ライフサイエンスコーパス: CBT

1 CBT (training to change pain-related thoughts and behavi

2 CBT as an adjunct to usual care that includes antidepres

3 CBT is effective in reducing fatigue severity in QFS pat

4 CBT seems to be effective in patients after self-harm.

5 CBT-I and TCC groups showed robust improvements in sleep

6 CBT-I may exert its treatment effects on PI by reducing

7 CBT-SAD and light therapy did not differ in remission ra

8 CBT-SAD was superior to light therapy two winters follow

9 osocial intervention group (n=6 [IQR 4-11]), CBT group (n=9 [5-14]), and short-term psychoanalytical

10 randomly assigned to receive MBSR (n = 116), CBT (n = 113), or usual care (n = 113).

11 the brief psychosocial intervention (n=158), CBT (n=155), or short-term psychoanalytical therapy (n=1

12 reat analysis (doxycycline, 52; placebo, 52; CBT, 50).

13 nt trial aimed to investigate whether adding CBT to standard treatment prolongs remission in IBD in c

14 ons while groups F and B received additional CBT of the modified Fones or the modified Bass technique

15 response to visual SS in PI before and after CBT-I.

16 predict improvement in social anxiety after CBT.

17 hieved by reducing the exposure to ATG after CBT.

18 26% for every 10-point increase in AUC after CBT (hazard ratio [HR], 0.974; P < .0001).

19 BOLD changes after CBT-I in patients were also examined.

20 ts >50 x 10(6)/L twice within 100 days after CBT.

21 .26; P < .0001) and lower ATG exposure after CBT (HR, 1.005; P = .0071).

22 Clinical improvements after CBT-I were correlated with BOLD reduction in the right i

23 sses the importance of early CD4(+) IR after CBT, which can be achieved by reducing the exposure to A

24 tigue severity was significantly lower after CBT (31.6 [95% CI, 28.0-35.1]) than after placebo (diffe

25 U was associated with higher mortality after CBT, which may be a useful criterion for CBU selection,

26 brain hyper-responses were normalized after CBT-I.

27 increased responses to SS were reduced after CBT-I.

28 sleep onset (WASO), and its reduction after CBT-I was associated with improvements in WASO.

29 1-year relapse rate and worse survival after CBT than did HLA-C1/C1 or HLA-C1/C2 (HLA-C1/x) patients:

30 molecular aggregates comprising H-aggregated CBTs are formed.

31 choice for psychological therapy, alongside CBT, for adolescents with moderate to severe depression

32 ith nonrecurrence at the second winter among CBT-SAD participants (relative risk=5.12) compared with

33 gher for those who received MBSR (60.5%) and CBT (57.7%) than for usual care (44.1%) (overall P = .04

34 or joint treatment with both fluoxetine and CBT.

35 ant differences in outcomes between MBSR and CBT.

36 randomized between oral study medication and CBT (2:1) for 24 weeks.

37 The combination of sertraline and CBT significantly reduced clinician-reported primary anx

38 f threat predict better response to SSRI and CBT treatment in anxious youth and that neuroimaging may

39 representatives of psychodynamic therapy and CBT, the main rival psychotherapeutic treatments (advers

40 e counselling using the Life-Steps approach (CBT-AD) has an emerging evidence base.

41 psychoanalytical therapy was as effective as CBT and, together with brief psychosocial intervention,

42 e psychosocial depression treatments such as CBT-AD to people living with HIV/AIDS and examine the co

43 ho were screened, 90 were randomly assigned (CBT-I: n = 45; TCC: n = 45).

44 first time a sustained increased astronauts' CBT also under resting conditions.

45 Relapsed/refractory disease at CBT, recipient HLA-C2/C2 genotype, and donor HLA-KIR gen

46 To evaluate a web-based, automated CBT-I intervention to improve insomnia in the short term

47 Conclusion Internet-based CBT has salutary effects on sexual functioning, body ima

48 y assigned 169 BCSs to either Internet-based CBT or a waiting-list control group.

49 ceived a previously validated Internet-based CBT protocol over 12 weeks and were randomized to receiv

50 -intensity computerised CBT (cCBT; web-based CBT materials and limited telephone support) through "OC

51 treatment response from data acquired before CBT.

52 orm, and posterior cingulate cortices before CBT-I.

