ライフサイエンスコーパス: CA1 region

1 cellular model for memory formation, in the CA1 region.

2 ) facilitated electrically evoked LTD in the CA1 region.

3 glial cells were observed in the hippocampal CA1 region.

4 ial information may flow into and out of the CA1 region.

5 ion (LTP), and NADH metabolic imaging in the CA1 region.

6 istributed across the transverse axis of the CA1 region.

7 ns (twin and single apical dendrites) in the CA1 region.

8 ous epileptiform activity in the hippocampal CA1 region.

9 g-term potentiation (LTP) in the hippocampal CA1 region.

10 mushroom dendritic spines in the hippocampal CA1 region.

11 hibition in the dentate gyrus but not in the CA1 region.

12 wer occurred, and was synchronous across the CA1 region.

13 ces in the magnitude of CBF decreases in the CA1 region.

14 tine modulates DP and LTD in the hippocampal CA1 region.

15 erience-induced structural plasticity in the CA1 region.

16 g-term potentiation (LTP) in the hippocampal CA1 region.

17 , and were distributed evenly throughout the CA1 region.

18 d functional capabilities of the hippocampal CA1 region.

19 ic potential recorded in the rat hippocampal CA1 region.

20 development of epileptiform activity in the CA1 region.

21 timulation or theta burst stimulation in the CA1 region.

22 and NMDAR signaling in the mouse hippocampal CA1 region.

23 icity in slice recordings of the hippocampal CA1 region.

24 ntorhinal cortex (MECIII) to the hippocampal CA1 region.

25 s in the stratum radiatum in the hippocampal CA1 region.

26 ns recorded in the stratum pyramidale of the CA1 region.

27 gamma synchrony among the dentate, CA3, and CA1 regions.

28 in the dentate gyrus and another in the CA3-CA1 regions.

29 SPW-R-related population activity in CA3 and CA1 regions.

30 ctions compared with distal CA3 and proximal CA1 regions.

31 mpal region, specifically the cornu Ammonis (CA1) region.

32 was prominent in NeuN-positive layers in the CA1 region 6-12 h following ischemia.

33 VI neurons while stimulating the Sb near the CA1 region, a low dose of nicotine not only enhanced syn

34 dopsin in pyramidal cells in the hippocampal CA1 region, achieving high quality recording, activation

35 d that IR stress induced SG formation in the CA1 region after 3-day reperfusion, consistent with TI a

36 fail to activate CRE-binding protein in the CA1 region after restraint stress.

37 Synchrony between anatomically distant CA1 regions also shifts to higher gamma frequencies as r

38 d inhibitory microcircuit in the hippocampal CA1 region, an increased excitability of pyramidal neuro

39 pal fissure and stratum (str.) oriens of the CA1 region and (2) between CA1 stratum radiatum and the

40 s and increased apoptosis in the hippocampal CA1 region and cortex.

41 ntrated in stratum oriens of the hippocampal CA1 region and dentate inner molecular layer and hilus.

42 munoreactivity (-ir) most prominently in the CA1 region and diffuse ERbeta-ir primarily in the CA1 py

43 acetyltransferases (HATs) in the hippocampal CA1 region and find that K-acetyltransferase 2a (Kat2a)-

44 n decreased numbers of spine synapses in the CA1 region and impaired performance of mice in behaviora

45 n vivo network activities in the hippocampal CA1 region and impaired synaptic function.

46 d in the principal cell layer in the CA3 and CA1 region and in the dentate gyrus of the hippocampus.

