ライフサイエンスコーパス: CBF

1 CBF and CMRO2 were quantified before, during and after d

2 CBF and relative CBF in the most perfused area of each n

3 CBF at rest was quantified on a voxelwise basis using ar

4 CBF change in the bilateral parietal cortices also corre

5 CBF is correlated with tumor microvascular density.

6 CBF reduction required protein kinase C but was not asso

7 CBF responses to neural activity (functional hyperemia),

8 CBF was significantly restored with ECFCs and almost tot

9 CBF was strongly correlated with microvascular density (

10 CBFs and CBF regulon genes were down-regulated in rdm4 b

11 (27,467 vs. 20,841 mm Hg/min(-1); p = 0.02), CBF was similar in both groups.

12 In addition, CBF-independent classes of BR-regulated COR genes are id

13 e ameliorated white matter lesions, although CBF responses were unchanged.

14 eported together as core binding factor AML (CBF-AML).

15 Decreased amygdala CBF correlated with reduced depressive symptoms.

16 t on BBB disruption, cerebral apoptosis, and CBF were evaluated by SPECT/CT up to 14 d after MCAO.

17 ial impact on BBB disruption, apoptosis, and CBF.

18 The combination of BOLD-based and CBF-based fMRI can provide a signature of dopaminergic n

19 patial agreement between concurrent BOLD and CBF responses to median nerve stimulation, with primary

20 This dissociation of BOLD and CBF suggests that dopamine increases energy metabolism b

21 CBFs and CBF regulon genes were down-regulated in rdm4 but not nr

22 NA start site by Tup family corepressors and CBF/NF-Y proteins.

23 C-REPEAT BINDING FACTOR (CBF)-dependent and CBF-independent pathways.

24 ons between patterns of Abeta deposition and CBF or neural activity.

25 Robust associations between DVR and CBF at negligible Abeta levels, together with similar sp

26 tive voxelwise relationships between DVR and CBF or R1 were observed.

27 A more robust relationship between DVR and CBF was observed in the lower tertile of DVR, that is, n

28 show both resting-state field potentials and CBF were depressed after cocaine administration (19.8+/-

29 Correlations between LFP power and CBF, used to estimate NVC, were enhanced under high ACh

30 as specific associations between puberty and CBF.

31 -skull electroencephalography recordings and CBF with laser Doppler flowmetry in the rat's somatosens

32 ype Atf1, C(2)H(2)zinc finger-type Rst2, and CBF/NF-Y-type Php5 proteins.

33 FA in the SN and CBF in the caudate nucleus were inversely correlated wit

34 d artery (ICA) and vertebral artery (VA) and CBF velocity at the middle cerebral artery (MCA).

35 induced CBF increases than to the background CBF should be considered when interpreting functional MR

36 of understanding the interplay between basal CBF and resting neuronal activity.

37 from the mechanism of HIV-1/SIV Vif by being CBF-beta independent and requiring different ubiquitin l

38 he unique cofactor core binding factor beta (CBF-beta) and canonical ligase components Cullin 5 (CUL5

39 The correlation between CBF and microvascular density was analyzed in specimens

40 tudies that reported no relationship between CBF changes and the occurrence of AMS.

41 With CVR = BP/CBF, Slope-CVRICA , Slope-CVRVA and Slope-CVRiMCA were d

42 ld signaling is integrated into the clock by CBF-mediated regulation of LUX expression, thereby defin

43 ion (amyloid-beta load) may be influenced by CBF, in a cohort of controls and patients with mild cogn

44 ed by rdm4 overlapped with those affected by CBFs.