53 differences in adherence were noted between CBT-AD and ISP-AD (97 assigned, 87 completed assessment)

54 The largest mediated effect for both CBT and GET and both primary outcomes was through fear a

55 beliefs were the strongest mediator for both CBT and GET.

56 Brief CBT was effective in preventing follow-up suicide attemp

57 w-up assessment, eight participants in brief CBT (13.8%) and 18 participants in treatment as usual (4

58 eat=3.88), suggesting that soldiers in brief CBT were approximately 60% less likely to make a suicide

59 sual (N=76) or treatment as usual plus brief CBT (N=76).

60 rtain effect on cardiovascular outcomes, but CBT combined with antidepressant medication produces mod

61 insula connectivity that are not targeted by CBT alone.

62 positive adults with depression, we compared CBT-AD with information and supportive psychotherapy plu

63 ntly in randomized clinical trials comparing CBT and pharmacotherapy for depression.

64 Sixteen randomized clinical trials comparing CBT and pharmacotherapy for unipolar depression in 1,700

65 ents were more apparent for those completing CBT sessions over a shorter period of time, but were unr

66 s received 1 of 2 low-intensity computerised CBT (cCBT; web-based CBT materials and limited telephone

67 Conclusion CBT-I and TCC produce clinically meaningful improvements

68 criteria for noninferiority to conventional CBT at week 16.

69 ort the discovery of a 2-cyanobenzothiazole (CBT)-reactive peptide tag, CX10R7, from a cysteine-encod

70 Internet-delivered CBT-I has shown promise as a method to overcome this obs

71 fficacy and availability, internet-delivered CBT-I may have a key role in the dissemination of effect

72 nts to receive brief guided parent-delivered CBT (n=68) or solution-focused brief therapy (n=68).

73 hildren in the brief guided parent-delivered CBT group versus 47 (69%) children in the solution-focus

74 However, guided parent-delivered CBT is likely to be a cost-effective alternative to solu

75 ol, to receive brief guided parent-delivered CBT or solution-focused brief therapy, with minimisation

76 However, brief guided parent-delivered CBT was associated with lower costs (mean difference - p

77 superiority of brief guided parent-delivered CBT.

78 28.0-35.1]) than after placebo (difference, CBT vs placebo, 6.2 [97.5% CI, .5-11.9]; P = .03).

79 ssigned 3755 participants to receive digital CBT for insomnia (n=1891) or usual practice (n=1864).

80 ment and attrition more frequently than does CBT.

81 Given their similar efficacy, CBT and antidepressants are both viable choices for init

82 aseline and 12 weeks later, following either CBT or SSRIs in the patient sample.

83 amygdala, has been associated with enhanced CBT outcome for OCD among adults but requires evaluation

84 We here show that during exercise CBT rises higher and faster in space than on Earth.

85 Face-to-face CBT leads to quicker and greater reductions in the numbe

86 icipants received 20 individual face-to-face CBT sessions of 50 minutes each or sequentially complete

87 orporated the primary tenets of face-to-face CBT-I.

88 ed with traditional, individual face-to-face CBT.

89 All pediatric patients receiving a first CBT between 2004 and 2015 at the University Medical Cent

90 R genotyping in graft selection criteria for CBT.

91 treatment, suggesting greater durability for CBT-SAD.

92 oney, clinicians should discuss referral for CBT with all those for whom antidepressants are not effe

93 vs CBT, 0.95 [95% CI, 0.77-1.18]; and RR for CBT vs usual care, 1.31 [95% CI, 1.01-1.69]).

94 vs CBT, 1.03 [95% CI, 0.78-1.36]; and RR for CBT vs usual care, 1.69 [95% CI, 1.18-2.41]).

95 However, aside from CBT, there were few trials of other promising interventi

96 Apparently, they do not profit from CBT of the techniques studied here.

97 differ significantly between patients given CBT and those given short-term psychoanalytical therapy

98 sized pattern of response: joint treatment > CBT or fluoxetine treatment > placebo treatment.

99 e of the 7-week intervention conditions (ie, CBT-I, A, or both), when compared with a placebo capsule

100 in donors and recipients that could improve CBT outcome.

101 In CBT-I and TCC, TELiS promoter-based bioinformatics analy

102 could predict aGVHD and TRM after day 28 in CBT recipients.

103 response at 15 months was 43.7% and 46.7% in CBT-I and TCC, respectively.

104 s, insomnia remission was 46.2% and 37.9% in CBT-I and TCC, respectively.

