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1 ay contribute to the aggressive virulence of C. psittaci.
2 solates of C. pneumoniae, and 20 isolates of C. psittaci.
3 er C. pneumoniae strains, C. trachomatis, or C. psittaci.
4 thogen C. pneumoniae, and the zoonotic agent C. psittaci.
5 ead birds from pet stores for infection with C. psittaci.
6 strains of C. trachomatis and two strains of C. psittaci.
7 ig inclusion conjunctivitis (GPIC) strain of C. psittaci.
8 d within 1 h of infection of host cells with C. psittaci 6BC but that protein quantity peaks during t
14 d 169 animal specimens; 98 were positive for C. psittaci (71.4% genotype A, 3.1% genotype B, 4.1% gen
15 rted; however, two other chlamydial species, C. psittaci and C. abortus, are known zoonotic pathogens
16 ng and weak homologies were also detected in C. psittaci avian and feline pneumonitis strains, respec
17 plates, the synthetic peptides reacted with C. psittaci B577 antisera, but not with sera from specif
19 As were compared to those from the reference C. psittaci B577 elementary body (EB) ELISA and the Chla
20 blems, were screened for antibodies by these C. psittaci B577 peptide ELISAs and an ELISA with recomb
25 een developed to rapidly detect and genotype C. psittaci by light-upon-extension chemistry and high-r
30 Srp, whose sequence is 64% identical between C. psittaci GPIC and C. trachomatis, is partially trunca
31 uring outbreaks of psittacosis to infer that C. psittaci had been transmitted from birds purchased in
33 trachomatis IncA is structurally similar to C. psittaci IncA and is also localized to the inclusion
35 ci, uniquely targets the nuclear envelope of C. psittaci-infected cells and uninfected neighboring ce
38 e cells migrated identically to that seen in C. psittaci-infected cells, indicating the host cell was
41 onstrated that these proteins are present in C. psittaci-infected HeLa cells but are absent or below
42 rescently labelled anti-IncA antibodies into C. psittaci-infected HeLa cells resulted in immunostaini
44 the interpretation of results obtained with C. psittaci models of infection and immune resolution, p
49 three species of Chlamydiaceae; LcrH-2 from C. psittaci reacted with LcrE from C. pneumoniae but not
51 of EUO, a previously described ORF of avian C. psittaci strain 6BC which is preferentially transcrib
52 antiserum raised against a recombinant ovine C. psittaci strain POMP, and two possessed surface-expos
54 on at the periphery of aberrant RB, while in C. psittaci, treatment causes SEP to localize to distinc
55 ment of the 16S rRNA gene were observed when C. psittaci was electroporated with the recombination su
56 Additionally, target cells infected with C. psittaci were lysed by C. trachomatis-elicited immune
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