ライフサイエンスコーパス: CAM kinase IV

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1 d CaMKIK beta are also capable of activating CaM kinase IV.

2 CaM kinase IV activates the transcription factor CREB, a

3 These mechanisms, which implicate CaM kinase IV and CREB in the control of BDNF expression

4 We report that CaM kinase IV and CREB play a critical role in mediating

5 n of a transcriptional program that involves CaM kinase IV and CREB-mediated signaling to the nucleus

6 ng via calcium/calmodulin-dependent protein (CaM) kinase IV and microtubule-associated protein (MAP)

7 n be induced by activation of CaM kinase II, CaM kinase IV, and protein kinase A, but not by activati

8 Both CaM kinase I and CaM kinase IV are able to phosphorylate this Ser residue

9 A constitutively active mutant of CaM kinase IV, but not of CaM kinase II, leads to activa

10 a recombinant CaM-KK activates in vitro both CaM-kinase IV (CaM-K IV) and CaM-K I.

11 synapse, and activation of transcription by CaM kinase IV (CAMKIV) and MAPK.

12 ible for persistent CREB phosphorylation and CaM kinase IV (CaMKIV) responsible for phosphorylating t

13 ivation of Ca(2+)/CaM kinase kinase beta and CaM kinase IV (CaMKIV).

14 Type I adenylyl cyclase has two CaM kinase IV consensus phosphorylation sequences near i

15 that type I adenylyl cyclase is inhibited by CaM kinase IV in vivo.

16 nt negative form of CREB blocks calcium- and CaM kinase IV-induced dendritic growth.

17 inhibitors, a constitutively active form of CaM kinase IV induces dendritic growth in the absence of

18 ession of constitutively active or wild-type CaM kinase IV inhibited Ca2+ stimulation of adenylyl cyc

19 r serine to alanine by mutagenesis abolished CaM kinase IV inhibition of adenylyl cyclase.

20 We propose that CaM kinase IV may function as a negative feedback regula

21 Activation of CaM kinase I and CaM kinase IV occurs via phosphorylation of an equivalen

22 The effects of CaM kinase IV on the promoter require an intact CRE.

23 of this enzyme may be directly inhibited by CaM kinase IV phosphorylation.

24 esults in the calcium-mediated activation of CaM kinase IV, phosphorylation of CREB, increased expres

25 Neuronal depolarization and CaM kinase IV signaling alter the splicing of multiple e

26 titutively active mutants of CaM kinase I or CaM kinase IV specifically blocks nuclear targeting of C

27 lular stimulation, and a kinase-dead form of CaM kinase IV suppresses dendritic growth induced by cal

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