47 the cAMP pathway reduced PP1 activity in the CA1 region and increased the active form of inhibitor-1,

48 uency ripple oscillations in the hippocampal CA1 region and is linked to experience, but the mechanis

49 hippocampal primary culture, neurons of the CA1 region and the dentate gyrus (DG) express high Np65

50 ductions induced by LFPI and CFPI within the CA1 region and this SI-induced energy reduction appears

51 entate gyrus and the stratum radiatum of the CA1 region and were dependent on UCP2 expression, becaus

52 develop RIDNs, initially in the hippocampal CA1 region, and later in other hippocampal and cortical

53 lpha3 were accentuated in the dentate gyrus, CA1 region, and subiculum, whereas alpha5 expression was

54 fast ("ripple") oscillations (SPW-Rs) in the CA1 region are among the most synchronous physiological

55 In contrast, pyramidal cells in the CA1 region are not extensively interconnected.

56 Neurons in the hippocampal CA1 region are particularly sensitive to oxidative stres

57 ced activation of caspase-3, not only in the CA1 region as expected, but also in the dentate subgranu

58 mpaired synaptic function in the hippocampal CA1 region as the result of epigenetic-dependent alterat

59 psilateral septal hippocampus in the CA3 and CA1 regions associated with atrophy.

60 Cell counts in the CA1 region at 7 days post-injury revealed no significant

61 us, alpha5 transcripts first appeared in the CA1 region at E18.

62 1 phosphorylation at T840 in the hippocampal CA1 region, bath application of NMDA induced a strong, p

63 localized the main group differences to the CA1 region bilaterally and the CA2 and CA3 region on the

64 udy show that ERalpha in the rat hippocampal CA1 region but not the uterus undergoes enhanced interac

65 omatic layers of the dentate gyrus, CA3, and CA1 regions by using high-density, 96-site silicon probe

66 rol synapse numbers in the entorhinal cortex/CA1 region circuit by acting as a domain-specific postsy

67 lls in the stratum oriens of the hippocampal CA1 region confirmed that these cells were interneurons.

68 The level of expression in the CA1 region decreased dramatically between 24 and 72 h af

69 cingulate, basolateral amygdala, hippocampal CA1 region, dentate gyrus, nucleus accumbens shell and c

70 al synaptic transmission was observed in the CA1 region despite treatment with kynurenate.

71 ckdown of synaptotagmin-1 in the hippocampal CA1 region did not impede acquisition of recent contextu

72 tiviral shRNA-HCN1 in the dorsal hippocampal CA1 region displayed antidepressant- and anxiolytic-like

73 tamate were not increased in the hippocampal CA1 region during a 2-h postmortem interval, although in

74 that PI3-kinase activity was induced in the CA1 region during LTP of field EPSPs (fEPSPs) and that t

75 nd that MEK binding and residues in the KSR1 CA1 region enable KSR1 to form a ternary complex with B-

76 Previous work in the CA1 region has shown that the response to stimulation of

77 of EC and hippocampal dentate gyrus, CA3 and CA1 regions have revealed their distinct functions in le

78 numbers of a specific subset of synapses in CA1-region hippocampal neurons, suggesting that Lphn2 ac

79 transcriptome plasticity in the hippocampal CA1 region in aging and AD models and suggests that hist

80 ed an increase of the synapse density in the CA1 region in knockouts as well as control littermates.

81 in the pyramidal neurons of the hippocampal CA1 region in mice, blocking postsynaptic expression of

82 with learning and memory in the hippocampal CA1 region in rats.

83 ty of axospinous synapses in the hippocampal CA1 region in young rats, yet this is attenuated in aged

84 a showing a dissociation between the CA3 and CA1 regions in their responses to this local-global conf

85 Behaviorally, loss of Lphn2 from the CA1 region increased spatial memory retention but decrea

86 The hippocampal CA1 region is crucial for converting new memories into l

87 that the induction of LTD in the hippocampal CA1 region is dependent on ionotropic, rather than metab

88 The hippocampal CA1 region is highly vulnerable to ischaemic stroke.

89 ndocytosis of AMPA receptors (AMPARs) in the CA1 region is protein synthesis dependent.