45 Transcript levels of candidate CBF target genes, COR14B and DHN5 were increased in the

46 n diminishes global and individual capillary CBF responses to neuronal stimuli, resulting in neurovas

47 o gain mechanistic insight into HvCBF4/CBFIV CBFs we overexpressed Hv-CBF2A in spring barley (Hordeum

48 terial CO2 are particularly potent to change CBF (1 mmHg variation in arterial CO2 changes CBF by 3%-

49 BF (1 mmHg variation in arterial CO2 changes CBF by 3%-4%), the coupling mechanism is incompletely un

50 ed that exchange transfusions would decrease CBF and OEF by increasing CaO2, thereby relieving cerebr

51 Decreased CBF in the right amygdala and hippocampus correlated wit

52 Decreased CBF only was observed after MDMA, and this was localized

53 An acute hypoxic challenge decreased CBF (P < 0.05) in untrained VEGF(f/f) , untrained VEGF(H

54 Our results show significantly decreased CBF and CMRO2 during hypothermic CPB.

55 he ability of this approach to differentiate CBF in different locations of the oviduct at different p

56 his study demonstrate common and dissociable CBF abnormalities across neuropsychiatric disorders in y

57 tary to PANX1, in ATP release and downstream CBF modulation following a mechanical stimulus in airway

58 Several DREB/CBF TFs directly promote transcription of the IAA5 and I

59 8 families, including 26 members of the DREB/CBF family.

60 DVR-CBF associations in individuals with a higher DVR are mo

61 er with similar spatial distributions of DVR-CBF and DVR-R1 correlations, suggest that regional distr

62 The spatial distributions of the DVR-CBF and DVR-R1 correlations showed similar patterns.

63 and stroke, will give rise to dysfunctional CBF regulation and may result in subsequent neuronal dam

64 rease in the thiol containing compounds in E-CBF and E-MBF were attributed to their oxidation to othe

65 compounds of the two encapsulated samples (E-CBF and E-MBF) was investigated.

66 SIL, producing divergent influences on early CBF deficits and later white matter lesions.

67 Reduction of elevated CBF and OEF, both globally and regionally, suggests that

68 roinjection of glycine into the SCN elevated CBF in a dose-dependent manner, whereas no effect was ob

69 r astrocytes in the regulation of CO2-evoked CBF responses.

70 onic ACh deprivation hindered whisker-evoked CBF responses and the amplitude and power in most freque

71 one significantly potentiated whisker-evoked CBF responses through muscarinic ACh receptors and concu

72 r domain (NICD) and the transcription factor CBF-1/RBP-j, Su(H), Lag-1 (CSL) is a key event in Notch

73 n for regulators of C-REPEAT BINDING FACTOR (CBF) gene expression (RCFs), we identified RCF2 and foun

74 ssion in the poplar C-repeat binding factor (CBF) gene family.

75 n that includes the C-repeat binding factor (CBF) locus.

76 The C-REPEAT-BINDING FACTOR (CBF) pathway has important roles in plant responses to c

77 olves action of the C-repeat binding factor (CBF) regulatory pathway.

78 Expression of the C-repeat-binding factor (CBF) transcription factors is induced by cold stress, wh

79 g downstream of the C-repeat binding factor (CBF) transcription factors to recruit the core Mediator

80 angements involving the core-binding factor (CBF) transcriptional complex.

81 HYDRATION-RESPONSIVE ELEMENT BINDING FACTOR (CBF) transcriptional regulators that control cold respon

82 nder cold stress by C-REPEAT BINDING FACTOR (CBF)-dependent and CBF-independent pathways.

83 (16.1%) of aberrant non-core-binding-factor (CBF) karyotype patients.

84 RUNX1/CBFbeta (core binding factor [CBF]) is a heterodimeric transcription factor complex th

85 C-Repeat Binding Factors (CBFs) are DNA-binding transcriptional activators of gene

86 nscription factors C-REPEAT BINDING FACTORS (CBFs).