105 treatment decisions regarding engagement in CBT or SSRIs, especially among individuals with an enhan

106 Since even minor increases in CBT can impair physical and cognitive performance, both

107 HLA genotypes, and NK-cell reconstitution in CBT patients (n = 110).

108 ere more frequent in pharmacotherapy than in CBT.

109 BT was planned to be about one-third that in CBT.

110 ut how specific cell types within individual CBT brain regions support the generation, propagation, a

111 m cognitive behavioral therapy for insomnia (CBT-I) alone was unsuccessful.

112 e Cognitive behavioral therapy for insomnia (CBT-I) and Tai Chi Chih (TCC), a movement meditation, im

113 e cognitive behavioral therapy for insomnia (CBT-I) as the initial treatment for chronic insomnia dis

114 ugh cognitive behavior therapy for insomnia (CBT-I) has been established as the first-line recommenda

115 Cognitive behavioral therapy for insomnia (CBT-I) improved posttreatment global and most sleep outc

116 , cognitive behavioral therapy for insomnia (CBT-i) is now commonly recommended as first-line treatme

117 o cognitive-behavioral therapy for insomnia (CBT-I), tai chi chih (TCC), or sleep seminar education a

118 y cognitive behavioral therapy for insomnia (CBT-I).

119 ncerns the level of access to high-intensity CBT before the primary outcome assessment.

120 icant reductions in uptake of high-intensity CBT over 12 months; 86% of the patients allocated to the

121 cated to the waiting list for high-intensity CBT started treatment by the end of the trial, compared

122 of low-intensity CBT prior to high-intensity CBT, in adults meeting the Diagnostic and Statistical Ma

123 al effectiveness of 2 forms of low-intensity CBT prior to high-intensity CBT, in adults meeting the D

124 The internet CBT-I (Sleep Healthy Using the Internet [SHUTi]) was a 6

125 omized clinical trial comparing the internet CBT-I with internet patient education at baseline, 9 wee

126 A)-might be as effective and cheaper than is CBT.

127 nts were on a waiting list for therapist-led CBT (treatment as usual).

128 efits but may reduce uptake of therapist-led CBT.

129 BA was non-inferior to CBT (mITT: CBT 8.4 PHQ-9 points [SD 7.5], BA 8.4 PHQ-9 points [7.0]

130 ntribute to OCD pathophysiology and moderate CBT outcomes.

131 , compared with placebo, fluoxetine (but not CBT) was significantly more effective at week 24 in redu

132 Here, we present two novel CBT trisamides with (S)- or (R)-chiral side chains which

133 d-cycloserine augmentation of CBT did not confer additional benefit relative to placeb

134 Pooled results for the benefit of CBT for pregnant and postpartum women with screen-detect

135 was noninferior to a conventional course of CBT that provided over 8 additional hours of therapist c

136 A significant effect of CBT during continuation of antidepressant drugs compared

137 Evidence supports the effectiveness of CBT and SSRIs for reducing childhood anxiety symptoms.

138 The beneficial effects of CBT and GET seen at 1 year were maintained at long-term

139 analyses, DCS did not augment the effects of CBT compared with placebo (mean [SD] clinician-rated Y-B

140 im of this study was to test the efficacy of CBT-AD.

141 The sequential integration of CBT and pharmacotherapy is a viable strategy for prevent

142 ipants were randomly assigned to 3 months of CBT-I or TCC and evaluated at months 2, 3 (post-treatmen

143 itive outcomes but do not guide selection of CBT versus pharmacotherapy.

144 We found no evidence for the superiority of CBT or short-term psychoanalytical therapy compared with

145 We found no evidence for the superiority of CBT or short-term psychoanalytical therapy compared with

146 inal analyses also showed the superiority of CBT over GSH-I by the 6-month (adjusted effect, 0.36; 95

147 Use of CBT-I improved several sleep outcomes in older adults (l

148 Use of CBT-I improves most outcomes compared with inactive cont

149 d trial-were re-scanned after 12-14 weeks of CBT or after 8 weeks of minimal-contact waitlist; waitli

150 nters after a randomized trial of 6 weeks of CBT-SAD (N=88) or light therapy (N=89).

151 third scan after crossover to 12-14 weeks of CBT.

152 23 (96%) patients offered CBT took up the intervention.

153 vity, clothing, and environmental factors on CBT regulation under terrestrial conditions.

154 the effects of long-duration spaceflight on CBT at rest and during exercise are clearly lacking.

