ライフサイエンスコーパス: C-telopeptide

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1 act parathyroid hormone, and increased serum C telopeptide.

2 mor burden of Neo cells, the serum levels of C-telopeptide and TRAP-5b never increase above the level

3 obic interactions involving the short alpha2 C-telopeptide are crucial determinants of its azimuthal

4 asures were type I collagen crosslinked beta C-telopeptide (betaCTX), and type 1 procollagen-N-propep

5 ated peptides indicated that both the N- and C-telopeptides bound to a region between amino acid 776

6 (PTH), bone alkaline phosphatase (BAP), and C-telopeptide (CTX) in youth infected with human immunod

7 These animals have high serum levels of the C-telopeptide derived from type I collagen as well as si

8 This difference between N- and C-telopeptide docked structures demonstrates how unique

9 links at molecular sites (mediated by N- and C-telopeptide domains) found in collagen II fibrils proc

10 , aldosterone, endothelin-1, troponin I, and C-telopeptide for type I collagen levels, suggesting mor

11 h serum levels of the bone resorption marker C-telopeptide fragments of type I collagen (CTX), elevat

12 r-maximal reductions in mean levels of serum C-telopeptide from baseline were evident three days afte

13 d characteristics sex, serum type I collagen C-telopeptide, hip bone mineral density, urticaria pigme

14 hormone (PTH), osteocalcin, type I collagen C-telopeptide, hormones, and metabolic factors.

15 lagen triple-helical segment followed by the C-telopeptides in their docked conformation, and then th

16 rone dosage decreased, the percent change in C-telopeptide increased.

17 th an increase in bone resorption, the serum C-telopeptide level was 25% higher in transgenic mice th

18 ls of bone resorption (serum type I collagen C-telopeptide), low hip bone mineral density, absence of

19 linked alpha1(I) chains as a result of their C-telopeptide lysines being more completely oxidized to

20 e alkaline phosphatase (b-ALP), and terminal C telopeptide of collagen Type I (CTX) were analyzed.

21 ed with the systemic marker of bone turnover C-telopeptide of type 1 collagen (r=0.6; P<0.001).

22 h continuing alendronate: 55.6% (P<.001) for C-telopeptide of type 1 collagen, 59.5% (P < .001) for s

23 Serum levels of C-telopeptide of type I collagen (CTx), a marker of bone

24 sient decrease in the bone resorption marker C-telopeptide of type I collagen (CTX-1) were observed.

25 propeptide of type I procollagen (PINP) and C-telopeptide of type I collagen (CTX-I) are markers of

26 raphy and a significant decrease in systemic C-telopeptide of type I collagen, suggesting decreased o

27 Urinary C-telopeptide of type II collagen (CTX-II) levels were m

28 inoline, glycosylated Pyr (Glc-Gal-Pyr), and C-telopeptide of type II collagen (CTX-II) was assessed

29 for helical peptide of type II collagen and C-telopeptide of type II collagen, were observed after r

30 in the collagen triple helix or in the N- or C-telopeptides of collagen I.

31 ns of 2.5 mM, peptides with sequences in the C-telopeptides of the alpha1(I) and alpha2(I) chain inhi

32 C-telopeptide pyridinoline cross-links (ICTP), a host-de

33 cular fluid (GCF) analysis for the levels of C-telopeptide pyridinoline cross-links (ICTP), and genet

34 ecifically evaluate the relationship between C-telopeptide pyridinoline cross-links and periodontal d

35 idinoline differed between N-telopeptide and C-telopeptide sites, and between the individual intercha

36 ins was cross-linked by pyridinolines at the C-telopeptide to helix site, [alpha1(I)C]2alpha1(I)helix

37 were identified, N-telopeptide to helix and C-telopeptide to helix.

38 Docking of the heterotrimeric C-telopeptide to its receptor showed that hydrophobic in

39 analysis of kinetics of binding of alpha1(I) C-telopeptide using an optical biosensor.

40 ncentrations of collagen type 1 cross-linked C-telopeptide was significantly decreased in the RCE gro

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