancrofti and Brugia malayi.
17 in two nematodes; Caenorhabditis elegans and Brugia malayi.
18 ge larvae (L3) or live microfilariae (Mf) of Brugia malayi, a causative agent of human lymphatic fila
19 has been isolated from the filarial nematode Brugia malayi, a causative agent of human lymphatic fila
20 of almost all the major mammalian stages of Brugia malayi, a causative agent of lymphatic filariasis
21 ertaken the first study of heme transport in Brugia malayi, a causative agent of lymphatic filariasis
22 nts generated from MF patients, filarial Ag (Brugia malayi adult Ag (BmA))-stimulated supernatants fr
24 Brugian filariasis (caused by the nematodes Brugia malayi and B. timori) is an important cause of di
25 During our investigations into the course of Brugia malayi and Brugia pahangi infections in immunodef
27 e (Mb) genome of the human filarial parasite Brugia malayi and predict approximately 11,500 protein c
28 ical to the MIFs from the parasitic nematode Brugia malayi, and 22-35% identical to mammalian MIFs.
29 cells were exposed to microfilariae (mf) of Brugia malayi, and their phenotypic and functional chara
30 enomes to that of another filarial parasite, Brugia malayi, and to those of several other nematodes,
31 duals (n = 11) were stimulated in vitro with Brugia malayi antigen (BMA) or mycobacterial purified pr
32 association between ratios of polyclonal to Brugia malayi antigen (BmAg)-specific IgE (range, 14:1 t
33 (-/-) mice were first immunized with soluble Brugia malayi antigens and then inoculated intravenously
34 d secondary murine and human infections with Brugia malayi are characterized by substantial increases
35 s brenneri, using Pristionchus pacificus and Brugia malayi as outgroups, and identified numerous oper
36 chocerca volvulus, Wuchereria bancrofti, and Brugia malayi available, new drug targets have been iden
37 on passive transfer, led to the clearance of Brugia malayi (Bm) microfilariae (mf) from infected jird
39 response to live, infective-stage larvae of Brugia malayi but not to microfilariae of this parasite.
40 rgillus nidulans, Schizosaccharomyces pombe, Brugia malayi, Caenorhabditis elegans, Trypanosoma cruzi
41 uble extracts of the human filarial parasite Brugia malayi can induce potent inflammatory responses,
43 sponding genes in Caenorhabditis elegans and Brugia malayi contained a cysteine codon in place of TGA
49 e heme; however, the human filarial parasite Brugia malayi has acquired a bacterial gene encoding fer
50 sponse to the infective-stage larvae (L3) of Brugia malayi has not been well-characterized in vivo (b
51 he genomic environment of a newly identified Brugia malayi Hox6-8 ortholog (Bm-ant-1) revealed that i
53 We have shown that live microfilariae of Brugia malayi induce caspase-dependent apoptosis in huma
55 luated the contributions of T and B cells in Brugia malayi infection by utilizing knockout mice on a
56 after treatment with diethylcarbamazine for Brugia malayi infection, and recorded the severity of an
62 tics and anti-Wolbachia efficacy in a murine Brugia malayi model of minocycline versus doxycycline.
64 del of alternative activation in which adult Brugia malayi nematodes are implanted surgically in the
65 bitor of NO synthase abrogated resistance to Brugia malayi, one of the causative agents of human lymp
66 interactions between the infective stage of Brugia malayi--one of the extracellular parasites respon
67 termine, via preclinical infection models of Brugia malayi or Onchocerca ochengi that elevated exposu
69 IL4Ralpha(-/-) mice elicited by the nematode Brugia malayi, or via intraperitoneal thioglycollate inj
70 ells (PEC) from mice transplanted with adult Brugia malayi parasites suppress the proliferation of ly
71 a donovani, Toxoplasma gondii) and helminth (Brugia malayi) parasites were examined, each of which pr
72 ocheilonema viteae, Onchocerca volvulus, and Brugia malayi, strongly supporting the concept that the
73 ympahtic filariasis is the nematode parasite Brugia malayi that requires a competent mosquito vector
74 cation of binding protein(s) of the helminth Brugia malayi to CaGC and the ability of binding complex
76 pod Drosophila melanogaster and the nematode Brugia malayi, together with the partial genome sequenci
77 e, C. remanei, C. brenneri, C. japonica, and Brugia malayi using some of the best-performing gene-fin
78 e infective-stage larvae or microfilariae of Brugia malayi, we found significant impairment of both T
80 t the filarial nematodes responsible for LF (Brugia malayi, Wuchereria bancrofti) or onchocerciasis (
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