87 ention has been paid to cerebral blood flow (CBF) alterations in IGE detected by arterial spin labell

88 Here, cerebral blood flow (CBF) and blood oxygen-level dependent (BOLD) resting-sta

89 rel cortex, measured by cerebral blood flow (CBF) and neurophysiological recordings (local field pote

90 es in the regulation of cerebral blood flow (CBF) and neurovascular coupling remains, however, under

91 Cerebral blood flow (CBF) and oxygen extraction fraction (OEF) are elevated i

92 study aims to evaluate cerebral blood flow (CBF) and oxygen metabolism (CMRO2) intraoperatively in n

93 al correlations between cerebral blood flow (CBF) and quantitative histologic microvascular data.

94 s displaying changes in cerebral blood flow (CBF) and RSFC after MDMA administration.

95 Resting cerebral blood flow (CBF) and task-related activation of the amygdala were me

96 cological reductions in cerebral blood flow (CBF) at baseline would lower the 'CBF reserve', and ulti

97 on of glycine elevated cutaneous blood flow (CBF) at the plantar surface in a dose-dependent manner,

98 r response and improves cerebral blood flow (CBF) by activating the rostral ventrolateral medulla.

99 Resting-state regional cerebral blood flow (CBF) can be measured noninvasively with magnetic resonan

100 sis of the ASL relative cerebral blood flow (CBF) data, receiver operating characteristic (ROC) curve

101 d phenotypes, including cerebral blood flow (CBF) deficits, white matter lesions, and Notch3(ECD) dep

102 rain physiology such as cerebral blood flow (CBF) have never been investigated.

103 measured resting-state cerebral blood flow (CBF) in 29 adult smokers across four conditions: (1) nic

104 regional reductions in cerebral blood flow (CBF) in response to decreased oxygen supply (hypoxia) at

105 and instant increase of cerebral blood flow (CBF) in response to neural activation.

106 sly been shown to evoke cerebral blood flow (CBF) increases via the release of the vasodilator PgE2 W

107 ur study, we found that Cerebral Blood Flow (CBF) is 90.91% sensitive and 100% specific in examining

108 Cerebral blood flow (CBF) is controlled by arterial blood pressure, arterial

109 ls suggest that reduced cerebral blood flow (CBF) is one of the most enduring physiological deficits

110 Cessation of cerebral blood flow (CBF) leads to cell death in the infarct core, but tissue

111 Changes in cerebral blood flow (CBF) may occur with acute exposure to high altitude; how

112 , fiber number (FN) and cerebral blood flow (CBF) measurements.

113 nfluenced by changes in cerebral blood flow (CBF) or radiotracer clearance.

114 aine-induced changes in cerebral blood flow (CBF) reflect neuronal activation or its vasoactive effec

115 d TIMP3 and compromised cerebral blood flow (CBF) remains unknown.

116 nses decreased, whereas cerebral blood flow (CBF) responses increased.

117 les, increasing in turn cerebral blood flow (CBF) to areas with increased metabolic needs.

118 Cerebral blood flow (CBF) was measured at the internal carotid artery (ICA) a

119 ing period, hippocampal cerebral blood flow (CBF) was measured by functional magnetic resonance imagi

120 Cerebral blood flow (CBF) was measured using colour-coded duplex ultrasonogra

121 al and regional resting cerebral blood flow (CBF), (2) oxygen extraction fraction (OEF), and (3) cere

122 tructural connectivity, cerebral blood flow (CBF), and corticospinal excitability, respectively, befo

123 ood density, lower mean cerebral blood flow (CBF), and significant cerebral circulatory delay compare

124 onships between DVR and cerebral blood flow (CBF), as well as relative CBF (R1), in nondemented older

125 ted with a reduction in cerebral blood flow (CBF), but regulation of CBF during strenuous exercise in

126 vascular reactivity and cerebral blood flow (CBF), but, to our knowledge, abnormalities in cerebral b

127 Increased visual cortex cerebral blood flow (CBF), decreased visual cortex alpha power, and a greatly

128 rapid identification of cerebral blood flow (CBF), prediction of infarct area and hemoglobin oxygenat

129 as surrogate indices of cerebral blood flow (CBF), with a secondary goal of directly examining the ex

130 e (Pbto2), and regional cerebral blood flow (CBF).