90 wn of HCN1 protein in the dorsal hippocampal CA1 region is sufficient to produce antidepressant-like

91 derived from AC8 KO mice fail to demonstrate CA1-region long-term depression after low-frequency stim

92 In the hippocampal CA1 region, low-frequency stimulation (LFS; 200 pulses a

93 form of CREB, in hippocampal neurons of the CA1 region lowers the threshold for eliciting the late,

94 r, conditional knockout of beta-neurexins in CA1-region neurons impaired contextual fear memories.

95 In CA1-region neurons in vivo, Lphn2 was specifically targe

96 bodies of the dentate gyrus and the CA3 and CA1 regions, neurons that are involved in the major inpu

97 antly increased in dorsal but not in ventral CA1 region/neurons following chronic unpredictable stres

98 g-term potentiation (LTP) in the hippocampal CA1 region of 28-day-old control animals.

99 s) and field potentials were recorded in the CA1 region of acute hippocampal slices.

100 Inhibitory synapses in the rat hippocampal CA1 region of acute slices were studied using whole-cell

101 vels of all CK1 isoforms are elevated in the CA1 region of AD hippocampus relative to controls, with

102 these neurons (granule cell layer), and the CA1 region of Ammon's horn, which is the principal site

103 experiment 2, temporary inactivation of the CA1 region of anterior dorsal hippocampus by microinfusi

104 nd cortical regions of APP23 mice and in the CA1 region of control mice.

105 ned on two memory tasks, and the hippocampal CA1 region of each was analyzed on an individual microar

106 00 cells/microl) of cells dissected from the CA1 region of foetal brain at embryonic day 94-96, or of

107 term potentiation (LTP) were detected in the CA1 region of hippocampal brain slices of HIVE mice.

108 -induced long-term potentiation (LTP) in the CA1 region of hippocampal slices but had no significant

109 one train of tetanic stimulation (TS) in the CA1 region of hippocampal slices from both nulliparous f

110 r NR2A and NR2B subunits was elevated in the CA1 region of hippocampal slices from CIE-treated animal

111 ow that long-term potentiation (LTP), in the CA1 region of hippocampal slices from this mouse, is imp

112 voked potentials (EP) were determined in the CA1 region of hippocampal slices harvested from animals

113 theta frequency synaptic stimulation in the CA1 region of hippocampal slices maintained in vitro.

114 tory postsynaptic potentials (fEPSPs) in the CA1 region of hippocampal slices prepared from control r

115 In the CA1 region of hippocampal slices prepared from juvenile

116 In the CA1 region of hippocampal slices, briefly elevating extr

117 ough extracellular field recordings from the CA1 region of hippocampal slices, whereas behavioral tec

118 e-cell configuration from pyramidal cells of CA1 region of hippocampal slices.

119 nterior hypothalamus, centromedial thalamus, CA1 region of hippocampus and dentate gyrus, substantia

120 In both the CA1 region of hippocampus and the dentate gyrus, MOR-lik

121 ompared the distribution of cadherins in the CA1 region of hippocampus at postnatal days 2, 3 (P2-3)

122 bserved that the dendritic complexity in the CA1 region of hippocampus but not in CA2 is significantl

123 er)-N,N,N',N'-tetraacetic acid (EGTA) in the CA1 region of hippocampus in vivo.

124 Animals with cannulae implanted in the CA1 region of hippocampus were subjected to extinction o

125 nxiolytic effects of NPY are mediated in the CA1 region of hippocampus, and NPY injection into hippoc

126 sion, histological damage was evident in the CA1 region of hippocampus, correlated with a high expres

127 In the CA1 region of hippocampus, septal GABAergic projections

128 dal cells and subsets of interneurons in the CA1 region of hippocampus.

129 ng patterns of place cells recorded from the CA1 region of its hippocampus is used to develop a two-s

130 corded from either the dentate gyrus (DG) or CA1 region of mouse hippocampal slices in vitro.

131 s on ERK activation and LTP induction in the CA1 region of mouse hippocampal slices.

