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1 cilitate safer application of effective CD19 CAR T-cell therapy.
2 ncing CRS and other adverse events following CAR T-cell therapy.
3 inued for 7.5 months after the initiation of CAR T-cell therapy.
4 ed and refractory MLL-B-ALL who receive CD19 CAR-T-cell therapy.
5 nical trials and inform the design of future CAR T cell therapies.
6 ective strategy for improving the potency of CAR T cell therapies.
7 data illustrate the potential use of SLAMF7-CAR T-cell therapy as an effective treatment against mul
9 unexpected organ damage and deaths following CAR T-cell therapy first highlighted the possible danger
11 inical success of chimeric antigen receptor (CAR) T cell therapy for B cell malignancies represents a
14 the bone marrow by flow cytometry after CD19 CAR-T-cell therapy; however, within 1 month of CAR-T-cel
16 ese devices may improve the effectiveness of CAR T cell therapy in solid tumors and help protect agai
18 i-CD19 chimeric antigen receptor-modified T (CAR-T) cell therapy in patients with chronic lymphocytic
20 e, as well as the unaddressed integration of CAR T-cell therapy into conventional anticancer treatmen
22 ever, in solid tumors, the full potential of CAR T cell therapy is limited by the availability of cel
25 efficacy, the general application of current CAR-T--cell therapy is limited by serious treatment-rela
26 dicate that FRbeta is a promising target for CAR T-cell therapy of AML, which may be augmented by com
28 ologous anti-CD19 chimeric antigen receptor (CAR) T-cell therapy, showed efficacy in patients with re
30 s the efficacy of chimeric antigen receptor (CAR) T cell therapies, which redirect T cells to solid t
31 efractory large B-cell lymphoma who received CAR T-cell therapy with axi-cel had high levels of durab
32 esized that a combination of alpha-4-1BB and CAR T-cell therapy would result in improved antitumor re
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