131 cerebral apoptosis, and cerebral blood flow (CBF).

132 celerate and decelerate coronary blood flow (CBF).

133 performed, in which 3D cerebral-blood-flow (CBF) networks in mouse brain over a large field-of-view

134 ancy may be attributed to the use of ASL for CBF measurement at both sea level and high altitude in t

135 lts delineate sex-specific growth curves for CBF during youth and for the first time to our knowledge

136 esting a potential prognostic indication for CBF as a biomarker.

137 o to assess CMBs, arterial spin labeling for CBF, and T1- and T2-weighted imaging for atrophy, white

138 ated gene loci, indicating a requirement for CBF heterodimers in EBNA3 recruitment during target-gene

139 degeneration of cholinergic basal forebrain (CBF) neurons.

140 ATP, which increases ciliary beat frequency (CBF) and speeds up mucociliary clearance.

141 the cilia location and cilia beat frequency (CBF) in the intact mouse oviduct with micro-scale spatia

142 ial cells, we imaged ciliary beat frequency (CBF), intracellular calcium, and nitric oxide (NO).

143 ezing tolerance is dependent on a functional CBF-CRT/DRE regulatory module.

144 Global CBF, intra-cranial arterial blood velocities, extra-cran

145 st analyses (percentage difference in global CBF, -25.3%; P = .0003), with the largest reductions in

146 pared by conventional and microwave heating (CBF and MBF) of enzymatic hydrolysate of mushroom protei

147 -grade pediatric brain tumors display higher CBF than do low-grade tumors, and they may be accurately

148 Regionally, we found higher CBF in the right pallidum/putamen of the cannabis users

149 rcent increase of normocapnic to hypercapnic CBF normalized by the change in end-tidal carbon dioxide

150 Hyperemic CBF was less in AS compared with controls (2,170 vs. 2,7

151 Cold temperatures trigger the ICE1-CBF-COR transcriptional cascade in plants, which reprogr

152 Importantly, CBF in the dorsal midinsular cortex was both decreased a

153 ated the use of electrical TNS for improving CBF and brain oxygen tension (PbrO2), with the goal of d

154 raving positively correlated with changes in CBF from the denicotinized cigarette smoking conditions

155 t had a minimal relationship with changes in CBF.

156 sion, dehydration accelerated the decline in CBF by decreasing P aCO 2 and enhancing vasoconstrictor

157 Notably, within these regions the decline in CBF was similar between males and females in early puber

158 nalyses revealed post-treatment decreases in CBF in the temporal cortex, including the amygdala.

159 An increase in CBF was observed in wild-type ALIs following mechanical

160 in newly diagnosed AML, and particularly in CBF-AML, incited us to retrospectively investigate the i

161 viously unappreciated cooperating pathway in CBF-AML.

162 potential to recover if local reductions in CBF are restored.

163 Reductions in CBF were seen in brain regions typically associated with

164 Regional reductions in CBF, and associated vasoconstriction, within the default

165 ly compensated without an additional rise in CBF and associated with brain tissue hypoxia, or higher-

166 icuously divergent nonlinear trajectories in CBF evolution with development as modeled by penalized s

167 Increased CBF was also evident in the right amygdala but did not r

168 tion of D-serine into the SCN also increased CBF, whereas these effects were blocked in the presence

169 support that AMS may be related to increased CBF rather than vasodilation; these results contradict m

170 t increased DVR is associated with increased CBF in the frontal, parietal, temporal, and occipital co

171 ctions by cocaine to the stimulation-induced CBF increases than to the background CBF should be consi

172 diverse cooperating mutations may influence CBF-AML pathophysiology as well as clinical behavior and

173 To investigate CBF changes common across psychiatric disorders, we capi

174 Moreover, translocations involving CBFs are not sufficient to induce AML on its own and the

175 core-binding factor-acute myeloid leukemia (CBF-AML) (t[8;21] or inv[16]/t[16;16]) represents a favo

176 These effects were not related to local CBF changes in response to PIDs.

177 ION: Thrombectomy salvaged tissue with lower CBF, likely attributed to earlier reperfusion.

178 ges in cerebrovascular resistance (CVR = MAP/CBF) and MAP relative to baseline values.