155 rticipants assigned to wCBT completed online CBT modules and those assigned to ACG received emails wi

156 for significant attrition when using online CBT.

157 s a surface temperature proxy, or MBT and/or CBT for reconstructing pH, in anoxic or euxinic lakes, e

158 roscopy (MRS) was associated with OCD and/or CBT response.

159 erated code, to receive either usual care or CBT in addition to usual care.

160 10-20 mg/day), duloxetine (30-60 mg/day), or CBT (16 50-minute sessions).

161 hronic low back pain, treatment with MBSR or CBT, compared with usual care, resulted in greater impro

162 dicated whether they preferred medication or CBT or had no preference.

163 ) to BA from junior mental health workers or CBT from psychological therapists.

164 The combined OCD participants (CBT-only plus waitlist-CBT) also showed a 16.2% (p=0.004

165 contribute to improved outcomes in pediatric CBT.

166 assigned to one of four treatments-placebo, CBT, fluoxetine, or joint treatment with both fluoxetine

167 verity indicated that the d-cycloserine plus CBT group and the placebo plus CBT group declined at sim

168 to either 10 sessions of d-cycloserine plus CBT or placebo plus CBT.

169 a double-blind fashion to d-cycloserine plus CBT or placebo plus CBT.

170 loserine plus CBT group and the placebo plus CBT group declined at similar rates per assessment point

171 ns of d-cycloserine plus CBT or placebo plus CBT.

172 on to d-cycloserine plus CBT or placebo plus CBT.

173 ring SMC alone or SMC plus APT with SMC plus CBT and SMC plus GET for patients with chronic fatigue s

174 list-CBT) also showed a 16.2% (p=0.004) post-CBT decrease in pACC Glu.

175 CC, lower pre-CBT Glu predicted greater post-CBT improvement in symptoms (CY-BOCS; r=0.81, p=0.00025)

176 Q-9 points [95% CI -1.3 to 1.5], p=0.89; PP: CBT 7.9 PHQ-9 points [7.3]; BA 7.8 [6.5], mean differenc

177 e combined OCD group, within vPCC, lower pre-CBT Glu predicted greater post-CBT improvement in sympto

178 Cognitive-behavioural-based psychotherapy (CBT; comprising cognitive-behavioural and problem-solvin

179 N=85) were then randomly assigned to receive CBT paired with either active or placebo ABMT.

180 ts were randomly assigned (2:2:1) to receive CBT-AD (one Life-Steps session plus 11 weekly integrated

181 All patients received CBT of the basics of toothbrushing.

182 ety symptoms within individuals who received CBT but not SSRIs.

183 o treatment, particularly those who received CBT plus placebo ABMT.

184 rder, or separation anxiety and who received CBT, pharmacotherapy, or the combination.

185 ata on cognitive-behavioral therapy for SAD (CBT-SAD) are promising but preliminary.

186 gnitive-behavioral therapy tailored for SAD (CBT-SAD) than light therapy.

187 g and after completing a standard 30-session CBT, including exposure and response prevention, and hea

188 omly assigned to either 16 weeks of standard CBT (up to 20 sessions of 50 minutes each) or CCBT using

189 Humans' core body temperature (CBT) is strictly controlled within a narrow range.

190 Core body temperature (CBT), salivary melatonin, subjective alertness, and poly

191 This randomized clinical trial compared TF-CBT and treatment as usual (TAU) (varying by site) for c

192 to -0.29), a reduction of 68.3%, for the TF-CBT and TAU groups, respectively.

193 to -1.27), a reduction of 81.9%, for the TF-CBT group and -0.37 (95% CI, -0.57 to -0.17), a reductio

194 Cortico-basal ganglia-thalamic (CBT) neural circuits are critical modulators of cognitiv

195 costly training, with no lesser effect than CBT.

196 no evidence that IPT was less effective than CBT.

197 treatment outcomes in cancer survivors than CBT-I alone.

198 t BA, a simpler psychological treatment than CBT, can be delivered by junior mental health workers wi

199 This study suggests that CBT can improve quality of life for adults with persiste

200 The CBT consisted of weekly therapist-guided sessions, with

201 groups (intent-to-treat rates, 41.6% for the CBT group and 42.9% for the CCBT group).

202 ed with the CBT plus placebo ABMT group, the CBT plus active ABMT group exhibited less severe anxiety

203 f-harmed) and eight (4%) participants in the CBT group (seven [4%] who overdosed and one [1%] who sel

204 s completed the 16-week protocol (79% in the CBT group and 82% in the CCBT group).