132 bited long term potentiation recorded in the CA1 region of mouse hippocampus slices.

133 ed events accompanying SD in the hippocampal CA1 region of murine brain slices, using whole-cell reco

134 rst stimulation, is perturbed in hippocampal CA1 region of old but not young htau mice.

135 that neuronal activity in the plaque-bearing CA1 region of older mice is profoundly altered.

136 y demonstrated a preserved morphology of the CA1 region of preconditioned rats, whereas pyknosis was

137 sal synaptic transmission in the hippocampal CA1 region of PSD-93 and PSD-95 mutant mice and compared

138 sure adenosine release during hypoxia in the CA1 region of rat hippocampal slices in conjunction with

139 anolone, we found that immunostaining in the CA1 region of rat hippocampal slices was confined to pyr

140 In the CA1 region of rat hippocampal slices, we observed that l

141 rom three different types of neurones in the CA1 region of rat hippocampal slices: pyramidal cells, f

142 anisms of NMDA receptor-dependent LTD in the CA1 region of rat hippocampus but there is still conside

143 upling of theta and gamma oscillators in the CA1 region of rat hippocampus during maze exploration an

144 trodes in the dorsal and ventral hippocampal CA1 region of rats and recorded the neuronal activity du

145 term potentiation (L-LTP) in the hippocampal CA1 region of rodents.

146 Here we examine the participation of the CA1 region of the dorsal hippocampus (CA1) and the basol

147 Estrogens induce synaptogenesis in the CA1 region of the dorsal hippocampus during the estrous

148 nd progestins regulate synaptogenesis in the CA1 region of the dorsal hippocampus during the estrous

149 combination in comparison to vehicle in the CA1 region of the dorsal hippocampus.

150 a, or green fluorescent protein (GFP) in the CA1 region of the dorsal hippocampus.

151 pyramidal neurons acutely isolated from the CA1 region of the early postnatal hippocampus, l-glutama

152 -induced long-term potentiation (LTP) in the CA1 region of the hippocampal slice is not altered in Dy

153 obal brain ischemia causes cell death in the CA1 region of the hippocampus 3-5 days after reperfusion

154 orebrain that caused selective damage in the CA1 region of the hippocampus also enhanced the producti

155 ed to abnormal long-term potentiation in the CA1 region of the hippocampus and consequently to defici

156 ings of putative excitatory neurons from the CA1 region of the hippocampus and the deep layers of the

157 s in place-specific firing of neurons in the CA1 region of the hippocampus and the relationship betwe

158 Here, we assess how dendritic spines in the CA1 region of the hippocampus are affected by morphine-c

159 nd learning-induced activation of ERK in the CA1 region of the hippocampus are attenuated in mice lac

160 Neurons within the CA1 region of the hippocampus are co-activated during hi

161 Neurons within the CA1 region of the hippocampus are co-activated during hi

162 and local field potential activity from the CA1 region of the hippocampus as rats performed a contex

163 In vivo field potential recordings in the CA1 region of the hippocampus before, during and after t

164 ependent long-term potentiation (LTP) in the CA1 region of the hippocampus can be divided into an ear

165 Interestingly, inactivation of the dorsal CA1 region of the hippocampus did not affect relative re

166 Glutamate released at synapses in the CA1 region of the hippocampus escapes the synaptic cleft

167 urons involved in feedback inhibition in the CA1 region of the hippocampus exhibit an unusual form of

168 Place cells in the CA1 region of the hippocampus express location-specific

169 phosphorylation was seen to increase in the CA1 region of the hippocampus following memory acquisiti

170 In the CA1 region of the hippocampus from rats, we found that b

171 glutamatergic NMDA receptors, in the dorsal CA1 region of the hippocampus hinders retention of long-

172 ncy (100-200 Hz) oscillatory patterns in the CA1 region of the hippocampus in the absence of theta ac

173 of GABA-mediated synaptic inhibition in the CA1 region of the hippocampus in Ts65Dn mice, a model sy