179 Results Mean CBF was significantly higher for high-grade than for low

180 The mean CBF of IGE patients was significantly increased in the l

181 continuous arterial spin labeling to measure CBF at normocapnia (ie, breathing room air) and hypercap

182 and asymmetric spin echo sequences measured CBF and OEF, respectively, which were compared pre- and

183 lance of TIMP3 and ADAM17 activity modulates CBF through regulation of myocyte KV channel number.

184 Here we show that cold induction of most CBF regulon genes is lower in IT plants compared with SW

185 h a poorer prognosis compared with other non-CBF aberrant karyotypes and led to lower remission rates

186 r transcranial Doppler for the assessment of CBF.

187 2 genomic segment in the proximal cluster of CBF elements, a negative role of HvCBF3 in the distal cl

188 hts into the pathogenesis and development of CBF-AML, while highlighting dramatic differences in the

189 tions were performed to assess the effect of CBF and radiotracer clearance changes on SUVRs and nonin

190 vs 1 month postinjury; P < .001) evidence of CBF recovery in the right insular and superior temporal

191 mpk6 mutants display increased expression of CBF genes and enhanced freezing tolerance, whereas const

192 scade in plants causes reduced expression of CBF genes and hypersensitivity to freezing, suggesting t

193 tion factor that regulates the expression of CBF genes, and the phosphorylation promotes the degradat

194 A family of CBF transcription factors plays a major role in reconfig

195 iphoton microscopy, laser speckle imaging of CBF, and electrophysiological recordings in a mouse mode

196 Simulations showed a limited impact of CBF and radiotracer clearance changes on multilinear ref

197 plicity and, second, to assess the impact of CBF changes and radiotracer clearance on SUVRs and nonin

198 a potent vasodilator causing an increase of CBF.

199 id artery (CCA) haemodynamics (indicative of CBF and extra-cranial blood flow), middle cerebral arter

200 Inducer of CBF expression 1 (ICE1) is a master regulator of CBFs, a

201 Inducer of CBF expression 1 (ICE1) mediates the cold stress signal

202 that the cold signaling activator INDUCER OF CBF EXPRESSION 1 (ICE1), FLC and the floral promoter SUP

203 The influence of CBF and radiotracer clearance changes on amyloid-beta lo

204 o better understand the genomic landscape of CBF-AMLs, we analyzed both pediatric (n = 87) and adult

205 Measurements of CBF, metabolism, and Abeta were compared with the presen

206 gulatory element present in the promoters of CBF-regulated genes.

207 ary density pertaining to defined regions of CBF in response to ischemia.

208 ia could lead to dysfunctional regulation of CBF and subsequent neuronal damage.SIGNIFICANCE STATEMEN

209 cerebral blood flow (CBF), but regulation of CBF during strenuous exercise in the heat with dehydrati

210 K homology (KH) domain protein REGULATOR OF CBF GENE EXPRESSION 3 (RCF3) as a cofactor affecting miR

211 The resolution of CBF abnormalities closely mirrors previous reports from

212 signal thereby contributes to the rhythm of CBF expression and the downstream COLD RESPONSIVE expres

213 EBNA3C-conditional LCL confirmed the role of CBF in the regulation of EBNA3C-induced and -repressed g

214 and FLT3) were frequent in both subtypes of CBF-AML.

215 4 constitutively increased the expression of CBFs and regulon genes and decreased cold-induced membra

216 expression 1 (ICE1) is a master regulator of CBFs, and ICE1 stability is crucial for its function.