205 ic findings were significantly higher in the CBT group than in the GSH-I group at 6-month follow-up (

206 y in the BA group and one [1%] cancer in the CBT group) and 15 depression-related, but not treatment-

207 events (three in the BA group and 12 in the CBT group) occurred in three [2%] participants in the BA

208 was 43.6% in the MBSR group and 44.9% in the CBT group, vs 26.6% in the usual care group (overall P =

209 the BA group compared with 189 (86%) in the CBT group, whereas 135 (61%) were assessable in the PP p

210 the BA group compared with 151 (69%) in the CBT group.

211 terneurons in supporting oscillations in the CBT network that are closely related to movement and par

212 onstrated a significant phase advance of the CBT but not melatonin rhythms, as well as an advance in

213 ction of oscillatory dynamics throughout the CBT circuit or how specific oscillatory dynamics are rel

214 uency (15-30 Hz) oscillations throughout the CBT network.

215 Compared with the CBT plus placebo ABMT group, the CBT plus active ABMT gr

216 Cognitive behavior therapy (CBT) among youth with obsessive-compulsive disorder (OCD

217 I) sertraline or cognitive behavior therapy (CBT), participants completed an emotional faces matching

218 combination of cognitive behavioral therapy (CBT) and antidepressant medication improved depression s

219 ngth evidence, cognitive behavioral therapy (CBT) and antidepressants led to similar response rates (

220 erse events of cognitive behavioral therapy (CBT) and pharmacotherapy for childhood anxiety disorders

221 he efficacy of cognitive-behavioral therapy (CBT) and two antidepressant medications (escitalopram an

222 he efficacy of cognitive-behavioral therapy (CBT) for adolescents with anxiety.

223 exposure-based cognitive behavioral therapy (CBT) for obsessive-compulsive disorder (OCD).

224 eness of brief cognitive-behavioral therapy (CBT) for the prevention of suicide attempts in military

225 the benefit of cognitive behavioral therapy (CBT) in pregnant and postpartum women.

226 doxycycline or cognitive-behavioral therapy (CBT) in reducing fatigue severity in patients with QFS.

227 than those of cognitive-behavioral therapy (CBT) in the acute phase of treatment.

228 Cognitive-behavioral therapy (CBT) is effective for pediatric obsessive-compulsive dis

229 apy (ABMT) and cognitive-behavioral therapy (CBT) may have complementary effects by targeting differe

230 Internet-based cognitive behavioral therapy (CBT) on sexual functioning and relationship intimacy (pr

231 choice between cognitive-behavioral therapy (CBT) or an antidepressant medication for treatment-naive

232 oderately from cognitive-behavioral therapy (CBT) or pharmacotherapy, some experience divergent outco

233 change during cognitive-behavioral therapy (CBT) or selective serotonin reuptake inhibitors (SSRIs).

234 Although cognitive behavioral therapy (CBT) represents the criterion standard for treatment of

235 armacotherapy, cognitive-behavioral therapy (CBT), and psychodynamic therapy are most frequently appl

236 nt response to cognitive behavioral therapy (CBT).

237 udies involved cognitive-behavioral therapy (CBT).

238 uction through cognitive-behavioral therapy (CBT).

239 herapy and for cognitive-behavioral therapy (CBT).

240 on to standard cognitive-behavioral therapy (CBT).

241 High-intensity" cognitive-behaviour therapy (CBT) from a specialist therapist is current "best practi

242 cing worry with cognitive behaviour therapy (CBT) would reduce persecutory delusions.

243 Integrating cognitive behavioural therapy (CBT) for depression with adherence counselling using the

244 ceive digital cognitive behavioural therapy (CBT) for insomnia or usual care, and the research team w

245 Cognitive behavioural therapy (CBT) is an effective treatment for people whose depressi

246 e, to receive cognitive behavioural therapy (CBT) or short-term psychoanalytical therapy versus a ref

247 ent-delivered cognitive behavioural therapy (CBT) would be associated with better clinical outcomes t

248 enced therapy-cognitive behavioural therapy (CBT)-is complex and costly.

249 However, cognitive behavioural therapy (CBT)-the best evidence-based treatment for insomnia-has

250 aches such as cognitive-behavioural therapy (CBT).

251 21 (50%) participants to BA and 219 (50%) to CBT.