174 or the activity of PERK, specifically in the CA1 region of the hippocampus in young adult male mice,

175 anic stimulation of axons terminating in the CA1 region of the hippocampus induces oscillations in th

176 The CA1 region of the hippocampus is particularly vulnerable

177 The CA1 region of the hippocampus is split into two well-org

178 affer collateral pathway from the CA3 to the CA1 region of the hippocampus is thought to involve post

179 excitatory glutamatergic synapses within the CA1 region of the hippocampus may play a role in storing

180 AR)-induction of long term depression in the CA1 region of the hippocampus of mice.

181 lly, the reduction of PERK expression in the CA1 region of the hippocampus of middle-aged male mice u

182 harp-wave ripple events were examined in the CA1 region of the hippocampus of old (n = 5) and young (

183 the induction of NMDAR-dependent LTD in the CA1 region of the hippocampus of postpubescent mice (pos

184 d with bilateral guide cannulae aimed at the CA1 region of the hippocampus one week prior to measurin

185 In the CA1 region of the hippocampus pyramidal neuron basilar d

186 The CA1 region of the hippocampus receives distinct patterns

187 modeling efforts suggest that rhythms in the CA1 region of the hippocampus that are in the beta range

188 hways, and modulate the vulnerability of the CA1 region of the hippocampus to the episodic hypoxia th

189 increase in paired-pulse facilitation in the CA1 region of the hippocampus to the level observed in R

190 gth of dendrites in pyramidal neurons of the CA1 region of the hippocampus using microtubule-associat

191 asured the activity of single units from the CA1 region of the hippocampus while GluA1 knock-out (Glu

192 s for imaging the activity of neurons in the CA1 region of the hippocampus with subcellular resolutio

193 en, bed nucleus of the stria terminalis, and CA1 region of the hippocampus).

194 ve interneurons in the stratum oriens of the CA1 region of the hippocampus, accompanied by nonsignifi

195 Here we report that in the CA1 region of the hippocampus, agonists of native P2Y(1)

196 gray matter pathology was restricted to the CA1 region of the hippocampus, and consisted of edematou

197 cohol consumption increased DAT sites in the CA1 region of the hippocampus, basolateral nucleus of th

198 olin in cultured cortical neurons and in the CA1 region of the hippocampus, both the Syt12 knock-out

199 e hypothalamus, the arcuate nucleus, and the CA1 region of the hippocampus, but not in two thalamic n

200 is region evokes excitatory responses in the CA1 region of the hippocampus, but nothing is known abou

201 -induced long-term potentiation (LTP) in the CA1 region of the hippocampus, but selectively abolished

202 the motor cortex, piriform cortex, striatum, CA1 region of the hippocampus, dentate gyrus, and basola

203 ng from NMDA-type glutamate receptors in the CA1 region of the hippocampus, GRF1 promotes LTD, wherea

204 The results showed that in the CA1 region of the hippocampus, hypothyroid or stress par

205 titutive nuclear localization of MKL1 in the CA1 region of the hippocampus, in the deep layers of the

206 best characterized form of LTP occurs in the CA1 region of the hippocampus, in which LTP is initiated

207 Specifically, in slice preparations from the CA1 region of the hippocampus, KCNH2-3.1 transgenic mice

208 actory bulb, corpus striatum, dentate gyrus, CA1 region of the hippocampus, layers I and II of the ce

209 ort here that, unlike LTP in the more mature CA1 region of the hippocampus, LTP in neonatal rodent hi

210 In the CA1 region of the hippocampus, LTP is thought to be init

211 Evidence suggests that, in the CA1 region of the hippocampus, NR2A-containing NMDARs pr

212 regions, including the striatum, cerebellum, CA1 region of the hippocampus, or deep layers of cerebra

213 Here we show that, in the CA1 region of the hippocampus, reduction of postsynaptic

214 In the CA1 region of the hippocampus, stimulation of the Schaff

215 In the CA1 region of the hippocampus, the combined activation o

216 One example--long-term potentation in the CA1 region of the hippocampus--has been studied extensiv

217 h the vesicular GABA transporter VGAT in the CA1 region of the hippocampus.