217 ncluding genes that are not known targets of CBFs.

218 1)C-PiB retention did not strongly depend on CBF across primary cortical regions.

219 The DVR images were regressed on CBF images on a voxel-by-voxel basis using robust biolog

220 se A3 proteins, whereas knockdown of CUL5 or CBF-beta did not.

221 on also increased transcript levels of other CBF genes at FROST RESISTANCE-H2-H2 (FR-H2) possessing C

222 ent the first in vivo mapping of the oviduct CBF in its native context, and demonstrate the ability o

223 dinal Study of Aging underwent (15)O-H2O PET CBF and dynamic (11)C-PiB PET.

224 tified FA in the SN(FA-SNAv), CBF in the PFC(CBF-PFCAv) and FA in the parietal white matter(FA-PWMAv)

225 Some Poplar CBF homologs exhibited patterns consistent with historic

226 sodilators, and decreases in blood pressure (CBF autoregulation) were similarly reduced in TgNotch3(R

227 ificantly increased systemic blood pressure, CBF and PbrO2 at the hyperacute phase of TBI.

228 . niger, and A. flavus cultures also reduced CBF by ~10% after 60 min exposure, but effects were bloc

229 de the first prospective evidence of reduced CBF in human concussion and subsequent recovery.

230 Bs was associated with significantly reduced CBF in multiple regions on voxelwise and region-of-inter

231 showed a nonsignificant trend toward reduced CBF.

232 Regional CBF may also provide a useful biological marker across d

233 Global resting CBF and regional CBF of right superior frontal cortex correlated positive

234 imaging (MRI) and abnormalities in regional CBF are present in many neuropsychiatric disorders.

235 udies documenting serial changes in regional CBF following human concussion have yet to be performed.

236 Increased regional CBF of 21.0 mL/100 g/min for 0 Hz, 25.9 mL/100 g/min for

237 destabilize ICE1, which negatively regulates CBF expression and freezing tolerance in plants.

238 CBF and relative CBF in the most perfused area of each neoplasm and contr

239 status (i.e., amyloid-beta load and relative CBF) through dynamic (11)C-PiB imaging.

240 rebral blood flow (CBF), as well as relative CBF (R1), in nondemented older adults.

241 omparison of ASL revealed decreased relative CBF in the dCON group compared with that in the sCON gro

242 ROC analysis of relative CBF in the PCC enabled discrimination of dCON (P < .001;

243 Analysis of relative CBF values extracted from the amygdala bilaterally revea

244 group and slightly more pronounced relative CBF in the MCI group compared with that in the sCON grou

245 is highly correlated with regional relative CBF, as measured by (15)O-water K1 normalized to cerebel

246 aging was used to collect voxelwise relative CBF at each visit.

247 th only the HIV-1/SIV Vif proteins requiring CBF-beta as a cofactor, HIV-1/SIV Vif using CUL5-RBX2, a

248 light (PIF3, HY5) and cold stress response (CBF).

249 Global resting CBF and regional CBF of right superior frontal cortex co

250 ng at rest and that the reduction in resting CBF reflected reduction in synchronized spontaneous neur

251 e results reveal an important role for RUNX3/CBF during B cell transformation and EBV latency that wa

252 rpus callosum was increased but sensorimotor CBF was decreased, particularly in the ipsilesional side

253 All six CBF genes were cold inducible, but showed varying patter

254 Of the 46 SNPs surveyed across the six CBF homologs, only CBF2_619 exhibited latitudinal differ

255 themia and hypoxemia, and lower and sluggish CBF compared to CMS patients without cerebral edema; but

256 validation identified FA in the SN(FA-SNAv), CBF in the PFC(CBF-PFCAv) and FA in the parietal white m

257 h SN in combination with changes in FA-SNAv, CBF-PFCAv and FA-PWMAv values might serve as potential m

258 Our results suggest that resting-state CBF is a marker of CMB-related small-vessel disease.

259 with widespread reductions in resting-state CBF.