252 Patients randomly assigned to CBT who had antidepressants tapered and discontinued wer

253 Patients who were assigned to CBT-AD (94 randomly assigned, 83 completed assessment) h

254 tcomes of remission and treatment failure to CBT and antidepressant medication and survived applicati

255 ly two-thirds of youths respond favorably to CBT.

256 BA was non-inferior to CBT (mITT: CBT 8.4 PHQ-9 points [SD 7.5], BA 8.4 PHQ-9 p

257 ntion-to-treat sample, GSH-I was inferior to CBT in reducing OBE days at the end of treatment (adjust

258 feriority showed that TCC was noninferior to CBT-I at 15 months ( P = .02) and at months 3 ( P = .02)

259 , we evaluated whether TCC is noninferior to CBT-I for the treatment of insomnia in survivors of brea

260 was found to be statistically noninferior to CBT-I, the gold standard for behavioral treatment of ins

261 remained stable among patients randomized to CBT and healthy controls.

262 from scan-to-scan for patients randomized to CBT, with minor increases (3.8%) for waitlist participan

263 eatening faces predicted greater response to CBT and SSRI treatment.

264 Predicting treatment response to CBT based on functional neuroimaging data in PD/AG is po

265 observation of enhanced clinical response to CBT plus active ABMT.

266 ysiology of OCD and may moderate response to CBT.

267 from the 1 year outcome and were similar to CBT and GET at long-term follow-up, but these data shoul

268 nd effectiveness of computer-based training (CBT) of different brushing techniques (Fones versus Bass

269 The ability of cord blood transplantation (CBT) to prevent relapse depends partly on donor natural

270 or limitation of cord blood transplantation (CBT), due in part to a defect in the cord blood (CB) cel

271 lowing pediatric cord blood transplantation (CBT).

272 ACT]; n = 18) or cord blood transplantation (CBT; n = 16).

273 after unrelated cord blood transplantations (CBTs).

274 was compared with wait-listing/no treatment, CBT significantly improved primary anxiety symptoms, rem

275 not significantly differ between treatments (CBT: 10.2, escitalopram: 11.1, duloxetine: 11.2).

276 not significantly differ between treatments (CBT: 41.9%, escitalopram: 46.7%, duloxetine: 54.7%).

277 The average annual cost of trial CBT per participant was pound343 (SD 129).

278 systems like carbonyl-bridged triarylamine (CBT) trisamides are known for their long-range energy tr

279 trols who had undergone double unmanipulated CBT.

280 re, 1.37 [95% CI, 1.06-1.77]; RR for MBSR vs CBT, 0.95 [95% CI, 0.77-1.18]; and RR for CBT vs usual c

281 re, 1.64 [95% CI, 1.15-2.34]; RR for MBSR vs CBT, 1.03 [95% CI, 0.78-1.36]; and RR for CBT vs usual c

282 ned OCD participants (CBT-only plus waitlist-CBT) also showed a 16.2% (p=0.004) post-CBT decrease in

283 Only within the oxycline were CBT-based pH values close to in situ pH.

284 When CBT was compared with wait-listing/no treatment, CBT sig

285 et rapid, implicit threat reactions, whereas CBT may target slowly deployed threat responses.

286 l intervention, we first established whether CBT was inferior to short-term psychoanalytical psychoth

287 In this study, we evaluated whether CBT-I, in combination with the wakefulness-promoting age

288 easons for their deficits and to explore why CBT does not work in this group of patients.

289 At the second winter, CBT-SAD was associated with a smaller proportion of SIGH

290 Improvements associated with CBT were found on the primary outcome measures relating

291 hreshold treatment alternative compared with CBT for adults with BED.

292 y and cost-effectiveness of BA compared with CBT for adults with depression.

293 een various medications and comparisons with CBT represent a need for research in the field.

294 ion to medication and treatment failure with CBT.

295 ts were common with medications but not with CBT and were not severe.

296 s site-specific labeling of the protein with CBT both in vitro and on the surface of E. coli cells.

297 nectivity was associated with remission with CBT and treatment failure with medication, whereas negat

298 pleted 12 weeks of randomized treatment with CBT or antidepressant medication.

299 ugh "OCFighter" or guided self-help (written CBT materials with limited telephone or face-to-face sup

300 magnitude for GET (standardised effects x10, CBT vs APT, fatigue -1.22, 95% CI -0.52 to -1.97, physic