218 ighly enriched in inhibitory synapses in the CA1 region of the hippocampus.

219 ransmission and short-term plasticity in the CA1 region of the hippocampus.

220 ls of the AMPA receptor subunit GluR1 in the CA1 region of the hippocampus.

221 ency and amplitude of miniature EPSCs in the CA1 region of the hippocampus.

222 ribution of the place fields recorded in the CA1 region of the hippocampus.

223 the central nucleus of the amygdala, and the CA1 region of the hippocampus.

224 del was developed for L-LTP induction in the CA1 region of the hippocampus.

225 romedial nucleus of the hypothalamus and the CA1 region of the hippocampus.

226 to basal dendrites of pyramidal cells in the CA1 region of the hippocampus.

227 odulators of interneuron excitability in the CA1 region of the hippocampus.

228 tate gyrus and long-term potentiation in the CA1 region of the hippocampus.

229 xcitability and synaptic transmission in the CA1 region of the hippocampus.

230 otein (CREB) within the hypothalamus and the CA1 region of the hippocampus.

231 7a labeled many second-order neurons in the CA1 region of the hippocampus.

232 ation in diverse interneuron subtypes in the CA1 region of the hippocampus.

233 Cs participate in synaptic plasticity in the CA1 region of the hippocampus.

234 postsynaptic potentials were recorded in the CA1 region of the hippocampus.

235 ngs to measure dendritic excitability in the CA1 region of the hippocampus.

236 memory decline has focused primarily on the CA1 region of the hippocampus.

237 ynapses is given, focusing on a study in the CA1 region of the hippocampus.

238 nance of normal spine synapse density in the CA1 region of the male rat hippocampus.

239 ime course and postsynaptic responses in the CA1 region of the mouse hippocampus during this critical

240 components of inhibitory transmission in the CA1 region of the mouse hippocampus.

241 ons-theta-driving neurons in the hippocampal CA1 region of the mouse-to characterize the interactions

242 in synaptic function has been studied in the CA1 region of the rat hippocampal slice.

243 Here bilateral recordings in the dorsal CA1 region of the rat hippocampus show that retrospectiv

244 ues and confocal microscopy were used in the CA1 region of the rat hippocampus to compare acute slice

245 In the CA1 region of the rat hippocampus, long-term potentiatio

246 In the CA1 region of the rat hippocampus, metabotropic glutamat

247 dendritic spines of pyramidal neurons in the CA1 region of the rat hippocampus.

248 ng back from reference neuronal units in the CA1 region of the simulated hippocampus to all of the sy

249 We investigated this issue in the CA1 region of urethane-anesthetized male rats using ampe

250 ic density and plasticity in the hippocampal CA1 region of young female rats but fails to do so in ag

251 xospinous synapse density in the hippocampal CA1 region of young female rats but fails to do so in ag

252 rmed that the AMPA/NMDA ratio was reduced in CA1 regions of hippocampal slices from lithium-treated a

253 10 mRNA 2 kb form in the ipsilateral CA3 and CA1 regions of LTP animals.

254 ission potentiates synaptic responses in the CA1 regions of rat and mouse hippocampus.

255 s compared in the entorhinal cortex (EC) and CA1 regions of the hippocampus perfused with Mg(2+)-free

256 howed trends toward larger dentate gyrus and CA1 regions of the hippocampus.

257 nization of pyramidal neurons in the CA3 and CA1 regions of the hippocampus.

258 iation was examined in the dentate gyrus and CA1 regions of the hippocampus.

