ライフサイエンスコーパス: C57BL mice

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1 n the brains of SAMP8 and SAMR1 mice than in C57BL mice.

2 ed 99Tc-G extravasation in the dura mater of C57BL mice.

3 Eight male C57BL mice, 8 weeks old, were submitted to intraperitone

4 Nontransgenic (NTg) C57BL mice and apoA-I-transgenic (apoAI-Tg) mice, with g

5 killing by AMs may explain the resistance of C57BL mice and the susceptibility of C3H mice to mycopla

6 ly decreased in both Swiss-Webster (S-W) and C57bl mice (approx. cell loss of 40% and 45%, respective

7 ere that B10.BR mice, which are derived from C57BL mice but have the same MHC locus (H-2(k)) as susce

8 ed in increased dura mast cell activation in C57BL mice, but not in NK-1R-/- mice.

9 AM depletion exacerbated the infection in C57BL mice by reducing killing of the organism to a leve

10 On the other hand, C57BL mice do not produce IL-4 and, in the absence of bo

11 sent studies the immune response in BALB and C57BL mice during the acute phase of E-55+ murine leukem

12 oE synthesis were also observed in castrated C57BL mice given either physiological or pharmacological

13 The results showed that visceral AT from old C57BL mice had significantly higher mRNA expression of t

14 inal-RPE separation was created in wild-type C57BL mice, IL-6(-/-) mice, and Brown Norway rats by sub

15 especially in aging control and 22A-infected C57BL mice, predominantly in the stratum radiatum of the

16 Under normoxic conditions, macrophages from C57BL mice produce low levels of vascular endothelial gr

17 Young adult male C57BL mice received whole brain irradiation, and 6-48 h

18 R mutant of Ec1a was similarly attenuated in C57BL mice, regardless of the p47(phox) genotype, as in

19 This locus is not sufficient to make C57BL mice resistant to coccidioidomycosis.

20 Finally, embryos from two substrains of C57BL mice that differ in susceptibility to ethanol tera

21 -10 and IL-4 adversely affect the ability of C57BL mice to resist infection with C. immitis, but IL-1

22 Young and old C57BL mice were fed diets containing 30 (control) or 500

23 Although C57BL mice were less susceptible to experimental UTI tha

24 than CBA/J mice, wild-type and p47(phox-/-) C57BL mice were similarly susceptible, and the oxyR muta

25 duce both IL-4 and IFN-gamma, in contrast to C57BL mice, which produce only IFN-gamma.

26 C57BL/6J-scid/scid (C57-SCID), and C57BL/6N (C57BL) mice with Mycoplasma pulmonis and at 14 and 21 da

27 J mice had higher peak parasitemias than did C57BL mice, with mortality rates of 40, 50, and 50%, res

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