260 ced baseline (~6-12%) and agonist-stimulated CBF.

261 on of these compounds (13.84 +/- 0.06%) than CBF (10.74 +/- 0.06%).

262 The CBF locus includes three genes - CBF1, CBF2 and CBF3 - t

263 The CBF transcription factors alter the expression of more t

264 lators of sub-zero acclimation, although the CBF signal transduction pathway seems to be less importa

265 at with acute exposure to high altitude, the CBF in acute mountain sickness (AMS) subjects was higher

266 ression of more than 100 genes, known as the CBF regulon, which contribute to an increase in freezing

267 ow-temperature regulatory network beyond the CBF pathway is complex and highly interconnected; and th

268 The RSFC results complemented the CBF results, with decreases in RSFC between midline cort

269 regulate a group of more than 100 genes, the CBF regulon, which impart freezing tolerance.

270 How the CBF genes themselves are activated after cold acclimatio

271 However, the CBF genes accounted for only a small amount of the varia

272 cute exposure to high altitude; however, the CBF of the brain parenchyma has not been studied to date

273 trol, thirteen subjects were included in the CBF analysis.

274 ovide evidence that natural variation in the CBF pathway has contributed to adaptive evolution in the

275 expression at low temperature, including the CBF pathway in Arabidopsis.

276 receptor antagonist strychnine inhibited the CBF increase caused by glycine injection into the brain.

277 our data suggest a major involvement of the CBF genes located in the proximal cluster, with no appar

278 Moreover, the dynamic changes of the CBF networks elicited by acute cocaine such as heterogen

279 results indicate that cold regulation of the CBF regulon involves extensive co-regulation by other fi

280 ed the extent to which cold induction of the CBF regulon is regulated by transcription factors other

281 nant negative effects on the function of the CBF-CRT/DRE regulatory module, and 11 transcription fact

282 osterior fossa tumors, additional use of the CBF-to-contrast enhancement ratio yielded sensitivity an

283 climation is only partially dependent on the CBF-CRT/DRE regulatory module.

284 by a disproportionate amount relative to the CBF response, causing the reduced BOLD response.

285 ot changed by cocaine (P=0.244), whereas the CBF to the stimulation was reduced 49.9+/-2.6% (P=0.028)

286 S) subjects was higher (P < 0.05), while the CBF of non-AMS subjects was lower (P > 0.05) compared wi

287 lood flow (CBF) at baseline would lower the 'CBF reserve', and ultimately reduce OT.

288 These CBF responses were almost abolished (-70%, p < 0.001) by

289 Poaceae contain three CBF subfamilies, two of which, HvCBF3/CBFIII and HvCBF4/

290 o show that RNA polymerase II recruitment to CBF-responsive cold-regulated genes requires MED16, MED2

291 scription of genes known to be regulated via CBFs binding to the C-repeat motif/drought-responsive el

292 ld-induced in parallel with the 'first-wave' CBF genes, and determined the effects that overexpressin

293 particularly for stimulation conditions when CBF might be insufficient to cover for the energetic dem

294 ampus (Hip) and the NMSS-Mood score, whereas CBF in the Hip and the prefrontal cortex(PFC) correlated

295 e full spectrum of mutations coexisting with CBF translocations has not been elucidated.

296 uberty and only diverged in midpuberty, with CBF actually increasing in females.

297 lin 2 (CUL2), ELOB/C, and RBX1, but not with CBF-beta or CUL5, to form a CRL2 E3 ubiquitin ligase and

298 ly analyzed the outcome of 145 patients with CBF-AML (59 t[8;21], 86 inv[16]/t[16;16]) in first relap

299 h-throughput sequencing in 215 patients with CBF-AML enrolled in the Phase 3 Trial of Systematic Vers

300 In study 2 (n = 10; aged 25 +/- 4 years), CBF was pharmacologically reduced by administration of i