259 In dentate and CA1 regions of the lesioned hippocampus receiving grafts

260 tic spine plasticity in the cornu ammonis 1 (CA1) region of the hippocampus, and GPR30 (G-protein cou

261 tamate-mediated synaptic transmission in the CA1 region or dentate gyrus was unaffected in hippocampa

262 y anterior cingulate) to hippocampus (CA3 to CA1 region) projection in mice, and find that optogeneti

263 bpopulation of astrocytes in the hippocampal CA1 region prominently expressed nAChRbeta4 (and nAChRal

264 e reverses stabilized LTP in the hippocampal CA1 region, providing the first evidence that consolidat

265 % above control levels, most notably, in the CA1 region pyramidal cell layer.

266 %) increase in NMDA-induced neurotoxicity in CA1 region pyramidal cells.

267 expression of Cre-recombinase in hippocampal CA1 region pyramidal neurons at postnatal day 0 (P0) or

268 In synapses formed by CA1-region pyramidal neurons onto burst-firing subiculum

269 in the number of synapses (seen by EM in the CA1 region), reduction of synaptic proteins, and localiz

270 ubunits in the hippocampal dentate gyrus and CA1 regions, respectively.

271 ophysiological recordings in the hippocampal CA1 region revealed that short-term and long-term synapt

272 scope analysis of hippocampal tissues in the CA1 region showed an increase in the number of mitochond

273 y alter neurophysiological properties of the CA1 region, such as long-term potentiation.

274 ame transitions in the dentate gyrus and the CA1 region suggests that this process probably occurs be

275 Thus in the CA1 region synaptic plasticity is likely to be regulated

276 expression in the dentate gyrus, hippocampal CA1 region, thalamus, and striatum but not in the cerebr

277 Specifically, in the hippocampal CA1 region, the NL3(R451C) mutation caused an approximat

278 city of pyramidal neurons in the hippocampal CA1 region to maintain an appropriate homeostatic state

279 rmal long-term plasticity in the hippocampal CA1 region together with deficits in learning and memory

280 We found that LTP induced in the CA1 region using theta-frequency stimulation (5 Hz) is s

281 omatic layers of the dentate gyrus, CA3, and CA1 regions using high-density, 96-site silicon probes.

282 the CA3 recurrent system, which entrains the CA1 region via its interneurons.

283 Intracellular Ca(2+) in CA1 region was measured with fura-2 during preconditioni

284 al dendritic spine density on neurons in the CA1 region was not enhanced by E treatment, a finding th

285 Subsequent cell death in the CA1 region was quantified using propidium iodide stainin

286 ted synaptic potentiation in the hippocampal CA1 region was significantly reduced compared with the c

287 ransfer of neuronal patterns from the CA3 to CA1 region was studied by simultaneous recording of neur

288 ells and field recordings in the hippocampal CA1 region, we investigated the specific contribution of

289 tioxidant action, and neuroprotection in the CA1 region were lost after long-term E(2) deprivation, a

290 duced levels of synaptic transmission in the CA1 region when compared with wild-type littermate contr

291 l neurodegenerative changes were seen in the CA1 region when the slices were treated with Abeta plus

292 ion of amyloid deposition in the hippocampal CA1 region, where RIDNs predominantly formed before amyl

293 supports L-type channel-dependent LTP in the CA1 region, whereas nmdaLTP depends solely on MMP-9.

294 ings suggest that fast ripples emerge in the CA1 region, whereas slow (100-130 Hz) oscillatory patter

295 or long-term potentiation in the hippocampus CA1 region, which is thought to underlie learning and me

296 selective death of pyramidal neurons in the CA1 region, which was prevented by treatment with an NMD

297 ies were primarily detected in the subiculum/CA1 region, which was therefore the focus of analysis.

298 al model by probing interburst epochs in the CA1 region with local dentate gyrus stimulation just sup

299 Finally, rats infused in the dorsal CA1 region with thapsigargin, an irreversible inhibitor

300 of long-term potentiation in the hippocampal CA1 region without affecting basal synaptic transmission