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1 sis; 16 (100%), 4 (95.5%), and 4 (98.9%) for C. guilliermondii.
2 r more of the echinocandins, most (68%) were C. guilliermondii.
3 e not significantly different from those for C. guilliermondii.
4 ogy and antifungal susceptibility profile of C. guilliermondii.
5 is to 100% for C. krusei, C. lusitaniae, and C. guilliermondii.
6 ranged from 0% for C. lusitaniae to 100% for C. guilliermondii.
7 or C. glabrata to 100% for C. lusitaniae and C. guilliermondii.
8 .0), 1 (92.7)/-, and 1 (100.0)/2 (100.0) for C. guilliermondii.
9 0.12; for C. lusitaniae, 0.03 and 0.007; for C. guilliermondii, 0.12 and 0.03; and for C. kefyr, 0.06
11 ; C. kefyr, 0.06/0.06; C. parapsilosis, 1/2; C. guilliermondii, 0.5/1; C. lusitaniae, 0.12/0.25; othe
12 ), 8 (95.0), 0.5 (97.5), and 0.25 (98.0) for C. guilliermondii; 0.25 (98.9), 0.03 (98.0), 0.12 (97.5)
13 67 isolates of C. lusitaniae, 48 isolates of C. guilliermondii, 10 isolates of C. famata, 10 isolates
14 mong the emerging fluconazole-R species were C. guilliermondii (11.4% R), C. inconspicua (53.2% R), C
15 mutant strain), 25 of C. parapsilosis, 19 of C. guilliermondii, 12 of C. tropicalis (2 mutant strains
16 ng methods demonstrated that 19 strains were C. guilliermondii, 14 were C. parapsilosis, 5 were C. lu
17 abrata (2 fluconazole-resistant isolates), 5 C. guilliermondii (2 fluconazole-resistant isolates), 10
18 olates were C. parapsilosis, 6 isolates were C. guilliermondii, 2 isolates were C. rugosa, and 1 isol
23 . lusitaniae, 0.5 (98.9) and 0.25 (98.3) for C. guilliermondii, and 0.25 (100.0) and 0.015 (100.0) fo
25 2.0% were due to C. krusei, 0.7% were due to C. guilliermondii, and 5.8% were due to Candida spp.
26 picalis, 270 C. krusei, 99 C. lusitaniae, 88 C. guilliermondii, and 61 C. kefyr isolates) were obtain
27 5 isolates of C. lusitaniae, 177 isolates of C. guilliermondii, and 93 isolates of C. kefyr were obta
31 to detect seven additional Candida species (C. guilliermondii, C. kefyr, C. lambica, C. lusitaniae,
32 lates include Candida krusei, C. lusitaniae, C. guilliermondii, C. kefyr, C. rugosa, C. lipolytica, C
34 , C. parapsilosis, C. krusei, C. lusitaniae, C. guilliermondii, C. lipolytica, C. rugosa, and C. zeyl
35 nce over time was seen with C. parapsilosis, C. guilliermondii, C. lusitaniae, C. sake, and C. pellic
36 ion and antifungal susceptibilities of 1,029 C. guilliermondii clinical isolates collected from 127 m
37 tion, with some species (e.g., C. rugosa and C. guilliermondii) demonstrating reduced susceptibilitie
40 r, extensive environmental sampling revealed C. guilliermondii in an anaerobic holding jar in the cli
43 C. krusei (n = 143), C. lusitaniae (n = 54), C. guilliermondii (n = 39), C. pelliculosa (n = 17), C.
44 C. parapsilosis (n = 39), C. krusei (n = 5), C. guilliermondii (n = 6), and C. lusitaniae (n = 2).
46 15 with low confidence between C. famata and C. guilliermondii or C. parapsilosis, displaying only 56
48 C. tropicalis, C. krusei, C. lusitaniae, and C. guilliermondii) simultaneously with very high specifi
50 oriconazole was more active in vitro against C. guilliermondii than fluconazole (91% susceptible; ran
52 lbicans, C. parapsilosis, C. tropicalis, and C. guilliermondii were all highly susceptible to both fl
53 ml), and C. parapsilosis, C. lusitaniae, and C. guilliermondii were the least susceptible (MIC(90), 0
54 rapsilosis and variants of C. lusitaniae and C. guilliermondii were the most resistant, demonstrating
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